General surgery (2) (Common conditions) Flashcards

1
Q

intestinal obstruction summary

A

Passage of food, fluids and gas become blocked.

  • Small bowel more common than large bowel
  • Results in build up of gas and faecal matter proximal to obstruction
    • Back pressure- vomiting and dilatation of proximal intestines
    • Surgical emergency
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2
Q

how can intestinal obstruction cause dehydration

A
  • Obstruction also reduces fluid reabsorption in the colon- fluid loss from intravascular space – hypovolaemia
    • This is called third-spacing
    • Higher up the obstruction the worse the fluid losses- less bowel where the fluid can be reabsorbed
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3
Q

3 main causes of intestinal obstruction

A

The big three (HAM)
• Hernias (small bowel)
• Adhesions (small bowel)
• Malignancy (large bowel)

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4
Q

causes of small bowel obstruction

A
  • Hernias
  • Adhesions
    • Scar tissue that binds abdominal contents together
    • Causing kinking or squeezing of the bowel
    • Causes
      • Surgery
      • Peritonitis
      • Infection
      • Endometriosis
  • Stricture
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5
Q

causes of large bowel obstruction

A
  • Malignancy
  • Volvulus- bowel twists on self
  • Diverticular disease
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6
Q

causes of small and large bowel obsturction

A
  • Stricture e.g. secondary to Crohns
  • Intussusception (in young children 6m-2y)
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7
Q

RF for obstruction

A
  • Abdominal surgery
  • Cancer
  • Hernias
  • Crohns disease
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8
Q

presentation of obstruction

A
  • Vomiting (particularly green bilious vomiting)
    • Early- upper
    • Later- lower
  • Abdominal distention
  • Diffuse abdominal pain
  • Absolute constipation and lack of flatulence (early in lower,

Late in upper)

  • “Tinkling” bowel sounds may be heard in early

bowel obstruction

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9
Q

investigations of obstruction

A
  • Abdominal X-ray – distended loops of bowel
    • Upper limits of normal diameter of bowel
      • 3cm small. Bowel
      • 6cm colon
      • 9cm caecum
  • May skip straight to contrast abdominal CT- confirm diagnosis and establish site and if perforation has occurred- modality of choice
  • Blood tests: U and E (electrolytes), venous blood gas (metabolic alkalosis due to loss of HCL in vomit), vowel ischaemia- raised lactate)
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10
Q

small bowel AXR

A
  • Dilated >3cm
  • Central, valvulae conniventes
  • String of pearls sign
  • Paucity of gas in large bowel (i.e. not in the rectum)
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11
Q

large bowel obstruction AXR

A
  • Colon >6cm, caecum >9cm
  • Peripheral and haustra (don’t extend full width)
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12
Q

initial management of obstruction

A
  • ABCDE
    • Hypovolaemia shock due to third spacing
    • Bowel ischaemia
    • Bowel perforation
    • Sepsis
  • Nill by mouth
  • Analgesia, catheterise
  • IV fluids to hydrate and correct electrolyte imbalances
  • NG tube with free drainage to allow stomach contents to freely drain and reduce risk of vomiting and aspiration
  • Conservative treatment if adhesions or volvulus, where this fails- surgery
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13
Q

surgical intervention for obsturction

A

intervention (closed loop or ischaemia- pain)

  • Definitive management – either laparoscopy or laparotomy
    • Exploratory surgery in patients with an unclear underlying cause
    • Adhesiolysis to treat adhesions
    • Hernia repair
    • Emergency resection of the obstructing tumour- may need stoma
      • Stents may be inserted- holds tumour out of the way
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14
Q

volvulus on x-ray

A
  • Sigmoid volvulus- coffee bean appearance
  • Caecal volvulus- fetal appearance
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15
Q

Closed loop obstruction

A

Where there are two points of obstruction along the bowel; meaning that there is a middle section sandwiched between two points of obstruction.

Result: contents of section of bowel does not have open end to drain- bowel continues to expand- ischaemia and perforation

This might happen with:

  • Adhesions that compress two areas of bowel
  • Hernias that isolate a section of bowel blocking either end
  • Volvulus where the twist isolates a section of intestine
  • A single point of obstruction in the large bowel, with an ileocaecal valve that is competent
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16
Q

what is the significance of a competent ileocaecal valve

A
  • A competent ileocaecal valve does not allow any movement back into the ileum from the caecum. When there is a large bowel obstruction and a competent ileocaecal valve, a section of bowel becomes isolated and the contents cannot flow in either direction.
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17
Q

The complications of bowel obstruction include:

*

A
  • Bowel ischaemia
  • Bowel perforation leading to faecal peritonitis (high mortality)
  • Dehydration and renal impairment
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18
Q

hernias (summary)

A

Occur when there is a weak point in a cavity wall, affecting muscle or fascial- this weakness allows body organ e.g. bowel that would. Normally be contained within that cavity to be passed through the cavity wall

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19
Q

types of hernia

A
  • Inguinal
  • Femoral
  • Umbilical
  • Incisional
  • Hiatus hernia

Specific abdominal hernias

  • Richter’s hernia
    • Specific situation
    • Only part of bowel wall and lumen herniate through defect, other side of that section of bowel remains within peritoneal cavity
    • Risk of strangulation
    • Surgical emergency
  • Maydls hernia
    • Two diff loops of bowel are contained within hernia
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20
Q

assessing a hernia

A
  • does it go back in by itself or is it reducible
  • get patient to cough and feel over
  • get patient to lie down
  • pain?

