LA MS 7: LA OCD Flashcards

1
Q

OCD PP

A

Failure of endochondral ossification

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2
Q

Vascular Failure

A

Cartilage gets traumatized while soft so doesn’t turn into bone
Loss of vasculature before have opportunity to turn it into bone

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3
Q

Implicated etiologies for vascular failure

A

Genetics
Growth rate - linked
Nutrition: low copper, excess zinc or energy
Gender: questionable
Exercise: excess likely involved - more important in propagation than development
All linked together –> genetically predisposed to grow lrg and fast, fed more, large foals have increased trauma to growth cartilage

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4
Q

Exercise and OCD

A

OCD fragment there - during exercise can wobble –> see CS

Doesn’t mean that exercise caused the OCD!!!

  • -didn’t form because started training too young
  • -Training led to OCD fragment becoming wobbly and dislodging
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5
Q

CS

A

Articularepiphseal growth cartilage
Physis
Remodeling, closure of growth plate

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6
Q

Physeal OC

A

“Physitis, epiphysitis” - growing pains
Most commonly dx’d in distal radius during growth spurt of lrg, fast-growing foals
Uncertain link to OC

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7
Q

Physeal OC CS

A
Variable Lameness 
Enlarged physes, tender to palp 
Look sore 
Nobly knees 
Holding the leg up
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8
Q

Physeal OC Dx

A

Rads - wide, irregular physes, metaphyseal flaring

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9
Q

Physeal OC Tx

A

NSAIDS, rest - avoid running
None
Self-limiting dz
Good prognosis

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10
Q

Articularepiphseal Complex OC - CS

A
Synovitis 
--+/- Lameness 
--eventually lead to O 
Lameness
--increased after work/flexion 
Synovitis +/- Lameness typically dramatic w/ free fragments - not necessarily related to prognosis
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11
Q

Synovitis

A

Tibiotarsal jt effusion “bog spavin”

FP jt effusion “gonitis”

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12
Q

Most Freq OC Lesions - femoropatellar

A

Lateral trochlear ridge

Patella

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13
Q

Most freq OC locations - hock

A

Distal intermediate ridge of the tibia
Lat and med trochlear ridges of talus
Medial malleolus

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14
Q

Most freq OCD locations - shoulder

A

NIGHTMARE
Humerus
Glenoid
Mostly go undx’d until too late because not on people’s lists and shoulder RADS difficult to obtain
–prog terrible if have issues on both sides of the jt

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15
Q

Most freq OC locations - MC/MTphalangeal

A

Sagittal ridge

Condyles of MCIII and MTIII

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16
Q

Other OCD Locations

A

Cervical vertebrate - Wobbler’s (OCD in neck facets of C2, C3, and C4)

17
Q

Tx OC Lesions

A

Single or multiple fragments

Standard of care: take out fragment, debride bed so that don’t get recurrent jt filling, and fill with graft or MSCs

18
Q

Lesions that are prob not OC

A

Medial femoral condyle cysts

Axial palmar/plantar prox phalanx fragments aka PP1

19
Q

Medial Femoral Condyle Subchondral Bone Cysts

A

Horses 1-8yrs (DT trauma in younger horses)

QH predisposed - conformational predisposition

20
Q

Medial Femoral Condyle Subchondral Bone Cysts CS

A

Effusion
Variable severe lameness - can come and go because has a ball valve. When ball valve closes, synovitis resolves
–pain DT synovitis
Can be pos to flexion tests

21
Q

PP1 Fragments

A

Occur at insertion of short distal sesamoidean ligament on P1
–avulsion fracture
–no histologic evidence of retained cartilage
Occurs with greatest freq in STB
Clean articular break (vs OCDs which have a crumbly, brown sugar retention of cartilage)

22
Q

Tx PP1 Fragments

A

Fix
Remove
Leave alone
Arthroscopy - frag removal and lesion debridement

23
Q

PP1 Prognosis

A

Age of horse
Age of lesions
Extent of lesion
Pretense of OA on RADS or intraop

24
Q

OCD In cattle

A

Surgical tx necessary for long term success

Clinicially manifests in 1 jt
Stifle > tarsi > carpi > shoulder > digit
Majority have concurrent OA at time of dx