HNP and Low Back Pain 1/21 Flashcards

1
Q

Spondylosis

A

bone spurs

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2
Q

spondylolysis

A

pars fracture

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3
Q

spondylolisthesis

A

anterior deviation

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4
Q

what are most often cause of lower back pain?

A

Psoas/Illiacus muscle

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5
Q

osteomyelitis

A

infection of vertebrae

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6
Q

MI

A

can have referred pain to lower back

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7
Q

Aortic Abdominal Aneurism

A

pulsatile mass can be detected in periumbilical/epigastric region

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8
Q

Major causes of pain from GI system?

A

constipation
flatus = gas
necrotizing enterocolitis
perforation

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9
Q

L4/L5 herniation

A

Would affect L5 nerve root

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10
Q

If pain is above knee

A

primarily direct injury to a specific area:

  1. Most likely a non-specific muscular or ligamentous injury
  2. Somatic Dysfunction
  3. Degenerative Disease
  4. Fracture
  5. Spondylolysis
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11
Q

pain radiates below the knee

A
- normally due to nervous innervation 
Pain radiates below the knee:
1. Radiculopathy
2. Spinal Stenosis
3. ***Cauda Equina Syndrome (medical emergency)
4. Associated Piriformis Syndrome
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12
Q

Cauda Equina syndrome

A
  • compression of nerves from L1-S5 normally due to massive disc protrusion, fracture, or tumor that causes bowel or bladder dysfunction
  • Medical Management and Surgical decompression required within 48 hours to prevent permanent neurological damage
  • Check for saddle/perineum anesthesia and decreased anal sphincter tone (loss of bowel and bladder control) in suspected cases
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13
Q

Radiculopathy

A
nervous like symptoms due to : 
Herniated Nucleus Pulposus
Tumor
Exostoses 
Spinal stenosis
Infection
Conditions that compromise intervertebral foramen, canal, disc
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14
Q

What can lead to decrease in the size of the intervertebral foramen?

A
Arthritis
Ligament hypertrophy
Disc degeneration
Muscle imbalance
Tissue quality
SD
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15
Q

What do GAG’s do?

A
  • Negatively charged glycosaminoglycan molecules on proteoglycan chains attract fluid into the extracellular space
  • help vertebral disc maintain water (the more negative the space, the more inhibitory pressure is created within the disk)
  • Anulus fibrosis can adapt, but does not deform very much
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16
Q

Aging and vertebral disc degeneration:

A
  • age-related changes in discs begin in adolescence:
  • Diminished blood flow to the disc
  • Nucleus Pulposus gets smaller and more fibrotic during adult years and blood vessels disappear, cracks appear in the lamellae and annulus
  • in the elderly, the annulus becomes stiff fibrocartilage and the nucleus pulposus has few viable cells
17
Q

Protruded disc

A

type of disc herniation where the annulus remains intact

18
Q

Extruded disc

A

disc herniation through the annulus but posterior longitudinal ligament maintains disc within vertebral space

19
Q

sequestered disc

A
  • disc herniation where annulus is free to roam the spinal canal
  • can cause AI disorder
20
Q

Where do most disc herniations occur?

A
  • Most herniations occur in the Lumbar Spine
  • More stress in the lumbar region compared to other regions of the spine
  • Thoracic spine is protected from extreme motion by the thoracic cage, meanwhile the lumbar spine is left without boney connections to stabilize it.
  • most commonly affected is S1 due to disc herniation from L5, and second most common is hernation from L4
21
Q

Where does L4 go?

A
  • herniation of disk L3,4
  • dermatome crosses over knee medially, and the medial side of the leg
  • patellar tendon reflex lost
  • medial portion of foot
  • motor affected: tibialis anterior - INVERSION
22
Q

Where does L5 go?

A
  • herniation of L4/5 disc
  • lateral side of the leg
    (higher up in the spine starts anterior and superior, and then will rotate down.)
  • there is no reflex
  • extensors affected
  • anterior portion of leg: extensor digitorum longus: flexion of foot
23
Q

S1?

A
  • herniation at L5/S1
  • peroneal/ fibular muscles: eversion
  • Achilles reflex
  • lateral side of foot parasthesia
24
Q

where do disc herniations normally occur?

A
  • most often occur posteriorly, because the anterior long. ligament is thicker and stronger
  • the post. long. lig. is at its narrowest in the lower portion of the spine
  • results in the herniation occurring posteriorly or off to the side (paracentrally)
  • keeps herniation from occurring anteriorly
25
Q

causes of pain in HNP?

A
  1. mechanical compression of nerve root
  2. chemical mediators: such as inflammatory agents
  3. irritation of highly innervated posterior longitudinal ligament
26
Q

Why do we do OMT to treat HNP?

A
  • SDs pre-dispose or aggravate radiculopathy when chronic processes are present within a disc
  • SDs alter mechanical relationships which can change the forces directed toward the disc

OMT goals:

  • Stabilize and decrease impingement locally
  • Address any SD contributing to mechanical instability in the area
  • Treat SDs that may have been created by the HNP
  • Reduce viscerosomatic and Chapman’s reflexes
  • Reduce sympathetic tone
  • Improve lymphatic and venous drainage
27
Q

Acute HNP

A
  • Indirect techniques (HVLA & MUSCLE ENERGY ARE CONTRAINDICATED, as is any technique that exacerbates symptoms)
  • Patient may not tolerate much
  • May need to treat areas distal to the herniation first
28
Q

Subacute/Chronic HNP

A
  • May use direct treatments with care (Personally I would still shy away from direct techniques if at all possible)
  • Patient normally tolerates more
29
Q

Treatment for HNP

A

treat affected extremities and sacrum/pelvis:

  • Type II/Type I mechanics
  • ribs and diaphragm (L1-L3)
  • latissimus dorsi (seen in cases with low back pain associated with shoulder pain)
  • quadratus lumborum: becomes hip stabilizer with weak gluteus medius, results in LBP
  • Iliopsoas: results in majority of back pain coming in - due to people seated at most times, results in constant stretch of muscle and increased gamma gain.
30
Q

paracentral disk herniation

A
  • will result in posterior herniation to the side

- causes unilateral symptoms