Fluid and Electrolytes Flashcards

1
Q

metabolic acidosis

A

decreased pH <7.35
decreased HCO3 <22
possible normal PaCO2. Respiratory compensation may occur, causing a decrease in the PaCO2 level.
Urine pH <6

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2
Q

Respiratory acidosis

A

decreased pH <7.35
increased PaCO2 >45

compensation: kidneys conserve HCO3; eliminate H to increase pH
common causes asphyxia, respiratory and CNS depression

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3
Q

Metabolic alkalosis

A

increase pH >7.45
increased HCO3 >28
compensation: hypoventilation to increase CO2 kidneys keep H and excrete HCO3
common causes: hypercalcemia, alkaline (antacid) overdose.

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4
Q

respiratory alkalosis

A

increased pH >7.35
decreased PaCO2 <35
compensation: kidneys eliminate HCO3; conserve H and decrease pH
common causes: hyperventilation, anxiety, diabetic ketoacidosis.

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5
Q

hypercalcemia >10.5

A

signs & symptoms: weakness, fatigue, anorexia, nausea, vomiting, constipation, polyuria, tingling lips, muscle cramps, confusion, hypoactive bowel tones.
common causes: hyperparathyroidism or malignancies, thiazide diuretics, lithium, renal failure, immobilization, metabolic acidosis.

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6
Q

hypocalcemia < 9

A

signs & symptoms: anxiety, irritability, twitching around mouth, convulsions, tingling/numbness of fingers, diarrhea, abdominal/muscle cramps, arrhythmias.
common causes: inadequate vitamin D intake, low albumin, renal failure, lactose intolerance, Crohn;s disease, hyperthyroid, increased magnesium, acute pancreatitis.

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7
Q

hyperkalemia >5

A

signs & symptoms: weakness nausea, diarrhea, hyperactive GI, muscle weakness and paralysis, arrhythmias, dizziness, postural hypotension, oliguria
Common Causes: Potassium-sparing diuretics, NSAIDS, renal failure, multiple transfusions, decreased renal steroids, OD of potassium supplements.

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8
Q

hypokalemia <3.5

A

signs & symptoms: anorexia, nausea, vomiting, fatigue, decreased LOC, leg cramps, muscle weakness, anxiety, irritability, arrhythmias, postural hypotension, coma
common causes: anorexia, fad diets, prolonged NPO status, alkalosis, transfusion of frozen RBCs, prolonged NGT suctioning

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9
Q

hypermagnesemia >2.1

A

signs & symptoms: muscle weakness and fatigue are most common, nausea vomiting, flushed skin, diaphoresis, thirst, arrhythmias, palpitations, dizziness.
common causes: increased magnesium intake, chronic renal disease, pregnant women on parenteral magnesium for pre-eclampsia, addison’s disease.

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10
Q

hypomagnesemia <1.3

A

signs & symptoms: diarrhea, anorexia, arrhythmias, lethargy, muscle weakness, tremors, nausea, dizziness, seizures, irritability, confusion, psychosis, decreased BP, increased HR
Common Causes: prolonged NGT suctioning, diarrhea, laxative abuse, malnutrition, alcoholism, prolonged diuretic use, DKA, digoxin

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11
Q

hypernatremia >145

A

common signs & symptoms: confusion, fever, tachycardia, low BP, postural hypotension, dehydration, poor skin turgor, dry mucous membranes, flushed.
Common Causes: fever, vomiting, diarrhea, ventilated Pts, severe burns, profuse sweating, diabetes insipidus, diuresis

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12
Q

hyponatremia < 136

A

signs & symptoms: nausea, vomiting, abdominal cramps, diarrhea, headache, dizziness, confusion, flat affect, decreased DBP, increased HR, postural hypotension, decreased deep tendon reflex.
common causes: diuretic uses, vomiting, diarrhea, burns, hemorrhage, fever, diaphoresis, CHF, renal failure, hyperglycemia, increased ADH

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13
Q

conditions that cause combined acidosis

A

decreased respiratory rate, kidney dysfunction (oliguria), and dehydration (dry mucous membranes).

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14
Q

After successful resuscitation of cardiopulmonary arrest, the nurse views these arterial blood gases: pH 7.28; CO 2 52; HCO 3 – 16. What is the interpretation of these values?

