Final study guide Flashcards

1
Q

what are the Phases of healing and what days do the correspond to?

A
  • Hemostasis 0-3 days
  • inflammation 4-6 days
  • Proliferation 4-24 days
  • remodeling/maturation 3weeks to 2 years
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2
Q

Hemostasis

A
  • 0-3 days
    -o Vasocontriction
    o Platelet aggregation
    o Fibrin seals lymph vessels
    o Migration of leukocytes
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3
Q

Inflammation

A
4-6 days
o	Release of histamine
o	Neutrophils early
o	Macrophages late
o	Phagocytosis: removal of bacteria, debris, and foreign bodies
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4
Q

Proliferation

A
-	4-24 days
o	Fibroblast proliferation
o	Fibroplasia
o	Angiogenesis
o	Reorganization of extracellular matrix
o	epithelialization
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5
Q

remodeling/maturation

A

3weeks to 2 years
- o Collagenase breaks down inappropriately oriented collagen mollecules
o New collagen, initially laid down in a chaotic disorganized way, becomes oriented along the lines of contour stress; has more tensile strength
o Scar tissue is = 80 % as atrong as original tissue

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6
Q

Stages of a wound

A

1: non-blanchable erythema of intact skin
2: partial -thickness skin loss with exposed dermis
3: full-thickness skin loss
4: full-thickness skin and tissue loss

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7
Q

Pressure injury stage 1

A

: Pressure injury: non -blanchable erythema of intact skin.
o Intact skin with a localized area of non-blanchable erythema, which may appear differently in darkly pigmented skin. Presence of blanchable erythema or changes in sensation, temperature, or firmness may precede visual changes. Color changes do not include purple or marron discoloration; these may indicate depp tissue pressure injury

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8
Q

Pressure injury stage 2

A

partial-thickness skin loss with exposed dermis
o The wound bed is viable, pink or red, moist and may also presen as an intact or ruptured serum-filled blister. Adipose (fat) is not visible and deeper tissues are bot visible.granulation tissue, slough and eschar are not present. These injuries commonly result from adverse microclimate and shear in the skin over the pelvis and shear in the heel.

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9
Q

Pressure injury stage 3

A

full-thickness skin loss
o Adipose tissue is visible in the ulcer and granulation tissue and epibole are often present. Slough and/or eschar may be visible. The depth of tissue damage varies by anatomical location;areas of significant adiposity can develop deep wounds. Undermining and tunneling may occur. Fascia, muscle, tendon, ligament, cartilage and/or bone are not exposed. If slough or eschar obscures the extent of tissue loss this is an unstageable pressure injury

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10
Q

Pressure injury stage 4

A

o Full-thickness skin and tissue loss with exposed or directly papable fascia, muscle, tendon, ligament, cartilage or bone in the ulcer. Slough and/or eschar may be visible. Epibole, undermining and /or tunneling often occur. Depth varies by anatomical location. If slough or eschar obscures the extent of tissue loss this is an unstageable pressure injury

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11
Q

Define sharp debridement

A
  • Performed by PT, PTA, MD,PA,RN
  • Requires training and skill. Must have goof knowledge of anatomy and ability to differentiate viable and nonviable tissue.
  • Sharp debridement tools: scapel, forceps, scissors, curette
  • Important to know lab values before debridement. May indicate that debridement is contraindicated. Such as condition that will delay healing.
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12
Q

Precautions for sharp debridement

A
  • Prior to debridement look for: Decr Hemoglobin (<12) , Incr INR (>2), Decr platelets (<130)
-	PRECAUTIONS
o	Low RBC count, hematocrit, or hemoglobin
o	Marginal platelets level
o	Poor medical status/poor prognosis
o	On coumadin with marginal INR
o	Near deep structures
o	Hands, genital area
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13
Q

COntraindications for sharp debridement

A
-	CONTRAINDICATIONS
o	Stable eschar
o	Poor medical status/ poor  prognosis
o	Patient on IV Heparin
o	Low platelet count
o	INR above 2.0
o	Gangrene of toes or fingers
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14
Q

Arterial ulcer possible locations

A

over toe joints, anterior shin, over malleoli, under heel

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15
Q

arterial ulcer predisposing factors

A
  • PVD, smoking,diabetes, advanced age, male, hypertension
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16
Q

Arterial ulcer appearance

A

o Thin, shiny, dry skin, hair loss on ankle and foot, thick toenail, elevation pallor, dependent rubor, decreased temperature, absent or diminished pulses, cyanosis, ischemic pain

17
Q

Arterial ulcer wound characteristics

A

o Well-defined wound margins “punched-out”, pale or necrotic, gangrene may be present, minimal exudate, painful, infections are common, blanched or purpuric periwound

18
Q

ABI readings with compression measurements

A
  • Important measurement to screen for: arterial insufficiency, safe level of compression and wound healability.
  • 0.8-1.2 = 30-40 mmHG compression
  • 0.6-0.8 = 20-30 mmHg compression
  • <0.6= no compression
  • CONTRA
    o ABI less than 0.6
19
Q

Long stretch bandage type

A

Ace wrap, used for more aggressive compression. High resting pressure an dlow working pressure.

