Heart Failure - Dr. Miller Flashcards
sx of HF
- SOB
- fatigue
- edema + rales
HF 2 types
- normal Ejection Fraction (LV ejects 50% or more of blood that filled it) (HFPEF)
- reduced EJ : LV can eject 40% or less of blood that filled it (HFrEF)
preserved EF HF what happens
wall of LV concentric thickening = unable to relax (ejects normal onlt doesnt fill to its capacity)
reduced EF HF what happens
wall thinning = dilation of LV (can fill normally only cant eject all blood)
HF reduced EF usually from what event and what 2 things happen right after
MI, lowering CO
1. sympathetic NS
2. RAAS
= vasoconstriction, increase HR
disease not associated with heart that can cause HF reduced EF
- thyroid problem
- SLE, Sarcoidosis
- alcohol high consumption, drugs
- chemotherapy
what has been easier to tx HF reduced or preserved EF
HF with reduced EF
HF EF reduced risk
= male = LV hypertrophy (dilating) = smoking = MI = bundle block
HF EF preserved risk
= old age
= F
= HTN
= atrial Fib
HF SX
- CONGESTION : sob, paroxysmal nocturnal dyspnea, orthopnea, edema, weight (2-3lb a dat fluctuation, or 6lb a week)
- HYPOFERFUSION : exercise intolorance, fatigue, cold intolorance,
how to assess congestion
- S3 gallop
- orthopnea
- edema
- ascites
- JVP
(DRY OR WET)
how to assess for perfusion
= cool extr = renal dysfunction = narrow pulse = hypotension = altered mental status (WARM OR COLD)
cardiac biomarker most helpful to see HF in a pt
BNP : brain natriuretic peptide (rules out HF only)
= can be elevated in (COPD, anemia, renal insuff, old age, pul htn)
imaging fo HF *
Echocardiography :2D TTE (transthoracic)
LV EF is low
cardiomyopahty imaging
MRI
ACC/ AHA staging for HF and % in population (PROGRESSIVE)
A : no sx or HD, however high risk HF (22%)
B : HD only no sx HF (34%)
C : HD with prior or current sx HF (12%)
D : HF require intervention (0.2%)
NYHA stage classification of HF
I : no limitation in activity
II : slight limitation in activity
III: moderate limitation in activity
IV : cant do activities without sxs (unable to converse either)
TX HF EF reduced
- improve sx : diuretics*****
- prevent heart remodeling : (ACE Inh. and BB) + minerocoritcoroids antagonist (MR) needed if needed *****
- prevent hosp.
TX HF EF preserved
no improvement in meds
= only tx htn + diuretics *****
= tx other comorbidities
tx hypoperfusion
fluid and inotropic agent
congestion tx
diuretics and help to tx renal replacement therapy
vasodilators
Cor Pulmonale is from what
pulmonary disease (COPD, pulm htn, chronic bronchitis) = RV hypertrophy
Cor pulmonale SX
- SOB
- LOWER Extr edema
- abd girth increases = ascites
- elevated JVP
- S3 gallop, Tricuspid murmur
- hepatosplenomegally, pulsatile liver
Cor pulmonale EKG
right axis deviation , RV hypertrophy
Cor pulmonale CXR
enlarged main pulmonary As and hilar As
Cor pulmonale 2D TTE (echocardiogram)
hard to see , use MRI and cardiac catheterization with this
Cor pulmonale TX
- maintain O2 : give O2
- Diuretics and lower fluid and Na
- IV inotropes (hemodynamic support)
- manage arrhythmia
- restrict activity while sx, then improve it
prevent HF
htn, hyper lipids, DM, obesity, smoking = PREVENT + control
= diet, exercise and counseling my patients