Leaking Abdominal Aneurysm, Mesenteric Embolism or Thrombosis Flashcards

1
Q

Where do leaking abdominal aneurysms usually occur? What are the two types?

A

Below the renal arteries (abover the bifurcation of the common iliac)

Can be saccular (outpocketing) or fusiform (diffuse swelling)

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2
Q

What is often the cause of a leaking abdominal aneurysm? When and where do they rupture ?

A

Usually due to a thrombus on top of a plaque (atheromatous vessel wall with overlying mural thrombi)

They may rupture into the peritoneal or retroperitoneal tissue which is almost always fatal - prone to rupture when they reach 6-7cm

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3
Q

How are aortic aneurysms surgically fixed?

A

Most aneurysms are conveniently located below the renal arteries so that surgical resection can be performed with placement of a dacron graft

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4
Q

Describe the clinical presentation of a leaking abdominal aneurysm.

A
  • Severe central abdominal pain, commonly radiating to the back and may be to the groin along the course of the genito-femoral nerve.
  • The patient may collapse from the accompanying hypotensive shock or suddenly die.
  • Often the patient is a smoker with a history of angina, MI, intermittent claudication, TIA or stroke.
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5
Q

What are the degrees of severity of ischaemic bowel disease?

A
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6
Q

List some causes of ischaemic bowel disease.

A
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7
Q

Describe the pathology of transmural infarction in ischaemic BD.

A

Arterial or venous side is occluded –> infarction which appears haemorrhagic because of blow reflow to the damaged area

Within 1-4 days intestinal bacteria produce gangrene and perforate the bowel

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8
Q

Describe the clinical presentation of mesenteric embolism or thrombosis.

A
  • Severe and continuous pain which quickly develops all the all the hallmarks of peritonitis
  • Generalised pain accompanied by vomiting
  • History of angina, MI, intermittent claudication, TIA or stroke.
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9
Q

What is Virchows Triad?

A

Factors that predispose to thrombosis:

  1. Changes to the constitution of the blood - hypercoagulability
  2. Changes to the blood flow - haemodynamic changes
  3. Changes to the wall - endothelial dysfunction/injury
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