Head and Spinal Cord Injury Flashcards
How many deaths per year via TBI?
52,000
What percent has long lasting symptoms after TBI?
10%
What are the basic mechanisms of brain injury?
primary (localized at impact site, rotational forces, axonal shearing)
secondary (hypotension, hypoxia, anemia, sepsis)
What are secondary causes of TBI? HASH
hypotension
hypoxia
anemia
sepsis
concussion-
reversible traumatic paralysis of nervous function, immediate effect. Caused by sudden change in movement of head
diffuse axonal injury caused by shearing motion
What is the mechanism of a concussion?
rotational motion of the cerebral hemispheres in the anterior posterior plane, around the fulcrum of the fixed-in-place upper brain stem.
maximal rotational forces exerted around the midbrain and diencephalic region
What are the clinical manifestations of a concussion?
immediate loss of consciousness supressed reflexed fall in BP transient arrest of respiration convulsive activity retrograde amnesia or anterograde amnesia
Management of concussion:
ABC's Imaging recommended in 60 drunk bleeding tendencies headache and vomiting
What are the grades of concussions in athletics?
1- no loss of consciousness, transient confusion, resolution 15 min
3- loss of consciousness
Post concussion syndrome:
interval between head trauma w/ loss of consciousness and development of symptoms, <4 weeks
contusions:
heterogenous lesions consisting of areas of punctate hemorrhage, edema and necrosis
coup lesion-
contusions of the surface of the brain beneath the point of impact
contrecoup lesions-
contusions on the opposite side of impact
cooup/contrecoup lesions-
frontal and temporal poles
Types of intracranial hematomas?
epidural
subdural
Epidural hematoma:
temporal parietal fracture, laceration of the middle meningeal artery or vein, bleeding into epidural space, lens shaped
hematoma on side of trauma
“lucid moment”
Subdural hematoma:
tearing of the bridging vein, blood in subdural space
a/w atrophy, coagulopathy, CSF shunts
typically extended over most of cerebral convexity
acute or chronic
Acute SDH:
in combination with epidural hemorrhage or contussion
tearing of bridging vein
evolution over many hours
drowsiness, coma; pupillary dilation w/ contralateral weakness then bilateral limb wakness; progressive stupor then coma
Chronic SDH:
minor trauma, a/w coagulopathy and brain atrophy
usually in elderly
evolution over days to weeks
headache, progressive alteration in behavior +/- neurological symptoms
Itraparenchymal hematoma:
after severe head injury
location: frontal temporal
evolution over 12-48 hours
stupor- coma, dilated pupil, progressive hemiplegia