ADHD and disruptive behaviour disorders Flashcards

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1
Q

What is ADHD?

A
  • attention and hyperactivity disorder
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2
Q

Bridgett and Walker (2006)

A
  • 50% have symptoms persisting into adulthood
  • 50% children worldwide and 2.5% adults
  • comorbid
  • consequences of ADHD are often left unsupported e.g. accident by injury, peer rejection, antisocial behaviour etc
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3
Q

Farane et al (2005)

A
  • adoption study
  • genetic support
  • 76% heritable
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4
Q

Pine et al (2001)

A
  • structural magnetic resonance imaging
  • differences between ADHD patients and control groups
  • dorsal lateral PFC, basal ganglia and cerebellum … BUT there is considerable overlap between the groups
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5
Q

Durston et al (2004)

A
  • children with ADHD have smaller brains compared to healthy children - 3.2% reduction in grey matter
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6
Q

Johnstone and Marsh (2007)

A
  • the learning theory of ADHD
  • parent child interactions may cause ADHD
  • positive reinforcement maintains and does not cause
  • e.g. may be used as a tool - ‘my child has ADHD which is why he doesn’t listen’ - need to ensure that you reward good behaviour
  • maybe ADHD caused by rewarding bad behaviour or not punishing bad behaviour
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7
Q

Barkleys (2003)

A
  • ADHD is not a deficit, BUT a difference in delayed gratification
  • need to have the reward immediately, low dopamine levels, so need new things to increase the dopamine
  • this could be genetic?
  • maybe cultural and learned behaviour to prevent delayed gratification e.g. Buddhism
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8
Q

Murray (2003)

A
  • Westerners are at a possible disadvantage. Most jobs are office jobs, and school is always indoors at a desk
  • those with ADHD need constant stimulation
  • a lot of children with ADHD go on to become creative performers or do laborious work as they can work outside in open spaces with more cognitive variation in the environment
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9
Q

Rutler et al (1997)

A
  • whether a child with ADHD becomes an adolescent with comorbid CD is modulated by environmental factors
  • parental hostility or whether a child is integrated into a peer group
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10
Q

Findling et al (2000)

A
  • parental training to deal with oppositional aspects of CD
  • targets symptoms of physical aggression, destruction of property, rule-violations and socialisation
  • in comorbid cases - hyperactivity, inattention, socialisation and aggression
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11
Q

Spencer et al (1996)

A
  • psychopharmacological treatment for ADHD
  • MPH and amphetamines - no significant difference between safety and efficacy of these two psychostimulants
  • improve inattention, hyperactivity, impulsivity and improve oppositional behaviour and social interactions
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12
Q

Swanson et al (1998)

A
  • 31 children with ADHD participated in a double-blind cross over study of four conditions, where the children were observed over an 11 hour day, and ratings of classroom behaviours were recorded
  • found that psychostimulants increased child academic productivity and accuracy - BUT yet to see if psychostimulants alone can yield academic gains e.g. school grades
    • loss of appetite, insomnia, head and stomache ache, weight gain
    • when medication is stopped abruptly, withdrawal reactions occur and 4-12hours after last dose
    • not effective beyond period administered
    • may not be dosed appropriately to achieve full day coverage of medication e.g. may be effective during school hours but not after

+ available in many countries - lasts for 8 hours e.g. Ritalin L A.
+ long acting, so no need for a midday boost in school hours - good because less stigmatisation and removed the possibility of diversion and allows all medication to be delivered by the parent
+ smoother ascending and descending pharmacological profile

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13
Q

What is conduct disorder?

A
  • persistant, aggressive and destructive
  • child/adolescent
  • behaviour has to be pervasive in all different situations e.g. in school and at home
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14
Q

Loebar et al (2002)

A
  • 4-16% M
  • 1.2-9% F
  • criminality, economic cost and social
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15
Q

Moffit (2005)

A
  • early onset = low IQ, reading differences, teenage single parent, poor discipline, increased family conflict , low SES
  • adolescent onset = no evidence of a deficit, less discipline, low SES, jobless, decrease in parental monitoring
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16
Q

Moffit (2005) - biological model

A
  • 100+ adoption and twin studies and found that it is 50% heritable
  • MAOA genotype regulates aggressive behaviour, and breaks down 5HT - the ‘warrior gene’
17
Q

Lynham and Henry (2001)

A
  • associated with deficits in executive functioning, verbal IQ, and memory
  • strong association with reading difficulties
  • BUT rare to find executive functioning deficits in children without comorbid ADHD