When assessing a hernia, always comment on the size of the neck/defect (narrow or wide), as this will help formulate a risk assessment (risk of strangulation reduced if wider neck) and management plan for the hernia (such as how urgently they need to be operated on).

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21
Q

causes of hernias

A

anything which increases pressure in abdomen

  • Lifting heavy objects
  • Diarrhoea or constipation
  • Persistent coughing
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22
Q

risk factors of henrias

A
  • Pregnancy
  • Weight-lifting
  • Constipation
  • Obesity
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23
Q

presentation of hernia

A
  • A soft lump protruding from the abdominal wall
  • The lump may be reducible (it can be pushed back into the normal place)
  • The lump may protrude on coughing (raising intra-abdominal pressure) or standing (pulled out by gravity)
  • Aching, pulling or dragging sensation
24
Q

complications of hernias

A

incarceration

obstruction

strangulation

25
Q

incarceration vs strangulation

A
  • Incarceration
    • Where hernia cannot be reduced back into proper position (irreducible)
    • Can lead to obstruction and strangulation of hernia- bowel ischemia
  • Strangulation
    • Non reducible and base of hernia becomes so tight it cuts of blood supply- ischaemia
    • Significant pain and tenderness
    • Surgical emergency
26
Q

obstruction caused by hernia

A
  • Blockage in passage of faeces through bowel
  • Vomiting, pain and absolute constipation
27
Q

investigation fo hernia

A
  • Clinical examination
  • Ultrasound or CT
  • If negative but high clinical suspicion- MRI
28
Q

conservative management of hernia

A
  • Leaving hernia
  • Appropriate when hernia is wide neck and if pt not a good candidate for surgery
29
Q

hernia surgical repair : tension free repair

A
  • Placing mesh over the defect in the abdominal wall
  • Mesh is sutured to the muscles and tissues on either side of the defect, covering and preventing herniation of the cavity contents
  • Over time tissue grows into mesh- more support
    • Complications- chronic pain due to mesh
    • Lower recurrent rate than tension repair
30
Q

hernia surgical repair : tension repair

A
  • Suture the muscles and tissues on either side of the defect back together
  • Rarely performed
  • Complications – pain
  • High recurrence rate of hernia
31
Q

gallstones

A

Stones form in the gallbladder and are made from concentrate bile from the bile duct (mostly cholesterol). Leads to complications such as:

  • Biliary colic
  • Acute cholecystitis
  • Acute cholangitis
  • Acute pancreatitis (when stones block the pancreatic duct)
32
Q

risk factors for gallstones

A
  1. Fs
    * Fat, Fair, Female,Forty
33
Q

presentation of gallstones general

A
  • Asymptomatic (sometimes)
  • Biliary colic
    • After meal
    • RUQ pain/ N+V
    • 30-8 hours
  • Acute cholecystitis
    • Positive murphy’s sign
  • Ascending cholangitis
    • Charcots triad-jaundice
34
Q

basic anatomy of bile duct

A
  • The right hepatic duct and left hepatic duct leave the liver and join together to become the common hepatic duct.
  • The cystic duct from the gallbladder joins the common hepatic duct halfway along.
  • The pancreatic duct from the pancreas joins with the common bile duct further along.
  • When the common bile duct and the pancreatic duct join they become the ampulla of Vater, which then opens into the duodenum.
  • The sphincter of Oddi is a ring of muscle surrounding the ampulla of Vater that controls the flow of bile and pancreatic secretions into the duodenum.
35
Q

investigations for gallstones

A
  • LFTS
  • US
  • MRCP
  • ERCP
  • CT scan
36
Q

liver function tests and gallstones

A
  • Bilirubin
    • Raised bilirubin (jaundice) due to blockage in the in common bile ducts
    • Pale stool and dark urine
    • Causes: gallstones, chlangiocarcinoma or tumour of pancreas
    • Alkaline phosphatase
      • Biliary obstruction
      • Also- liver or bone problems and pregnancy
      • Gamma-glutamyl transferase GTT to check biliary problem
    • Alanine aminotransferase (ALT) and Aspartate aminotransferase (AST)
      • Hepatocellular injury
      • ALT better
37
Q

