General Surgery Flashcards
Define the acute abdomen.
What physical examination signs suggest its presence?
Acute abdomen = an inflamed peritoneum (peritonitis), usually exhibited by rebound tenderness and involuntary guarding (uncontrolled muscle spasms)
What should you do if you are not sure whether a stable patient has an acute abdomen?
use minimal as needed pain medications (to avoid masking symptoms before you have a diagnosis), perform serial abdominal examinations, and consider a CT scan.
If the patient becomes unstable, proceed to laparoscopy and/or laparotomy.
Name 3 causes of peritonitis that do not require laparotomy or laparoscopy.
Pancreatitis
many cases of diverticulitis
spontaneous bacterial peritonitis.
Specify which conditions are associated with pain and peritonitis in the:
URQ
ULQ
LRQ
LLQ
Epigastric area
What are the classic symptoms and signs of gallstone disease?
What are some physical findings to look for?
postprandial, colicky RUQ pain (usually 15-60 min after a fatty meal)
bloating
+/- N/V
Look for Murphy sign (palpation of the RUQ under the rib cage causes inspiratory arrest as a result of pain)
What are the six Fs of cholecystitis?
How are the demographics of patients with pigment stones different from those with cholesterol stones?
fat, forty, fertile, female, and flatulent
the sixth F is febrile (indicates that patients has developed acute cholecystitis)
pigment (i.e., calcium bilirubinate) stones are classically young patients with hemolytic anemia (e.g., sickle cell disease, hereditary spherocytosis).
How is a clinical suspicion of cholecystitis confirmed and treated?
What are some confounders of the diagnostic tests?
US + Murphy’s sign (variant anatomy + significant obesity can create uncertainty)
Nuclear hepatobiliary scintigraphic study (e.g., hepato-iminodiacetic acid [HIDA] scan) clinches the diagnosis with nonvisualization of the gallbladder
Treatment: pain control + cholecystectomy (antibiotics indicated if infection is suspected)
Define cholangitis and cholecystitis.
How does one differ from the other in terms of cause + presentation?
How is it treated?
Cholangitis - inflammation of the bile ducts; usually caused by biliary obstruction with subsequent bile stasis and infection. Managed with biliary stent placement for unresectable cases. Causes of obstruction include:
- Choledocholithiasis
- malignancy
- Autoimmune cholangitis (e.g., sclerosing cholangitis)
- primary infection (e.g., Clonorchis sinensis) are other causes.
Cholecystitis - inflammation of the gallbladder; usually caused by gall stones; presents with Charcot triad: (1) RUQ pain, (2) fever or rigors, and (3) jaundice. Managed with cholecystectomy
Describe the classic presentation of appendicitis.
How is it diagnosed and treated?
presents in 10-30 yo with a history of crampy, poorly localized periumbilical pain followed by N/V. Pain localizes to the RLQ with peritoneal signs and worsening of N/V
Look for:
- Rovsing sign: when a different quadrant is palpated and then quickly release, patient feels pain at McBurney point (2/3 of the way from the umbilicus to the ASIS).
- McBurney point: area of maximal tenderness in the RLQ and the site where an open appendectomy incision is made.
CT is increasingly used to confirm the diagnosis before surgery in stable patients
What is the cause of LLQ pain and fever in a patient older than 50 years until proved otherwise?
How is it treated?
Diverticulitis
treat with broad-spectrum antibiotics (e.g., ciprofloxacin plus metronidazole), NPO, and a NGT if N/V are present.
What tests should and should not be done to confirm possible cases of diverticulitis?
What test does every patient need after a treated episode of diverticulitis?
should: CT
should not: colonoscopy (due to increased risk of colon rupture), barium enema
Describe the typical history, physical examination, and laboratory findings of pancreatitis.
How is it treated?
What 2 complications should you be concerned about?
- epigastric pain that radiates to the back in an alcohol abuser or a patient with a history (or risk factors) of gallstones.
- elevated serum amylase and/or lipase
- decreased bowel sounds, localized ileus (“sentinel” loop of bowel on abdominal radiograph) and nausea, vomiting, and/or anorexia
Treatment: supportive: pain control (hydromorphone or fentanyl), NPO, NGT if N/V present, IVF
Complications: pseudocyst and pancreatic abscess
Describe the usual history of a perforated ulcer. How is it diagnosed and treated?
look for a history of peptic ulcer disease
diagnosis: KUB, may cause increased amylase and lipase.
treat with surgery
What are the hallmarks of small bowel obstruction?
How is it diagnosed and treated?
hallmarks: bilious vomiting (early symptom), abdominal distention, constipation, hyperactive bowel sounds (high-pitched, rushing sounds), and usually poorly localized abdominal pain, history of abdominal surgery
diagnosis: multiple air-fluid levels on Xray, CT scan to confirm if diagnosis is uncertain
treatment: ex-lap
What are the common causes of a small bowel obstruction?
adhesions secondary to prior surgery
Crohns
incarcerated hernia
Meckels diverticulum
intussusception
Describe the signs and symptoms of large bowel obstruction.
What are the top 4 causes? How is it treated?
gradually increasing abdominal pain, abdominal distention, constipation, and feculent vomiting (late symptom).
causes: diverticulitis, colon cancer, volvulus, Hirschsprung
treatment: NPO, NGT for N/V, endoscope decompression if it is a sigmoid vovulus, surgery to relief the decompression if it is refractory
List and differentiate the three common types of groin hernias.
Of the three, which one is the most susceptible to incarceration and strangulation?
3 = most susceptibe to incarceration and strangulation; all treated with elective surgical repair if symptomatic
- Indirect hernias - most common in both sexes and all age groups; (+) hernia sac travels through the inner + outer inguinal rings (protrusion begins lateral to the inferior epigastric vessels) and into the scrotum or labia because of a patent processus vaginalis (congenital defect).
- Direct hernias (ø sac) - protrude medial to the inferior epigastric vessels because of weakness in the abdominal musculature of Hesselbach’s triangle.
- Femoral hernias - common in women; (no sac) goes through the femoral ring onto the anterior thigh (located below the inguinal ring).