Gen Surg: Intestinal obstruction Flashcards

1
Q

What is the definition of intestinal obstruction?

A

Failure of downward passage of intestinal contents

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2
Q

What is meant by dynamic intestinal obstruction?

A

There is increasing peristalsis working against an obstructing agent

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3
Q

What is adynamic intestinal obstruction?

A

Peristalsis is absent or ineffective and there are no effective propulsive waves

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4
Q

What is a simple obstruction?

A

Obstruction of the intestinal lumen without interference with its blodd supply

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5
Q

What happens above/below the site of obstruction in someone with a simple obstruction?

A

Peristalsis + distention (due to gas, GI content and fluid build up)

Below the obstruction - collapsed, immobile, pale

@ obstruction = perforation can occur

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6
Q

What are causes of death in simple bowel obstruction?

A
  • Fluid & electrolyte imbalance - from third space loss and dehydration
  • Peritonitis - proliferation of bacteria proximal to obstruction leading to bacterial translocation
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7
Q

Causes of simple bowel obstruction

A

Inside the lumen - gallstones, impaced faeces

In the wall - strictures, tumours, CD, diverticulitis

Outside the wall - ADHESIONS, hernias

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8
Q

What is strangulation?

A

Intestinal obstruction with persistent interference of the blood supply

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9
Q

What are causes of intestinal strangulation?

A
  • Strangulated hernia
  • Intussuception
  • Adhesive intestinal obstruciton
  • Volvulus
  • Vascular occlusions
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10
Q

What are the pathophysiological consequences of strangulation?

A
  • Venous return is impaired - venous supply lower pressure than arterial supply, strangulated bowel and its mesentery look congested
  • Serosanguinous fluid formation - accumulated inside the peritoneal cavity
  • Arterial supply is impaired - colour of the affected segment becomes black
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11
Q

When would you suspect strangulation?

A

Internal strangulation

  • Severe pain thats never completely absent between attacks
  • Shock
  • Tenderness and rigidity with rebound tenderness
  • GI suction fails to releive pain

Strangulated external hernia

  • Hernia swelling that is tense, tender, irreducible and no expansable impulse on cough (hernias are painless unless complicated)
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12
Q

What are causes of death in strangulation?

A
  • Peritonitis due to perforation - bacterial translocation and impairment of barrier function of intestinal mucosa
  • Hypovolaemic shock - third space loss
  • Sepsis
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13
Q

What is a closed loop obstruction?

A

When some part of the gut is closed at both ends - THIS IS DANGEROUS AND IS A SURGICAL EMERGENCY eg competant ileocaecal valve

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14
Q

What is typically the cause of a closed loop obstruction

A

Sigmoid volvus

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15
Q

At what diameter would you be concerned of caecal perforation?

A

10cm

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16
Q

What are the complications of bowel obstruction?

A
  1. SEPSIS - venous compression, bowel cells die, decreased peristalsis, bacteria enter circulation
  2. SHOCK (hypotension) - venous compression, fluid secretion, decreased H2O, decreased electrolytes, hypotension
  3. SHOCK (hypovolaemia) - bowel distension, decreased vomitting response, decreased H2O, decreased fluids

Bowel ischaemia from venous compression

Perforation due to increased air

Sepsis due to perofation/dissemination

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17
Q

What are causes of small bowel obstruction?

A
  • Adhesions
  • Hernias
  • Malignant tumours
  • Crohn’s disease
  • Intussusception
  • Gallstone ileus
  • Paralytic ileus
  • Miscellaneous (bezoars)
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18
Q

What is the most common cause of small bowel obstruction?

A

Adhesions

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19
Q

What are abdominal adhesions?

A

Adhesions are bands of ‘scar’ tissue in various degrees of development. They are part of a normal intra-abdominal repair process following a variety of insults - handling of bowel, contact of internal organs with foreign objects, cuts involving internal organs.

Can also result from appendix rupture, infection, radiation, abdo infections

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20
Q

Indications for surgery in adhesive intestinal obstruction

A

Suspected strangulation

Failure of conservative management

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21
Q

What is the pathophysiology behind abdominal adhesion formation?

A

Peritoneum is ‘injured’ -> reparative process similar to that seen following the formation or in prevention of a thrombus.

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22
Q

What types of internal hernias can occur which can lead to small bowel obstruction?