A

with a pH of 7.28 (acidosis), there is evidence of a respiratory component (CO 2 > 45) and a metabolic component (HCO 3 – < 21). This is therefore combined respiratory and metabolic acidosis, which would likely follow a cardiopulmonary arrest (CO 2 retention, lactic acidosis). Compensation in respiratory acidosis is demonstrated by an elevated HCO 3 –. In metabolic acidosis, there is very little, if any, change in CO 2. Full compensation would be demonstrated by a normal pH.

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15
Q

Kussmaul respiration

A

In metabolic acidosis, the rate and depth of breathing increase as the hydrogen ion levels rise. The breathing pattern becomes deep and rapid and not under voluntary control. This type of breathing is known as Kussmaul respiration, which is not present in respiratory alkalosis, respiratory acidosis, or metabolic alkalosis.

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16
Q

Which skin and mucous membrane assessments will the nurse use to evaluate a patient for dehydration?

A

color, turgor, moisture

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17
Q

insensible water loss

A

skin, lungs and stool

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18
Q

An older adult patient with a history of renal failure is brought in to the emergency department with sudden onset of acute confusion, worsening muscle weakness in the extremities, abdominal cramps, and a weak, rapid, and thready pulse. What are the immediate nursing interventions to stabilize the patient? Select all that apply.

A

Administering diuretics
Administering 0.9% saline
Administering 5% dextrose in 0.45% sodium chloride

The patient’s symptoms indicate hypernatremia. Administering diuretics that promote sodium loss and administering fluids such as 0.9% saline and 5% dextrose in 0.45% sodium chloride to restore the fluid balance are the immediate interventions needed to stabilize the patient. The patient’s acute confusion is caused by high sodium levels, so anti-psychotic drugs should not be administered. Sodium decreases the heart contractibility by retarding the movement of calcium into the heart cells. Therefore calcium channel blockers should not be administered.

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19
Q

The nurse is administering sodium chloride 0.9% (normal saline) intravenously to a patient who is dehydrated. Which assessments does the nurse perform to evaluate the effectiveness of rehydration therapy? Select all that apply.

A

Urinary output
Pulse rate and quality

Pulse rate and quality as well as urinary output best reflect improving volume status with rehydration therapy. Temperature, level of consciousness, and bowel sounds are not indicators of an improving volume state.

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20
Q

A patient is admitted to the intensive care unit with a diagnosis of syndrome of inappropriate antidiuretic hormone secretion (SIADH). Which treatments does the nurse anticipate for this patient? Select all that apply.

A

Furosemide (Lasix)
Tolvaptan (Samsca)
Conivaptan (Vaprisol

The nurse anticipates the health care provider will prescribe furosemide, tolvaptan, and conivaptan to manage SIADH. Furosemide is a loop-diuretic that helps rid the body of excess fluid. Tolvaptan is used to manage hyponatremia with SIADH. Conivaptan acts as an antidiuretic hormone (ADH) inhibitor to help resolve fluid volume overload. Digitalis is a cardiac glycoside that slows and strengthens myocardial contraction, but it has no effect on fluid volume. Vasopressin is contraindicated with SIADH since it is the synthetic form of ADH. Norepinephrine is a powerful peripheral vasoconstrictor used to increase blood pressure.

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21
Q

Which assessment data is the best indication of perfusion after a surgical procedure?

A

urine output
The renin-angiotensin II pathway is highly stimulated whenever the patient is in shock or when the stress response occurs. This is why urine output is used as an indicator of perfusion adequacy after surgery or any time the patient has undergone an invasive procedure and is at risk for hemorrhage. Although heart rate, blood pressure, and pulse oximetry are also useful when monitoring perfusion, they are not the most important indicator of perfusion adequacy.

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22
Q

Which situation can cause a patient to experience “insensible water loss?” Select all that apply.

A
fever
diarrhea
dry hot weather
mechanical ventilation
increased respiratory rate

Insensible water loss occurs through the intestinal tract as diarrhea. It can be caused and/or influenced by dry, hot weather. Insensible water loss occurs through the skin, lungs (increased rate of respirations), and intestinal tract. It is increased in patients who are mechanically ventilated, and it is increased by the presence of fever. Nausea with no accompanying vomiting would not cause insensible water loss.

