HEENT Flashcards
Hordeolum (“Stye”)
most common causative organism
Staphylococcus abscess
Hordeolum (“Stye”) - external vs. internal
external - glands in eyelash follicle or lid margin
internal - inflammation of Meibomian gland
Hordeolum (“Stye”) - symptomology
- localized edema (“bump”) and redness
- acutely tender
- pain proportional to amount of edema
Hordeolum (“Stye”) - exam findings
- erythema
- edema
- tender
Hordeolum (“Stye”) - management
- most resolve spontaneously without intervention over several days
- warm, moist compresses 5-10 min. 3-5x/day
- don’t wear eye makeup
- possibly refer to ophthalmology
Hordeolum (“Stye”) - why refer to ophthalmology
- no start to resolution in 1- weeks
- bacitracin or erythromycin eye drops
- incision and drainage
Chalazion - what is it?
granulomatous inflammation of Meibomian gland
Chalazion - symptomology
- may be asymptomatic
- itchy
- flesh-colored “bump”
- vision changes if “bump” is large
Chalazion - exam findings
- flesh-colored, hard, swollen/indurated area
- NON-tender
- adjacent conjunctival injection
Chalazion - management
- may resolve spontaneously over days or weeks
- warm compresses 10-15 min. few times/day
- possible referral to ophthalmology
Chalazion - why refer to ophthalmology
- if eyelid is swollen causing drooping or obstruction of vision
- corticosteroid injections
Cataracts - what are they?
- abnormal, uniform opacity
- leading cause of blindness
- chronic, progressive
Cataracts - symptomology
- may have increase in near-sightedeness before lens opacity starts to appear
- progressive loss of vision
- glare
- NO pain
Cataracts - exam findings
- loss of red reflex! (or darkening of red reflex)
- opacity on fundoscopic exam
Cataracts - management
- glasses/magnifying glass
- contact lenses
- home safety
- surgical tx to remove opacity
Age Related Macular Degeneration - what is it?
- acute/chronic deterioration of central vision
- older ages, white/Caucasian, female > male
- irreversible
Age Related Macular Degeneration - 2 types
- non-exudative (dry)
- exudative (wet)
Age Related Macular Degeneration - Nonexudative (dry) symptomology
- slow, progressive loss over span of years
- visual fluctuation
- difficulty with night vision
- distortion
Age Related Macular Degeneration - Exudative (dry) symptomology
- progressive loss over span of months
- acute or insidious
- painless
Age Related Macular Degeneration - management
- antioxidants (vit. A & E, copper, zinc, carotenoids) can help reduce speed of progression
- VEFT inhibitors (ophthalmology rx)
Conjunctivitis (“pink eye”) - most common causes
- bacterial or viral
- can also be allergic or contact (chemical irritants)
Conjunctivitis (“pink eye”) - general symptomology
- NO effect on vision
- diffuse conjunctival injection
- mild pain possible (more discomfort or annoying sensation)
- very itchy = allergic
Conjunctivitis (“pink eye”) - viral common cause
Adenovirus
Conjunctivitis (“pink eye”) - viral symptomology
- typically bilateral
- discharge = copious, watery
- marked foreign body sensation
- associated with UTI, pharyngitis, fever, malaise
Conjunctivitis (“pink eye”) - viral management
- symptomatic (cold compresses)
- artificial tears
- antihistamine/ decongestant drops
Conjunctivitis (“pink eye”) - viral exception!
Herpes Simplex Virus (HSV)
- unilateral
- lid vesicles
- possible acute hemorrhagic conjunctivitis
- tx = topical antiviral (ganciclovir) and/or systemic (acyclovir)
- REFER!
Conjunctivitis (“pink eye”) - bacterial symptomology
- uni- or bilateral
- discharge = copious, mucopurulent, possible eyelash matting
- self-limiting (10-14 days without tx)
Conjunctivitis (“pink eye”) - bacterial common cause
Staphylococci (MSSA, MRSA)
Conjunctivitis (“pink eye”) - bacterial management
- erythromycin ointment or trimethroprim- polymyxin B drops
Conjunctivitis (“pink eye”) - bacterial exception!
