Functional decline and falls Flashcards

Pressure sores Reduced mobility Falls

1
Q

Patient presents to A&E with a fall. Which bloods should you send for?

A

FBC, U+E, haematinics, bone, vit D

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2
Q

Patient presents to A&E with a fall. Which investigations/exams/reviews do you want to carry out?

A

ECG, L+S BP, AMT4, vision, med review, bone health

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3
Q

What are categories of causes of falls?

A
  1. mental health
  2. vision defects
  3. MSK
  4. Neuro
  5. CVS
  6. Environment
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4
Q

Which of the following is not typically a cause of a fall: incontinence, polypharmacy, or hyperglycaemia?

A

hyperglycaemia (hypo=risk for falls), incontinence-> rushing to bathroom

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5
Q

What are the autonomic symptoms of hypoglycaemia?

A

trembling, sweating, hunger, anxiety, palpitations, nausea, tingling

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6
Q

What are the neuroglycopaenia symptoms of hypoglycaemia?

A

confusion, weakness, drowsiness, headaches, dizziness, visual changes, speech changes

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7
Q

Which questions make up the AMT?

A
  1. what year is it
  2. How old are you
  3. what is your dob?
  4. where are you?
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8
Q

What are symptoms of mild hypoglycaemia?

A

non-specific symptoms, generally feeling unwell (nauseous)- check BM

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9
Q

What are symptoms of moderate hypoglycaemia?

A

confusion, disorientation, aggression

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10
Q

What are symptoms of severe hypoglycaemia?

A

seizure, unconscious, very aggressive

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11
Q

What is the treatment for mild hypo?

A

15-20g fast-acting CHO e.g. luocozade 170 ml, pure fruit juice 150-200 ml, glucotabs, followed by long-acting CHO e.g. two biscuits, slice of bread

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12
Q

What is the treatment for moderate hypo?

A

if cooperative, same treatment as mild, escalate to i mg IM of glucagon

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13
Q

What is the treatment for severe hypo?

A

IV glucose or IM glucagon

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14
Q

List examples of drugs that can cause falls

A

drugs that cause postural hypotension: nitrates, ACEi, diuretics, L-dopa, SSRIs

Benzos, opiates, oral hypoglycaemia agents

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15
Q

What is the definition of postural hypotension?

A

fall in systolic BP of at least 20mmHg (30 in HTN) and/or fall in diastolic BP at least 10mmHg within 3 min of standing.

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16
Q

What is the management of postural hypotension?

A

med r/v, slow from lying to standing, small frequent meals, raise head of bed, mineralcorticoid

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17
Q

Which blood test is important to do in patient who has been lying down for a long time?

A

creatinine! risk of rhabdo. Muscle can start to break down during this time frame, and this rhabdomyolysis leads to release of haem which is nephrotoxic and can cause an acute kidney injury. Rhabdomyolysis is assessed by measuring creatine kinase (CK) - a raised level suggests the patient requires IV fluids to increase urinary flow rates and reduce the risk that the kidneys become damaged by intratubular cast formation.

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18
Q

How are the descending motor tracts divided?

A

pyramidal and extrapyramidal http://freeosmosis.com/learn/Pyramidal_and_extrapyramidal_tracts?section=Physiology

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19
Q

What is the function of pyramidal tracts?

A

voluntary movement, carry motor fibres from the cerebral cortex to the spinal cord and brainstem. Pyramidal tract lesions can occur from any type of damage to the brain or spinal cord, for example a stroke.

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20
Q

What is the function of extrapyramidal tracts?

A

responsible for involuntary control of the muscles, including posture and balance adjustments. They descend from the brainstem to the spinal cord. Damage to the extrapyramidal tracts results in dyskinesias and disorders of involuntary movement. Parkinson’s disease is an example of this

21
Q

What are four classical features of idiopathic parkinson’s disease?

