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AUDE 6220 (TIN, MIS, & HYPER) > Final Exam > Flashcards

Final Exam Flashcards

1
Q

what is tinnitus

A

Tinnitus is the perception of sound occurring in the ear or head when no external sound is present
Phantom auditory perception
It is a symptom, not a disease/disorder

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2
Q

what are the theories of peripheral mechanisms

A

cellular mechanisms
edge theory
discordant damage of IHC & OHC
tectorial membrane displacement
NTs & receptors
Synaptopathy

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3
Q

what are the theories of central mechanisms

A

auditory deprivation
inhibitory gating mechanism
hyperactivity & hypersynchrony
neural crosstalk
MOC dysfunction

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4
Q

complex sensory network that allows our brain to perceive and interpret sensations from the body

A

Somatosensory mechanisms

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5
Q

triggers for tinnitus of Somatosensory mechanisms

A

temporomandibular joint syndrome or whiplash
Anything related to the head and neck

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6
Q

what is the limbic system

A

Involved in our behavioral and emotional responses - feeding, reproduction, caring for our young, and fight or flight responses

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7
Q

what is the limbic systems role in tinnitus perception

A

Mediates the emotional response to tinnitus
Those with strong emotional responses often show enhanced sympathetic nervous system activity - fight or flight response

When it is heard there is a reaction and the reaction is controlled by this system and it can make it worse which explains why some cannot ignore it while others can

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8
Q

what can make the tinnitus worse in regards to the limbic system

A

Anatomical or physiological abnormalities in the limbic system - lead to emotional reactions to the tinnitus
if there is any lesion or damage, limbic system will not block the tinnitus signal meaning that all of it reaches the cortex and perceived causing it to be loud and clear

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9
Q

Greater brain connectivity between auditory and limbic areas =

A

more distress levels

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10
Q

subjective tinnitus

A

most common
heard only by the PT
can be bothersome or nonbothersome

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11
Q

common causes of subjective tinnitus

A

Ototoxicity - high-pitched sound
Meniere’s - LF tinnitus with vertigo & HL that fluctuates
OTSC - high-pitched, white noise and can initially be pulsatile

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12
Q

ototoxicity tinnitus

A

high pitched

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13
Q

meniere’s tinnitus

A

LF tinnitus with vertigo & HL that fluctuates

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14
Q

OTSC tinnitus

A

high-pitched
often white noise
can initially begin as pulsatile

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15
Q

how does OTSC cause tinnitus

A

CHL causing deafferentation (reduced auditory information going to the brain)
Reduction of masking effect - ex if you cover your ears you miss hearing the environmental noise
Environmental noise can mask the tinnitus but if you have CHL this is not the case so you hear it more
Rich blood supply causing pulsatile
Arteriovenous malformations
Cochlear tinnitus caused by toxic enzymes produced by otosclerotic bone, bony invasion of the cochlea, and damage to the cochlear blood supply

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16
Q

What is the common description of tinnitus reported by patients with otosclerosis?

A

Usually the first symptom noted is pulsatile in nature.

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17
Q

Tinnitus is a common symptom in otosclerosis patients and sometimes it can appear as the first symptom, explain.

A

If tinnitus is the initial symptom, it suggests that the patient hasn’t yet experienced conductive hearing loss. This means there’s no auditory deprivation triggering the brain to compensate for the reduced auditory input.

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18
Q

objective tinnitus

A

heard by the PT and examiner; sounds generated within the body and can be audible to another person
Rare
Vascular or mechanical in origin - Usually due to vascular disturbances or muscular spasms in the head/neck

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19
Q

causes of objective tinnitus

A

Glomus tumors
ME muscle spasms
Palatal myoclonus
ETD

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20
Q

acute vs chronic tinnitus

A

acute - short duration, <6 mos, associated w/ recent exposure triggers, high chance of spontaneous recovery

chronic - persistent, >6mos, involved in sustained neurobiological changes, less chance of spontaneous recovery

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21
Q

bothersome vs nonbothersome tinnitus

A

bothersome - significantly impacts quality of life

nonbothersome - has minimal impact

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22
Q

primary tinnitus

A

Tinnitus that is idiopathic & may or may not be associated with SNHL (source is unknown)
If we do not know the cause or disorder that caused it

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23
Q

secondary tinnitus

A

Tinnitus that is associated with a specific underlying cause (other than SNHL) or an identifiable organic condition
If we do know the cause of the tinnitus

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24
Q

auditory and non-auditory cauess of secondary tinnitus

A

Auditory causes: impacted cerumen, ME diseases, cochlear abnormalities, auditory nerve pathology
Non-auditory causes: vascular anomalies, myoclonus, intracranial hypertension, tonic tensor tympani syndrome, TMJ disorder