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23
Q

The nurse is caring for a patient with an oxygen saturation of 88% and use of accessory muscles for breathing. The nurse provides oxygen and anticipates which of these health care provider orders?

A

Intubation and mechanical ventilation

Support with mechanical ventilation may be needed for patients who cannot keep their oxygen saturation at 90% or who have respiratory muscle fatigue. Signs of hypoxemia and work of breathing are present, requiring correction with intubation and mechanical ventilation. Sodium bicarbonate is used to treat metabolic acidosis; this patient displays hypoxemia. Although the underlying reason for this patient’s hypoxemia may eventually require a diagnostic study such as a CT scan of the chest, the priority is to restore oxygenation. There is no clinical evidence of hypokalemia, so administration of potassium chloride is not indicated.

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24
Q

Which patient would be appropriate to assign to the new nurse working on the medical-surgical unit?

  1. Patient with emphysema and cellulitis with a PaCO 2 level of 58 mm Hg
  2. Patient with reactive airway disease, wheezing, and a PaO 2 level of 62 mm Hg
  3. Patient with diabetic ketoacidosis and change in mental status who has a pH of 7.18
  4. Patient with a small bowel obstruction and vomiting with a bicarbonate level of 40 mEq
A

Patient with emphysema and cellulitis with a PaCO 2 level of 58 mm Hg

A PaCO2 level of 58 mm Hg in the patient with emphysema and cellulitis, although abnormal, is anticipated for a patient with chronic obstructive pulmonary disease (COPD) and is stable for assignment to a new graduate. The patient with diabetic ketoacidosis and change in mental status is unstable and requires care by a more experienced nurse. The patient with reactive airway disease is still wheezing and requires experienced nursing care. The patient with a small bowel obstruction is unstable and may likely require surgery, which requires more experienced nursing care.

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25
Q

Which acid-base imbalance does the nurse anticipate the patient with morbid obesity may develop?

A

Respiratory acidosis

Respiratory acidosis is related to CO 2 retention secondary to respiratory depression, inadequate chest expansion, airway obstruction, or reduced alveolar-capillary diffusion. Respiratory acidosis is common in morbidly obese patients who experience inadequate chest expansion owing to their size and work of breathing. Metabolic acidosis is related to overproduction of hydrogen ions, underelimination of hydrogen ions, underproduction of bicarbonate ions, and overelimination of bicarbonate ions. Metabolic alkalosis is related to loss of bicarbonate or buffers (i.e., vomiting or nasogastric suction). Respiratory alkalosis usually is caused by excessive loss of CO 2 through hyperventilation secondary to fever, central nervous system lesions, and salicylates.

26
Q

A patient currently taking acetazolamide develops metabolic acidosis. Which nursing interventions are appropriate for the nurse to include in the plan of care? Select all that apply.

Monitoring electrolyte levels

Administering prescribed diuretics

Administering intravenous (IV) fluids

Placing the patient on fall precautions

Administering prescribed antiemetics

A

Monitoring electrolyte levels
Administering intravenous (IV) fluids
Placing the patient on fall precautions
Administering prescribed antiemetics

For a patient with metabolic acidosis due to diuretic therapy, the patient’s diuretics should be stopped, the nurse should administer IV fluids and monitor electrolyte levels, and the patient should be placed on fall precautions because hypotension and muscle weakness are common with metabolic acidosis. Antiemetics are used if metabolic acidosis is caused by vomiting.

27
Q

Which electrolyte abnormality does the nurse anticipate when reviewing laboratory data for a patient admitted with metabolic acidosis?

A

hyperkalemia

Serum potassium (hyperkalemia) occurs during metabolic acidosis as the body attempts to maintain pH by moving potassium ions from the cell in exchange with hydrogen ions moving into the cell. Hypokalemia may occur as the cause of the metabolic acidosis is corrected. Sodium concentrations (hypernatremia and hyponatremia) are not affected in the buffering process of acid-base balance.

28
Q

The nurse is caring for a patient with metabolic alkalosis. What manifestations of metabolic alkalosis is the nurse likely to assess? Select all that apply.

