Foot penetrations and conditions of the hoof wall Flashcards

1
Q

List the visible structures on the bottom of a horses hoof

A
  • Hoof wall
  • White line
  • Sole
  • Frog apex
  • Bars
  • Central sulcus
  • Collateral sulcus
  • Heel bulb
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2
Q

Hoof cracks usually run in which direction?

A

Proximo-distal

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3
Q

What are transverse hoof cracks associated with?

A

Coronary band injury

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4
Q

What are the consequences of hoof cracks

A

Instability -> shear forces -> further separation -> infection and pain

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5
Q

How should you assess hoof cracks before treatment?

A
  • Characterise depth, direction,
  • Determine sensitive/insensitive parts (do not nerve block)
  • Incomplete often just need trimming/shoeing
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6
Q

How are hoof cracks treated?

A
  1. Farriery
    - Debride/dremmel all necrotic tissue
    - Filler to stabilise (plate; wire)
    - Trim foot/unload crack/bar shoe/quarter clips
  2. Identify underlying cause (and treat)
  3. Antibiotics (local/systemic) - Flush via catheter/tubing
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7
Q

What is white line disease?

A

Progressive, crumbling, poor quality hoof wall with separation at the white line
- Non-pigmented portion of the stratum medium and the laminar horn

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8
Q

Which factors can act as risk factors for white line disease?

A

Warm, wet weather
Biotin/methionine/zinc/selenium deficiency
Bacterial infection common

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9
Q

What are the clinical signs of white line disease?

A

+/- lameness
Separation of hoof wall esp at toes/quarters
Grey/black crumbly horn

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10
Q

How is white line disease treated?

A
  • Remove abnormal horn
  • Support remaining horn: Bar shoe+clips, Hoof acrylic
  • Prevent progression: Environmental factors, Topical povidone/iodine, Feed supplementation
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11
Q

Wire lacerations/foot trapped (e.g. gate/fence)/overreach injuries most commonly cause injuries to which part of the hoof?

A

Coronary band and hoof wall

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12
Q

Why must you assess if there is damage to the coronary band?

A

Damage to the coronary band can affect how the hoof wall grows

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13
Q

List the clinical signs of damage to the coronary band and hoof wall

A
  • Avulsion/disruption to the hoof wall +/- coronary band
  • Lameness: Moderate/severe
  • Haemorrhage++
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14
Q

Which other structures may be involved when there is damage to the coronary band and hoof wall?

A

Synovial structure = DIP/PIP/Navicular bursa/DFTS
Tendons/ligaments (DDFT/SDFT/extensor tendons/collateral ligaments)

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15
Q

How are injuries to the coronary band and hoof wall treated?

A
  • Primary or secondary intention healing
  • Preserve coronary band if you can
  • Antibiotics
  • NSAIDs
  • Bandaging
  • Cast
  • Flush
  • Shoes
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16
Q

Why is it important to flush synovial structures?

A

Treat sepsis early and aggressively

17
Q

How should puncture wounds of the foot be assessed and managed?

A
  • Common cause of lameness
  • Most are managed conservatively with a good prognosis
  • All foot penetrations have the potential to involve deeper structures resulting in life threatening complications
  • Need to know what kind of object is involved
18
Q

What is nail bind?

A

Nail close to sensitive structures
Mild lameness
Pain around nail

19
Q

What is shoeing prick?

A

Nail into sensitive structures
Immediately painful/blood
May develop into subsolar abscess if left

20
Q

How do subsolar abscesses occur?

A

Penetration of bacteria results in abscess formation and pressure on sensitive hoof lamina

21
Q

What are the clinical signs of subsolar abscessation?

A
  • Usually acute lameness: SEVERE!
  • Increased digital pulse
  • Increased hoof temperature
  • Sensitive to hoof testers
22
Q

How are subsolar abscesses treated?

A
  1. Key: DRAINAGE
    - Remove shoe/nail (if present)
    - Pare foot: follow tracts and remove all necrotic/underrun horn
  2. Poultice or tub
  3. Bandage - Protect foot/apply poultice
  4. (NSAIDs/antibiotics)
  5. Check tetanus status
23
Q

Which structures could be damaged when there are foot penetrations affecting synovial structures?

A

Navicular bone/bursa
DDFT
Distal sesamoidean impar ligament – joins the bottom of the navicular bone to the back of P3
DIP joint
DFTS

24
Q

How are foot penetrations affecting synovial structures diagnosed?

A
  • Moderate to severe lameness
  • Presence of nail/foreign body in foot, particularly the middle third
  • Puncture wound – can be difficult to find sometimes
  • Distal limb swelling/DIP effusion/DFTS effusion
  • Increased digital pulse to foot
  • Sensitive to hoof-testers over tract
  • Radiography
  • Synoviocentesis
  • MRI
25
How are foot penetrations affecting synovial structures treated?
- Debridement of infected tissue - Removal of necrotic horn/tendon - Flushing of affected synovial structures - Bandage - NSAIDs
26
Describe the prognosis following foot penetration that has damaged synovial structures?
- Infection: fair (56% survival to discharge) - Return to athletic function: guarded (due to involvement of DDFT/impar ligament) (36% return to pre-injury function)
27
List some possible underlying causes of chronic foot abscesses
- Immunocompromise e.g Cushing’s - Keratoma - Sequel to laminitis (poor quality laminae) - Bone sequestrum/collateral cartilage infection - Infective (pedal) osteitis
28
What is a keratoma?
Benign tumour of the hoof/solar horn
29
How does a keratoma present?
- Intermittent lameness/discharge - Characteristic circular area of abnormal keratinisation with discharging tract - Radiography may show smooth, radiolucent defect in P3
30
How is a keratoma treated?
Surgical resection under GA
31
What is quittor?
Infection of the collateral cartilages Trauma/wound Swelling/chronic discharge from coronary band
32
How is quittor treated?
Surgical debridement of infected tissues Be careful of the DIPJ!
33
What is canker?
Chronic condition associated with hypertrophy of the germinal layer of the epithelium of the frog - May affect frog, bars, heels and sole
34
What agent is linked to cause canker?
Fusobacterium/Bacteriodes spp
35
What are the consequences of canker?
- Infection leads to dyskeratosis of the keratin producing cells - Results in abnormal hyperkeratotic horn with keratolysis and fronds of unconnected intertubular horn
36
How can early/mild cases of canker be managed and treated?
- Improve environment - Debride abnormal areas - Apply metronidazole bandages +/- systemic abs - Astringents: picric acid (5%) and benzoyl peroxide (10%)
37
How can advanced/severe cases of canker be managed and treated?
Aggressive surgical debridement Bandaging/shoeing