Gonoccocal Conjunctivitis
- EMERGENCY!
- copious purulent drainage
- corneal involvement may lead to perforation
- tx: ceftriaxone 1g IM, topical (erythromycin, bacitracin)
- REFER to ophthalmology
Corneal Abrasion - symptomology
- tearing
- blephorospasm
- severe pain, foreign body sensation
- watery or purulent discharge
- blurry vision common
Corneal Abrasion - exam findings
- Fluorescein stain - dye uptake at site of corneal defect (using slit-lamp)
- circumcorneal injection
- consider retained foreign body
Corneal Abrasion - managment
- cycloplegic drops (1%) for exam only
- simple/clean - topical erythromycin
- dirty/contacts - ophthalmic ciprofloxacin
- tetanus booster
- patching and steroid preps contraindicated
- ophthalmology follow-up within 24 hours
Corneal Foreign Body - symptomology
- foreign body sensation
- pain
- tearing
- redness
- photophobia
Corneal Foreign Body - exam findings
- normal or decreased visual acuity
- conjunctival injection
- visible foreign body
- rust ring
- epithelial defect with fluorescein stain
- corneal edema
Corneal Foreign Body - management
- cycloplegia drops (1%) for exam
- remove foreign body
- antibiotic drops (tobramycin, polymyxin B)
- ophthalmology follow-up
Diabetic Retinopathy - 2 types
- non-proliferative
- proliferative (less common, more severe)
Diabetic Retinopathy - non-proliferative phases
- mild: at least 1 microaneurysm
- moderate: microaneurysms and hemorrhages
- severe (4-2-1): hemorrhages and microaneurysms in 4 quadrants, venous bleeding in 2+ quadrants, and intraretinal abnormalities in 1+ quadrants
Diabetic Retinopathy - proliferative hallmark sign
Neovascularization
Diabetic Retinopathy - symptomology
- early/initial: asymptomatic
- advanced: floaters, blurry vision, progressive visual acuity loss
- visual acuity and symptoms are poor guides to presence of diabetic retinopathy
Diabetic Retinopathy - exam findings
- microaneurysm: earliest, sign, looks like small dots
- dot/blot hemorrhages: look similar to microaneurysms but in deeper layers
- flame-shaped hemorrhage
- cotton-wool spots
- venous looping/ beading: significant predictor of proliferative from non-proliferative
Diabetic Retinopathy - workup/ diagnostics
- lab: diabetes workup
Diabetic Retinopathy - management
- control diabetes #1 (control sugar, BP, lipids)
- REFER to ophthalmology (retina specialist)
- annual screenings
Retinal Detachment - what is it?
- separation of inner layers of retina from underlying retinal epithelium
- can be spontaneous or penetrating/ blunt trauma
- spontaneous generally affects > 50 years
Retinal Detachment - symptomology
- photopsia: perceived flashes of light
- rapid loss of vision in curtain-like fashion
- may describe vision loss as a shadow
- floaters
- NO pain or redness
Retinal Detachment - workup / exam findings
- ask about previous eye trauma, surgeries, conditions
- visual acuity
- external signs of trauma
- assess pupil reaction
- intraocular pressure (tonometry)
- ophthalmoloscopic exam
Retinal Detachment - management
- emergent referral to ophthalmology
- NPO
- protect globe if traumatic
- avoid pressure on eye, limit activity
Central/Branch Retinal Artery Obstruction - most common cause
Embolus (cholesterol)
Central/Branch Retinal Artery Obstruction - risk factors
- same as CVD
- smoking, HTN, CAD, high cholesterol, hx of TIA
Central/Branch Retinal Artery Obstruction - symptomology
- acute, painLESS partial loss of vision
- “descending nightshade”
- monocular
- central or sectoral visual deficits
- may be asymptomatic
- may c/o amaurosis fugax (transient loss of vision in one eye)
Central/Branch Retinal Artery Obstruction - workup / exam findings
- ESR
- consider coag panel: PTT, fibrinogen, CBC, lipid
- ophthalmoscopic exam: box cars (segmented or narrowed flow) or cherry spots
- CV work-up