Hypertension Flashcards

1
Q

What is HTN a risk factor for?

A
Chronic Kidney Disease
Stoke
Heart Failure
Ischaemic Heart Disease (MI)
Vascular Damage
Cognitive Decline
Premature Death
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2
Q

How much does risk increase with blood pressure?

A

For every 2mm of increase in BP, risk of IHD increases by 7% and risk of stroke increases by 10%

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3
Q

How much do HTN cost the UK in drugs?

A

1 billion pounds

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4
Q

What should you tell patients who have to monitor their blood pressure at home with HBPM?

A
  • Get comfy and sit for 5 minutes
  • Place cuff on upper arm skin
  • Rest arm on surface level with heart
  • Sit still and don’t talk during the reading
  • Use same arm each time / note which arm
  • Keep written record
  • Don’t change your treatment without talking to the doctor
  • Take 2-3 measurements with 1min gap between and record all of them
  • Do this in the morning BEFORE meds and in the evening
  • If BP changes by more than 10mmHg between readings, next time wait more than 5mins before starting.
  • Overall 15mins.
  • Record BP for 7 days
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5
Q

When would you send someone home with A/HBPM?

A
  • clinic BP of +140/90
  • unusual BP variability
  • pregnancy
  • whitecoat syndrome
  • nocturnal HTN
  • drug efficacy
  • drug-resistant HTN
  • symptomatic HypoTN
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6
Q

What should you tell patients who are being send home with ABPM?

A
  • Wear the cuff under your clothes
  • Attach the monitor to a waistband etc or under the pillow at night
  • Will record every 15/20mins during the day and 30/60mins at night
  • Machine might repeat a measurement
  • Try to let your arm hang motionless is possible
  • Can take it off for showers etc
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7
Q

How would you take a BP reading in pregnant women?

A

Use specific monitor approved for pregnancy as normal monitors can underestimate BP. Go manual!
Have patients lying on their RHS if in 2/3 trimester as supine is not a good position.
If diaBP is heading towards 0, can measure and record Korotkov sound 4 and 5

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8
Q

What are Korotkov sounds?

A
Phases of BP
1 - first appearance of sound that increases in volume over 2 consecutive beats = sysBP
2 - sounds may then go faint
3 - sounds can get very crisp and loud
4 - distinct sudden muffling of sounds
5 - sounds stop = diaBP
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9
Q

When would HTN be drug-resistant?

A

If it has been untreated due to a slow progressive unsymptomatic rise leading to vascular and kidney damage to the extent that it is non-treatable

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10
Q

How many adults have HTN, and which kind?

A

More than 25% and over 50% of those over 60.
Under 50 years, DBP is more commonly high
Over 50 years, SBP is more commonly the problem.

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11
Q

How would you diagnose HTN?

A

APBM: Use the average value of at least 14 measurements taken during the person’s usual waking hours. If +140/90 then diagnose.
HBPM: Discard the first day’s measurements then take an average of the remaining measurement to confirm diagnosis

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12
Q

What do you have to do before stating that clinic BP is +140/90?

A

Check both arms, if one arm is +20mmHg more than the other in SBP, redo both arms. If this change is consistent then use the arm that gives the higher reading.
Take a least 2 measurements and if the 2nd is substantially different, take a third.
Record the lower of the last two measurements as the clinic BP.

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13
Q

When would you give HBPM instead of APBM?

A

If the person cannot tolerate ABPM

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14
Q

What are the stages of HTN?

A
Grade 1 (mild) : 140-159 & 90-99
Grade 2 (moderate) : 160-179 & 100-109
Grade 3 (severe) : >180 & >110
Isolated SHTN : >140 &amp; <90
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15
Q

What is a hypertensive crisis?

A

This is when BP rises quickly and severely. There are two types, HT urgency and HT emergency.

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16
Q

What is hypertensive urgency?

A

This is a raise in BP to SBP +180mmHg OR DBP +110mmHg.
In HTU there is no damage to target organs.
Symptoms include headache, nosebleed, SOB and anxiety.
Treat with oral anti-hypertensives in outpatient setting.

17
Q

What is hypertensive emergency?

A

This is a raise in BP to SBP +180mmHg OR DBP +120mmHg.
There is damage to target organs: heart, kidneys, brain.
This needs urgent treatment.
Symptoms can include SOB, chest pain, back pain, numbness/weakness, vision change, difficulty speaking.
CAN LEAD TO ENCEPHALOPATHY due to cerebral arterioles not regulating capillaries, blood flowing into IS space causing ICP and dysfunction
Treat with IV vasodilator/CCB/BB in ICU setting

18
Q

What tests would you do for target organ damage?

A

ECG
CXR
Urine
Bloods

19
Q

What is the QRISK2/3 calculator?

A

It calculates a person’s risk of developing a heart attack or stroke over the next 10 years.

20
Q

What is the difference between QRISK2 and QRISK3?

A

QRISK3 uses more parameters (CKD, migraine, steroids, SLE, mental illness, atyp antipsychs, erectile dys, standard deviation of SBP) so should be more accurate.

21
Q

What happens if blood flow to the kidneys is reduced?

A

Kidneys will release renin. Renin converts angiotensinogen (produced by liver) to AT1. AT1 then gets converted to AT2 by ACE (produced by lungs). AT2 then affects the blood vessels and the adrenal gland. AT2 causes vasoconstriction of blood vessels. The adrenal glands produce aldosterone due to the AT2. Aldosterone causes the kidney to increase NaCl and H2O reabsorption, increasing blood volume and pressure.

22
Q

What three things regulate short term blood pressure?

A

CNS
Baroreceptors
Chemoreceptors

23
Q

How do you calculate CO?

A

HR x Stroke Volume

24
Q

How do you calculate BP?

A

CO x Peripheral Resistance

25
Q

What is the main cause of increasing the BP in regards to COxPR?

A

Increased PR mainly due to increased arteriolar vasoconstriction

26
Q

What physiology can cause increased arteriolar vasoconstriction?

A

SNS activation, myogenic reflexes

27
Q

Why does blood flow to the kidneys reduce in HTN?

A

Due to increased peripheral resistance.