Inner Ear Disease Flashcards

1
Q

What needs to be excluded for LMN CNV11 palsy?

A

-Herpes Zoster (Ramsay-Hunt Syndrome- vesicles in conchal bowl, vesicular rash around ear, facial nerve palsy, auricular pain, lower recovery, can affect more than one cranial nerve, oral Aciclovir and corticosteroids)
-Middle ear pathology (cholesteatoma= otoscopy)
-Parotid tumour (masses)
-Trauma
-CPA tumours

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2
Q

Treatment and assessment of Bell’s palsy

A

-Idiopathic!! acute unilateral LMN CNV11 palsy
=Peak incidence 20-40, pregnant women
=Forehead involved, dry eyes, altered taste, post-auricular pain, hyperacusis
-Extent assessment= House-Brackmann grade
-Prednisolone 50mg od 10/7 if <3/7 onset +/- antivirals (within 72 hours). Combination
-Eye protection (lacrilube, patch/ tape)

-Follow-up
if the paralysis shows no sign of improvement after 3 weeks, refer urgently to ENT
a referral to plastic surgery may be appropriate for patients with more long-standing weakness e.g. several months

Prognosis
most people with Bell’s palsy make a full recovery within 3-4 months
if untreated around 15% of patients have permanent moderate to severe weakness

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3
Q

Causes of sensori-neural hearing loss

A

-Presbyacuasis/ age related hearing loss
=slow gradual hearing loss usually bilateral
=normal otoscope, audiometry high frequency
– Iatrogenic (Surgery/Drugs)
– Congenital
- Infective
– Autoimmune
-Acoustic neuroma
– Idiopathic

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4
Q

Treatment of sensori-neural hearing loss

A

-Hearing tactics
-Conventional hearing aids
-Implantable hearing aids
-Cochlear implant

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5
Q

Causes of conductive hearing loss

A

-EAM (external auditory meatus)
=Wax (rare unless occlusive) impaction
=Atresia of ear canal (rare)

-Middle ear disease
=OME
=Ossicular discontinuity (COM)
=Ossicular fixation (otosclerosis/ COM)

-Otitis external
-Foreign body
-Tympanic membrane perforation
-Cholesteatoma

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6
Q

General categories of vertigo

A

-Central (brain issue)
-Peripheral (vestibular)

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7
Q

Overview of BPPV (benign paroxysmal positional vertigo)

A

The average age of onset is 55 years and it is less common in younger patients

-Transient (seconds) vertigo with movement (sudden onset), may be associated with nausea
-Following other inner ear disease/ head trauma
-Self-limiting

-Hall pike test= rotatory nystagmus and vertigo

-Epley manoeuvre treatment and vestibular rehabilitation (Brandt-Daroff)

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8
Q

Overview of Vestibular Neuronitis/ Labyrinthitis

A

-Vertigo pattern (long-term)
=Prostrating 3/7 (recurrent vertigo attacks lasting hours or days)
=Housebound 3/52
=Not right 3/12
-Nausea and vomiting
-Horizontal nystagmus
-NO HEARING LOSS OR TINNITUS

-Vestibular sedatives (prochlorperazine/ cinnarizine- buccal or intramuscular) for 3/7, physiotherapy (vestibular rehabilitation exercises)

-Differential diagnosis
viral labyrinthitis
posterior circulation stroke: the HiNTs exam can be used to distinguish vestibular neuronitis from posterior circulation stroke

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9
Q

Overview of viral labyrinthitis

A

-Inflammatory disorder of membranous labyrinth; affects vestibular and cochlear end organs
-Labyrinthitis should be distinguished from vestibular neuritis as there are important differences: vestibular neuritis is used to define cases in which only the vestibular nerve is involved, hence there is no hearing impairment; Labyrinthitis is used when both the vestibular nerve and the labyrinth are involved, usually resulting in both vertigo and hearing impairment.

-40-70 y/o
-Acute onset: vertigo not triggered but exacerbated by movement), N&V, HL (uni/bilateral, varying severity), tinnitus, preceding or concurrent URTI
-Spontaneous unidirectional horizontal nystagmus towards unaffected side, SNHL, abnormal head impulse test (impaired vestibulo-ocular reflex), gait disturbance (fall towards affected side)

-Self-limiting, prochloperazine or antihistamines

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10
Q

Overview of Meniere’s Disease

A

-Episodic vertigo mins-hrs
-Fluctuating sensorineural hearing loss
-Fluctuating tinnitus (ALL HAPPENS TOGETHER)
-Aural pressure
-Low frequency SNHL (stabilises over years)
-Nystagmus and positive Romberg test
-Unilateral, bilateral develop after number of years

-Treat with low salt/ caffeine, betahistine, intratympanic injection
-Acute attacks: buccal or intramuscular prochlorperazine, betahistine prevention, vestibular rehabilitation exercises, DVLA
-Resolve 5-10 years, hearing loss, psychological distress