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25
Q

pulsatile tinnitus

A

characterized by the sound that is rhythmic and resembles the heartbeat

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26
Q

somatosensory tinnitus

A

Tinnitus caused or influenced by sensory input in the body
apply pressure or clench jaw etc. and they notice the tinnitus

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27
Q

Somatic and pulsatile could be classified as secondary because we know what is causing it

A

true

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28
Q

what are ways to classify tinnitus

A

temporal characteristics, duration, impact

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29
Q

what are temporal characteristics

A

how often they experience the tinnitus
broken down into spontaneous, temporary, occasional, intermittent & constant

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30
Q

spontaneous

A

sudden sound, usually unilateral lasting 2-3 min

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31
Q

temporary

A

lasts minutes to days often after noise exposure or medications and can accompany TTS

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32
Q

occasional

A

occurs less than weekly (e.g., every few weeks, months or every few months) and lasts at least 5 minutes

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33
Q

intermittent

A

occurring regularly (daily or weekly) and lasts at least 5 minutes

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34
Q

constant

A

continuous sound

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35
Q

what is duration

A

how long you had it

classified:
recent/acute: experienced less than 6 months
persistent/chronic: experienced for >/= 6 months

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36
Q

impact

A

how does it affect their life or interfere
classified: bothersome or nonbothersome

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37
Q

pulsatile tinnitus

A

In synch with the heartbeat
Rhythmical noise usually the same beat as the heart
Usually caused by blood flow changes near the ear or by a change in awareness of blood flow
whooshing sound

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38
Q

causes of pulsatile tinnitus

A

Hypertension
Hyperthyroidism
Vascular stenosis
Unilateral head or neck pain with abrupt tinnitus onset = carotid dissection
Tinnitus intensity changes with head movement = venous source

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39
Q

how to determine if it is pulsatile tinnitus

A

Complete audio testing - comprehensive audio, tymps, reflexes
DPOAEs
Find pulse to determine if pulsing matches tinnitus
Tinnitus of venous origin can be suppressed by compressing the jugular vein
Use 2 fingers to press firmly on SCM muscle & carotid artery for 10 sec. On the right and left sides of the neck
Ask PT if pulsing changes in any way (may stay, go away or change to a different sound)
If it does change, refer for medical workout of vascular pathways

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40
Q

glomus jugulare symptoms

A

HL is the main presentation - unilateral CHL or mixed
Pulsatile tinnitus (decreases when you push on carotid), vertigo, otorrhea, CN involvement
Red ™ - rising sun appearance

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41
Q

what is clicking tinnitus

A

Rare
Can be objective; somatosounds
Not all are detectable by examiner
appears to be due to contractions of the tensor tympani or the nasopharyngeal muscles controlling the patency of the ET
Or a symptom of ME myoclonus (jerking of a muscle group)
Bilateral clicking = palatal myoclonus

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42
Q

bilateral clicking

A

palatal myoclonus

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43
Q

clinical manifestations of clicking tinnitus

A

May be seen on tymps
Detectable using ARTs or decay
Reflexes sometimes are too short
Have PT tap their leg when they hear it and look for corresponding results to the taps

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44
Q

how to find clicking tinnitus

A

Have PT tap their leg when they hear it and look for corresponding results to the taps on reflex decay

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45
Q

should you refer for clicking or pulsatile tinnitus

A

yes

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46
Q

clinical manifestations of somatosensory tinnitus

A

Normal hearing
Symptoms - High-pitched constant ringing
No other hearing or vestibular complaints & neurological exams show no abnormalities

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47
Q

how to determine if it is SS tinnitus

A

different maneuvers and holding for 5-10s

Temporomandibular joint
Have PT clench their jaw for 10 s. Any change in tinnitus?
Have PT open jaw wide/protrude jaw for at least 5s. Any change in tinnitus?

Maneuvers involving the neck
Active neck movements - with and without resistance

Passive muscular palpation in order to search for myofascial trigger points (MTP) or tender points

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48
Q

Characteristics/Causes of SS tinnitus

A

Muscle tension, TMJ/jaw issues, dental disorders, head injuries, cervical spine issues, chronic stress, teeth clenching, etc

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49
Q

tinnitus red flags to refer for

A

Pulsatile tinnitus
Tinnitus in association with vertigo
Unilateral tinnitus or hearing loss
Examination showing abnormalities of the ears
Tinnitus in association with asymmetric hearing loss
Psychological distress
Significant associated sleep/concentration problems
Anxiety regarding possible underlying pathology
Distressing tinnitus not improving, despite initial treatment
Patient request for ENT/audiological assessment
Tinnitus in association with significant neurological symptoms

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50
Q

An 87-year-old woman had a year of intermittent severe hissing tinnitus that could be left ear only or bilateral. It followed a cyclical pattern. Following a day or two of no tinnitus, it would begin softly in the left ear but over the next two days become progressively louder as it spread to both ears. It would then lessen over the next two days or so, until the tinnitus was heard only in the left ear and ultimately would disappear completely. She had a long history of bruxism and muscle contraction headaches (tightness over her temples and jaw). When her tinnitus was severe her headaches were severe.
Her audiogram showed symmetric mild sloping to moderately severe SNHL, bilaterally.
At a visit when her tinnitus was extremely loud (10/10) and bilateral, somatic testing was performed.
Her tinnitus loudness decreased to 4/10 and became unilateral (left ear only).
These changes in her tinnitus persisted for the remainder of that visit (20 minutes).
By the next day, her tinnitus had disappeared completely, but the usual cycle then returned within a day or two.