A

Numbness around the mouth
Hyperactivity of deep tendon reflexes

Hypercalcemia occurs with alkalosis, which can cause hyperactivity of deep tendon reflexes. Alkalosis overexcites the nervous system leading to tingling or numbness around the mouth. Overstimulation of the nerves may cause contraction of skeletal muscles, but the contractions are weaker because of hypokalemia. Therefore there is a decrease in handgrip strength. Alkalosis increases myocardial irritability and increases the heart rate. Kussmaul respiration (deep and rapid involuntary breathing) is seen in metabolic acidosis with respiratory compensation.

29
Q

Common causes of metabolic acidosis

A
DKA (diabetic ketoacidosis) most common
lactic acidosis (shock, respiratory or cardiac arrest)
renal failure
severe diarrhea
salicylate toxicity
starvation
GI fistulas
liver failure
30
Q

signs & symptoms of metabolic acidosis

A

kussmaul respirations (deep, rapid respirations)
confusion, disorientation progressing to coma
headache and lethargy
hypotension
dysrhythmias secondary to hyperkalemia
warm flushed skin (peripheral vasodilation)
abdominal pain and nausea and vomiting

31
Q

Nursing Management of Metabolic Acidosis

A

renal function-check BUN, creatinine and hgb and hct levels
monitor hydration status for problems with fluid balance.
support respiratory function- turn, cough, and deep breathe.
check ABGs and assess for Kussmaul respirations.
check electrolyte levels. K usually goes up in acidosis; Ca usually goes down.
Assess for cardiac dysrhythmias.
Assess blood sugar levels, they may need to be corrected.
antidiarrheal medications (antimetic for vomiting) and soda bicarbonate may be given to correct acidosis. (IV HCO3- in severe)
correct the precipitating cause of acidosis.
administer fluid replacement (0.9% or 0.45% sodium chloride for hydration therapy).

32
Q

metabolic acidosis compensation

A

If the CO2 or HCO3 that does not match the direction of the pH is moving in the opposite direction, then compensation is occurring. Kidneys will compensate acidosis by increasing the HCO3-; lungs will compensate acidosis by decreasing the CO2.

33
Q

isotonic fluids osmolarity 250-375

A
0.9% NaCl
lactated ringers solution
5% dextrose in water
ringer's solution
plasmalyte
34
Q

hypotonic solutions

A
  1. 45% NaCl
  2. 33% NaCl
  3. 225% NaCl
  4. 5% dextrose in water
35
Q

hypertonic solutions

A
3% NaCl
5% NaCl
Dextrose 5% in 0.45% NaCl
Dextrose 5% in 0.9% NaCl
Dextrose 5% in lactated ringer's
10% dextrose in water
20% dextrose in water
36
Q

hypokalemia ekg

A

st depression
shallow, flat or inverted T wave
prominent U wave

37
Q

hyperkalemia ekg

A

tall peaked t waves
flat p waves
widened QRS complexes
prolonged PR intervals

38
Q

hypocalcemia

A

prolonged ST segment

prolonged QT interval

39
Q

hypercalcemia

A

shortened ST segment

widened T wave

40
Q

hypomagnesemia

A

tall t waves

depressed st segment

41
Q

hypermagnesemia

A

prolonged pr interval

widened QRS complex

42
Q

S&S of respiratory acidosis

A

drowsiness
disorientation
dizziness
headache
coma
decreased bp
dysrhytmias (related to hyperkalemia from compensation)
warm, flushed skin (related to peripheral vasodialation
seizures
hypoventilation with hypoxia (lungs are unable to compensate when there is a respiratory problem)

43
Q

Respiratory Acidosis interventions

A
  • monitor for signs of respiratory distress
  • administer O2 as prescribed
  • place the client in a semi-fowler’s position
  • encourage and assist the client to turn, cough and deep-breathe.
  • encourage hydration to thin secretions
  • reduce restlessness by improving ventilation rather than by administering tranquilizers, sedatives, or opioids because these medications further depress respirations.
  • prepare to administer respiratory treatments as prescribed.
  • suction the client’s airway if necessary.
  • monitor electrolyte values, particularly the potassium level and arterial blood gas levels.
  • administer antibiotics for respiratory infection or other medications as prescribed.
  • prepare for endotracheal intubation and mechanical ventilation if CO2 levels rise above 50 mm Hg and if signs of acute respiratory distress are present.
44
Q