20
Q

short stretch bandage type

A

Unna boot. Safer than long stretch due to less extensibility and therefore less tension to limb. Squeezes tissue in between muscle and bandage to push fluid out. Low resting pressure and high working pressure

21
Q

Initial first aid for burns in general

A

o Stabilize airway, breathing and circulation
 Check for respiratory distress and evidence of smoke inhalation. Check carotid pulse.
o Evaluate depth and extent of burn injury
o Remove all burnt clothing and foreign material
o Cold water may be indicated, max 20-30 minutes, start within 2 minutes of burn. No ice
o Transfer to burn unit if indicated
o Appropriate antibacterial dressings as needed
o Fluid resuscitation for burns within 2 hours for burns >20% TBSA, using Parkland or Brooke formula

22
Q

first aid for chemical burns

A

usually copious irrigation, Phenols are irrigated with polyethylene glycol. Keep victim warm/calm, monitor vitals often. Monitor vitals frequently

23
Q

Superficial 1st degree

A

o Damage of epidermis. Presents dry, red and blanches. Painful. Resolves in 3-6 days without scarring

24
Q

Superficial partial thickness

-superficial 2nd degree

A

o Damage into papillary dermis. Present with blisters, moist red, weeping, and blanches. Severe pain to touch. Resolves in 1-3 weeks

25
Q

Deep partial thickness

- Deep 2nd degree

A

o Damage into reticular dermis, most skin appendages destroyed. Presents with blisters, wet or dry wax with poor blanching. Decreased sensation to light tough but intact to deep pressure. Usually scars, likely to need surgical incision and possible grafting

26
Q

Full thickness (3rd degree)

A
  • o Damage into subcutaneous tissue. Presents waxy white to leathery dry and inelastic (eschar). Does not blanch. Absent sensation to light pressure, intact to deep pressure. Will need surgical excision and grafting
27
Q

fourth degree

A

o Damage into fascia, muscle and/or bone. Presents usually with eschar. Pain with deep pressure. Will need surgery

28
Q

Rule of nines

A
  • Based on anatomic region. Burn area is calculated to estimate the extent of injury and the prognosis. Superficial burns are not included in the calculation. Less accurate for children
  • Arms are each 9 front and back, legs are each 18 front and back, head is 9, chest is 9 front 9 back, abdomen is 9 front 9 back.
29
Q

Saftey issues when splinting a burn

A
  • Check splints daily for skin breakdown, pressure points.
  • Prevent pressure injury when splinting
  • Educate nursing and family on splint fit , placement and schedule
  • Straps- limit or don’t use due to infection risk; use wrapping instead
  • Neck conformers- custom-made from thermoplast, keeping head in slight extension preventing contractures and webbing of the neck. MUST be in full contact with neck
  • Chest and abdomen: best position- trunk extension, shoulder retraction.
  • Shoulder axilla: shoulder flexion/AB, horizontal ADD, ER
  • Elbow / forearm: full elbow extension forearm neutral
  • Wrist and hand: wrist extensionMP flexion, PIP and DIOP in ful ext. thumb in radial extension and palmar abduction, putting ligaments on stretch
  • Hip: flexion and AB
  • Knee: knee full extension
  • Ankle / foot : neutral alignment with 0 degree or greater of DF
30
Q

Lymph nodes action/purpose

A
  • Filter the lymph fluid, removing bacteria, dead cells, and toxins using macrophages. Produce lymphocytes. Usually located in adipose; only inguinal LN should be palpable
31
Q

Lymph nodes location

A
  • Locations: cervical, axillary, cisternal chyli, iliac and lumbar lymph nodes, inguinal. Also cubital, mediastinal, mammary, shoulder/head/neck, subscapular , popliteal
32
Q

Causes of lymphedema

A
  • Low output failure: damages lymphatic system can’t handle normal interstitial fluid.
  • Chronic venous insufficiency, edema caused by cardiac, liver or renal dysfunction
33
Q

Risk factor of lymphedema

A
    • Risk factors: CA, surgery, trauma, radiation, chronic edema, obesity, immobility, mosquito exposure
34
Q

stages of lymphedema

A
  • Stage 0: no swelling
  • Stage 1: mild edema, indents with pressure
  • Stage 2 : skin thickening
  • Stage 3: extreme edema, skin hardening
35
Q

What does lymph drainage do?

A
  • Stimulates lymph nodes. Stimulates lymphatic vessels to pump fluid in greater volume. Directs stagnant lymph to enter lymphatic vessels for removal. Moves fluid around damaged or impaired lymph areas.
36
Q

Transport capacity in lymphatic system

A
  • Transport capacity: max amount of fluid lymphatic system can carry. Normal lymph time volume is approx. 10% of the transport capacity. The difference between the two is called functional reserve
37
Q

Pracatutions for lymphedema

A
  • Active cancer: no MLD, bandages may be ok
  • CHF: no lymphedema treatment if acute
  • Arterial disease: ok for bandaging at 0.8+ ABI, light bandaging ABI between 0.6 and 0.8
  • Peripheral neuropathy or paralyzed limb
    o Check the skin after pt in bandaging for 1 hr OR use light bandaging. Check capillary nail refill: <2 sec normal.
  • Diabetes; use caution with small vessel disease and sensory impairment
  • Acute DVT
  • Hypertension: monitor BP pre and post bandaging
  • Active cellultis/infection: MLD and bandaging may resume after this is treated