US and and gallstones

A
  • Can locate gallstone
  • Limited by: pt weight, gaseosus bowel obstructing view and discomfort
38
Q

MRCP

A

Magnetic resonance cholangio-pancreatography (MRCP)

  • MRI scan- detailed image of biliary system
39
Q

ERCP

A
  • Endoscopic retrograde cholangio- pancreatography
    • Involves inserting endoscope down oesophagus, past stomach and into the sphincter of oddi up into common bile duct
    • Main indication: clear stones in bile duct
    • Allows operator to:
      • Inject contrast and take x-rays
      • Clear stones
      • Insert stents
      • Biopsy of tumour
    • Complications
      • Excessive bleeding
      • Cholangitis (infection)
      • Pancreatitis
40
Q

CT scans and gallstones

A
  • Less useful
  • Good for differentiations and complications
41
Q

key definitions for gallstones

A
  • Cholestasis: blockage to the flow of bile
  • Cholelithiasis: gallstone(s) are present
  • Choledocholithiasis: gallstone(s) in the bile duct
  • Biliary colic: intermittent right upper quadrant pain caused by gallstones irritating bile ducts
  • Cholecystitis: inflammation of the gallbladder
  • Cholangitis: inflammation of the bile ducts
  • Gallbladder empyema: pus in the gallbladder
  • Cholecystectomy: surgical removal of the gallbladder
  • Cholecystostomy: inserting a drain into the gallbladder
42
Q

three different and worsening presentations of gallstones

A

biliary colic → acute cholecystitis → ascending cholangitis

43
Q

biliary colic pathophysiology

A
  • Due to cholecystokinin (CCK) release after meal, which causes the gall bladder contract and push a gallstone up against the neck of the gall bladder- temporary obstruction of biliary duct
  • Gall stones happily sitting within gall bladder, but can cause sudden onset of RUQ pain typically a few hours after eating a fatty meal
44
Q

presentation of biliary colic

A
  • Severe, colicky epigastric or right upper quadrant pain
  • Radiates to back
  • Often triggered by meals (particularly high fat meals)
  • Lasting between 30 minutes and 8 hours
  • May be associated with nausea and vomiting
45
Q

treatment of biliary colic

A

pain relief and elective cholecystectomy removal

46
Q

acute cholecysittis pathophysiology

A

Inflammation of gallbladder caused by full impaction of stone in cystic duct- preventing gallbladder draining→ can cause dilatation of the gall bladder

47
Q

presentation of acute cholecysitits

A
  • RUQ pain
  • Fever, N and V
  • Tachycardia
  • Raised CRP
  • Positive Murphy signà place a hand on right side of the patients stomach and ask them to take a deep breathe in- will push gall bladder down and cause them to take a sharp breathe in pain (wont happen on left hand side)
  • Pain which radiates to shoulder (shared phrenic nerve innervation)
  • Imaging
    • MRCP or US
48
Q

treatment for acute cholecysitits

A
  • pain relief and Ab
  • Cholecystectomy
49
Q

gallbladder empyema

A

infected tissue and puss in gallbladder- IV antibiotics and surgery

50
Q

ascending cholangitis

A

Infection and inflammation in bile duct- high mortality rate

51
Q

causes of ascending cholangitis

A
  • Gallstone in CBD or infection due to ERCP
  • E.coli, klebsiella
52
Q

presentation of ascending cholangitis

A

charcots triad

53
Q

charcots triad

A
  • Inflammation (pyrexia and rigors)
  • RUQ pain,
  • jaundice (when stone reaches common bile duct)
54
Q

management of ascending cholangitis

A
  • acute management of sepsis and acute abdomen (BUFALO)
  • imaging to diagnose CBD stone and cholangitis
    • most sensitive MRCP or endoscopic ultrasound
  • ERCP required for stone removal
  • Percutaneous transhepatic cholangiogram
55
Q

if no symptoms of gall stones

A

no intervention required

56
Q

cholecystectomy

A
  • Surgical removal of the gall bladder
  • When pts symptomatic or stone leading to acute cholecystitis
  • Stones removed by ERCP or during surgery
  • Laparoscopic cholecystectomy (keyhole) preferred) to open (kocher incision)
    • Faster recovery less complications
      • Bleeding, infection, pain scars
      • Damage to bile duct- stricture
      • Stones left In bile duct
      • Anaesthetic risk
      • VTE
      • Post cholecystectomy syndrome
57
Q

post-cholecystectomy syndrome

A

involves a group of non-specific symptoms that can occur after a cholecystectomy. They may be attributed to changes in the bile flow after removal of the gallbladder. Symptoms often improve with time. Symptoms include:

  • Diarrhoea
  • Indigestion
  • Epigastric or right upper quadrant pain and discomfort
  • Nausea
  • Intolerance of fatty foods
  • Flatulence