Causes of Respiratory Acidosis

A
  • asthma
  • atelectasis
  • brain trauma
  • bronchiectasis
  • brochitis
  • CNS depressants (sedatives, opioids
  • emphysema and COPD
  • administering high O2 levels per NC to clients who are CO2 retainers )copd and emphysema)
  • hypoventilation
  • pneumonia
  • pulmonary edema
  • pulmonary emboli
45
Q

uncompensated respiratory acidosis

A

ph: decreased
HCO3: normal
PaCO2: increased

46
Q

partially compensated respiratory acidosis

A

pH decreased
HCO3 increased
PaCO2 increased

47
Q

fully compensated respiratory acidosis

A

pH normal
HCO3 increased
PaCO2 increased

48
Q

uncompensated respiratory alkalosis

A

pH increased
HCO3 normal
PaCO2 decreased

49
Q

partially compensated respiratory alkalosis

A

pH increased
HCO3 decreased
PaCO2 decreased

50
Q

fully compensated respiratory alkalosis

A

pH normal
HCO3 decreased
PaCO2 decreased

51
Q

uncompensated metabolic acidosis

A

pH decreased
HCO3 decreased
PaCO2 normal

52
Q

partially compensated metabolic acidosis

A

pH decreased
HCO3 decreased
PaCO2 decreased

53
Q

fully compensated metabolic acidosis

A

pH normal
HCO3 decreased
PaCO2 decreased

54
Q

uncompensated metabolic alkalosis

A

pH increased
HCO3 increased
PaCO2 normal

55
Q

partially compensated metabolic alkalosis

A

pH increased
HCO3 increased
PaCO2 increased

56
Q

fully compensated metabolic alkalosis

A

pH normal
HCO3 increased
PaCO2 increased

57
Q

Causes of Respiratory Alkalosis

A
  • fever
  • hyperventilation
  • hypoxia: causes hyperventilation
  • hysteria: causes hyperventilation
  • overventilation by mechanical ventilators
  • pain: overstimulation of the respiratory center in the brain stem results in a carbonic acid deficit.
58
Q

S&S of Respiratory alkalosis

A
  • lethargy
  • lightheadedness
  • confusion
  • tachycardia
  • dysrhythmias (related to hypokalemia from comprensation)
  • nausea
  • vomiting
  • epigastric pain
  • tetany
  • numbness
  • tingling of the extremities
  • hyperreflexia
  • seizures
  • hyperventilation (lungs are unable to compensate when there is a respiratory problem)
59
Q

Interventions for Respiratory Alkalosis

A
  • monitor for signs and symptoms of respiratory distress
  • provide emotional support and reassurance to the client
  • encourage appropriate breathing patterns
  • assist with breath techniques and breathing aid as prescribes: encourage voluntary holding of the breath as appropriate, provide use of a rebreathing mask as prescribed, provid CO2 breaths as prescribed( rebreath into a paperbag)
  • provide cautious care with ventilator clietns so that they are not forced to take breaths too deeply or rapidly.
  • monitor electrolyte values, particularly potassium and calcium levels; monitor ABG levels.
  • prepare to administer calcium gluconate for tetany as prescribed.
60
Q

Causes of metabolic acidosis

A
  • diabetes mellitus or diabetic ketoacidosis
  • excessive ingestion of acetylsalicylic acid (aspirin)
  • high fat diet
  • insufficient metabolism of carbohydrates: lactic acid production…..lactic acidosis
  • malnutrition
  • renal insufficiency
  • severe diarrhea
61
Q

S&S of metabolic acidosis

A
  • drowsiness
  • confusion
  • headache
  • coma
  • decreased blood pressure
  • dysrhythmias (related to hyperkalemia from compensation)
  • warm, flushed skin (related to peripheral vasodilation)
  • nausea, vomiting, diarrhea, abdominal pain
  • deep rapid respirations (compensatory action by the lungs); known as Kussmaul’s respirations