Intro to Pain Management (Week 4--Pham) Flashcards

1
Q

Definition of pain

A

Pain is an unpleasant sensory and emotional experience associated with actual or potential tissue damage or described in terms of such damage

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2
Q

Acute pain

A

Adaptive response necessary for the preservation of tissue integrity

Short in duration, well characterized and requires treatment of underlying disease or process

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3
Q

Chronic pain

A

Pain that has outlived its usefulness

Cause of pain is often unknown and has persisted for greater than 3 months (used to be 6 months)

Treatment involves managing underlying idsease and optimizing patient’s function

Often associated with confounding psychological factors and these should be addressed

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4
Q

Malignant/cancer pain

A

Pain due to tissue injury/invasion from cancer, HIV, etc

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5
Q

Non-malignant pain

A

Pain due to tissue damage or perceived tissue damage that is not related to malignancy

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6
Q

Nociceptive pain

A

Pain associated with tissue injury (fracture, joint inflammation, muscle trauma)

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7
Q

Neuropathic pain

A

Pain associated with peripheral nerve injury (carpal tunnel syndrome, diabetic neuropathy) or central nervous system injury (stroke, spinal cord lesion, etc)

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8
Q

Visceral pain

A

Poorly localized pain due to organ injury such as MI, pancreatitis, appendicitis

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9
Q

Barriers to pain management

A

1) Clinician’s poor ability to assess pain
2) Clinician’s poor knowledge about pain management
3) Patient’s and Clinicial’s fear of using opioids and fear of addiction
4) Various attitudes about pain

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10
Q

How do we assess pain?

A

Visual analog scale: patient mark on scale of 0-10

Verbal analog scale: patient verbally give number from 0-10

Faces: point to face scale 0-10 at increments of 2 (usually used for kids)

Pain assessment in chronic conditions should also be based on function, since traditional assessment may not be accurate

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11
Q

How is pain subjective?

A

Cultural/gender differences in expression and report of pain

Vital signs may not reflect intensity of pain experienced by patient (especially in chronic pain)

Patients react differently to pain depending on their coping skills, social/economic factors, cultural beliefs, etc

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12
Q

How is pain managed in acute vs. chronic pain?

A

Acute: NSAIDs, opioids, adjuvants (only for neuropathic pain), modalities (ice/heat/ultrasound), bracing/splinting, interventional procedures (injections/surgery)

Chronic: NSAIDs, opioids, adjuvants (only for neuropathic pain), modalities (ice/heat/ultrasound), therapeutic exercises (stretching/strengthening/aerobic), psychological management (biofeedback, coping skills, relaxation training), interventional procedures (injections/surgery)

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13
Q

WHO stepwise approach to pain treatment

A

Step 1: mild pain; non-opioid (acetaminophen/NSAIDs) +/- adjuvant

Step 2: mild to moderate pain; mild opioid + non-opioid (hydrocodone/acetaminophen (Vicodin), codeine/acetaminophen (Tylenol #3, 4), etc) +/- adjuvant

Step 3: moderate to severe pain; strong opioid (morphine, fentanyl, methadone, etc) + non-opioid +/- adjuvant

Note: can always use adjuvants if neuropathic pain

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14
Q

Patient controlled analgesia (PCA)

A

PCA is a medication-dispensing unit (pump attached to IV lines)

Patient can self-administer doses on as-needed basis

Often given to patient post-op or terminal illness

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15
Q

What does physician determine with PCA?

A

Type of medication

Max dosage given over 24 hours

Max dosage in single dose

Frequency medication can be given

If medication needs to be given as continuous drip

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16
Q

Indications for PCA

A

Moderate to severe pain

Patients needing rapid opioid titration

Patients needing pain meds >48 hours

Patients with changing pain intensity

17
Q

Why PCA instead of IM/SQ injections?

A

Improved pain relief

Greater patient satisfaction

Lower level of drug consumption

Fewer post-op complications

Less painful than IM/SQ injections

Variable absorption with IM/SQ routes

18
Q

When to convert from PCA to oral opioids?

A

At least 24 hours before discharge

If pain has improved or is 1-4 on Visual analog scale

Tolerating clear liquids

19
Q

Adjuvants

A

Anti-epileptic (anti-seizure) medications for neuropathic pain, headaches, fibromyalgia (ex: gabapentin, carbamazine, topiramate, etc)

Antispasmodics/muscle relaxants (ex: Baclofen, Tizanidine)

Antidepressants for neuropathic pain, sleep disorder (ex: TCA)

20
Q

How should we use NSAIDs?

A

Avoid chronic regular use of NSAIDs due to side effects

Use for acute exacerbations of symptoms when there is inflammatory component

Good for bone pain

Chronic daily use may lead to rebund headaches

21
Q

Antispasmodics

A

AKA muscle relaxants

Most are centrally acting

For chronic use, select those that are not habit forming (baclofen, tizanadine)

Muscle spasm in chronic MSK pain often due to chronic deconditioning and overstrain, so rehabilitation (rather than medication) and modalities are key to management

22
Q

Concerns with using opioids

A

Expect tolerance (loss of efficacy of medication over time) and dependence (development of adverse physical symptoms without the drug)–these happen to everybody

Watch for addiction and drug diversion (inappropriate use of prescribed medication)–don’t necessarily happen

23
Q

Therapeutic exercise

A

Since muscles shorten to protect during acute injury, need therapeutic exercise to return muscle and connective tissue to normal length and increase endorphin release

Passive and active stretching

Relaxation

Strengthening

Aerobic

This takes time, and many people get frustrated

24
Q

TENS (transcutaneous electroneurostimulation)

A

Stimulates large afferent fibers to inhibit pain impulses (low intensity, high frequency)

Increase endogenous opioid production (high intensity, low frequency)

Proposed similar mechanism of action as with accupuncture

25
Q

Cold therapy

A

Causes vasoconstriction, reduces edema and inflammation

Slows nerve conduction velocity

Reduces pain

Reduces muscle spasm

Contraindicated in sensory impairment, circulatory problems

26
Q

Heat therapy

A

Increases blood flow and metabolic rate

Increases collagen extensibility

Decreases joint stiffness and muscle spasm

Contraindicated in sensory impairment, circulatory problems

Do not use in acute injury phase due to risk of increasing inflammation

27
Q

Injections/interventions

A

Trigger point injections for myofascial pain (lidocaine, botox, dry needling)

Intra-articular injections for joint inflammation/arthritis (corticosteroids vs. hyaluronic acid derivatives)

Epidural injections for radiculopathy (corticosteroids and lidocaine)

Spinal cord stimulator for neuropathic pain (electrodes implanted to dorsal column to suppress pain signal from periphery)

Implantable medication pump for cancer pain or severe spasticity (medication continusously released by a pump: morphine, dilaudid, baclofen)