L4 - Omasal/Abomasal Disorders Flashcards
Where is the omasum located?
R of the median plan and opposing the 7th and 11th ribs
The omasum provides large surface area for the absorption of _____.
- Volatile fatty acids
- Electrolytes
- Water
What role does the omasum have in regards to feed particles?
It reduces them
Omasal impaction is a _____ disorder.
primary/secondary
Primary
TRUE/FALSE
The omasum is typically never involved with other fore stomach primary diseases.
FALSE.
It can be secondarily involved
What is the cause of omasal impaction?
- Feeding rough fibrous feed during droughts
- Feeding machine-made wheat
What are the CS of omasal impaction?
- Anorexia
- Dehydration
- Abdominal distention
- Ruminal Hypomotility
- No palpable abnormalities of intestines
- Empty rectum (no feces)
How is omasal impaction usually diagnosed?
Found on exploratory rumenotomy
What are the treatment options for omasal impaction?
- Fluid therapy
- Supportive care
- Change diet
What is the role of the abomasum?
Glandular organ
Secretes HCl acids and enzymes
TRUE/FALSE
Age is an important risk factor for abomasal ulcers (i.e. young > adult)
FALSE
it can affect cattle of all ages
-Periparturient cows, dairy calves, pre-weaned beef calves
What kind of stress factors may lead to abomasa ulcers?
- Higher stocking rate
- Parturition → retained fetal membranes, mastitis, metritis, hypocalcemia
What organisms are thought to play a role in abomasa ulcers?
- Clostridium
- Campylobacter
- Streptococci
- Fungi
- C. Perfringens type A
- Not really H. pylori
What are the types of abomasal ulcers?
- Non Perforating ulcers → types I & II
- Perforating ulcers → types III & IV
What are the types of Non-peforating abomasal ulcers?
- Type I: Non-bleeding ulcers
- Type II: Major bleeding
What are the types of perforating ulcers?
- Type III: local peritonitis
- Type IV: diffuse peritonitis
Type ____ abomasal ulcers are associated with periparturient period, LDAs, coliform mastitis, and metritis.
Type I
What are the CS of Type I abomasal ulcers?
- Not severely affected → ↓ feed intake, ↓ milk production
- Darkened, soft to fluid feces, minimal anemia
How are type I abomasal ulcers diagnosed?
Only at necropsy
Type ____ abomasal ulcers are associated with black tarry feces and anemia.
Type II
What are the CS of type II abomasal ulcers?
- Black tarry feces
- Anemia
- Sharp decline in milk production, depression, +/- appetite
- Rumen motility depressed (strength and rate)
How are type II abomasal ulcers diagnosed?
- profouNd anemia (PCV <15%)
- Guaiac fecal occult blood test → detects 75 mL blood loss/day
Type ___ abomasal ulcers are associated with lymphosarcoma.
What is seen because of this?
Type II
- more gradual blood loss
- Detetable weight loss
- may have enlarged LN
TRUE/FALSE
Type II abomasal ulcers can affect cows >5yrs at any stage of gestation and lactation.
TRUE
What are the treatment for Type I and II abomasal ulcers?
- Correct concurrent disease
- Reduce stress
- Correct dietary problems
- Severe anemia → restore blood volume
- IV fluids
- Whole blood → min 5 L recommended
- Not surgery
- Acidity modifiers (Ranitidine IV)
TRUE/FALSE
A cow comes in with a Type II abomasal ulcer and needs a blood transfusion. The donor can donate up to 8 L of blood and must be cross matched.
FALSE
The healthy donor can donate up to 8L but no cross matching is needed.
TRUE/FALSE
Oral medications are effective in ruminants.
FALSE
meloxicam is one of the only things you can give orally to ruminants.
Type ____ abomasal ulcers are associated with early postpartum.
What does it resemble?
Type III
-Resembles TRP → moderate febrile, anorectic, acute ↓ in milk
TRUE/FALSE
Type IV abomasal ulcers are medical emergencies.
TRUE
What are the CS associated with perforating abomasal ulcers?
- Tachycardia (>120/min)
- complete rumen stasis
- severe dehydration
- recumbency with cold extremities
How are perforating abomasal ulcers diagnosed?
-Abdominocentesis → toxic changes in cells + intracellular bacteria
What clinicopathological changes are seen with Type III abomasal ulcers?
- Neutrophilic leukocytosis and hyperproteinemia
- Hyperglobulinemia
- Hyperfibrinogenemia
What clinicopathological changes are seen with Type IV abomasal ulcers?
- Severe neutropenia
- Severe hemoconcentration (>40%)
- Hypoproteinemia
What is the treatment for for perforating ulcers?
- Broad spectrum antibiotics
- Restricted exercise (allows firm adhesion to develop)
Displaced abomasum occurs mostly on the ____ side.
Left
Describe a Left displaced abomasum
180˚ torsion WITHOUT volvulus
Rotation along its long axis ventral to the left of the rumen
little/no outflow obstruction occurs
Describe a Right displaced abomasum
180˚ torsion +/- volvulus
Rotation about the mesenteric axis
outflow obstruction and schema if gastric arteries or veins obstructed
What are the predisposing factors for displaced abomasum
- Genetics (females> males)
- 4-7 y/o dairy cows → 2 weeks lactation
- Periparturient disease
- Hypocalcemia
- Ketosis fatty
- Ineffective fiber
- Metritis
- Mastitis
- Dystocia
- Liver disease
What are the prognoses of the different abomasal ulcers?
I-III: Fair unless associated with lymphosarcoma
IV: GRAVE
What test predicts for LDAs?
BHB
How is Displaced abomasum diagnosed?
- Ping & auscultation
- Abdominal Ultrasound
- Definitive diagnosis: abdominal exploratory
- Liptack test
What happens if you hear a ping on the right?
A Ping on the Left?
Right: Gas within spiral colon or cecum
Left: rumen gas
What do you after you obtain a ping on the left side, indicating LDA?
Liptack test
What is the Liptack test?
- Performed to diagnose displaced abomasum.
- Centesis of the area below the gas ping “abomasum”
- Fluid pH <4.5 = abomasum
- Burnt almond odor of gas
What are the medical treatments for displaced abomasum?
- Usually in combination with surgical correction
- Correct underlying cause
How is displaced abomasum surgically corrected?
- Roll and toggle
- Right flank omentopexy (most common)
- Right flank omentoabomasopexy
- Left flank abomasopexy
- Right paramedian abomasopexy
- Laparoscopic abomasopexy