Exotics Flashcards

1
Q

Marek’s disease what birds affected, clinical signs and aetiology

A
  • Only chickens are affected, fairly rare due to vaccine
  • Insignificant in terms of the poultry industry
  • Sulkies particularly infected
    Signs
  • Usually 3-4 months (point of lay)
  • Paresis/paralysis - unable to walk or lameness
    ○ wing, leg, neck
    ○ respiratory, GIT - if these nerves are affected
  • Non-specific signs
    Aetiology
  • Herpesvirus
  • Three serotypes -> evolving and increasing in virulence over the years
    ○ serotype I -> pathogenic, a spectrum of virulence
    ○ serotypes II & III (HVT) -> ubiquitous but avirulent
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2
Q

Lymphoid leucosis what age, clinical signs, transmission and mechanism

A
  • Naturally occurs in chickens
  • Usually > 5 m
  • Clinical Signs - non-specific
    ○ Reduced weight, weakness & anorexia
    ○ Egg parameters, hatchability & fertility
    ○ Non-specific mortality
    Transmission
  • Virus generally comes from the parents
    ○ Chicks hatch with viraemia and shed from all systems (respiratory, gastrointestinal, reproductive)
    Mechanism
  • Affect B cells within the cloacal bursa and B cells become tumour and metastasise to other organs
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3
Q

List some immunosuppressive conditions

A
  • Infectious bursal disease
  • Chicken Infectious Anaemia
  • MD, LL & RET
  • Pigeon circovirus infection (any circovirus)
  • Psittacine Beak & Feather Disease
  • Polyomavirus
  • Mycotoxicosis
  • Nutritional deficiencies
  • Stress
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4
Q

What is important to do when clipping bird wings

A
  • Clip last 10 primary flight feathers
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5
Q

What is good alignment for v/d and lateral projections at radiographs

A

○ Good alignment results in the marrow cavity of the sternum being clearly marked and directly over the backbone.
○ Good alignment of the lateral x-ray will have the acetabulum directly overlying each other and gizzard in direct line to acetabulum (pathology may change this)

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6
Q

What are the 3 main blood vessels to take samples, what is important and how much to take

A
  1. Jugular vein -> large right jugular and smaller left -> common
    ○ Large blood vessels, need to control the head to ensure not bitten
  2. Wing vein
  3. Medial metatarsal vein -> better for ducks and chickens
    - Long bleeding time -> need to be careful with the amount of blood you are taking
    ○ Rule of thumb 1% of body weight
    - Generally don’t need to anaesthetise however if not confident anaesthetising can be done
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7
Q

Haematology/biochem what tube used, what % of immature RBCs is normal and what does a general blood exam include

A
  • Needs to be done manually as no counters for nucleated RBC cells - may estimate which is not accurate
  • 5% presence of immature RBC’s normally as high turnover rate - every 30 days
  • A general blood examination includes CBC
    ○ Should include TP, Uric acid, AST, glucose, Haematology, PCV, WCC and blood film
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8
Q

Haematology/biochem what are important molecules in the liver, kidney, GI and blood glucose

A
  • Liver
    ○ AST (hepatocellular damage), GLDH (sig liver damage if increase), LDH (mostly muscle), ALT (not helpful in birds), bile acids (decreased liver function), CK (is AST liver or not? - could muscle disease or VitE/selenium deficiency)
    § Most common bile pigment in birds is biliverdin but no test available for it
  • Kidney
    ○ Uric acid - main excretory product so good kidney function, Urea - dehydration, Creatinine- not helpful in avian med
  • GI
    ○ Amylase and Lipase, Electrolyte changes, CK, AST and LDH
  • Blood glucose
    ○ Don’t regulate mainly via insulin but by glucagon
    ○ much higher normal blood glucose
    ○ Treatment -> insulin not as good as glucagon
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9
Q

What are 7 factors that can lead to polyuria

A
  1. Stress or excitement
  2. Egg laying
  3. Species - lorrikeets and other nectar feeders
  4. Diet - increase green food or vegetables
  5. Toxicity - heavy metals
  6. Diabetes mellitus
  7. Multiple systemic disease
    Usually associated with LOW INTAKE
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10
Q

what nutritional value is within seeds and pellets and what natural supplements are important

A

Seeds
- Really low in essentials nutrients -> vitamins, minerals and amino acids
- Extremely high in fat
- Birds will pick out favourite seeds -> such as sunflower seeds
Pellets
- Not full nutritional value
- But can mix with vegetables - better base diet than seeds
Natural supplements
- Gum leaves, nuts and flowers
- Other native flowers eg Banksia, bottle brush etc
- Seeding grasses
- Milk thistle
- Dandelions