A
TRAP
Tremor (asymmetric and resting)
Rigidity 
Akinesia
Postural instability
22
Q

Name examples of exclusion criteria for parkinsonian syndrome

A

MPTP exposure, fam hx, babinski sign, unilateral features, negative response to levodopa

23
Q

What is the diagnostic criteria for parkinson’s?

A

bradykinesia + 1

  • muscular rigidity
  • 4-6 Hz rest tremor
  • primary instability not caused by primary visual, vestibular, cerebellar, or proprioceptive dysfunction
24
Q

Name four non-motor symptoms of parkinson’s?

A

constipation, sleep disorders, depression, anxiety, erectile dysfunction, urinary frequency/incotinence/nocturia, altered sense of smell

25
Name two facial features of parkinson's disease
infrequency of blinking, hypomimia (reduction in facial expression), hypophonia (soft, monotonous voice), lips are parted
26
How to describe parkinsonian tremor?
asymmetrical 4-6 Hz, enhanced by anxiety, pill rolling
27
What are features of parkinsonian gait?
reduced arm swing, shuffling gait, freezing, stooped posture
28
Name four differentials for parkinsonism
1. idiopathic parkinson's disease 2. drug induced parkinson's 3. progressive supranuclear palsy 4. parkinson's plus syndromes 5. lewy body dementia
29
What is multiple system atrophy?
parkinsonism, cerebellar and autonomic dysfunction- orthostatic HTN (lying/standing BP)
30
What is progressive supranuclear palsy?
axial imbalance, oculomotor disturbance, early bulbar involvement (speech and swallow)
31
Which assessments would you conduct for supranuclear palsy?
eye movements and cranial nerve exam
32
Which assessment would you carry out to support lewy body dementia?
cognitive assessments
33
A patient has skin changes on back. What are five differentials for this?
1. Pressure sores 2. Staph infection 3. BSC 4. SCC 5. contact dermatitis
34
Name 4 factors that increase the risk of pressure sore development
1. immobility 2. cognitive impairment 3. neuropathy 4. age and frailty 5. incontinence
35
How would you describe a pressure sore in lower back region?
This patient’s sacral region bears a well-demarcated area of erythema around 14x 6cm with superficial skin loss. There is no clear evidence of slough, bruising, discharge or sinus formation. This is in keeping with a pressure ulcer.
36
What are the four grades of classifying pressure sores?
``` I-IV I: non-blanching erythema II: partial thickness III: full-thickness skin loss IV: full-thickness tissue loss ```
37
What is the management of pressure ulcers?
1. Positional changes 2. Mattress support 3. Dressings 4. Barrier creams 5. Abx 6. Dietary support 7. debridement
38
What are some long term consequences of pressure sores?
bedbound potential, nursing home, infection- sepsis, pain
39
What is an acronym to remember the causes of falls?
``` DAME drugs (plus alcohol) age medical- CVD, PD environmental ```
40
Which drugs are associated with falls?
sedatives, antihypertensives, antiarrhythmic, polypharmacy, extrapyramidal side effects
41
Name examples of gait abnormalities
PD, hemiplegic gait, cerebellar disease, antalgic gait, foot drop (common peroneal nerve)
42
What is acronym for taking history from patient who has fallen
``` SPLATT symptoms previous falls location activity time trauma ```
43
Which examinations/assessments should you carry out in patient who has fallen
cardio, CNS, vision, hearing, AMT, bloods, ecg
44
Differentials for joint related causes of immobility?
osteoarthritis, RA, gout, psuedogout, infection
45
Differentials for muscular related causes of immobility?
Myositis, myxoedema, PD, polymyalgia rheumatica
46
Reduced effort tolerance differentials for immobility?
dyspnoea, anaemia, reduced CO
47
What are complications of immobility?
1. social 2. psychological 3. physical- muscle wasting, osteoporosis, pneumonia, constipation, incontinence
48
What is a festinating gait?
parkinsonian shuffle
49
After several months of treatment, L-DOPA does not seem to be effective in PD patient. How should you modify their treatment?
add a dopamine agonist (continue with L-DOPA)