Interventions Final Flashcards

1
Q

grade 1 tissue injury

A
  • pain at time of injury or within first 24 hours
  • mild swelling local tenderness
  • pain when tissue stressed
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2
Q

grade 2 tissue injury

A
  • moderate pain, requires stopping of activity
  • stress and palpation causes pain
    • ligaments – some fibers may be torn, possibly increased joint mobility
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3
Q

grade 3 tissue injury

A

near complete or complete avulsion/ tear with severe pain

stress to torn tissue usually painless - torn ligament may result in instability of joint

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4
Q

goal of acute stage and how long the stage lasts

A

protection, allowing healing process to occur
~1-6 days
UNLESS perpetuation of injury

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5
Q

acute stage tissue response and characteristics

A
vascular changes
exudation of cells and chemicals
clot formation
phagocytosis, neutralization of irritants
early fibroblastic activities
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6
Q

acute stage tissue response and characteristics

A
vascular changes
exudation of cells and chemicals
clot formation
phagocytosis, neutralization of irritants
early fibroblastic activities
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7
Q

clinical signs of acute stage

A

inflammation and pain before tissue resistance (empty end feel)

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8
Q

signs of inflammation

A

swelling, redness, heat, pain at rest (irritating nerves), loss of function

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9
Q

phase 1 of acute stage

A

control effects of inflammation (PRICE)
prevent deleterious effects of rest
- nondestructive movements

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10
Q

what interventions can be done in the acute stage?

A

PROM
massage
muscle setting (w caution)

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11
Q

what interventions can be done in the acute stage?

A

PROM
massage
muscle setting (w caution)

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12
Q

how to control effects of inflammation

A
  • if it hurts, don’t do it*
  • patient education
  • protection with splint/ sling, AD for walking
  • 24-48 PRICE
  • edema control
  • massage
  • gentle grade 1 joint mobilization
  • facilitate wound healing
  • maintain normal function of unaffected tissues
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13
Q

strain

A

stretching or tearing of muscle fibers or tendon

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14
Q

sprain

A

tearing of ligament fibers

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15
Q

subluxation

A

partial dislocation

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16
Q

muscle/tendon rupture or tear

A

partial or complete tear

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17
Q

tendinopathy/ tendinous lesions

A

degeneration of collagen protein that forms tendon

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18
Q

synovitis

A

inflammation of synovial membrane

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19
Q

hemarthrosis

A

bleeding in joint cavity

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20
Q

ganglion

A

abnormal benign swelling on tendon sheath

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21
Q

bursitis

A

inflammation of bursae in joints – painful

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22
Q

contusion

A

region of damaged tissue where blood vessels have ruptured; bruise

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23
Q

contracture

A

condition of shortening and hardening of muscles, tendons, or other tissues – makes joint and other structures rigid and deformed

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24
Q

adhesion

A

bands of scar-like tissue that form between 2 surfaces inside the body and cause them to stick together

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25
Q

purpose of PROM in acute

A

maintains mobility of tissue and fluid dynamics in joint

26
Q

purpose of massage in acute

A

moves fluid and prevents adhesions

27
Q

purpose of muscle setting in acute

A

pumping action increases circulation without causing joint compression
gentle, low intensity isometric contraction

28
Q

low dosage joint mobilization - purpose in acute

A

grade 1-2 distraction and glide
improves fluid movement and maintains cartilage health
reflexively inhibits pain and decreases muscle guarding

29
Q

precautions in acute stage

A

if movement causes pain, either too high of dosage and/or too high intensity

30
Q

contraindications in acute stage

A

NO stretching

NO resistance exercises for inflamed or swollen tissue

31
Q

subacute stage - goals and how long it lasts

A

proliferation, repair, and healing (moderate protection and controlled motion)
~7-21 days post injury, could last up to 6 weeks

32
Q

tissue proliferation, repair and healing in subacute

A
  • collagen synthesis and deposition – bonds created are becoming stronger
  • tissue granulation
  • excessive fibroblast activity
  • new immature CT is FRAGILE but needs (safe) loading
33
Q

clinical signs of 2nd phase subacute

A
  • decreasing inflammation
  • pain with stress or at end range
  • ROM restrictions due to immobilization (might have muscle guarding)
  • muscle weakness and functional restrictions may be present due to disuse or pain (bc limited by healing of tissues)
34
Q

management guidelines subacute

A
  • moderate protection
  • controlled motion
  • non-destructive activities
  • patient education
  • management of pain and inflammation
  • initiation of active exercises
  • initiation and progression of stretching
  • correction of contributing factors
35
Q

criteria for initiating active exercises and stretching

A
  • decreased swelling
  • no constant pain
  • pain not exacerbated by motion in available range
36
Q

what should you educate pt on during the subacute stage?

