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EKG > Intraventricular conduction defects > Flashcards

Intraventricular conduction defects Flashcards

1
Q

in ___BBB the ST-T is opposite the direction of the QRS

A

RBBB

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2
Q

the atrioventricular pathway is through the Bundle of Kent

A

Wolff Parkinson White (WPW)……….. FYI: only a portion goes through the Bundle of Kent, the rest goes through the AV node

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3
Q

if you see a LAD, you should infer

A

LAHB (left anterior hemiblock)

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4
Q

wide QRS + wide/ notched/flattened R (in lead 1 and V6)

A

LBBB (this is the M in WiLliaM)

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5
Q

how do you determine LVH in the limb leads

A

R (in lead 1) + S (in lead 3) > 25

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6
Q

pre excitation syndrome with a normal QRS

A

LGL

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7
Q

LBBB

A

QRS > 0.12 sec, deep/ wide S in V1/V2, wide R in leads 1 and V6, R waves may be prolonged/ notched/ flattened (WiLliaM in V1 and V6)

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8
Q

RBBB + LPHB

A

bifascicular block

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9
Q

M shape in V1

A

RBBB

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10
Q

if EKG looks like BBB, but the QRS is normal, it can be referred to as

A

incomplete BBB pattern AND either hypertrophy OR cannot r/o hypertrophy

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11
Q

tall R in lead 3, deep S in lead 1, normal QRS, strong RAD

A

LPHB

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12
Q

tall R in lead 1, deep S in lead 3, normal QRS, strong LAD

A

LAHB

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13
Q

RBBB

A

QRS > 0.12 sec, M shaped RR’ in V1, wide/slurred S in V6 (MaRroW in V1 and V6)

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14
Q

patients with WPW are vulnerable to

A

PSVT

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15
Q

how do you determine LVH in the augmented leads

A

R (in aVL) > 11

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16
Q

how can you determine a BBB

A

QRS greater than 0.12 sec and RR’ configuration in chest leads

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17
Q

pre excitation syndrome with a wide QRS

A

WPW

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18
Q

RBBB + LAHB

A

bifascicular block

19
Q

where are you looking for atrial enlargement

A

lead 2 and V1

20
Q

LPHB goes with

A

RAD

21
Q

how do you determine LVH in the precordial leads

A

S (in V1 or V2) + R (in V5 or V6) > 35

22
Q

how can you determine a hemiblock

A

a change in the QRS axis but the QRS duration is NOT prolonged

23
Q

causes of nonspecific IVCD

A

ventricular hypertrophy, MI (peri-infarction blocks), antiarrhythmics (quinidine, flecainide), hyperkalemia, paced complexes

24
Q

if EKG looks like BBB, but the QRS is normal, think

A

hypertrophy

25
Q

when I say hypokalemia, you think

A

U waves

26
Q

QRS greater than 0.12 sec

A

BBB

27
Q

why is the QRS wide in WPW

A

premature activation

28
Q

the delta wave occurs b/c

A

a small area of myocytes are depolarized separately from the rest of the ventricles

29
Q

if you see RAD, you should infer

A

LPHB (left posterior hemiblock) aka LPFB

30
Q

RR’ in chest leads (rabbit ears)

A

BBB with the delayed ventricle representing R’

31
Q

wide QRS + broad deep S waves (in V1-3)

A

LBBB (this is the W in WiLliaM)

32
Q

the atrioventricular pathway is through the James fibers

A

Long Ganong Levine syndrome

33
Q

short PR interval (less than 0.12)

A

pre-excitation syndromes (accessory conduction pathways that exist between atria and ventricles)

34
Q

tall R in lead 1, deep (neg) S in lead 3, normal QRS

A

LAHB (plus you’ll prob see LAD)

35
Q

where are you looking for BBB

A

V1 and V6

36
Q

a slurring in the initial portion of the QRS with a wide QRS and short PR interval

A

delta wave seen in WPW

37
Q

prolonged QRS without features of RBBB or LBBB

A

nonspecific IVCD (intraventricular conduction delay)

38
Q

deep (neg) S in lead 1, tall R in lead 3, normal QRS

A

LPHB (plus you’ll prob see RAD)

39
Q

wide QRS + wide S (in lead 1 or V6)

A

RBBB

40
Q

how do you determine RVH

A

RAD or R>S (in V1) or S>R (in V6)

41
Q

LAHB goes with

A

LAD

42
Q

wide QRS + RR’ in V1

A

RBBB

43
Q

a change in the QRS axis but the QRS duration is NOT prolonged

A

hemiblock

EKG (8 decks)

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