fluids and electrolytes Flashcards

1
Q

what is the path of extracellular volume regulation

A
BARORECEPTORS
• SYMPATHETIC NERVOUS SYSTEM
• RENIN ANGIOTENSIN SYSTEM
• VASOCONSTRICTION AND RELEASE OF ALDOSTERONE
• ALDOSTERONE (CONSERVES NA++ AND WATER)
• ANTIDIURETIC HORMONE
• ATRIAL NATRIURETIC PEPTIDE (ANP)
• THIRST- WATER LOSS >2%
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2
Q

Thirts mechanism kicks in at what point

A

water loss great than 2%

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3
Q

hypovolemia know diffrence in subjective and objective finding

A

Ojective

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4
Q

vomting and diuretics can lead to meabolic shift

A

Alkalosis

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5
Q

diarrhea, DKA, shock can lead to what type of metabolic shift

A

acidosis

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6
Q

Patient with WEIGHT GAIN
• INCREASED BP
• EDEMA
• ASCITES
• DECREASED HG, HCT, RBC
• RESPIRATORY ALKALOSIS (PH >7.45, PACO2 < 35)
• CHEST X-RAY-PULMONARY CONGESTION you suspect what type of fluid volume and treat with

A
hypervolemia
FLUID AND NA RESTRICTION
• DIURETICS
• DIALYSIS
• OXYGEN VIA NASAL CANNULA
• FOLLOWING SUCCESSFUL TREATMENT
• CLEAR LUNGS
• NO S3 OR S4
• NORMAL URINARY OUTPUT
• NORMAL URINE SPECIFIC GRAVITY
• NO EDEMA, NORMAL BP, HR, CVP, AND PCWP
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7
Q

FIRST STEP IN MANAGEMENT hyponatremia

A
URINE SODIUM (NORMAL 10-20 MEQ/L)
• SERUM OSMOLALITY (USUALLY 2X NA = 280)
• CLINICAL STATUS
• MEASURING URINE SODIUM HELPS TO DETERMINE RENAL FROM NONRENAL
CAUSES
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8
Q

Monitor serum Na how often in symptomatic patients needing sodium correction

A

every 2-4 hours

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9
Q

normals for urine osmolality

A
URINE SODIUM (NORMAL 10-20 MEQ/L)
•
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10
Q

Normals for serum osmolality

A

SERUM OSMOLALITY (USUALLY 2X NA = 280)

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11
Q

Management of Hypernatremia SEVERE HYPERNATREMIc HYPOVOLEMIa

A

NS iv

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12
Q

1/2 ns and or D5W will allow for what

A

fluid into cells

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13
Q

treatment for hypervolemic hypernatriemia

A

TREAT WITH D5W AND LOOP DIURETICS

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14
Q
which has the highest potassium; 
Seaweed
avocado
bananas
tomatoes 
lamb
A

seaweed

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15
Q

normal calcim

A

8.5 to 10.5mg/dL

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16
Q

normal ionized Ca

A

4.5 to 5.5mg/dL

17
Q

elderly person comes in with low albumin what else would you expect ot be low and what the best way to check this level

A

Ca will be low so need to check ionized ca

18
Q

Acidemia increase or decrease ionized calcium

A

increases ionized calcium

19
Q

patient getting multiple blood transfusions you expect ca to go

20
Q

Over use of thiazide diuretics affect ca in what way

A

hypercalcemia

21
Q

ca greater than 12mg?dl

A

IF >12 MG/DL –NS INFUSION WITH LOOP DIURETICS
• TEACHING ABOUT DIET, FLUID INTAKE, SIGNS AND SYMPTOMS, PAIN
CONTROL

22
Q

Hypercalcemia management medication for impaired renal or cardiovascular problem

A

calcitonin

23
Q

Hypercalcemia can be managed with Dialysis true or False

24
Q

chronic alcoholic suspect impaired intestinal absorbtion leading to low VitD and lead you to suspect low

A

phosphorus, Hypophosphatemia

25
Causes of hypophosphatemia
phosphorus <2.8mg/dL refeeding syndrome ( insulin spike hungrey bone syndrome ``` Increased renal secreation with high PTH osmotic diuretic rickets renal proximal tubular disorder Fanconi syndrome inpaired intestinal absorption vit D deficiency chronic alcoholism ```
26
Phosphorus level <1mg/dL cause end organ effects due to lack of
adenosin triphosphate; effects all muscles
27
urine phosphorus excretion of >100mg in 24 hours mean
high loss of phosphorus hypophosphatemia
28
what is molarity
moles per liter of solution
29
what is molality
moles per liter of solvent
30
what is diffusion
movement of particles or molecules from an area of high concentration to an area of low concentration
31
Membrane that allows certan materials to pass throughbut not other
selective permeable membrane
32
A dexcription of the relative solute concentration in a soultion as compred to another solution
tonicity
33
solution with higher concentratoin than other
hypertonic
34
solution with lower concentration than other
hypotonic
35
diffusion regulated by protein channels in the membran and requires no energy input from cell
facilitate diffusion
36
movement of molecules or ion into or out of cells against it concentration gradietn from low to high and requires an input of enrey
active transport