Introduction (overview of theories) Flashcards

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1
Q

History

A
  • Hippocrates - brain is source of mental disorders
  • middle ages 0 conflicts between god and devil
  • 16th century asylums
  • until 1950s - psychosurgery and ECT
  • 1950s - drugs used to alter mood and revolutionised psychiatry
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2
Q

The medical model

A
  1. diagnosis using DSMV
  2. pharmacological intervention
  3. maybe used alongside psychometric treatments
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3
Q

Pros of diagnosis

A
  • labelled as a disorder so more treatments available
  • common language between doctors and psychologists
  • helps suggest the correct level of treatment
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4
Q

Cons of diagnosis

A
  • discrete entity vs. dimensional
  • co morbidity
  • people stereotype and incorrectly believe they are a danger to society
  • patient self report may be inaccurate
  • interpretation-based criteria
  • pharmaceutical industry may be bias in pushing drugs for: sales, money, professional gains
  • interest in an increasing sense of abnormality e.g. women have sexual desire problems (seen as abnormal)
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5
Q

Cultural differences

A
  • Cochrane (1997) - West Indian immigrants in the UK - most were hospitalised and diagnosed with SCZ than non immigrants
  • causes may be a lack of knowledge about the social norms of the immigrants country of origin - e.g. excessive staring may be normal
  • or religion - i.e. believing that someone is a God may be reasonable
  • Rosenthat and Bervan (1999) - effects of client race on clinical judgement - African American clients, and differences persisted after receiving subsequent information
  • social stigma
  • attitudes towards mental illness
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6
Q

The psychodynamic model

A
  • Freud (1856-1939) - 3 forces - Id, Ego, and Superego
  • defence mechanisms
  • e.g. repression, projection, regression, displacement
  • psychosexual stages, oral, anal, phallic, latency
  • treatment - understand unconscious and conflict free association

+ meanginful to meaningless behaviour e.g. Freudian slips
+ support attachment
+ effective with a range of problems e.g. personality and anxiety

    • not cost effective
  • -untestable hypothesis
  • -falsifiable
    • not generalisable
    • mainly social abuse
    • expensive and time consuming
    • subjective therapy
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7
Q

The behavioural model

A
  • learnt behaviour
  • CC/OC reinforcement
  • flooding
  • response retention
  • behavioural activation
  • function analysis

+ effective for a range of mental health problems

    • simple learning presses
    • reductionist
  • -difficult to trace back to learning experiences
    • do not take into account the biological and cognitive models
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8
Q

The biological model

A
  • explains psychopathology disorders as being due to: genetics, neuroanatomy, neurotransmitters etc.

+ relevant
+ explains why some people are more likely to get if they are in similar environments
+ scientific evidence to support - can use fMRIs etc.
+ clear indication for treatment

    • reductionist
    • stigma
    • variation
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9
Q

The cognitive model

A
  • Ellis and Beck
  • psychological problems are the result of acquiring irrational beliefs, a dysfunctional way of thinking and processing information in a biased way
  • how you think determines what you do
  • development: early experiences –> core beliefs and assumptions –> critical incidence –> thoughts
  • maintenance: thoughts feelings behaviour

+ applied to explaining SCZ
+ brief and effective approach to problems
+ evidence based movement in mental health

    • risk of trivialising psychological problems
    • disregarding role of organic factors
    • not taking into account the social context
    • ignores biological
    • biased thoughts
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10
Q

The biosocial model

A
  • genetic influence
  • biochemical imbalance
  • structural abnormalities
  • social: poor association, stressful life circumstances, cultural and social inequalities
  • psychological: maladaptive learning and coping, cognitive biases, dysfunctional attitudes, interpersonal problems
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