A

Type: somatosensory (changes intensity when things change - she has jaw problems, clenching causes migraine,s and comes and goes with certain things)
Maneuver relaxed the muscle which is why it could’ve decreased the intensity

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51
Q

A 33-year-old man was seen because of non-lateralized tinnitus. His mother had been diagnosed with terminal lung cancer about three weeks earlier, and he had been closely involved with her care. Two days before her death as he was trying to sleep, his tinnitus began. It was described as a high-pitched ringing similar in pitch but much louder than the transient tinnitus he had previously experienced following loud sound exposure. Clenching or turning his head would aggravate his tinnitus. His exam and audiogram were normal. Despite four years of a variety of treatments, his tinnitus has persisted, and he remains distressed. what is the tinnitus & what referrals should be made if any

A

Type: dishabituation because he experienced tinnitus before and it was gone (occurred due to some change, he habituates and then he is fine and it comes back and the brain perceives it as a new stimulus)
Value of digging into the history of PT
He has normal hearing

Mental health services to deal with the stress and this might change the vicious cycle

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52
Q

Most common sound a patient will report for tinnitus is

A

ringing

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53
Q

pushing/whoosing

A

pulsatile

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54
Q

Count the beats you feel and at the same time ask the PT to count the number of tinnitus beats they hear

A

technique for pulsatile tinnitus

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55
Q

Confirmed with tone decay because it provides a larger time window whereas a reflex is shorter (can se there too) and gives a better chance to see it
Every time you hear the clicking sound tap your foot and watch decay and changes confirm the clicking

A

clicking tinnitus

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56
Q

Related to muscle spasms, etc.
Do maneuvers to confirm this
Changes tinnitus (can be worse, different tone, different volume, etc.)
Change the head and jaw and have the PT tell whether there is a change in tinnitus
They can protrude the jaw or clench the teeth and ask if there is a change
Can do head movements

A

somatosensory tinnitus

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57
Q

Objectives of psychoacoustic assessments

A

Rule out or confirm disease or pathology underlying tinnitus
Document health conditions and factors influencing tinnitus perception
Evaluate auditory function to identify peripheral or central auditory dysfunction associated with mechanisms of tinnitus
Describe and quantify the severity of PT tinnitus
Define impact of tinnitus on quality of life
Contribute to decisions regarding an effective management plan

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58
Q

what is pitch masking and the goal

A

Estimate the general frequency region of the patient’s perceived tinnitus
Goal: to determine the frequency of a tone or noise that is closest to that of the PT’s most bothersome tinnitus

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59
Q

what should the presentation be for pitch masking

A

stimuli at 10 – 20 dB SL at frequencies with normal limits
Stimuli at 5 –10 dB SL at frequencies with hearing loss.

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60
Q

where do you present for pitch masking

A

Start by presenting 1000 & 2000 Hz tone to the “stimulus ear” (contra ear to the ear with tinnitus)
Unilateral tinnitus: present stimulus to the non-tinnitus ear (stimulus ear)

Bilateral tinnitus: present stimulus to the ear with the softest tinnitus. If tinnitus is perceived the same in both ears, test either ear or the ear with the better thresholds.

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61
Q

what is octave confusion

A

where a person incorrectly identifies the pitch of their tinnitus by choosing a frequency that is either one octave above or below the actual tinnitus pitch.

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62
Q

what is loudness matching & the goal

A

Obtain loudness match at the pitch-matched frequency
Goal: to determine the loudness of a tone or noise that is closest to that of the PT’s most bothersome tinnitus

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63
Q

where should you start for presentation for loudness matching

A

Start 5 dB below the AC threshold and then gradually increase the level in 1-dB steps until the patient hears the tone.

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64
Q

what is tinnitus quality match

A

Tinnitus Matching to Bands of Noise
Determine whether tinnitus sounds like a tone or like noise

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65
Q

what is MML

A

minimum masking level

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66
Q

what is MML & the goal

A

Identify the lowest level of BBN that completely masks the tinnitus
Goal: Determine the lowest level of BBN that completely masks the tinnitus (MML).
Determine whether patient’s tinnitus is bilateral or unilateral.

