Healing and Assessment Flashcards
stages of the healing process
- inflammation generic response to tissue threat, acute inflammation is exaggerated response lasting up to 72h post inj is generated by biochem mediators such as histamines, prostaglandin, and bradykinin (vasodialates, increase vascular permeability, and increase pain sensitivity of nerve endings to cause acute hyperalgesia)
- repair and proliferate from 2 days up to 3 weeks post inj, macrophages use phagocytosis to remove debris, fibroblasts lay down new foundation of weak and unorg collagen and build new BV to form granulation tissue; clinically feel better but pain, swelling, and alt func may occur but disappear by end
- remodelling and maturation from 3 weeks to 2 yrs post inj simultaneously break foundation of collagen and replace with strong collagen (no net gain) and BV stabilize with no new growth; can progressively stress tissue to regain strength and endurance
clinical presentation of inflammation
- swelling, can remain after inflammation over esp if immobile or inj has poor circulation for ext period, diff types have diff name (edema is fluid leak from vessels, hematoma is collection of blood which can present as bruise, hemarthosis is bleeding into joint cavity, joint effusion is excessive synovial fluid)
- heat due to vasodilation by biochem mediators but can depend on structure and location
- altered func related to pain and lack of stability
- redness due to vasodilation by biochem mediators but can depend on structure and location
- pain due to increased nerve ending (nociceptor) sensitivity by biochem mediators
injury assessment process overview
filter info to come to conclusion: take history, observe inj, note ROM, test resistance, perform special tests, palpation
taking history
- introduce self to patient and build trust
- guide into injury story focusing on how injury happened to determine acute or chronic, find timeline to know how far into the healing process
- clinical presentation of signs (visible diff and objective) and symptoms (feelings/exp and subjective) and functional impact of inj on daily life
- previous health history to know ideal state to set goals and factors contributing to problem, current health, and physical activity demands
measuring ROM
- record all types of motion for specific joint actively and passively while recording instances of pain
- measure degrees of movement with goniometer
- compare to avg ROM values keeping in mind health history (limited, excessive, painful) and interpret (what is being pulled apart, what is contracting that could contribute to pain)
- what structure (periosteum, tendon, ligament) is being pulled apart and is injured muscle contracting
observation of inj
- ask for consent
- use qualitative and bilateral observations to allow for comparison of inj v. control
- look for signs of swelling, deformity, and discolouration (look carefully on darker skin since it can make signs less visible)
resistance testing
use manual strength test grading (isometric test in mid range ROM where struc strongest)
1. starting in antigravity position, zero with no contraction
2. one if some contraction but no motion
3. two in gravity pos, some motion unable to overcome gravity
4. three able to overcome and hold pos against gravity
5. four able to meet external opp force with some resistance
6. full contraction, able to work as well as other side
special tests and palpation
- id potential structures dmg, test each structure
- reproduce mechanism of injury by applying same stress causing injury to test reproducibility of symptoms
- use a battery of tests to confirm, no test perfect and know anatomy for palpation
What is the problem with assessing an injury during the inflammation stage?
You get a lot of false positives, because nociceptors become significantly more active during an injuries inflammation stage. so everything hurts.
When testing range of motion, why must we perform all tests bilaterally?
To get a sense of what normal function is.
why is there more swelling with ligament than tendon injury
less blood supply in the ligaments, increase blood supply to ligaments to speed up healing process, tendons already have more blood supply thus don’t need to swell as much
relative risk
also called risk ratio, is the risk of a certain event happening in 1st group compared to the risk of the same event happening in 2nd group
odds ratio
the risk of a certain event happening if a specific factor is true
incidence risk ratio
the risk of an event happening in the 1st group divided by the risk of the 2nd group