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11
Q

Anaesthetic drugs what are some examples of premed and induction

A
Pre med 
1) butorphenol 
2) midazolam - sedative 
Induction 
-- Injectable drugs such as ketamine combinations (esp ket/ medetomidine or ket/diazepam) are useful in anseriformes and larger birds such as ostriches.
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12
Q

Euthanasia what are 4 ways of achieving

A

1) Halothane gas - ideal for small birds
2) lethobarb acceptable - Dilute 1:1 with water
○ It is not acceptable to inject lethabarb into the air sacs and the heart is often difficult to injected as it is well guarded by the sternum.
3) cervical dislocation - not in front of owner and consider doing this under GA
4) Can use IM pentobarbital, there could be considerable pain as the injection is highly irritant, use sedation with midazolam or butorphenol first.

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13
Q

Fluid treatment what is maintenance requirements and if 10% dehydration what fluid treatment needed over how many days and how given

A
  • Maintenance requirements of parrots and raptors; 50ml/kg/day or 5% body weight, small species (eg passerines), may require up to 8% body weight.
  • If a 10% dehydration is assumed, 50% of this deficit is replaced in the first 24 hours and the remainder over the following 2 days ie
    – Day 1 – 5% (maintenance)+5% (dehydration) = 10% of body weight.
    – Day2 – 5% +2.5% = 7.5% body weight.
    – Day3 – 5% +2.5% = 7.5% body weight
    Ongoing losses needs to be added onto this
  • total volume over a 24 hour period, every 4-6 hours - less often larger bolus, depends on the type of bird
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14
Q

oral fluids how much can give birds

A
  • 30ml/kg PO q 6-8h may be used in larger birds (parrots) such as waterfowl
  • In pssitacine (finches, magpies) birds the volume should not exceed 20ml/kg may repeat when the crop is empty.
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15
Q

Intraosseous fluids what bones used in, what good for, what bad about and needles used

A
  • Distal ulna or proximal tibia - not pneumatic bones
  • Good for shock treatment and surgical fluids.
  • Painful to insert, best to do under general anaesthetic
  • Use spinal needle as have stylet that prevent bone being stuck at the end of the needle
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16
Q

what are the common antibiotics used in birds, why and common analgesic drugs

A

antibiotics
1) tetracyclines (doxycycline) - psittacosis (oral or IM)
2) penicillin and clavulanic acid - 100mg/kg
- CHICKENS -> production animal -> shouldn’t use baytril
ANALGESIA
- Meloxicam and carprophen (chickens, ducks -> tablet form) have become popular drugs for short term or long term arthritis pain.

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17
Q

What are shock fluid rates for birds

A
  • Shock treatment is usually an infusion of 30ml/kg to 60ml/kg in the first hour with poor perfusion -
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18
Q

What antifungals and corticosteroids use in birds

A

1) Nystatin is great for GI infections.
2) Newer generation drugs such as voriconazole are giving better results for aspergillosis and seem to have better efficacy.
NO CORTICOSTEROID USE

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19
Q

Trichomoniasis (canker) what age, types of birds involved, transmission and treatment

A
• Common in pigeons in small numbers 
• Most common in young post-fledgling 
○ Pigeons, Magpies, Budgies. Raptors 
• Transmitted via feeding of young birds, possible sexual behaviour of regurgitation
treatment - ronidazole
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20
Q

Differentials for trichmoniasis for budgies, pigeons and raptors

A
- Budgies
○ Goitre and other obstructions causing regurgitation
○ Behavioral regurgitation
○ “Megabacteriosis” - a yeast 
○ Crop mycosis (regurgitation and lesions)
○ Vitamin A deficiency 
- Pigeons
○ Herpesvirus
○ Pox
- Raptors
○ Helminthiasis
○ Pox
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21
Q

Stomatitis in gallinacious birds (chickens, turkeys) what is not likely and what is likely

A
  • Trichomoniasis is very rare - NOT THIS ONE
  • Pox
  • ILT (infectious Laryngotracheitis virus)
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22
Q

Megabacteriosis - Avian Gastric Yeast what birds most susceptible, cause, signs, diagnosis and treatment/control

A

○ Budgies/canaries and finches particularly susceptible
○ Generally occurs with other diseases -> possibly a secondary disease to coccidiosis
§ Predisposing factors -> stress, viral infection, nutrition
○ Signs - weight loss, weakness, high morbidity, some mortality, regurgitation
○ Diagnosis - presence of the yeast rods via faecal smear, also can do via histopathology of proventriculus
Treatment/control
§ Amphotericin B (human or bird version) for a few days
§ Acidify drinking water
§ Balanced diet -> finches as above
§ Probiotics -> not cheap

