GI Section II: Peritoneal Cavity Flashcards

1
Q

What is the peritoneal cavity?

A

The peritoneal cavity is the space between the various coverings (parietal and visceral peritoneum) in the abdomen. It can be both a conduit for disease and a source of anatomic trivia.

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2
Q

What are the two primary “sacs” of the peritoneal cavity?

A

Greater and Lesser

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3
Q

The “sac” behind the stomach

A

Lesser sac

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4
Q

Lesser sac is a.k.a.?

A

Omental bursa

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5
Q

What is the conduit (channel) between the Greater and LEsser sac?

A

Epiploic foramen (of Winslow)

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6
Q

The greater sac can be divided into parts:

A

Above (supracolic)

——– TRANSVERSE MESOCOLON —-

Below (infracolic)

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7
Q

supracolic and infracolic compartments are connected by these

A

The right and left paracolic gutters along the lateral aspects of the ascending and descending colon

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8
Q

Which paracolic gutter is wider?

A

the right one

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9
Q

How does the femal peritenoeum communicate with the extraperitoneal pelvis?

A

Via the fallopian tubes

(male peritoneum does not communicate)

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10
Q

Barium in HSG =

A

peritonitis

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11
Q

Disease may spread through the abdomen and pelvis by

A

(1) the bloodstream
(2) Lymphatic extension
(3) Direct invasion
(4) Intraperitoneal seeding

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12
Q

allows spread from the stomach, esophagus, and liver

A

Gastrohepatic Ligament

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13
Q

allows spread from the stomach to the splenic hilum

A

Gastrosplenic Ligament

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14
Q

allows spread from the right colon to nodes around the duodenum / pancreas

A

Duodenocolic Ligament

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15
Q

occurs from spread along the peritoneal ligaments and mesenteries

A

(2) Lymphatic extension / (3) Direct invasion

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16
Q

this occurs via the natural flow of fluid

A

Intraperitoneal seeding

17
Q

The natural intraperitoneal fluid mostly flows upward from the pelvis to the upper abdomen via

A

The right (more than left) paracolic gutters

18
Q

Anywhere the fluid tends to spend more time / get caught up (shown by the stars) will be predisposed for serosal-based metastases in the setting o f peritoneal carcinomatosis.

A
19
Q

Pus and ascites tends to flow away from the liver (the opposite of normal flow). As a result the dependent areas

A

right posterior subphrenic recess (RPSA)
anterior subheptic space (ASH)
posterior sub hepatic space / hepatorenal recess / Morrison (M) and pelvic cavity

tend to be involved.

20
Q

This is a gelatinous ascites

A

Pseudomyxoma Peritonei

21
Q
A

Pseudomyxoma Peritonei

“scalloped appearance of the liver.”

22
Q

Causes of Pseudomyxoma Peritonei

A

a. Ruptured mucocele (usually appendix)
b. Intraperitoneal spread of a mucinous neoplasm (Ovary, colon, appendix, pancreas)

It’s usually the appendix (least common is the pancreas).

23
Q

dictates the location of implants

A

natural flow of ascites

24
Q

why is the retrovesical space is the most common spot for peritoneal carcinomatosis?

A

it’s the most dependent part of the peritoneal cavity.

25
Q

“posterior displacement of the bowel from the anterior abdominal wall.”

A

Omental Seeding/Caking

26
Q
A

Omental Seeding/Caking

27
Q

occurs 30-40 years after the initial asbestosis exposure.

A

Primary Peritoneal Mesothelioma

mesothehoma involving the pleura (and it does 75% of the time), but the other 25% of the time it involves the peritoneal surface.

28
Q

Barium Peritonitis

A

The pathology is an attack of the peritoneal barium by the leukocytes which creates a monster inflammatory reaction (often with massive ascites and sometimes hypovolemia and resulting shock).

29
Q

If barium ends up in the systemic circulation it kills via

A

Pulmonary embolism (50% of the time)

Risk is increased in patients with inflammatory bowel or diverticulitis (altered mucosa).

30
Q

Mesenteric Lymphoma

A

is usually NHL

31
Q
A

Mesenteric lymphoma

“Sandwich sign”

lobulated confluent soft tissue mass encasing the mesenteric vessels “sandwiching them.”

32
Q
A

Mesenteric panniculitis

+ Retroperitoneal nodes = lymphoma

Misty mesentery

increased mesenteric fat density centered around the jejunal root, often with a few small nodes

6 month follow up