Hip Biomechanics Flashcards

1
Q

What is the innominate bone a fusion of and when does it fuse?

A

The ilium, ischium, and pubis. At puberty.

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2
Q

Where do the right and left innominate bones connect and how do they contribute to a definition of the “pelvis”?

A

The right and left innominate bones connect at the pubic symphysis (anteriorly) and at the sacrum (posteriorly). The two bones + the sacrum makes up the whole pelvis.

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3
Q

What muscle attaches to the ASIS?

A

Sartorius

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4
Q

What muscle attaches to the pubic crest / tubercles?

A

The adductors (longus, brevis, magnus)

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5
Q

What muscles attach to the Ischial Tuberosity?

A

The hamstrings and adductor magnus.

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6
Q

In what direction does the proximal femur course and how does this affect gait?

A

Medially, which means it’ll lines up with the medial bias of the foot during gait for better overall efficiency.

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7
Q

What can we palpate on the proximal femur and what muscles attach here?

A

The greater trochanter, and the glute medius and minimus, and deep hip muscles (piriformis).

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8
Q

What is the angle of inclination @ the proximal femur? What are normal values and what are abnormal values called?

A

The angle of inclination is the angle of the femoral neck and femoral shaft when viewed in the frontal plane. Normal angles are 125 degrees. Coxa vara = anything less than 125 degrees. Coxa valgus = anything more than 125 degrees.

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9
Q

How does coxa vara vs. coxa valga affect femoral head loading? How about femoral neck loading? How is torque affected in either of these instances?

A

In coxa vara, the superior aspect of the head is overloaded. In coxa valga, the inferior aspect of the head is overloaded.

In coxa vara, the neck experiences more torsional stress. In valga, the neck experiences more compressive force.

In coxa vara, there is a longer moment arm so more of a mechanical advantage. In coxa valga, there is a shorter moment arm. Think weightlifters…

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10
Q

What is a SCFE?

A

A slipped capital femoral epiphysis. Often in teens and preteens, males, obese individuals - happens as a part of coxa vara. The head of femur falls off the neck and requires surgical stabilization.

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11
Q

How does coxa vara/valga affect the length of the limb? How does it affect joint contact force? How about hip stability?

A

Vara shortens the limb. Valga lengthens the limb.

People with valga have a higher joint contact force because the muscles have to pull harder to maintain the same torque.

You have more hip stability with vara. If valga goes into adduction it will be very unstable (because it has less coverage of the femoral head in the socket). It valga goes into abduction it’s more stable which is why it’s often set this way post-op.

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12
Q

What is a torsion angle of the femur? And what is considered normal, excessive, and retroversion?

A

It is the relative rotation between the shaft and the neck of the femur. Normal is 10-15 degrees. Excessive anteversion (the head is super anterior - the shaft is posterior) is greater than 15. Retroversion is less than 8 degrees.

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13
Q

What is excessive anteversion associated with?

A

Congenital dislocation.
Articular cartilage wear and tear.
Overtime you can lose ER and have to force IR to compensate - this means the tibia will ER… so the person will actually move the toe in to get more coverage of the head by the pelvis.

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14
Q

What is the “ACL and PCL” of the hip joint?

A

Ligamentum Teres

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15
Q

Where is the cartilage in the on the femoral head thickest?

A

“The lunate surface” - anterior and superior

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16
Q

Does the acetabular fossa contain any cartilage or have contact with the femoral head?

A

NO.

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17
Q

When in gait is the most joint reaction force from the hip required? How does the lunate surface respond to this?

A

Loading response to mid-stance. The lunate responds by widening to increase SA and reduce stress.

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18
Q

What is the acetabular labrum and what is its function?

A

It’s a fibrocartilage ring that acts as the meniscus of the hip. It’s chief role is stability which is creates by deepening the joint and increase the surface area gripping the femoral head.

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19
Q

What is the center-edge angle? How much is normal?

A

The angle that measures how much the acetabulum is covering the femoral head in the frontal plane. Normal is 35 degrees. The smaller this angle is the less stability you will have. There’s also more contact stress due to less contact area.

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20
Q

What is the acetabular anteversion angle? What is normal and risks does excessive anteversion impose?

A

How much the acetabulum surrounds the femoral head in a horizontal plane (looking down on it). Normal is 20 degrees. There is an anterior dislocation risk with this.

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21
Q

What are the 3 types of FAI?

A

CAM - which a bony growth on the head - more common in athletic males
PINCER - a bony growth on the acetabulum - more common in females
MIXED - is a bony growth on both sides

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22
Q

Which pelvic capsular ligament has the strongest tensile forces in the body? What motion does it limit? What role can it play in patients with SCI?

A

The Y ligament or iliofemoral ligament. It limits extension of the hip (and ER). In SCI patients it accounts for a lean back (they rely on their ligament to stabilize rather than their muscles.

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23
Q

Explain “hip close-packed position is not the position of maximal congruency”.

A

Hip close-packed position is full extension, with slight abduction and IR - congruency is 90 degrees of hip flexion, moderate abduction and ER.

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24
Q

What is typical hip flexion ROM - w/ a flexed vs. extended knee?

A

120 degrees vs. 80 degrees

25
Q

What is normal hip extension with an extended knee and with a flexed knee? What are the limiting structures?

A

Extended knee hip extension is 20 degrees, limiting structures are the iliofemoral, pubofemoral, and ischiofemoral ligament, and psoas.

Flexed knee hip extension is 0 degrees. Limiting structures are the rectus femoris.

26
Q

What is “normal” for hip abduction?

A

40 degrees.

27
Q

What is “normal” for hip adduction?

A

25 degrees

28
Q

How much IR @ the hip is “normal”? And what is one of the limiting muscle groups for IR?

