Fluid Imbalance Flashcards

1
Q

Fluid balance?

A

equilibrium in water exchange between internal fluid compartments of the body and between the body and the external environment

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2
Q

hypovolemia

A

Low blood volume
- extracellular volume depletion
- water and electrolytes lost in same proportion

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3
Q

Etiology for Hypovolemia

A

a. Decrease in intake of fluids = dehydration oral or Iv fluids

b. Decrease in extracellular volume = loss of blood, vomiting, diarrhea, excess swelling

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4
Q

pathophysiology for hypovalemia

A

decreased blood volume
= decreased Capillary hydrostatic pressure and filtration
- cells deprived of substances that they need to function

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5
Q

clinical manifestations of hypovalemia?

A
  • Tissue dehydration (tenting, dry mucus membrane)
  • decreased blood volume = decreased BP, decreased urine output, and increased heart rate
  • Decreased tissue perfusion = decreased 02 supply in blood
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6
Q

Hypervolemia

A

increased blood volume
- Excess extracellular fluid
- Water and electrolytes gained in the same proportion

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7
Q

Hypervolemia etiology (caused by)

A

increased administeration of isotonic fluids
- increased dietary intake of NaCl (pulls h2O with it)
- diseases: chronic renal failure, heart failure and liver failure hyperaldosteronism (RAAS system)
- Expansion of extracellular space and circulatory overload = because an increase in volume

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8
Q

Hypervolemia clinical manifestations

A
  • Edema = swelling of tissues
  • Increased BP and bounding pulse = opposite of hypovolemia, lot of fluid causes paulse
  • Urine output ↑
  • Weight gain (edema)
  • Shortness of breath = if around alveoli in lungs
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9
Q

Edema

A

expansion or accumulation of interstitial fluid volume (third space shift)
- Fluid shifts from plasma (intravascular) to interstitial spaces

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10
Q

Edema Etiology or caused by

A
  • inflammation
  • increased BV
  • venous obstruction
  • decreased serum albumin
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11
Q

Edema pathophysiology

A
  • Increased capillary permeability
  • decreased capillary osmotic pressure (pulls fluid back in)
  • increased capillary hydrostatic pressure (pushes fluid to tissue)
  • Lymphatic obstruction (lymph cannot absorb fluid)
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12
Q

Edema clinical manifestations

A

Pitting = depression
weeping = seeping through damaged skin
Dependent = body part on a dependent position (not using Atm)
generaized = edema all over (kindey failure)
Pulmonary = edema in the lungs (heart failure)

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13
Q

2 types of osmolar imbalances?

A

Hypotonic (hypo-osmolar Imbalance)
Hypertonic (hyperosmolar Imbalance)

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14
Q

Hypotonic?

A

Excess retention of free water or low solute intoxication

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15
Q

Hypotonic Etiology

A
  • Excess water intake
  • End stage renal failure = kidney cant excrete water
  • Inappropriate ADH release
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16
Q

Hypotonic pathophysiology

A
  • Movement of water to intravascular space and into cells
  • cellular swelling
17
Q

Hypotonic clinical manifestations

A
  • Decreased serum osmolarity
  • decreased hematocrit (diluted blood)
  • low urine specific (gravity urine is diluted)
  • central nervous system changes = headache, confusion because cells swell in brain
18
Q

Hypertonic (hyperosmolar imbalance)

A

excess concentration of extracellular fluid (water deficit, blood is concentrated)

19
Q

Hypertonic Etiology

A
  • Decreased water intake
  • Insufficient intake of free water
  • increased loss of free water
  • excess intake of hypertonic solutions
20
Q

Hypertonic pathophysiology

A

Hypertonic extracellular fluid environment
- shrinkage of cells

21
Q

hypertonic cellular manifestations

A
  • Elevated serum osmolarity
  • increased hematocrit (concentration of blood)
  • high urine specific gravity (more concentrated urine)
  • Tissue dehydration
  • polydipsia (more thirsty)
  • central nervous system changes: confusion from h20 being pulled out of the brain because they are dehydrated