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67
Q

how to present MML

A

The masking noise should be presented binaurally for patients with bilateral tinnitus, and monaurally for patients with unilateral tinnitus.

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68
Q

what is RI

A

residual inhibition

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69
Q

what is the goal of RI

A

To characterize the patient’s tinnitus as a supplement to MML testing and to demonstrate that BBN can have a positive effect.

70
Q

Negative RI

A

no change in tinnitus perception

71
Q

Positive - Complete RI

A

complete absence of tinnitus perception in both ears (0% of the usual tinnitus loudness)

72
Q

Positive - Partial RI

A

If there is a perceptible reduction in tinnitus loudness in one or both ears.
Ask the patient to estimate the current loudness as a percentage of their usual loudness (10%, 25%, 50%, 75%, or 90%).

73
Q

Rebound or Exacerbation

A

If there is an increase in the tinnitus loudness level

74
Q

Initial intake appointments are for most PTs to give brief education counseling & HA’s if needed what does this include

A

Includes basic case hx, audiological assessment, tinnitus screening questionnaires & tinnitus classification

75
Q

Comprehensive assessments are for PTs with severe symptoms & include

A

Comprehensive case hx
Tinnitus impact inventories
Psychoacoustic evals
Mental health screening

76
Q

role of the hypothalamus

A

homeostasis- creates & controls hormones, regulates body temp, thirst, emotions & appetite)

77
Q

role of the thalamus

A

egulates sleep cycles, every sensory function besides smell, thalamic nucleus that includes vision, hearing, and touch - somatosensory)

78
Q

role of amygdala

A

emotions - fear, pleasure, anxiety, anger), & hippocampus (memory conversion

79
Q

what are questionnaires used for

A

Used to assess the impact of tinnitus on a PT including
PTs reactions to it, tinnitus-related distress, the severity of tinnitus, the primary functions affected by tinnitus & other domains

80
Q

purpose of questionnaires

A

Help clinicians
Choose appropriate interventions or referrals
Identify areas that need to be addressed
Document changes when administered before, during, and after intervention
Did the recommendations work for the PT and was it appropriate for their tinnitus because now it is less

Quantify the impact on quality of life

Delineate PT reactions & psychosocial consequences

Identify potential psychological distress (depression & anxiety)

Provide metrics for referral decisionsGuide selection of appropriate management options

81
Q

Why do we do the assessment? (five applied benefit of a tinnitus assessment)

A

Improved patient -provider communication
Tinnitus patient reassurance
Establish a reference point
Basis for treatment
Documentation

82
Q

Score Interpretation of questionnaires

A

for us to figure out how bothersome the patient’s tinnitus is, and how it is impacting their daily life.
The higher the score, the worse it is.

83
Q

questionnaire examples

A

Tinnitus Functional Index (TFI)
Tinnitus Handicap Inventory (THI)
Tinnitus Reaction Questionnaire (TRQ)
Tinnitus & Hearing Survey

84
Q

what does TFI assess

A

used to assess the severity of tinnitus & its negative effects on daily functioning

85
Q

what does THI assess

A

assesses the impact of tinnitus on their daily life

86
Q

what does TRQ assess

A

assesses the psychological distress associated with tinnitus

87
Q

what does the tinnitus and hearing survey assess

A

assists in determining candidacy for tinnitus-specific interventions

88
Q

importance of mental health questionnaires

A

This is important because the stress of mental health can cause an impact on the vicious cycle and to help with tinnitus we need to also help their stressors

can identify suicidal ideation

89
Q

Suicidal ideation step

A

Do they have a plan to end their life?
Yes - suicidal intent, medical emergency
No - suicidal ideation, refers to mental health

90
Q

examples of mental health questionnaires

A

Patient Health Questionnaire (PHQ-9)
Generalized Anxiety Screener (GAD-7)

91
Q

what is the importance of case history

A

Understand the PTs problems related to tinnitus
Identify areas to address in counseling sessions
distinguish between bothersome and non bothersome
Educate PT on probable causes and consequences

92
Q

what elements should be included in case history

A

referral concerns
management history
medical/surgical history
perceptual features
factors that change tinnitus perception
psychosocial & fxnal impacts

93
Q

referral concerns & management hx

A

Who referred the patient?
Why is the patient seeking care?
What previous professional care, if any, has the patient received for his/her tinnitus)?
What were the recommendations?
Were they followed? Did they help?
What treatments/management strategies, if any, has the patient pursued on his/her own?
Did they help?