A
  • Paraduodenal
  • Transmesocolic
  • Transmesenteric
  • Omental
  • Retroanastomotic - bowel is trapped behind a surgical anastomosis
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23
Q

What types of benign tumours of the small intestine can cause SBO?

A
  • Hyperplastic polyps
  • Lipomas
  • Adenomas - including Peutz-Jeghers polyps
  • G/I stromal tumors
  • Hemangiomas
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24
Q

What secondary malignancies can present as SBO?

A
  • Ovarian
  • Stomach
  • Pancreas
  • Colonic
  • Malignant melanoma
  • Lung
  • Breast
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25
Q

How can crohn’s disease lead to SBO?

A

Can cause strictures. May also have adhesions from previous surgeries

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26
Q

What is paralytic ileus?

A

Cessation of peristalsis due to failure of neuromuscular mechanism of the intestine causing accumulation of fluid and gas in the intestine

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27
Q

What are causes of paralytic ileus?

A
  • Post surgery especially for peritoneal sepsis
  • Peritonitis
  • Drugs - TCAs, anticholinergic, opiod mets
  • Spinal injury
  • Electrolyte imbalance - hypokalaemia, hyponatraemia, uraemia (esp magnesium and potassium_
  • Extensive handling of the bowel at operation
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28
Q

How does paralytic ileus increase the risk of adhesion formation?

A
  • Intestinal segments have more prolonged contact, which allows fibrous adhesions to form
  • Intestinal distention causes serosal injury and ischemia
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29
Q

Presentation paralytic ileus

A

Progressive abdo distension, vominting, absolute constipation, failure to pass flatus.

Mild abdo discomfort.

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30
Q

Management paralytic ileus

A

TREAT UNDERLYING CAUSE

Gi suction

Restore fluid and electrolyte balance, monitor for AKI

Encourage mobilisation

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31
Q

What are the pathophysiological consequences of small bowel obstruction?

A
  • Proximal dilatation of intestine - due to accumulation of GI secretions and swallowed air
  • Stimulation of columnar cell secretory activity - increase in intra-lumenal fluid
  • Increased peristalsis above and below the obstruction -
  • Early frequent loose stools and flatus
  • Increased intraluminal pressure
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32
Q

What is the result of small bowel obstruction in terms of fluid balance?

A

Hypovolaemia - due to:

  1. Compression of mucosal lymphatics -> lymphoedema of the bowel wall
  2. High intraluminal hydrostatic pressures -> increased hydrostatic pressure in the capillary beds
  3. Massive loss into the third space - fluid, electrolytes, and proteins (into lumen)
  4. Vomiting + loss of normal fluid intake
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33
Q

How does intestinal obstruction affect intestinal flora?

A
  • Proliferation proximal to obstruction
  • Microvascular changes in bowel wall -> Translocation of bacteria to mesenteric lymph nodes
  • Resultant bacteraemia
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34
Q

What are symptoms of small bowel obstruction?

A
  • Pain
    • ​Peristaltic pain
    • Colicky initially, constant suggests strangulation/impending perforation
  • Vomiting
  • Abdominal distention
    • Jejunal obstruction - minimal
    • Ileal obstruction - central
  • Absolute constipation - late symptom
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35
Q

What are signs of SBO?

A
  • Features on inpection - surgical scars, visible peristalsis
  • Dehydration/Signs of shock
  • Abdominal distention
  • Bowel sounds
    • Increased/borborygmus - early dynamic
    • Decreased/absent - paralytic and late mechanical
  • May have signs of peritonism
  • Empty rectum on PR
  • May have herniation - non-reducible

Focal tenderness, guarding, rebound tenderness when ischaemic

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36
Q

What are the characteristic features of abdominal pain in intestinal obstruction?

A

Generalized abdominal colicky pain - Each attack lasts for few minuets then gradually disappears, with periouds of relief in between

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37
Q

When is distention less prominent in SBO?

A

When the obstruction is more proximal

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38
Q

When is distention more prominent in SBO?

A

In distal obstruction

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39
Q

How does the timing of vomiting roughly indicate where a SBO might be?

A

The more proximal the obstruction, the earlier vomiting will occur:

  • Jejunal Obstruction - Vomiting occurs with the first and each attack of pain
  • Ileal Obstruction - Vomiting is delayed for few hours; then it occurs with each attack of pain
40
Q

If vomiting occured early on in someone with suspected SBO, what might this suggest about where the problem is in the small intestine?