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23
Q

What are the 5 main bacterial enteritis and treatment

A

1) salmonellosis
2) campylobacterosis
3) necrotic enteritis - Clostridium perfringens type A and C (neomycin)
4) ulcerative enteritis (quail disease)
5) avian tuberculosis - mycobacterium avium - reportable

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24
Q

What are some common anti-coccidials

A

Sulfaquinoxaline, Amprolium (coccivet), Toltrazuril (baycox), Trimethoprim

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25
Q

In birds what are the 2 main things kidneys are associated with and describe the renal portal system

A
  • Kidneys associated with:
    ○ Sciatic nerve -> lesions on kidney may result in paresis of limb on same side
    ○ Abdominal air sac -> diseases of air sac may extend into the kidney
  • Renal portal system- valves associated with external iliac vein and vena cava therefore medication directed into vena cava may go into the renal circulation and excreted via kidneys before entering the body
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26
Q

List and describe the 6 parts of the reproductive system of a bird

A
  1. Ovary – yolk production - LEFT ONLY
  2. Infundibulum – fertilisation site and inner albumin layer and chalaza – 15mins
  3. Magnum – bulk of albumin (egg white) – 180 mins
  4. Isthmus – extra protein in albumin and shell membranes - 73 mins
  5. Uterus (shell gland) – ‘plumping’ w/ water and shell formation – 1200 minutes (20h)
  6. Vagina -> close to keep oviduct sterile, until lay then relax and open
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27
Q

What are the 3 main external parasites of birds and the types within

A

1) mites
1. knemidocoptes species
2. dermanyssus - red mite
3. ornithonyssus - northern fowl mite
2) ticks - argas persicus
3) lice

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28
Q

Knemidocoptes clinical signs in different bird species, diagnosis and treatment

A

§ Clinical signs
□ Scaly leg disease -> in chickens
□ In budgies leads to scaly face/scaly leg
□ Canaries and finches leads to tassel foot
§ Diagnosis
□ DEEP scrapings -> visualisation of the adult or immature mites
® Can get so deep done to the bone
§ Treatment
□ Easily treatable
□ Beware of withholding periods for commercial chickens
1) Malathion spray/dust
2) Ivermectin medications -> on the skin surface -
3) Moxidectin -> can be used in birds

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29
Q

Erysipelas cause, what most susceptible, zoonoses? and signs/lesions

A

Cause - Erysipelothrix rhusiopathiae
- Turkeys are susceptible
- ZOONOTIC - swelling of the limbs -> painful
○ If suspect use protective gloves
Signs/lesions
- Acute: sudden death, congested viscera, haemorrhages and fibrin exudates on epicardium with swollen liver/spleen
- Subacute/chronic: debilitation, poor condition, lameness/arthritis and endocarditis

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30
Q

Erysipelas transmission and treatment

A
  • Persists in the environment -> route of entry through skin trauma
    ○ Fighting, poor husbandry, overcrowding, artificial insemination
    § 12-20 weeks of age most common due to increased
    ○ FREERANGE CHICKENS -> generally more susceptible
  • Pigs and sheep can shed it
    Treatment - penicillin with vaccination
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31
Q

what is important about salmonella in chickens, which staph cause issues and 2 forms

A

REPORTABLE
- S. aureus (Coagulase positive -> ONLY ONE THAT CAUSES DISEASE)
- Opportunistic organism -> immunosuppressed,
Two forms
1. Septicaemic
2. Localised form
○ Arthritis, synovitis, osteomyelitis
§ Concrete hard floors -> breaks in skin -> “bumble foot”
§ Femoral head necrosis

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32
Q

Fatty liver when physiological and when pathological in birds

A

○ Normal -> physiological
§ Young newly hatched chicks that are reliant on metabolism of yolk - first 2 weeks of life
§ Laying hens active egg production have naturally fatty liver - yolk contains large lipid content and liver is the main producer of this
§ Nutritional -> large amount of calories without proper exercise
□ Is wanted in some restaurants -> force feed high calorie diets
○ Pathological -> due to fatty liver/haemorrhage syndrome and fatty liver/kidney disease

33
Q

What are the 5 main reasons for neurological signs in younger chicks

A
  1. Thiamine deficiency
  2. Aspergillosis - common
  3. Salmonellosis
  4. Entrococcosis
  5. Avian Encephalomyelitis
34
Q

