Fluid And Electrolytes/ ABG's Flashcards

1
Q

Dehydration Prevention and Interventions:

A

o Offer fluids regularly to patients unable to ask for or obtain fluids on their own.
o Identify and stop sources of fluid loss
o Oral fluids for mild to moderate fluid losses
o IV fluids for significant fluid losses

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2
Q

Daily weights

A

o Best indicator of fluid loss and gains

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3
Q

Hyponatremia S/S

A

Confusion, restlessness, lethargy, seizures, coma. May have fluid volume excess or deficit

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4
Q

Hyponatremia tx

A

Treat: Hypertonic solutions (0.9% NS, 0.3% NS); loop diuretics; fluid restriction

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5
Q

Hypernatremia S/S

A

Confusion, lethargy, seizures, coma, dry mucus membranes, postural hypotension, decreased skin turgor

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6
Q

Hypernatremia tx

A

Hypotonic solutions (D5W, 0.45% NS); low Na+ diet; Oral care for dry mucous membranes

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7
Q

Hypokalemia S/S

A

Fatigue, poor muscle contractions, skeletal muscle weakness, possible paralysis, hyporeflexia, decreased GI motility with anorexia, N/V, ileus, paresthesia, confusion, lethargy
o Cardiac dysrhythmias with Flat T waves
o Increased sensitivity to Digitalis – Risk for digitalis toxicity

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8
Q

Hypokalemia Tx

A

K+ replacement PO or IV

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9
Q

Hyperkalemia S/S

A

Cardiac dysrhythmias with tall/tent T waves, abdominal cramping, hyperactive bowel sounds, diarrhea, abdominal cramping, muscle twitching and contraction with ultimately weakness and paralysis

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10
Q

Hyperkalemia Tx

A

Lasix; Kayexalate (2 doses); Sorbital (promotes bowel elimination); Insulin with D50 to move K+ from ECF to ICF

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11
Q

Hypocalcemia S/S

A

Paresthesias, twitching, muscle cramps, Trousseu’s and Chvostek’s signs, tetany, hyperactive bowel sounds, seizures. Rickets in children.

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12
Q

Hypocalcemia Tx.

A

PO calcium replacement, vitamin D supplements, IV calcium for serious cases

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13
Q

Hypercalcemia S/S

A

Muscular weakness, fatigue, hyporeflexia, lethargy, confusion, coma dysrhythmias, slowed GI motility

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14
Q

Hypercalcemia Tx

A

Diuretics and IV fluids to excrete urine. IV Na+ or K+ is given for excessive calcium levels.

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15
Q

Hypomagnesaemia S/S

A

Muscle twitching and spasms, hyperreflexia, tetant, cardiac dysrhythmia, anorexia, N/V/D, altered mental status, lethargy, confusion, seizures

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16
Q

Hypomagnesaemia Tx

A

PO or IV magnesium; Dietary intake

17
Q

Hypermagnesaemia S/S

A

: Similar to hypercalcemia, paresthesias, muscle weakness, hyporeflexia, anorexia, naursea, constipation, cardiac dysrthmias

18
Q

Hypermagnesaemia Tx

A

Avoid meds with magnesium for pts at risk. IV calcium to counteract the effects of hypermagnesemia; Dialysis

19
Q

• Metabolic Alkalosis causes

A

Vomiting, loose acid, diuretics, too much sodium bicarbonate (Tums), NG tube suctioning

20
Q

Metabolic alkalosis lab values

A

Increased pH and HCO3

21
Q

Metabolic alkalosis S/S

A

CNS irritability, restlessness, tremors, seizures, cardiac monitoring for dysrhythmias

22
Q

Metabolic alkalosis Tx

A

Fix the underlying cause; Oral K+ supplements. Control of vomiting and avoid bicarbonate antacids are primary interventions

23
Q

• Metabolic Acidosis Causes:

A

Severe diarrhea, renal disease, Crohn’s disease, bypass surgery, aspirin overdose, untreated DM, DKA, starvation, lactic acidosis

24
Q

• Metabolic Acidosis lab values

A

Decreased pH and decreased HCO3

25
Q

Metabolic acidosis S/S

A

Lethargy, confusion, coma, dysrhythmias; deep, rapid respirations; Kussmaul respirations in people with DM

26
Q

Metabolic acidosis Tx

A

Fix underlying cause; bicarbonate IV or PO; Insulin for DM; dialysis for renal failure; O2 for lactic acidosis; antidiarrheals for diarrhea

27
Q

Respiratory Alkalosis:

Causes:

A

• Asthma, high altitude, fever, hypoxia, excessive mechanical ventilation, hyperventilation, anxiety attack

28
Q

Respiratory Alkalosis: Lab Values:

A

Increased pH and CO2

29
Q

Respiratory Alkalosis:S/S:

A

Tremors, restlessness, irritability, seizures (severe), hypocalcemia (muscle spasms, twitches, Trousseau’s and Chvostek’s sign).

30
Q

Respiratory Alkalosis:Treatment

A

: Fix underlying cause; Breath into paper bag to rebreathe CO2

31
Q

• Respiratory Acidosis: Causes:

A

Any condition that impairs gas exchange, pneumonia, COPD, chronic bronchitis, Rapid shallow breathing, brain stem injury, drug OD

32
Q

• Respiratory Acidosis:

Lab Values:

A

Decreased pH and CO2

33
Q

• Respiratory Acidosis: S/S:

A

Reduced LOC, confusion, lethargy, coma, dysrhythmias

34
Q

• Respiratory Acidosis: Treatment:

A

Respiratory support, breathing treatment, ventilator for patients with poo ventilation, IS use, bronchodilators for narrowed airways

35
Q

Total parenteral nutrition (TPN):

• Side Effects:

A

Hyperglycemia, hyperosmolar syndrome, electrolyte imbalances (hypophosphatemia, hypokalemia, hypomagnesemia, and hypernatremia)

36
Q

Total parenteral nutrition (TPN):

• Nursing Considerations:

A

o Glucose regulation (monitor BG level)
o TPN must run in PICC or central line
o Inform patient TPN does not make them diabetic but insulin may be needed during therapy to control BG levels
o Hypoglycemia can occur if the TPN is stopped abruptly; therefore, always wean patients from TPN.
o TPN protocols for laboratory study monitoring should be in place.

37
Q

Total parenteral nutrition (TPN):
Complications:

A

Can be life-threatening and include GI atrophy, fluid overload, hyperglycemia, allergic reactions, and sepsis