Have they seen a specialist for it? What have they tried for it? If anything

94
Q

Medical issues that can cause tinnitus

A

neurosurgery especially areas of the auditory cortex affected, brain tumors like acoustic neuromas, TBIs, concussions, & head trauma, cardiovascular diseases, strokes, TMJ, otologic diseases including ME disorders, autoimmune diseases, & metabolic diseases including abnormal thyroid function

95
Q

Medical issues that can impact how they experience the tinnitus

A

mental health disorders (anxiety, depression, PTSD, schizophrenia), brain injuries (affecting learning and memory that can impact tinnitus treatment), cognitive disorders, & chronic pain

96
Q

Medications that can cause temporary tinnitus

A

NSAIDs like aspirin in high risk, loop diuretics like furosemide, & quinine derivatives

97
Q

Medications that can lead to permanent tinnitus

A

ototoxic chemotherapy meds like Vincristine or nitrogen mustard, aminoglycoside antibiotics (erythromycin, streptomycin), and some antidepressants (Buproprion)

98
Q

perceptual features

A

What is the location of the tinnitus (left, right, bilateral, can’t locate)?
What does it sound like (e.g., hissing, ringing, pulsing, etc.)?
How loud is it (eg, 1-10 scale)?
How annoying is it in an average day (eg, 1-10 scale)?
Does the pitch, loudness, and/or quality change?

99
Q

Factors that change tinnitus perception

A

What factors, if any, worsen the patient’s perception of tinnitus?
What factors, if any, improve the patient’s tinnitus perception or reactions to their tinnitus?

100
Q

Psychosocial and functional impacts

A

Sleep disturbance, difficulty concentrating, speech understanding difficulties, avoiding social situations, increased mental/emotional stress, effect on family/friends/coworkers, & depression/suicidal ideation

101
Q

case history picture

A

History of tinnitus
Tinnitus severity
Factors related to its onset
Behavioral, social and interpersonal and emotional consequences of tinnitus
Descriptive characteristics of it
Factors that make it worse or better
Worse - caffeine, no sleep, alcohol etc.
Risk factors for it
co-morbidities

102
Q

order of testing for hearing assessment and why

A

avoid suprathreshold testing first to avoid exacerbating the tinnitus

otoscopy
pure tones
srt
tinnitus eval
HF audio
LDL
WRS
dpoaes
tymps
ARTs
reflex decay
quicksin

103
Q

screening questionnaires

A

Describes & quantifies the severity of PTs tinnitus
Defines the impact of tinnitus on quality of life
Contributes to decisions regarding an effective treatment plan

104
Q

case hx

A

Document health conditions & factors influencing tinnitus perception
Rule out or confirm disease pathology underlying tinnitus
Contribute to decisions regarding an effective management plan

105
Q

hearing assessment

A

Rule out or confirm disease or pathology underlying tinnitus
Evaluate auditory function to identify peripheral or central auditory dysfunction associated with the mechanism of tinnitus
Describes and quantifies the severity of the PT’s tinnitus
Contributes to decisions regarding an effective management plan

106
Q

Unilateral tinnitus
Secondary tinnitus (somatosounds)
Pulsatile tinnitus
Whether it is in beat or not with the heartbeat
Mental health professionals when warranted

A

refer

107
Q

when should you refer to ENT

A

Vestibular symptoms (vertigo/dizziness)
Otalgia & otorrhea
Tinnitus of somatic origin (pulsing w/ heart)

108
Q

when should you refer for ENT or emergency care

A

Tinnitus plus any of the following
Physical trauma - facial palsy
Sudden idiopathic HL

109
Q

when should you refer for mental health or emergency care

A

Tinnitus plus any of the following
Suicidal ideation
Obvious mental health concerns

110
Q

should you refer for SNHL & tinnitus findings

A

advised you do
further diagnostic testing is not needed but otology should rule out physical causes of tinnitus

111
Q

Tinnitus of sensorineural origin
Hx of noise exposure w/ concurrent or subsequent onset of tinnitus
Symmetrical, stable, and non-pulsatile tinnitus
Long duration tinnitus
Audio consistent w/ symmetrical SNHL

A

should refer

112
Q

decrease HL sensitivity, no red flags (it is symmetrical), suggests SNHL, what is the recommendation?

A

HAs
Needs medical clearance

113
Q

what can otoscopy reveal

A

can check for pulsatile tinnitus, might see tumors, cerumen impaction (can cause tinnitus)

114
Q

how can you verify thresholds with pure tones

A

present one after another to confirm, after establishing all of the thresholds for AC go down 5dB and present and there shouldn’t be a response but when you go back to the threshold there will be a response (threshold is 35 dB go to 30 and present and no response then go back to 35 and there should be a response

115
Q

why is SRT done

A

to cross check PTA & make sure pure otnes are reliable

116
Q

why include HF audiometry

A

Especially for those with hearing
Shows HF HL before pure tones will
Early indication there is damage in the IE

117
Q

why do we include LDL

A

Decides whether they have hyperacusis
Helps with HA’s to make sure we don’t exceed this level

118
Q

why is WRS included

A

Used for HA assessment
Shows if there is retro pathology (unilateral tinnitus, HL & poor wrs)