List the 7 main diseases of the trachea

A
  1. Infectious bronchitis (IB)
  2. Infectious laryngotracheitis (ILT)
  3. Newcastle disease (ND)
  4. Pigeon herpesvirus infection
  5. Cryptosporidiosis
  6. Parasites
  7. Pox
    vaccination important
35
Q

Infectious laryngotracheitis (ILT) what birds does it affect, caused by and transmission

A
  • Only in gallinaceous birds - chickens, pheasants
  • Caused by an alpha-Herpesvirus
  • Transmission
    ○ Carrier birds - FOR LIFE
    ○ Shed in exudates
    ○ Aerosols, fomites, beetles, etc
    ○ Long Incubation period (6-12 d) - can vaccinate in the face of outbreak
36
Q

Infectious laryngotracheitis diagnosis, signs

A
  • Diagnosis
    ○ Histopathology - merging epithelial cells with intra-nucleus inclusion bodies, heterophils
    ○ PCR
    ○ Notifiable disease
  • Signs
    ○ Severe form – acute haemorrhagic to necrotizing tracheitis – severe dyspnoea, gasping, open mouth breathing, cough up blood / blood-stained exudate, significant mortality.
    ○ Mild form – conjunctivitis, rhinitis, tracheitis, reduced production, predisposition to C.R.D.
37
Q

Newcastle disease cause, structures and significance

A
  • Cause - avulavirus (Paramyxoviridae)
  • Affects variety of bird species
  • High mortality and morbidity
  • Socioeconomic losses
  • OIE list A
    ○ An exotic disease to Australia - trade implications
38
Q

What are the 3 forms of newcastle disease, mortality and signs

A

a. Velogenic
§ High mortality
§ Nervous system -> partial or complete paralysis and twisting or the neck
§ Respiratory system - tracheitis, swelling of the face
§ Enteric - caecal lesions, diarrhoea
b. Mesogenic
§ Variable mortality
§ Respiratory
§ Nervous
§ Reduced egg production, quality (shell, pigment, albumen)
§ No enteric signs
c. Lentogenic
§ Mild-subclinical respiratory signs
§ Predisposition for CRD (chronic respiratory disease, like mycoplasma)

39
Q

air sac/trachea mites what birds common in, clinical signs, diagnosis, treatment and control

A
  • Common in canaries, finches, budgies
  • Clinical signs - slight cough, wheezing, dyspnoea in severe infections - open mouth breathing, fluffed up, stopping singing (one of the first signs - as trachea was severely damaged)
  • Diagnosis - clinical signs and response to treatment
  • Treatment - medication with ivermectin - single drop applied to back of neck or within water
  • Control - involve in elimination of carriers birds or by periodic strategic treatment to keep infection at low level
40
Q

Respiratory mycoses cause, age and clinical signs

A
  • Aspergillus fumigatus -> can get from mould in grain
  • Particularly in the first few days of life -> probably came from the hatchery
  • Clinical signs
    ○ Increase mortality (loss of hatchability) - especially in younger birds and in hatcheries
    ○ Respiratory difficulty - lesions in trachea and lungs and sometimes eye
    § Ophthalmitis if lesions in the eye
    ○ Lethargic, off food, loss weight, regurgitate food
    ○ Abnormal pasture, twisted neck, paresis, paralysis -> if brain affected
41
Q

Chlamydiosis what birds affect, stress, zoonosis and the 3 species

A
  • Affects - corallers, cockatiels, pigeons, ducks
  • Stress often leads to reactivation of latent infection
  • ZOONOTIC - notifiable
  • C. psittaci
    ○ Serious diseases in a rage of bird species and mammals - in Europe
    ○ Conflicting reports on prevalence and pathogenesis in chickens
  • C. avium
    ○ Respiratory disease
    ○ Found in pigeons, parrots and possibly wild birds
  • C. gallinacea - in Australia
    ○ Gallinaceous birds and ducks
    ○ Clinical signs in chickens and zoonotic potential to be investigated - no evidence of illness
42
Q

Chlamydiosis treatment and prevention and control

A

Treatment
- Warn clients of zoonotic potential and shedding of pathogen particularly in faeces of the recovered birds
- Supportive therapy
- Tetracyclines for a prolonged period - 6 week
○ Doxycycline is reportedly the most effective to use
Prevention and control
- Carrier birds
- Quarantine
- Hygiene
- stress
- Periodic medication

43
Q

What are the 3 important rule of the ventrodorsal and lateral view on radiograph

A

Ventrodorsal View
- Hourglass appearance of organs - should fit between lines between shoulders and hips
- Good alignment – keel bone directly over back bone
- Heart rule – measure width of heart, ratio with the width of the chest >60% - enlargement
Lateral View
- Good alignment - acetabulum directly overlying each other
- Gizzard is important landmark (can see with grit) needs to be in direct line with acetabulum
- Heart rule – measure length of heart against length of sternum >40% - enlargement

