Knee Pathology Flashcards
Ligament Injuries
Collaterals (MCL, LCL
In general the collaterals (MCL and LCL) have better blood supply and heal without injury.
Medial Collateral Ligament
Mechanism of Injury ia valgus stress typically to a flexed knee.
- ACL may also be torn
- Lateral meniscus may be torn as a result of valgus stress (increase compressive force in lateral compartment.)
MCL examination findings
Tender over entire ligament
pain at the end range extension and flexion with grade 1 sprains
may have significantly restricted ROM in both directions with grade II or III sprains
MCL Rehab notes
Grade II and III will require immobilization to protect the healing ligament
Encourage early WBAT to encourage quad activity
Avoif frontal and transverse plane stresses initially with grade II and III sprainds
Return to Activities with MCL
Return to activities –
•Grade I 1-2 weeks
•Grade II 4-6 weeks
•Grade III 6-10 weeks
ACL Injuries
•Individuals typically reports hearing or feeling a “pop”
•Various MOI
–Contact vs. Non-contact
•Gender Issue
–Females 2-8x’s as likely to tear ACL
•Generally requires reconstructive surgery for athletes and active patients or those with instability (“pivot shift”) with ADLs
ACL - Mechanisms of Injury: Non-contact Injury Mechanisms
Most ACL injuries occur secondary to non-contact mechanism
–Valgus collapse
–Internal rotation of the tibia on the femur
–Pivoting laterally on fixed foot
–Deceleration (quad active mechanism)
–Hyperextension (or more extended position)
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ACL - Mechanisms of Injury: Contact Injury Mechanisms
•Contact
–Valgus stress to flexed knee with foot fixed
–Forced hyperextension (may tear PCL and even popliteal artery if severe)
–Posterior blow to tibia causing excessive anterior tibia translation
ACL: Gender Issue
•Researchers have pointed to several factors to why female athletes are more prone to ACL injury. –Environmental –Anatomical –Hormonal –Biomechanical •neuromuscular
ACL Reconstruction
Bone-patellar tendon-bone (BTB) autograft is still gold standard, but other graft sources (eg, hamstrings and allografts) are becoming much more common.
•Rehab depends on which procedure was performed.
PCL Injuries
Silent group – diagnosis is often overlooked because they are functionally stable
•Various mechanisms of injury
–Posteriorly directed blow to anterior tibia (dashboard injury, soccer, baseball slide)
–Fall onto a flexed knee with planterflexed foot
–Hyperextension
–Hyperflexion
PCL Injuries
•Isolated PCL injuries do well treated nonoperatively (conservatively).
–Significant percentage (85%) do well.
•PCL has excellent blood supply, generous synovial envelope, and meniscofemoral ligaments
•PCL reconstruction is generally only considered when gross knee instability is present.
LCL Injuries
•Less common
•Grade III – may require surgery
•May also involve posterolateral instability – excessive external rotation of the tibia on the femur producing functional instability
– arcuate ligament/posterolateral capsule
– popliteal complex (popliteus and popliteal fibular ligament)
– Cruciate ligaments
Meniscal Injuries
MOI
Forced rotation on flexed knee
Deep flexion occupations
Twisting while WB
2. Medial meniscus more succeptible to injury secondary to being less mobile
3. Patient usually describes a “giving Feeling” and “deep “ pain
Meniscal Injuries
•Clinical tests have either poor specificity or sensitivity, and therefore a number of symptoms and positive clinical signs (ie, a cluster) should be present to indicate a probable meniscal tear.
–MOI
–Complaints of intermittent “locking, catching, clicking or buckling” in chronic tears
–Acute or chronic effusion
–Joint line tenderness
–Unable to fully extend the knee (+ bounce home test)
–Positive clinical testing (McMurray’s/Apley’s/Thessalys)