Eating Disorders Flashcards
DSM-V
AN
- significantly reduced calorie intake (BMI
Keele et al (2010)
AN
- prevalence of 0.4%
- 10:1 - F:M
- early onset in middle teens following a stressful life event
- normally goes undetected for the first five years
+ have strict rules, have a sense of control, positive sense of self and wellbeing
– if rules are broken, it can cause stress and panic
– whole world revolves around routine, and cannot have a normal life
– isolation, loneliness, lack of awareness about risky behaviour
– want to stay alive, but cannot give up risky behaviour
BN
- prevalence of BN = 1-3% females, 0.1-0.3% males (9:1 - F:M)
- 75% remission 20 years after diagnosis
- comorbidity - 36-63% major depression
+ excitement of binge eating, but a feeling of sadness and disgust after, out of control, therapy is aimed at gaining control
The Diana effect
- increase in mid 1990’s in BN diagnoses
- more people coming forward and saying they have BN because Diana came out as bulimic
- AN rate has always been lower than BN
Pomeroy et al (2002)
- consequential medical problems with AN and BN
- bloated
- poor sleep
- sensitivity to cold
- osteoporosis
- dry skin
- swelling of glands
Keele and Klump (2003)
- more variably and heritability for BN cross culturally than AN
Fairburn and Harrison (2003)
- migration across disorders e.g. those who are AN are likely to become BN… BUT BN rarely become AN
Historically
- was apparent in early times
- not just a cultural construct
- ideology for people to be smaller
- maybe thinking you’re closer to God by staring yourself - irrational beliefs
Hoek
- normally Westen countries
- white people
- female (90%)
- young adults and adolescents
- AN (high social class)
- BN (even distribution of social class)
- AN (0.7% in teenagers)
- BN (1-2% in 16-35 year old females)
- BUT not everyone comes to clinical services and there is little evidence on atypical disorders
Trace et al (2013)
- genetics
- twin studies (40-60%) but a small sample
Treasure and Holland
- 55-5% = AN —> MZ vs. DZ
- 35-5% = BN —> MZ vs. DZ
BUT
Bulick
- 80% of variance was genetic for BN
- shared environment and learned behaviour
Molecular genetics
- serotonin and polymorphism - occurs in different forms
- genetic variation in a population
- suggests that depending on how much serotonin is in your brain varies with the severity of eating disorder
- BUT Gorwood couldn’t confirm this hypothesis
Fairburn - molecular genetics
- ignores other factors e.g. anxiety, low self esteem, SES, cultural norms
Socio cultural factors
- media reliance
- BMI - playboy
- food and eating fashions - e.g. very popular to be vegan
- negative attitudes towards obesity
- gap between actual and ideal weight
Hill (1998)
- familial factors
- psychiatric problems in family
- excessively criticise daughters appearance