A
  • expectations (how long process will be, signs to be aware of)
  • HEP
  • resuming normal, non-exacerbating activities (ex: weening off ADs slightly)
37
Q

when you are initiating active exercises, what types of exercises should they be?

A
  • multiple angle, sub maximal isometric exercises (high reps, low load)
  • AROM
  • muscular endurance exercises
  • protected WB exercises
  • concentric only
38
Q

contraindications of active exercise in early subacute

A

do NOT do eccentric or heavy resistance exercises

39
Q

which type of muscle fibers atrophy faster?

A

slow twitch muscles

40
Q

what must be done during/ before stretching?

A

muscles must be warm
pt must relax
use the new ROM after stretching

41
Q

signs of excessive stress

A
  • soreness greater than 4 hours and not resolved after 24 hrs
  • pain earlier or increased
  • progressively increased stiffness (inc edema, muscle guarding) or decreased ROM between sessions
  • swelling, redness, warmth in healing tissue
  • decreased functional use
42
Q

chronic stage - goals and time frame

A
  • minimal to no protection/ return to function
  • maturation and remodeling
  • ~21 days to 18 months
43
Q

tissue response during chronic stage

A
  • collagen fibers become THICKER and reorient in response to stress — much stronger
  • improvement in collagen quality
44
Q

after _____, scars become resistant to remodeling

A

14 weeks

45
Q

clinical signs of 3rd phase chronic

A
  • no signs of inflammation
  • limitations in ROM due contractures/ adhesions not pain (not chronic if there is an empty end feel)
  • decreased muscle performance
  • decreased usage of involved part of inability to participate normally in expected activities
46
Q

chronic stage management guidelines

A
  • minimal protection
  • ** return to function
  • patient education
  • exercise progression
  • stretching progression
  • progressing muscle performance: control, strength, endurance
  • return to high - demand activities (athletics, job skills)
  • progressive exercise (flexibility, strength)
  • use controlled forces
  • monitor, progress, modify
47
Q

what should you be educating pt on during chronic stage?

A
  • HEP
  • self-monitoring **
  • establishment of guidelines to safe return to work/ play with PT
48
Q

if tightness persists in joint, do…

A

joint mobilization

49
Q

if there is a limitation in muscle length, do…

A

stretching

50
Q

if limitation in strength, do…

A

strengthening in controlled manner

51
Q

if limitation in higher level activities, do…

A

work or sport specific training

52
Q

ways of progressing exercises

A
  • from isolated, unidirectional –> complex and multidimensional
  • strengthening to simulate demands – OKC/ CKC, eccentric/ concentric
53
Q

phase IV is ____ and includes ____

A

return to high demand activities
includes plyometrics, agility training, and skill development
add distractions, doing many things at once, etc.

54
Q

cumulative trauma is…

A

“roundabout” of chronic recurring pain – injury, stress, inflammation

55
Q

tissue response for cumulative trauma

A

state of prolonged inflammation

56
Q

fibroblast activity during chronic inflammation

A

proliferation of fibroblasts with increased collagen production (dumping fibroblasts but they are not being laid down)
degradation of mature collagen – overall weakening of tissue
myofibroblastic activity may lead to progressive limitations in ROM

57
Q

causes of chronic inflammation

A

overuse, cumulative trauma, repetitive straining

  • trauma (i.e. surgery, not following WB restriction)
  • reinjury of “old scar”
  • contractures or poor mobility
58
Q

contributing factors to chronic inflammation

A
  • imbalance between length and strength
  • rapid or excessive repeated eccentric demand (doing too much too fast)
  • muscle weakness
  • bone malalignment or weak structural support
  • change in usual intensity or demands
  • returning to activities too soon after injury
  • sustained awkward postures or motions (ergonomics)
  • environmental factors
  • age related factors
  • training errors
59
Q

clinical signs of chronic inflammation

A
  • PAIN* in involved extremity during and after repetitive activity that may prevent completion and is continued and unremitting
  • contractures or adhesions that limit normal ROM or joint play
  • weakness and imbalance, lead to biomechanics dysfunction
  • faulty movement patterns (due to substitutions)
  • decreased use
60
Q

key things to look for during chronic inflammation

A
  • progressive loss of ROM
  • muscle guarding
    • pull back if there are any problems with interventions in subacute or chronic, or they do not improve symptoms