gingival disease Flashcards

1
Q

gingivitis associated with dental plaque only (or only with local contributing factors)

A
  • inflammation of the gingiva in the absence of clinical attachment loss
  • redness/edema/surface texture changes
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2
Q

clinical attachment loss (gingival diseases do not have this)

A

-detachment of PDL fibers so you can probe onto root surface

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3
Q

clinical features of gingivitis associated with dental plaque only (or local contributing factors)

A
  • changes in color (red/magenta)
  • bleeding on probing/spontaneous
  • changes in consistency, edema, fibroma
  • changes in CONTOUR: enlarged (marginal/papillary), blunted or cratered (papilary), rolled (marginal)
  • changes in texture: lose stipppling, interdental groove, and/or free gingival groove
  • changes in position: enlargement (pseudopocket)
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4
Q

changes in contour, gingivitis assoc.w/dental plaque

A
  • bulbous papilla
  • papilla should be concave, but become straight or even convex
  • pap and marginal: enlarged
  • pap: blunted/cratered
  • marg: rolled
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5
Q

2 types of plaque: which causes gingival inflammation

A

supra and sub gingival

**sub gingival causes it

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6
Q

gingivitis: radiograph shows what

A
  • no evidence of crestal bone loss
  • bone should be 1-2mm apical of CEJ
  • in that 1-2mm space, you have gingival CT and PDL fibers
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7
Q

health or plaque induced gingivitis on a reduced but stable periodontium

A
  • generally follows active perio therapy
  • tissue is healthy or has slight gingivitis
  • no evidence of progressive attachment loss/active disease (12 months)
  • must have longitudnal atatchment measurements to support this dx
  • could also be used in presence of recession from a non-perio etiology
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8
Q

clinical patterns of gingivitis - extent

A
  • probe 6 sites per tooth
  • old system: by % of sites affected
  • new: by % of teeth affected
  • 30% or less= localized
  • more than 30% = generalized
  • diagnose by sextant and whole mouth
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9
Q

clinical pattersn of gingivitis - severity

A
  • slight, moderate, severe

- more subjective

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10
Q

gingivitis - histology

A
  • many changes: CT breakdown leads to inflammation
  • no apical migration of ATTACHMENT
  • jxnal epithelium’s apical part can migrate apically, but the point of attachment (which is the coronal part at base of sulcus) does not move
  • attachment is still at CEJ

**jxnal epithelium gets longer before it moves apically from attachment at CEJ

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11
Q

gingival diseases modified by systemic factors

A
  • endocrine system changes: puberty, menstrual cycle, pregnancy, diabetes
  • blood dyscrasias
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12
Q

periodontitis associated with…

A
  1. hematologic disorfers
  2. genetic disorder

**NOT DIABETES - clinically looks the same as chronic, so there is no ‘diabetes assoc-periodontitis’

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13
Q

gingival enlargement

A

-frequently without primary inflammatory etiology

  • *often caused by drugs!
  • can occur in absence of attachment loss or superimposped on attach.loss
  • features = enlargement of papillary adn marginal gingiva AND increased probing depth, without change in attachment levels
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14
Q

drugs that cause gingival enlargement

A
  1. calcium channel blockers - nifedipine
  2. anti-convulsants - phenytoin (dilantin, anti-seizure)
  3. cyclosporin (kidney transplant)
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15
Q

non plaque induced gingival lesions - herpetic gingivitis

A

viral infection of oral mucosa/gingiva with clinical features…

  • generalized painful gingiva/mucosa
  • inflammation
  • vesiculation/ulceration
  • lymphadenopathy
  • fever
  • malaise
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16
Q

non plaque induced gingival lesions

A
  1. viral origin = herpetic gingivostomatitis
  2. gingival manifestations of systemic conditions
  3. gingival lesions of genetic origin (gingival fibromastosis)
17
Q

non plaque induced gingival lesions - gingival manifestations of systemic conditions

A
  1. mucotaneous disorders (pemphigus)
  2. allergic reactions (toothpaste, gum, mouthwash, metals)
  3. hereditary gingival fibromatosis