Facial Nerve Flashcards
How long does it take for tOO% Wallerian degeneration to occur after a compressive conduction block
14 - 21 days.
Defects greater than ___ cannot be rerouted and require cable grafting
15 - 17 mm.
What is Sutherland’s classification for nerve injury
1st degree: reversible conduction block. 2”d degree: Wallerian degeneration occurs but endoneurium stays intact and recovery is usually complete. Yd degree: endoneurium is destroyed but perineurium stays intact and recovery is incomplete. 4th degree: all is destroyed except for the epineurium; recovery is poor. 5th degree: complete nerve transection; untreated recovery is not expected.
Where on the lid is the implant placed
2 mm above the lash line.
What % of patients with Ramsay Hunt syndrome have Vlllth nerve involvement
20%.
What % of patients with Lyme disease have facial nerve paralysis as the sole manifestation
20%.
How long does it take for 100°/o Wallerian degeneration to occur after complete nerve transection
3 - 5 days.
What is the incidence of diabetes mellitus in patients with recurrent Bell’s palsy
31%.
What is the incidence of facial palsy as the presenting symptom of tuberculous mastoiditis
39%.
What is the minimal time for functional return of the facial nerve after anastomosis or grafting
4 - 6 months.
What is the outcome of these patients who are treated with steroids alone
42% have a good outcome.
After microneurovascular muscle transfer, what is the maximum muscle power attainable compared to normal
55%.
What is the incidence of recurrent facial palsy
5-7%.
Between which days after injury is the degree of axonotmesis and neurotmesis unclear
6 - 14.
What is the mean interval to the 1st recurrence
9.8 years.
What is the outcome of patients with Bell’s palsy who have 90% or more degeneration on ENoG within the 1st 14 days of onset and undergo decompression
91 % have a good outcome (House I or II) 7 months after paralysis.
What is the cog
A ridge of bone that extends inferiorly from the tegmen epitympanum and partially separates the anterior epitympanic compartment from the mesoepitympanum.
What causes hemifacial spasm
A vascular loop, most commonly of the anterior or posterior inferior cerebellar artery, impinging on the root of VI I.
What is the most commonly proposed theory of the etiology of Bell’s palsy
Activation of a latent virus present within the geniculate ganglion leading to entrapment, ischemia, and degeneration of the labyrinthine segment of VII.
What is the recommended treatment for Ramsay Hunt syndrome
Acyclovir 800 mg five times a day x I 0 days and prednisone taper x 14 days.
What is Tangier’s disease
Autosomal recessive disorder of lipid metabolism characterized by low apolipoprotein A-1 and H DL levels. Clinical features include facial diplegia, neuropathy, and coronary artery disease.
Which of these result in Wallerian degeneration
Axonotmesis and neurotmesis.
What is the initial treatment for hemifacial spasm
Baclofen.
When is ENoG evaluation meaningful
Between days 3 and 21 after complete loss of voluntary function.