GI Flashcards
IBS is
chronic idopathic intestinal inflammation
breakdown of IBS
- ulcerative colitis
- Crohn’s disease
- interminate colitis
IBD eiology
immune dysregulation
genetic susceptibility
environmental triggers
pattern of inflammation in ulcerative colitis
continouos and involves mucosa layer only
rectum and colon
crohn’s disease pattern
segmental and transmuaral layer involved
FISTULA PRESENT
can occur from any location from mouth to anus
oral manifestations of IBD
more common in CD
parallels the bowel activity
lip swelling, fissures, cobble-stoning, linear ulceration, mucossal tags, mucogingivitis, gingival hyperplasia, granulomatous inflammation on biopsies, tonsialr crohns disease
GI complications of IBD
colon cancer and risk related to extent and duration of disease
first line treatment in IBD
Mesalamine – anti-inflammatory
steroids and immunomodulators (second) –> NOT FOR MAINTENANCE THERAPY ( like Azathioprine)
biologics - third (stuff like TNF alpha inhibitors)
dental implications of IBD
Anemia – due to iron deficiency
risk of coagulation due to vitamin k defciency
cation with NSAIDS– may exacerabte IBD
therapy / tx modalities can increase risk of infection
medication for GERD
PPI’s – proton pump inhibitors – take before meal
inhibit proton pump of gastric parietal cells
management of dental GERD patietns
potential to aspirate so do not put in full supine position
ineractiono iwth drugs
- antacids - decrease fluoride abdorption and
oral manifestaions of gardners
- supernumerary teeth
- unerupted teeth
- odontomas
- osteomas
Peux Jegher’s associated with ?
multiple hamartomatous polpys in GI tract
oral signs of PJS
fla, blue, gray brown spots
most common on lips and perioral region
buccal mucosa as well
T/F CRC can metastisize to oral cavity
true