119
Q

why are OAEs included

A

Confirm cochlear origin by identifying absent or below-normal amplitudes in PTs with SNHL
correlatioin between perceived tinnitus and the low or abs amplitudes in OAEs
Identify cochlear dysfunction in PTs with normal hearing sensitivity, providing a physiological explanation for their tinnitus symptoms
if they have unilateral tinnitus this can show abnormal OAEs with normal OAEs in the other ear
Validates auditory basis, especially with normal audios

120
Q

why are tymps included

A

rules out conductive component

121
Q

why include ARTsx

A

Helps to rule out conductive components or if there is a lesion in the reflex arc (tumors)
If PT is sensitive here, you can try using BBN to give an idea of a reflex

122
Q

why do we include reflex decay

A

Would want to include if retro cochlear pathology is suspected (acoustic neuroma, vestibular schwannoma, other lesions in CANS)
Would also want to include if clicking tinnitus is suspected

123
Q

why would yo include quick-sin

A

If suspecting HHL
Individual has normal hearing sensitivity but they share issues with hearing speech in background noise
Shows SNR loss

124
Q

vernon’s 2dB rule

A

Loudness match - repeat 5-6x with other tests intertwined between the repeated tests
Each loudness match is obtained to the nearest 1dB
Presence of tinnitus is indicated if the repeated results within a session agree to be within 2 dB
Different trial runs have to be witin 2dB of each other
Recent studies challenge validity of 2dB rule demonstrating that test-retest reliability is similar for individuals with and without tinnitus

125
Q

what is the goal in those with legal claims

A

establish if the responses are consistent, plausible, & credible across all assessment components

126
Q

Legally, assessments focus on

A

reliability of psycnoacoustic measures , the consistencies of responses, & tinnitus duration determine its permanency

127
Q

what must you keep in mind with those loking for legal claims

A

Must stay alert to exaggerations in self-reports, aiming to confirm consistency, plausibility and credibility of responses

128
Q

case 1
Case hx
High pitched tinnitus in both ears
Began 8 mos ago
Hears it all the time
Believes it is causing hearing problems
Anxious and depressed
Med hx is unremarkable
Audio
Mild to moderate SNHL
Tinnitus & hearing survey
Tinnitus score - 14/16
Hearing score - 13/16
Sound tolerance score - 0/4

what is etiology, temp characteristics, impact, duration, and HL

A

Etiology
Primary
Temporal characteristics
constant
Impact
Bothersome - high tinnitus score with anxiety & depression
Duration
Chronic
HL
yes

129
Q

case 2
Case hx
Referral & Management History: Previous exposure to loud blasts; Initial management with combination instruments, informational counseling & relaxation ineffective.
Medical/Pharmacological History: No significant findings
Perceptual Features: Bilateral, symmetric high-pitched hissing tinnitus; loudness and annoyance rating 7/10
Factors that change tinnitus perception : No significant dietary or lifestyle factors; workplace noise reduces tinnitus perception.
Attitudes & Beliefs: Low self-efficacy for self-management
Psychosocial & Functional Impacts: Anxiety; suicidal ideations; denies suicidal intent.
Social support: Limited.
Tinnitus impact inventory - significant tinnitus
Mental health screening - severe anxiety
Audio
Normal hearing & normal OAEs

What factors in Janis’s history might be related to the etiology of her tinnitus?
What factors in Janis’s history might be exacerbating her tinnitus?
Does Janis require additional tinnitus intervention?
What referrals may be appropriate for Janis?

A

Noise exposure

Her stress & anxiety or poor sleep quality causing additional stress

yes

referral for counseling or mental health support for the PT

130
Q

Case 3
Case history
Referral: referred by his primary care physician.
The patient was employed in a post office where his duties included unloading and sorting mail. History was remarkable for chronic exposure to recreational noise (loud engines and sports events).
Perceptual Features: Bilateral, high-pitched ringing tinnitus. Tinnitus is noticed three weeks earlier after waking up one morning.
Patient had a history of bruxism. He reported trying two different over-the-counter mouth guards to minimize the condition. Despite wearing these mouth guards, the patient’s tinnitus became more noticeable. It was unclear if his bruxism intensified or the over-the-counter mouth guards caused the tinnitus to become more noticeable.
He reported an increase in anxiety in the past month with several panic attacks, for which he took Alprazolam, that was prescribed to him by another clinician on several occasions.
Management history: None
Tinnitus impact questionnaire
Severe
Audio results
Normal otoscopy, mild HF SNHL, mild ultra HF loss
What factors in Chris’s history might be related to the etiology of her tinnitus?
What factors in Chris’s history might be exacerbating her tinnitus?
Does Chris require additional tinnitus intervention?
What referrals may be appropriate for Chris?