A

High - e.g. jejunal obstruction

41
Q

If feculent vomiting occured late after someone presented with abdominal pain and marked abdominal distention, where might you suspect the obstruction is occuring?

A

Low - e.g. ileum

42
Q

If someone had jejunal obstruction, how distended would their abdomen be?

A

Minimally distended

43
Q

If someone had ileal obstruction, how much abdominal distention might they have?

A

Prominent central distention

44
Q

If someone had distention of the flanks, what might this indicate about where the obstruction is occuring?

A

Colonic distention only - LBO with competent ileocaecal valve

45
Q

If someone had generalised distention of the abdomen, what might this indicate as to where the obstruction is occuring?

A

Distended small bowel & colon - LBO with incompetent ileocaecal valve

46
Q

What featuress would make you think that someone had a stangulated obstruction?

A
  • Pain - more severe and never completely absent in between the attacks
  • Shock - usually present and progressive
  • Tenderness & Rigidity - Localized tenderness & rebound tenderness
  • Tense, tender, irreducible, non-expansile external hernia
47
Q

What are some causes of intestinal obstruction without absolute constipation?

A
  • Richter’s hernia
    • Involves bowel wall only
    • Strangulation without obstruction
  • Gallstone ileus
  • Mesenteric vascular occlusion
  • Pelvic abscess causing obstruction
48
Q

What is gallstone ileus

A

GS impaction at terminal ileum.

Repeated/ongoing inflammation makes walls of gallbladder oedematous so it adheres to duodenum (most commpnly) and fistula forms. Some gallstones pass fine but the big ones can get stuck in terminal ileum or ileocaecal valve (mechanical obstruction).

49
Q

Diagnosis of gallstone ileus

A

Rigler’s triad - penumobila (air in bile ducts), evidence SBO, gallstone outside gallbladder

50
Q

What are symptoms of LBO?

A
  • Absolute constipation
  • Lower abdominal pain
    • Flanks - colonic distension only, LBO with incompetant ileo-caecal valve
    • Generalised - distended small bowel and colon, LBO with incompetent ileo-caecal valve
  • Vomiting a late manifestation
  • Features of ischaemia/strangulation
    • Acute toxicity
    • Fever
    • Chills
    • Hypotension
    • Confusion
51
Q

What signs can occur in LBO?

A
  • Features on inpection - surgical scars, visible peristalsis, “step-ladder” appearance due to distended loops over each other
    • Dehydration/Signs of shock
    • Abdominal distention - Flanks/generalised
  • Abdominal Mass
  • Bowel sounds
    • Increased/borborygmus - early dynamic
    • Decreased/absent - paralytic and late mechanical
  • May have signs of peritonism
  • Tender LIF - diverticular disease
  • Tender RIF +/- mass - impending ischaemia
    • ​NO TENDERNESS or rigidity with simple obstruction (strangulation)
    • Focal tenderness, guarding, rebound tenderness with ischaemia
  • PR - rectal mass, blood, mucus
  • May have herniation - non-reducible
52
Q

What are the 2 most common causes of SBO?

A

Adhesions and hernias

53
Q

What are causes of large bowel obstruction?

A
  • Colon cancer
  • Constipation
  • Diverticular stricture
  • Volvulus
54
Q

What investigations would you perform if you suspected intestinal obstruction?

A
  • Bedside
    • NEWS score,
    • Fluid status
    • Urine output
  • Bloods
    • FBC (low Hb? anaemia suspect colon cancer) (raised WCC? occurs early on in strangulation - bacterial translocation),
    • U+E’s (dehydration due to 3rd space loss, electrolye imbalance),
    • LFTs (mets?),
    • consider ABG (high lactate eg ischaemia)
    • CRP
    • G+S
    • Calcium and TFTs to exclude pseudo-obstruction
  • Imaging - AXR, Erect CXR, Gastrografin follow through/enema (LBO), Consider CT
55
Q

What might you see on AXR in someone with suspected small bowel obstruction?

A
  • Dilated loops proximal to the obstruction - predominantly central dilated loops
  • Valvulae conniventes are visible
  • Air-fluid levels if the study is erect
  • Gasless bowel
    • Partial: gas throughout the abdomen and into the rectum.
    • Complete: no distal gas, and staggered air-fluid levels.
56
Q

What might you see on CXR in someone with an intestinal obstruction?

