Intracranial Haemorrhage Flashcards

1
Q

What are the types of intracranial haemorrhage

A

Extradural
Subdural
Subarachnoid

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2
Q

What is extradural haemorrhage

A

Collection of blood between inner surface of skull and dura mater (endosteal layer)

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3
Q

What vessels are involved in EDH

A

middle meningeal artery (90%)

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4
Q

What is the mechanism of EDH

A

Trauma - pterion fracture

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5
Q

What age group is EDH common in

A

Young: <20yo 50% cases

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6
Q

How might a patient with EDH present

A
  1. Transient LOC (due to concussion)
  2. Transient recovery period (Lucid interval)
  3. Rapid decline in consciousness (enlarging haematoma causing RICP)
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7
Q

What are the findings on CT in EDH

A

Intracranial lesion
Bioconvex and hyperdense lesion (clot is denser than brain tissue)
Ventricular compression
Midline shift

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8
Q

What is the management of EDH

A

ABCDE assessment
Small: observe + conservative Mx
Large: craniotomy + clot evacuation

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9
Q

What are the complications of EDH

A

Permanent brain damage
Coma: RICP - herniation - brainstem compression
Seizures
Arteriovenous fistula

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10
Q

What is subdural haemorrhage

A

Collection of blood between dura mater and arachnoid mater

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11
Q

What vessels are involved in SDH

What are they

A

Bridging veins

Veins draining cerebral venous blood into dural venous sinuses

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12
Q

What is the mechanism of SDH

A

Shearing force on bridging veins:
Trauma
Falls

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13
Q

What age groups are affected by SDH

A

Acute: any
Chronic: elderly - recurrent falls, cortical atrophy cause tension on bridging veins

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14
Q

How might a patient with SDH present

A

Acute: reduced GCS and neurological abnormalities following trauma

Chronic: insidious onset of neurological decline

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15
Q

What are the findings on CT for SDH

A

Acute:
Intracranial
Biconcave + hyperdense lesion
Midline shift

Chronic: hypodense lesion (clot liquefies)

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16
Q

What is the management of SDH

A

Acute:
Immediate neurosurgical intervention
Craniotomy
Burr holes

Chronic:
Burr holes

17
Q

What is subarachnoid haemorrhage

A

Collection of blood in subarachnoid space between arachnoid and pia mater

18
Q

What vessels are involved in SAH

A

Circle of Willis

19
Q

What is the mechanism of SAH

A
Berry aneurysm rupture 
At sites of bifurcation of circle of Willis 
95% anterior circulation: 
Bw ACA/Anterior communicating
Bw MCA/Posterior communicating 
Bw MCA/Branches
20
Q

What are risk factors of berry aneurysms

A

Hypertension
Family history
Abnormal connective tissue: Ehlers Danlos, Marfans
Autosomal dominant polycystic kidney disease

21
Q

What age group does SAH occur in

A

Middle aged (<60)

22
Q

How might someone with SAH present

A

Thunderclap headache

Meningism: photophobia, neck stiffness, headache, N+V

23
Q

How CNS you distinguish meningitis vs SAH

A

In meningitis:
Non-blanching petecheal rash
Fever, tachycardia (sepsis)

24
Q

What are the findings on CT for SAH

A

Intracerebral

Spider- shaped Hyperdense lesion

25
What is the management of SAH
Bedside - fundoscopy Bloods - serology, cultures Imaging - CT Head Procedures - Lumbar puncture Stabilise patient Prevent rebleeding Treat cerebral vasospasm - Ca channel blockers Correct hyponatraemia
26
What are findings on lumbar puncture for SAH
Presence of RBC | Xanthochromia - yellow tinge in CSF
27
What are complications of SAH
Hydrocephalus Focal neurological deficits Seizures Coma
28
What type of hydrocephalus can occur with SAH and why
Communicating Functional impairment of arachnoid processes due to fibrosis of subarachnoid space No obstruction to flow in ventricular system
29
What are contraindications of Lumbar puncture and why | How can you identify this
RICP - risk of uncal herniation Investigations to rule out RICP: Fundoscopy CT head