A

noise exposure, history of TMJ, and stress (possibly the medication could have made it worse but he had the tinnitus before the medication)

stress, jaw, possibly the medication

yes

referral to TMJ specialist because it is affecting his neck and everything and could be increasing his tinnitus

131
Q

what are fxnal effects of tinnitus

A

Sleep disturbances, difficulties concentrating and varying emotional impacts
There is no solution to fit everyone because the causes are all different from patient to patient and we do not know the exact mechanism that causes the tinnitus

132
Q

Multidisciplinary collab/referrals

A

Do the assessment to help them and as you identify certain issues you want to refer them to specialties to help manage what could be the cause
Otolaryngology
Psychology
Cardiologist
TMJ specialist
Massage therapist: Get a massage to relieve stress and tension
Neurologist
Sleep specialist - for those that are unable to fall asleep

133
Q

what are managment options

A

HAs
Sound therapy
Masking devices
Music therapy
Sound apps
Lifestyle modifications
Education & counseling
TRT
CBT
Mindfulness
Physiotherapy
Transcranial magnetic stimulation
Bimodal neuromodulation
Drug therapies
Supplements
Low-level laser therapy (LLLT) - a light therapy that has been used to treat tinnitus, with some studies showing positive results

134
Q

If they have bothersome tinnitus with mild HL would you recommend HA’s?

A

yes

135
Q

how do HA’s help

A

Improves hearing-related quality of life
Reduces attention to innitus
Reduces stress & fatigue related to straining to hear
Stimulates the auditory system
Might prevent maladaptive neuroplastic changes in the auditory system

136
Q

what do HA work best with for tinnitus

A

LF hearing is good
Tinnitus pitch below 8kHz (within HA fitting range)
Strong reactions to tinnitus

137
Q

which Rx method is best for tinnitus

A

DSL has more gain for the LFs
Most of the ambient noise is LF so this helps to cover the tinnitus by giving the PT access to the LF soft sounds (ambient sound)

138
Q

what features are good for HA’s

A

Bilateral HL = bilateral amp
Open dome = less LF occlusion - allows more natural sound in to mask the tinnitus
Low compression TK = more audibility of soft sounds (adds more gain to low intesnsity sounds)
Turn off expansion = expansion reduces amplification of soft sounds to prevent them from distracting the wearer
But in tinnitus we want this
Omnidirectional mics = allows sounds from different directions to allow stimulation and ignore the tinnitus
Noise reduction off
Sound therapy program
BT connection to access therapy sounds
Frequency lowering - depends

139
Q

what is sound therapy

A

Uses sound to decrease the loudness or annoyance of tinnitus

140
Q

what are types of sound therapies used

A

music
noise - white, pink, brown, BB, NB
Relaxation or environmental sounds
Waves, rain, breezes, etc.

141
Q

Lit Candle in a Dark Room Analogy

A

In a dark space the candle is bright because the room is so dark and you focus more on the light
if you hear nothing and then there is tinnitus then they are paying more attention to it – gets louder in quiet – because they pay attention to it more in the presence of nothing
If you take that same candle and turn the lights on or put it in a lit room the candle is still there but it is less noticeable because you can focus on other things
if there are external sounds, the tinnitus is still there but you are not paing attention to it as much as before because there are other sounds that keep the PT busy

142
Q

Rationale for sound therapy

A

To reduce the audibility of tinnitus by replacing it with a pleasant sound
Provide stipulation of the auditory pathways to facilitate adaptation and replace the spontaneous activities
Help with relaxation to reduce stress

143
Q

education aspects to include

A

Simple way to share information to the PT so they understand what tinnitus is and the types
Reassure it is not dangerous or life threatening
Information between tinnitus and HL
Explaining that even though there is no cure there are management strategies to reduce the impact on the quality of life
Referral for resources

144
Q

counseling strategies

A

Helping them recognize how their beliefs impact their reactions
Providing coping/managment strategies
*those needing extensive counseling need to be referred to mental health professionals

145
Q

lifestyle modifications

A

Helping them recognize how their beliefs impact their reactions
Providing coping/managment strategies
*those needing extensive counseling need to be referred to mental health professionals

146
Q

mental state achieved by focusing one’s attention on the present moment while acknowledging and accepting one’s feelings, thoughts and bodily sensation

A

mindfulness

147
Q

how does mindfulness help with tinnitus

A

Technique to help manage tinnigus by teaching them to be more present and accepting of their experiences

148
Q

Originally for depression
Therapeutic approach combining mindfulness practices with elements of cognitive therapy to help manage psyhnological conditions

A

Mindfulness-Based Cognitive Therapy

149
Q

Magnetic coil placed near the scalp used to modulate brain activity and stimulate the areas of the brain where we regulate mood
Uses strong electromagnetic signals to reduce neural hyperactivity

A

Repetitive Transcranial Magnetic Stimulation (rTMS)