A

Free air under the diaphragm - due to perforation

57
Q

What is important to include in your examination of someone with features of intestinal obstruction?

A

PR and hernial orifice exam

58
Q

What is a gastrografin follow through study?

A

Involves administration of contrast material into the stomach. The subsequent assessment of degree of passage of this material, using serial x-rays, can provide information regarding the presence and location of the obstruction within the GI tract:

  • Partial SBO - medium passes into rectum.
  • Complete SBO - medium does not pass into rectum and is held up at site of obstruction.

Not done in acute presentations.

59
Q

What might you find on U+E’s in someone with bowel obstruction?

A
  • Electrolyte imbalance - hyponatraemia, hypokalaemia
  • Hyperuraemia - Renal failure
60
Q

What might you use a gastrografin enema to look for?

A

Lower bowel obstruction

61
Q

What might you use a CT to look for in the context of bowel obstruction?

A
  • Confirm the diagnosis if transition point is seen
  • Identify the level (SBO/LBO)
  • Find the cause
  • Staging in obstruction secondary to malignancy
62
Q

What are causes of obstruction in newborn babies?

A
  • Imperforate anus
  • Congenital atresia/stenosis of the gut
  • Volvulus
63
Q

What is the most common cause of obstruction in a child age 3-12 months old?

A

Intussuception

64
Q

Causes of intussusception in adults

A

Polyp

Submucous lipoma

Polypoidal tumours

Inverted merckel’s diverticulum

65
Q

Presentation intussusception chidlren

A

Recurrent episodes of screaming and drawin up of legs

Bilious vomiting

Redcurrent jelly stools

Sausage shaped mass

Most common @ ileocaecal valve

66
Q

How would you manage someone with complete/complicated/stangulated SBO?

A
  • ABCDE - give 100% oxygen
  • NBM
  • NG decompression (and to stop vomiting/prevent aspiration)
  • IV fluids - aggressive as pateint may be very dehydrated
  • Catheterise
  • Analgesia
  • Investigations - AXR, erect CXR, Bloods, monitoring/fluid status
  • Surgery/Emergency surgery - laparoscopy
    • Stangulation is an emergency
67
Q

What are indications for early surgery in bowel obstruction?

A
  • Obstructed hernia
  • Suspected strangulation
  • Small bowel obstruction in a ‘virgin abdomen’
  • Failure of conservative Rx in adhesive SBO (up to 72hr)
  • Obstructing tumours on CT
68
Q

How would you manage someone with complete/complicated/stangulation obstruction who wasn’t fit for surgery?

A
  • ABCDE
  • NG decompression + fluid resus
  • Antiemetics
  • Antispasmodics
69
Q

How would you manage partial SBO?

A
  • ABCDE
  • NG decompression
  • IV fluid resus
  • Analgesia
  • Anti-emetics
  • Consider surgery if not resolved within 48-72 hrs
70
Q

What are the differences in radiological appearence of the jejunum, ileum and colon?

A
71
Q

How would you treat intussusception in an adult?

A

Laparotomy - resection +/- anastamosis

72
Q

What is volvus?

A

A loop of intestine around its mesentery, resulting in a closed loop bowel obstruction. Affected bowel can become ischaemic due to compromised blood supply, rapidly leading to necrosis and perforation

73
Q

What are the different types of volvulus that can occur?

A
  • Volvulus neonatorum
  • Volvulus of small intestine
  • Caecal volvulus
  • Sigmoid volvulus
74
Q

Why is sigmoid volvus the most common volvus?

A

It has a v long mesentery

75
Q

Risk factors for development of volvus

A

Neuropsychaitric disorders

Laxative abuse

Male gender

DM

Increased age (especially common elderly male)

Chronic constipation

Previous abdo surgery

76
Q

What are features of a sigmoid volvulus?

A
  • Sudden left sided abdo pain
  • Adbo distention
  • Absolute constipation

(signs of bowel obstruction)

77
Q

What sign is seen on xray in sigmoid volvus?

A

Coffee bean sign

78
Q

How would you manage a sigmoid volvulus?