A

35 degrees, Piriformis or external rotators.

29
Q

What is “normal” for external rotation of the hip? And what are the limiting structures of ER?

A

45 degrees. Internal rotators, specifically, the adductors.

30
Q

What if a total arc of hip rotation motion is 90 degrees but IR is 80 and ER is 20? How do we know if this is bony or soft tissue (anteversion or excessive ROM)?

A

Craig’s Test - wherever you feel the greater trochanter is is where you measure IR. If this is greater than 8-15 degrees than this person is anteverted and will prefer to walk with their legs in IR. If aligned less than 8-15, then the person is retroverted and will prefer to walk with their legs in ER.

31
Q

When forward bending, about what is the distribution of degrees of flexion coming from the lumbar spine and hip?

A

40 degrees from the l-spine, 70 degrees from the hip. The l-spine proceeds the hip in this instance.

32
Q

Adduction of the pelvis causes…

A

Pelvic drop, with side bending of the l-spine toward the support hip.

33
Q

In the sagittal plane, any muscle anterior to the M/L axis of the hip is a…
Any muscle posterior to the M/L axis of the hip would be a…

A

Hip Flexor. Hip Extensor.

34
Q

Name the primary flexors of the hip. Howsabout secondary?

A
Iliopsoas
Sartorius
Rectus Fem
TFL
Adductor longus
Pectineus

Adductor brevis
Gracious
Glute min

35
Q

In anterior pelvic tilt what force couple is at work?

A

Hip flexors, low back Extensors.

36
Q

What are the primary hip Extensors? Secondary?

A
Glute Max
Hamstrings (except biceps femoris short head)
Adductor Magnus (post head)
Glute med
Adductor magnus (ant head)
37
Q

How do the hip adductors contribute to sagittal plane function?

A

Regardless of hip position, the adductor magnus is a hip extensor. All other adductors are flexors EXCEPT beyond 40 to 70 degrees of hip flexion - then they become extensors.

38
Q

What are the primary hip abductors? Howsabout secondary?

A

Glute medius, minimus, TFL

Piriformis, sartorius, rec fem.

39
Q

Describe the trendelenburgh sign.

A

An uncontrollable drop of the pelvis towards the swinging limb. The standing leg is displaying hip abduction weakness.

40
Q

Which muscle of the hip contributes the most to joint reaction force?

A

The glute medius.

41
Q

Why might a patient with pathology present with trendelenburg but have no loss of strength?

A

Guarding.

42
Q

During single limb support what direction are the moments working in?

A

BW creates a clockwise moment. The hip abductors create a counterclockwise moment.

43
Q

What are 2 ways that someone can reduce hip joint reaction force?

A
  1. Decrease Bodyweight

2. Decrease Hip Abductor Force (by trendelenburg)

44
Q

When do the hip abductors produce the most torque? What does this have to do with gait?

A

Like all other muscles, when elongated at 5-10 degrees of adduction. This position is the position of the hip in single limb support. The abductors are designed to produce peak torque when needed the most to create stability in the frontal plane when walking.

45
Q

In the horizontal / transverse plane, how do we know if something is an internal or external rotator?

A

An internal rotator would be a muscle whose line of force is anterior to the longitudinal axis. A muscle whose line of force is posterior would be an external rotator.

46
Q

What are the primary hip external rotator muscles? Howsabout secondary?

A
Glute max
Piriformis
Obturator internus
Gemellus inferior, superior
Quadratus fem 
Obturator externus 
Glute med (post head)
Glute min (post head)
Sartorius
Biceps Femoris (long head)
47
Q

In general, the glute max and glute med are external rotators, but both become internal rotators when…

A

You flex your hip.

48
Q

What are the main responsibilities of the piriformis? How does this effect how you stretch it?

A

ER when the hip is in neutral.
IR when the hip is flexed.
You must therefore stretch it in hip flexion and ER.

49
Q

What are the primary hip internal rotator muscles? Secondary?

A

THERE ARE NO PRIMARY HIP IR MUSCLES.

Glute minimus/med (ant fibers)
TFL
Adductor longus, brevis
Pectineus

50
Q

With more hip flexion the IR potential of the IR dramatically…

A

Increase.

51
Q

How do the Hip IR generally function during walking?

A

During stance phase as your lead foot is on the ground you are in ER. By the time your other leg is on the ground that lead foot hip is in IR.

52
Q

How does a cane opposite the involved limb work?

A

There is a 50% redux in how much the hip abductors have to work AND the cane also distributes your BW.

53
Q

Which side should you hold your briefcase on if you are having hip pain?

A

The ipsilateral side.

54
Q

In those with hip abductor weakness, what can cause an increase in risk of OA progression?

A

A contralateral pelvic drop (aka trendelenburg) can increase the distance between GRF (medial) and the knee joint. This results in an increase in an external knee adduction moment (the external moment arm).

55
Q

Who do ACL tears occur most commonly in and from what?

A

Females > Males

They are usually a noncontact mechanism. Abnormal hip mechanics are believed to contribute to ACL injury.

56
Q

What are the mechanisms for ACL injury? What are some proximal factors?

A

Hip Adduction and IR. Knee Valgus. Tibial ER. (Think of a picture here…)

Proximally, hip strength is redux and impairment of neuromuscular control of hip is likely.

57
Q

Where might you palpate for Iliotibial band syndrome? What is this syndrome the result of?

A

Gerdy’s tubercle.

Often the result of friction from the ITB as it slides over the lateral femoral condole. In general, knee IR and hip adduction increases the tension.

58
Q

What is patellofemoral pain syndrome the result of?

A

Poor patellar tracking. Usually from a Q angle issue which creates excessive hip adduction and hip IR.