L11: ECG Arrhythmias (Shih) Flashcards
CONS of monitoring ECG during anesthesia
- HR may be off by 10%
- no info on mechanical activity of heart
- additional set up time
- stable ECG signal can be challenging to get
- patient injury can occur from attachment sites
2 classes of antiarrhthmics
Ventricular
Supraventricular
P wave correlates to:
Sinus node and atrial tissue depolarization and atrial contraction
PR interval
AV node
QRS complex
Ventricular depolarization/contraction
If impulse comes from ventricle, QRS looks:
Wide and bizarre
If impulse comes from supraventricular, QRS looks:
Normal
P waves present
Faster HR
If impulse comes from junctional, QRS looks:
Normal
No p waves
Slower HR
Normal HR from supraventricular/junctional/ventricular
Supra: 100-120
Junctional: 80-90
Ventricular: 40-60***
sinus rate faster than ventricular rate
A fib characterized by what rhythm?***
Irregularly irregular
-atrial rate ~200 bpm, and ventricle just trying to keep up
When to tx A fib
- patient sluggish
- ventricular rate very fast
- tx: class 2 beta-blockers (esmolol) and class 4 Ca channel blockers (Ditiazem)
- do NOT tx if HR slow
Tx for 2nd degree AV block
- don’t tx if p otherwise ok
- remove primary cause
- tx: atropine, glycopyrrolate
Type I 2nd degree AV block
PR interval gradually increases before AV block occurs
Type II 2nd degree AV block
PR interval the same before AV block occurs (clinically the same as type I)
Junctional rhythm and tx
AV node taking over beat
Tx: atropine (facilitates comm. b/w SA node and AV node)
*if dz has mechanical origin, drugs won’t work and may need a pacemaker