Learning, Memory, & Language Flashcards

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1
Q

Classical Conditioning

A

CS + UCS (UCR) = CS – UCR (CR)

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2
Q

Instrumental (operant) Conditioning

A

Behaviour followed by reinforcement or punishment

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3
Q

Reinforcement

A

Increases behaviour

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4
Q

Punishment

A

Decreases behaviour

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5
Q

+ reinforcement

A

adding something to increase behaviour

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6
Q
  • reinforcement
A

taking something away to increase behaviour

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7
Q

+ punishment

A

adding something to decrease behaviour

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8
Q
  • punishment
A

taking something away to decrease behaviour

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9
Q

Lashley’s Engrams

A

physical representations of learning in the brain structure

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10
Q

Equipotentiality

A

Lashley’s idea that all areas of the cortex contrbute equally to learning and complex functioning behaviours

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11
Q

Mass action

A

The cortex works as a whole - the more cortex, the better

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12
Q

Thompson on Classical Conditioning

A

It occurs in the lateral interpositus nucleus (LIP) of the cerebellum, not the cortex

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13
Q

Hebb

A

Short term and long term memory

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14
Q

STM

A

7 +/- 2 - must be consolidated to get to LTM

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15
Q

Baddeley & Hitch

A

Working memory - temporary storage

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16
Q

Working memory happens in the

A

prefrontal cortex

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17
Q

Case of H.M.

A

had hippocampus removed to prevent epilepsy. -Anterograde amnesia

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18
Q

Anterograde amnesia

A

loss of ability to form new long-term memories

19
Q

Retrograde amnesia

A

loss of long-term memories from before surgery

20
Q

Declarative memory

A

Explicit. episodic, semantic

21
Q

Non-declarative memory

A

Implicit. Procedural, other

22
Q

Hippocampus is imperative for

A

declarative memory, esp. episodic, spatial, contextual

23
Q

Basal ganglia is imperative for

A

implicit learning

24
Q

Parkinson’s disease have difficulty with implicit memory because

A

they have damage to basal ganglia

25
Q

the weather task

A

three pictures, both implicit and declarative memory

26
Q

Alzheimer’s disease

A

Accumulation and clumping of Amyloid beta protein, abnormal form of the tau protein.

27
Q

clumping of Amyloid beta protein

A

widespread atrophy of hippocampus, cerrebral cortex, other areas

28
Q

tau protein

A

part of intracellular support system of neurons

29
Q

Accumulation of amyloid beta and tau proteins causes

A

plaques (structures caused by degenerating neurons) and tangles (degenerating structures within neuronal bodies).

30
Q

Treatment for Alzheimer’s

A

Increase acetylcholine, therefore increasing arousal; cumin spice - inhibits deposits and phosphate attachment to tau

31
Q

Amygdala and learning

A

fear learning

32
Q

parietal lobe

A

piecing info together

33
Q

Anterior and interior region of temporal lobe

A

semantic memory

34
Q

productivity

A

the ability to improvise new combinations of signals to represent new ideas

35
Q

two theories of how language evolved in humans

A
  1. language evolved as a by-product of overall brain development, 2. language evolved as a brain specialization
36
Q

Williams syndrome

A

impaired intelligence, strong language skills

37
Q

Language acquisition device

A

Chomsky - built in “something” that helps us learn language

38
Q

A mutation of FOXP2 gene can impair language development

A

it controls jaw and throat mechanisms

39
Q

Broca’s area

A

located in the frontal lobe of the cerebral cortex, near motor cortex

40
Q

Wernicke’s area

A

located near the auditory part of the left temporal cortex

41
Q

Broca’s (non-fluent) aphasia

A

difficulty with sentence structure, conjunctions; comprehension difficulty when sentence is complicated

42
Q

Wernicke’s (fluent) aphasia

A

fluent speech, but with pauses to find the word. Impaired language comprehension - verbs, nouns

43
Q

Dyslexia

A

more likely to have bilateral symmetrical cerebral cortex less arousal in parietal and temporal cortex while reading

44
Q

Dyslexia and auditory problems

A

people with dyslexia focus on different aspects of sound, also do not shift their attention in the same way