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11
Q

Overview of Vestibular Migraine

A

-Episodic vertigo mins-hrs
-No fluctuation in hearing/ tinnitus
-Other symptoms: headache, visual disturbance, numbness
-Personal/ FH of migraine
-Can be mistaken for Meniere’s disease
-Treat as per migraine

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12
Q

Describe tinnitus

A

-Sensation of sound in one or both ears
-Cause uncertain
-Most common with hearing loss (30% population)
-Drugs: aspirin/NSAIDs, aminoglycosides, loop diuretics, quinine
-Impacted ear wax
-If pulsatile vascular cause needs excluding
-If asymmetrical MRI scan for CPA tumour

-Treatment= reassurance, hearing aid/ therapy

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13
Q

Overview of Vestibular Schwannoma

A

-Unilateral sensori-neural HL, tinnitus, recurrent vertigo, absent corneal reflux
-Slow growing and benign
-MRI IAM cerebellopontine angle, audiogram
-1% of those with unilateral symptoms
-If growing and large= gamma-knife, surgery, radiotherapy, observation
-Bilateral vestibular schwannomas are seen in neurofibromatosis type 2.

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14
Q

What to examine if the ear is normal

A

-TMJ
-Throat
-Neck
-Nose
-Teeth

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15
Q

Describe Acoustic trauma

A

-Temporary threshold shift
=tinnitus, unilateral HL
=Resolves over hours-days
-Cumulative effect
=Permanent damage to hair cells
=4 kHz predominates

Control of noise at work regulations 2005

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16
Q

Sensorineural vs conductive hearing loss

A

-Rinne’s
=Conductive: bone>air

-Weber’s
=Conductive: sound louder on affected side
=Sensorineural: sound louder on unaffected side

17
Q

What is sensorineural hearing loss?

A

Inner ear, cochlear or auditory nerve pathology impaired neuronal transmission to the brain

=Distorted sound +/- tinnitus
=No features of external ear pathology
=Volume of voice is loud, hearing worsens in noisy environment and high frequencies lost preferentially

18
Q

What is conductive hearing loss?

A

External or middle ear pathology affects the conduction of sound into the inner ear

=Non-distorted sound
=Features of external ear pathology
=Volume of voice remains normal and hearing improves in noisy environment

19
Q

Sudden onset sensorineural hearing loss

A

When a patient presents with sudden onset hearing loss it is important to examine them carefully to differentiate between conductive and sensorineural hearing loss → sudden-onset sensorineural hearing loss (SSNHL) requires urgent referral to ENT.

The majority of SSNHL cases are idiopathic.

An MRI scan is usually performed to exclude a vestibular schwannoma.

High-dose oral corticosteroids are used by ENT for all cases of SSNHL.

20
Q

Suitability for cochlear implant

A

In children, audiological assessment and/or difficulty developing basic auditory skills.

In adults, patients should have completed a trial of appropriate hearing aids for at least 3 months which they have been objectively demonstrated to receive limited or no benefit from.

21
Q

Causes of severe-to-profound hearing loss in children

A

Genetic (accounts for up to 50% of cases).
Congenital e.g. following maternal cytomegalovirus, rubella or varicella infection.
idiopathic (accounts for up to 30% of childhood deafness).
Infectious e.g. post meningitis.

22
Q

Causes of severe-to-profound hearing loss in adults

A

Viral-induced sudden hearing loss.
Ototoxicity e.g. following administration of aminoglycoside antibiotics or loop diuretics.
Otosclerosis
Meniere disease
Trauma

23
Q

Contraindications to cochlear implant

A

Contraindications to consideration for cochlear implant:
Lesions of cranial nerve VIII or in the brain stem causing deafness
Chronic infective otitis media, mastoid cavity or tympanic membrane perforation
Cochlear aplasia

Relative contraindications:
Chronic infective otitis media or mastoid cavity infections
Tympanic membrane perforation
Patients that may be seen to demonstrate a lack of interest in using the implant to develop enhanced oral communication skills.

24
Q

Overview of presbycusis

A

-High frequency HL loss bilaterally
-Presbycusis progresses slowly, as sensory hair cells and neurons in the cochlea atrophy over time.

-Causes
=Arteriosclerosis (diminished perfusion and oxygenation of cochlea= damage)
=Diabetes
=Accumulated exposure to noise
-Drug exposure (salicylates, chemo)
=Stress
=Genetic

-Presentation
=Speech difficult to understand, loud telly, hard hearing on phone, loss of directionality of sound, worsening of symptoms in noisy environments, hyperacusis, sometimes tinnitus

-Otoscopy to rule out otosclerosis, cholesteatoma, conductive hearing loss
-Tympanometry
-Audiometry
-Blood tests (inflammatory, antibodies)