faecal incontinence and constipation Flashcards

1
Q

reversible causes of faecal incontinence

A

faecal loading (for more information see managing faecal incontinence in specific groups)

  • potentially treatable causes of diarrhoea (for example, infective, inflammatory bowel disease, irritable bowel syndrome)

warning signs for lower gastrointestinal cancer

rectal prolapse or third-degree haemorrhoids

acute anal sphincter injury including obstetric and other trauma

acute disc prolapse/cauda equina syndrome.

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2
Q

most common cause of feacal incontinence

A

faecal impaction with overflow diarrhoea

second most common cause is neurogenic dysfunction

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3
Q

medical management for faecal incontinence

A

Loperamide hydrochloride

CANT TOLERATE

Codeine phosphate or co-phenotrope

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4
Q

who do you not loperamide hydrochloride to

A

hard or infrequent stools
acute diarrhoea without a diagnosed cause
an acute flare-up of ulcerative colitis.

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5
Q

what should raise suspicios of impaction with overflow

A

smearing, small amount of type 1 stool or copious type 6/7 stool with no sensation of defaecation

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6
Q

risk of constipation

A

a risk of stercoral perforation and ischaemic bowel in those chronically constipated

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7
Q

define constipation

A

defecation that is unsatisfactory because of infrequent stools, difficulty passing stools, or the sensation of incomplete emptying.

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8
Q

define chronic constipation

A

least 12 weeks in the preceding six months.

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9
Q

define faecal loading/impaction

A

retention of faeces to the extent that spontaneous evacuation is unlikely.

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10
Q

social risk factors of constipation

A
  • Low fibre diet or low calorie intake.
  • Difficult access to toilet, or changes in normal routine or lifestyle.
  • Lack of exercise or reduced mobility.
  • Limited privacy when using the toilet.
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11
Q

psychological risk factors of constipation

A
  • Anxiety and/or depression.
  • Somatization disorders.
  • Eating disorders.
  • History of sexual abuse.
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12
Q

physical risk factors of constipation

A

Female sex.
Older age.
Pyrexia, dehydration, immobility.
Sitting position on a toilet seat compared with the squatting position for defecation

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13
Q

complications of chronic constipation

A

Haemorrhoids or anal fissure.

Progressive faecal retention, distension of the rectum, and loss of sensory and motor function.

Faecal loading and impaction.

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14
Q

complications of chronic faecal loading and impaction

A

Faecal incontinence, which can be embarrassing and distressing.

Chronic dilatation of the colon may cause megacolon.

Bowel obstruction, perforation, or ulceration.

Recurrent urinary tract infections, obstructive uropathy.

Rectal bleeding.

Rectal prolapse.

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15
Q

non specific symptoms seen in elderly constipated

A
  • Confusion or delirium, functional decline.
  • Nausea or loss of appetite.
  • Overflow diarrhoea.
  • Urinary retention.
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16
Q

medical management for constipation

A

bulk forming laxative - ispaghula

osmotic laxative - macrogol

INEFFECTIVE
lactulose

IF TWO LAXATIVES ARE USED FROM DIFF CLASSES FOR AT LEAST 6 MONTHS

Prucalopride - stimulates GI motility

17
Q

constipation criteria

A

frequency - passing stools less than once every 3 days

consistency - hard stool

difficulty

  • initiating evacuation despite regular bowel motions
  • feeling of incomplete evacuation
18
Q

stool hard then what can cause be

A

dehydration

19
Q

problems with act of defecation

A

muscular or neurological

20
Q

medications which affect transit time

A

pain killers - opiates/codeine

antispasmodics
aluminium containing antacids
anticholinergics
verapamil

21
Q

medications that cause water loss or reduce transit

A

diuretics

antihypertensives

22
Q

complications of constipation

A
urinary retention
overflow diarrhoea
bowel obstruction
rarely bowel perforation
faecal impaction
23
Q

non pharmacological management for constipation

A
  • increasing dietary fibre - decrease in portion sizes and lack of dietary fibre lead to reduce motility - 30MG of fibre per day
  • sorbitol - natural osmotic laxative - draws water into the gut leading to the softening of stools

ensure adequate fluid intake - maintain bowel contents and normal transit time

maintain mobility - immobility reduce muscle tone in the bowel and abdominal walls leading to constipation

review toileting conditions

  • lack of privacy
  • waiting for busy staff
  • position

regular toileting - making use of gastrocolic reflex

24
Q

pharmacological management for constipation

A

first line bulk forming laxative - fybogel -

osmotic laxatives - lactulose/macrogels/phosphate enemas

stimulant bisacodyl/senna

stool softener laxatives - docusate

25
Q

how do bulk forming laxatives work

A

Increase the bulk of stools by enabling fluid to be retained within the faeces. Increasing the mass increases peristalsis. Must have adequate fluid hydration to prevent intestinal obstruction. Caution with frail. Not suitable for those taking opioids.

26
Q

how do osmotic laxatives work

A

soften the stool makes them easier to pass by increasing the amount of water in your bowels.

27
Q

how do stimulant bisacodyl work

A

stimulating nerves that control the muscles lining the digestive tract.

28
Q

how does senna work

A

Stimulates muscle in the wall of the large bowel to squeeze harder than usual, pushing stools along and out. Their effect is usually within 8-12 hours. A bedtime dose is recommended so you are likely to feel the urge to go to the toilet sometime the following morning.

29
Q

how does stool softener laxatives work

A

Makes the surface of the stools permeable, water can be absorbed, which increases the fluid content of hard stools.These work by wetting and softening the faeces. The most commonly used is docusate sodium (which also has a weak stimulant action too).

30
Q

If someone has opiod induced constipation what do u do

A

do not prescribe bulk forming laxatives

Offer an osmotic laxative and a stimulant laxative (or docusate is an alternative which also has stool-softening properties).

31
Q

management of faecal loading and/or impaction

A

hard stools - oral macrogol

soft stools - oral macrogol plus oral stimulant laxatives

iNADEQUATE OR TOO SLOW
bisacodyl or mini enema

32
Q

How to examine faecal incontinence

A

Inspect the anus—and ask the patient to strain as if at stool. Look for inflammation, deformities, large haemorrhoids (internal or external), and prolapse

  • Rectal examination—assess anal tone by the pressure on the finger after asking the patient to ‘tighten’; feel for faeces and tumour; it is easy to miss even large internal haemorrhoids, unless proctoscopy is performed
  • Abdominal examination—feel for the descending colon. Work proximally to assess colonic faecal loading (this may be misleading)
  • Neurological examination—look for signs of a peripheral neuropathy and other neurological damage. Check perianal sensation (sacral dermatomes).
33
Q

cuases of faecal incontinece

A

disorders of the anal sphincter

  • sphincter laxity (from many causes)
  • severe haemorrhoids
  • rectal prolapse
  • tumours
  • constipation

any cause of fecal urgency - IBD

disorders of the neurological control of the ano-rectal muscle and sphincter:

  • LMN lesions (neuropathic incontinence)
  • spinal cord lesions
  • cognitive impairment (neurogenic incontinence)
34
Q

complications of faecal constipation

A

stercoral perforation

ischaemic bowel