A
  • General management
    • ​+ decompression with sigmoidoscopy
  • Surgery (if repeated failed attempts at decompression, necrotic bowel noted, suspected/proven peritonitis)
    • Emergency: Untwisting by flexible sigmoidoscopy -> Sigmoid resection in fit patients
    • Failed sigmoidoscopy -> open surgery
      • ​Primary anstomoses OR hartmans
79
Q

If someone was found to have a large bowel obstruction on AXR, what investigation would you consider doing next?

A

Gastrografin enema - determine whether carcinoma, ogilives syndrome or diverticular disease

80
Q

If someone was found to have free gas under the abdomen on AXR, How would you manage them?

A

Surgery

81
Q

What are contraindications for primary anastamosis?

A
  • Poor bowel preparation (on-table lavage *may be indicated)
  • Friable bowel
  • Circulatory instability
  • Synchronous or multiple tumours**
  • Peritonitis present
  • Immunocompromised patient
  • Previous radiation (often pelvic)
  • Pelvic abscess present (debatable)
82
Q

What is OGilvie syndrome?

A

Clinical syndrome with symptoms, signs & AXR appearance of LBO but with no identifiable mechanical obstruction

Thought to be an interruption of autonmic nervous system to colon, resulting in absense of smooth muscle action in bowel wall

83
Q

What are predisposing factors to developing Ogilivie syndrome?

A
  • Puerperium
  • Electrolyte imbalance
  • Pelvic surgery
  • Trauma
  • Malignancy
  • Cardiorespiratory disorder
  • Neurological disorder
  • Systemic infection
  • Medications
    • ​Opiods
    • Calcium channel blockers
    • Antidepressants
84
Q

How do individuals with Ogilvie syndrome present?

A

With features of mechanical bowel obstruction

85
Q

How would you manage Ogilvie syndrome?

A
  • Neostigmine - inhibits cholinesterase
  • Colonoscopic decompression
86
Q

What are the key things that you need to establish in suspected bowel obstruction?

A
  1. Is it small or large bowel?
  2. Is it dynamic/adynamic (ileus vs mechanical)?
  3. Is it simple/closed loop/strangulated?
87
Q

What is the cardinal sign of a strangulated obstruction?

A

Peristonism

88
Q

What is the pathophysiology of Ogilvie’s syndrome?

A

Most likely caused by a disturbance of the autonomic nervous system:

  • Reflex sympathetic stimulation inhibiting colon and/or
  • Interruption of parasympathetic influence of S2-S4

This leads to colonic dilatation and colonic atony

89
Q

What mnemonic can be used to remember the main symptoms of obstruction?

A

PV D+C

  • Pain
  • Vomiting
  • Distention
  • Constipation
90
Q

What mnemonic can be used to remember the causes of small bowel obstruction?

A

SHAVIT

  • Stones (gall stone ileus )
  • Hernias (always examine hernial orifices!)
  • Adhesions (can occur very early and very late after surgery)
  • Volvulus
  • Intusssusception / IBD
  • Tumour (1º adenocarcinomas rare in small bowel)

**Adhesions and hernias are the commonest causes of small bowel obstruction and should be considered before more unusual causes.

91
Q

Describe causes of bowel obstruction in terms of intraluminal, mural, extramural

A

Intraluminal

  • Gallstone ileus
  • Foreign body
  • Faecal impaction

Mural

  • Carcinoma,
  • Inflammatory strictures
  • Intussusception
  • Diverticular strictures
  • Meckles diverticulum
  • Lymphoma

Extramural

  • Hernias
  • Adhesions
  • Periteoneal masses
  • Volvus
92
Q

What is mesenteric vascular occlusion?

A

Occlusion of the superior mesenteric or one of it’s branches

93
Q

Risk factors for mesenteric vascular occlusion

A

Arterial embolism - AF, SBE

Arterial thrombosis - PCV, atherosclerosis, OCP

Venous thrombosis - PHT

MORE COMMON IN ELDERLY

94
Q

Presentation mesenteric vascular occlusion

A
  • Generalised abdo pain that is out of proportion to physical signs.
  • N+V
  • Shock
  • Abdo tenderness/rigidity
95
Q

What is seen on abdo xray in mesenteric vascular occlusion?

A

Thumb-printing sign

96
Q

Managment of mesenteric vascular occlusion

A

Early - embolectomy (preferably though angioplasty)

Late - bowel resection and anastomosis, managed in ICU