150
Q

are there drugs that help with tinnitus

A

No FDA approved drugs are available to treat tinnitus

151
Q

give ex of some off. label use that can help

A

Antidepressants - reduce loudness & improve quality of life
Anticonvulsants - stabalize neuronal activity; mixed results
Benzodiazepines - alleviates tinnitus related anxiety & insomnia
Glutamate receptor antagonists - reduce neuronal hyperactivity
Otoprotectants - explored for preventing NIHL & tinnitus; promising results

152
Q

what are the challenges to pharmacological treatment in tinnitus

A

Tinnitus is heterogenous so there is no one size fits all
Brain networks are complex making it hard to study and target effectively
Animal studies don’t fully mimic human tinnitus, which limits their usefulness in finding new treatments.

153
Q

supplements & tinnitus

A

clinicians should not recommend Ginkgo biloba, melatonin, zinc, or other dietary supplements for treating patients with persistent, bothersome tinnitus

154
Q

purpose of CBT

A

reduces negative responses to tinnitus by transforming negative thoughts into positive, realistic ones

Aims to decrease anxiety and depression enhancing overall quality of life
Doesn’t make the tinnitus go away but trains the brain to not treat it as a bad or threatening thing but to learn to ignore it

155
Q

Uses a variety of cognitive and behavioral techniques to help people identify and modify the relationships between their thoughts, behaviors, and emotions

A

CBT

156
Q

principle of CBT

A

based on the basic principle that what we think, how we feel, and how we behave are all closely connected, and each of these factors has a decisive influence on well-being.
Thoughts, emotions, behaviors

157
Q

goal of CBT

A

to alter maladaptive cognitive, emotional, and behavioral responses to tinnitus and not to abolish the sound itself.

158
Q

CBT aims to

A

Address psychological stress
Change negative thoughts about it with behavior modifications
Address emotions and problems to having it and not to tinnitus itself
Improve quality of life

159
Q

ABC model for tinnitus
A

A

A - activating/triggering event
Tinnitus starts

160
Q

ABC model for tinnitus
B

A

belief
Thoughts when they hear the tinnitus
blaming situations or themselves, blaming tinnitus keeping them up at night or etc.
overinterpreting all of these things - negative automatic thoughts

161
Q

ABC model for tinnitus
C

A

emotional consequences
Mad, stressed, depressed, hopeless, desperate

162
Q

what are the prinicples to CBT

A

Structured, set specific goals with time frame to complete
Active participation, homework between sessions, strategies for relapse prevention
Collaborative relationship to facilitate therapeutic process
Motivation for change and to modify behaviors
Strategies used aim to promote habituation to help them manage their perception & reaction to tinnitus

163
Q

components to CBT

A

cognitive & behavioral therapy

164
Q

cognitive therapy component

A

focuses on changing how they think about tinnitus
Teaches to replace negative thoughts with positive or neutral ones with counseling and cognitive restructuring

165
Q

behavioral therapy component

A

focuses on techniques like positive imagery, attention control to diver focus away, relaxation training to ease symptoms & exposure to stressful situations to lessen the impact

166
Q

Elements of cognitive-behavioral therapy

A

Relaxation
Muscle relaxation exercises to reduce tension
Cognitive restructuring
Identifying negative thoughts in response to life events or sources of distress
Attention control techniques
Learning to redirect attention from tinnitus to other environmental details
Smelling coffee, tasking honey etc.
Imagery techniques
PTs are guided to imagine what their tinnitus sounds like and being masked by waterfall or ocean waves without real sounds
Imagining scenarios walking in nature with different sounds or enjoying snow by a fire etc.
Sleep management
Bedtime, worry-time restriction, relaxation etc.

167
Q

Techniques to prevent relapses

A

Discussing risk factors for tinnitus getting worse and HL and creating a proactive plan to manage these symptoms

168
Q

Candidate selection for cbt

A

Ages 40-70yrs
With or without hearing loss
No other severe comorbid psychological conditions
Had tinnitus and/or suffered from it for at least 3 months
Wants to alleviate impact of tinnitus on their quality of life and daily activities

169
Q

Systems stimulated and advantages with bimodal neuromodulation

A

Auditory system & somatosensory system

Combines auditory stimulation via headphones with somatosensory stimulation through a tongue device, targeting both the trigeminal and auditory nerves to potentially alter tinnitus pathways in the brain.

170
Q

Combines auditory stimulation via headphones with somatosensory stimulation through a tongue device, targeting both the trigeminal and auditory nerves to potentially alter tinnitus pathways in the brain.

A

bimodal neuromodulation

171
Q

Indications for Lenire

A

18+yrs
Suffer from at least moderate chronic subjective tinnitus (indicated by THI)

172
Q
A

AUDE 6220 (TIN, MIS, & HYPER) (9 decks)

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