IBP Flashcards
Wheatstone Bridge
- Electrical circuit with one unknown resistor, one variable resistor, two unknown resistor
- Pulse into transducer causes mechanical deformation of strain gauge
- Pressure then sent to unknown resistor –> pressure gauge
Measuring Device
aneroid manometer, commercial transducer/physiograph
Transducer Level with Newer Monitors
internally compensate for vertical differences btw patient, transducer with ‘offset pressure’ – once zeroed, cannot change height of transducer without rezeroing
Transducer Level with Older Monitors
Older monitors: no offset feature, transducer/zeroing stopcock must be at level of RA
Frequency Response of Measuring System Determined By:
- Resonant Frequency
- Damping Coefficient
Resonant Frequency
rate of system oscillation IRT change in pressure
duration of one complete cycle (peak to peak, trough to trough)
Damping Coefficient
rate at which oscillations rest after change in pressure
Basics of IBP
–each cardiac contraction exerts pressure = mechanical motion of flow within catheter, transmitted to transducer via rigid fluid-filled tubing
–Transducer converts motion to electrical signials
–Monitor displays beat to beat arterial waveform as well as numerical pressures
Challenges with IBP in Cats
–Arteries = small, expertise required
–Constrict with cut down manipulation
–Collateral circulation spare: femoral, MT, coccygeal placement can lead to distal ischemia
Pressure Wave Analysis
Allows for better understanding of patient’s heart function as correlates to cardiac cycle
Systolic Phase
Rapid rise in pressure to peak followed by rapid decline
Opening of poetic valve, corresponds to LV ejection
Dicrotic Notch
Closure of aortic valves
Beginning of diastole
Turns into the dicrotic wave distally DT delay
Diastolic Phase
Run off of blood into peripheral circulation
Distal Systolic Pulse Amplification
Occurs further from aorta that measurement is occurring
SAP increases, MAP/DAP decreases
Dicrotic notch displaces R DT delay - transforms into dicrotic wave
Small, Weak Pulses
Pulse wave diminished, pulse feels weak/small
Upstroke may feel slowed, peak is prolonged
Causes:
–decreased SV
–Increased PVR: exposure to cold, severe heart failure
Normal Pulses
Pulse pressure ~30-40mm Hg
Pulse contour smooth, rounded - notch not palpable
Large, Bounding Pulses
Pulse pressure increased
Rise, fall may feel rapid - peak is brief
Causes:
–Increased SV: slow HR
–Decreased peripheral resistance: can occur with increased SV in fever, anemia, hyperthyroidism, aortic regurgitation, AV fistula, PDA
–Decreased aortic wall compliance: aging or athlerosclerosis in people, primates, birds
Bisferiens Pulse
Increased arterial pulse with double systolic peak
Causes:
–Pure aortic regurgitation
–Combined aortic stenosis, regurgitation
0-HCM
Pulses Alternans
Pulse alternates in amplitude from beat to beat even though rhythm basically regular
Can be caused by left ventricular failure or decreased ventricular filling