44
Q

What are treatment options for flagellates, trichomonas, budding yeast, fungal hyphae, avian gastric yeast

A
Protozoa
-Flagellates – Metronidazole 
-Trichomonas – Ronidazole 
Fungi 
-Budding yeast (snowmen)– Nystatin
-Fungal hyphae – Itraconazole 
-Avian gastric yeast – Amphotericin B
45
Q

hospital patients how feed and how much for bird, parrot, baby parrot and pigeons

A
  • Use crop feeding tube
  • Serve mixture warm (38-39 degrees) -> If it’s too cold -> Shock crop -> Crop stasis
  • Amount
    1. Unknown bird/species/requirements – 3% body weight is safe
    2. Parrot– 7% body weight
    3. baby parrots – 10%
    4. Adult pigeons – 10%
46
Q

caeca in chickens, parrots, pigeons and finches

A

chickens - caeca long
parrots - absence
pigeons and finches - short

47
Q

List 9 causes for swelling of reptile eyelids

A
  1. Hypovitaminosis A in chelonians
    ○ Hyperkeratosis, swollen eyes, digestive issues
  2. Blepharitis and conjunctivitis
    ○ Usually hygiene and foreign body related
  3. Foreign material/trauma
  4. Viral (pox/herpes)
  5. Bacterial (primary/secondary to sepsis)
  6. Periocular masses
    ○ Unilateral
  7. Neoplasia
  8. parasitic
  9. caseous abscess formation (tend to be harder than mammalian)
48
Q

What are 6 main issues that can arise of the spectacle

A
  1. Opacity prior to ecdysis (shedding)
  2. Retained spectacle due to dysecdysis (when haven’t shed properly -> lack of humidity or lack of abrasive items to rub against)
  3. Bullous spectaculopathy -> accumulation of clear fluid in Subspectacular space that presents as a bulging of spectacle
    ○ Aspirate to determine the cause but give pain relief as well
  4. Subspectacular abscess
  5. Retrobulbar abscess
  6. Trauma
49
Q

What are 5 common skin conditions of pythons

A
1. Mites 
○ Ophinoyssus natricis 
2. Trauma 
○ Burns, bites from prey, escape attempts, co-feeding, handling 
3. Scale rot 
○ Ventral dermatitis 
4. Blister disease 
○ Humidity too high/wet substrate 
○ Burns 
○ Migrating nematodes 
5. Dysecdysis 
○ Systemic disease 
○ Vivarium set up 
○ Overhandling
50
Q

What are the 4 main skin conditions of turtles

A
1. Shell rot 
○ Water quality/diet/UV
2. Oedema 
3. Scale and carapace deformities 
○ Malnutrition/injuries 
4. Trauma 
○ Co-inhabitants 
○ Tank design 
○ Gardening injuries
51
Q

Rabbit what are the 4 main ectoparasites, clinical signs and diagnosis/treatment for all

A

1) Fur mite – Cheyletiella parasitovorax - waling dandruff, mild pruritis and alopecia
2) Fur mite – Leporacarus gibbus - salt and pepper look
3) Ear mite – Psoroptes cuniculi - crusty exudate in ear, intensely pruritic and painful
4) Rabbit fleas (and cat and dog fleas) - pruritis
Dx -> sticky tape prep
Tx -> selamcentin (revolution) - advantage for flea
NO FIBRONIL IN RABBITS

52
Q

Guinea pigs what are the 2 main ectoparasites, clinical signs, diganosis and treatment

A
1) fleas - pruritis, dull coat, patchy alopecia
Dx - visualisation, flea dirt
2) scabies - pruritis and alopecia 
Dx - deep skin scrap, clinical signs 
Tx -> both selamectin (revolution)
53
Q

Ferrets what are the 3 main ectoparasites, clinical signs, diagnosis/treatment

A
1) Sarcoptic Mange “Foot rot” - alopecia, intense pruritus, pain, nails can slough
Dx - deep skin scrap 
Tx - ivermectin 
2) Ear mite – Otodectes cyanotis - head shake, scratch, pruritis 
Dx - smear
Tx - adovate (moxidectin)
3) Fleas (Dog & Cat)
Dx - visualisation
Tx - advantage (imidacloprid)
54
Q

Mice and rats what are the 3 main ectoparasites, clinical signs, diagnosis and treatment

A

1) Fur mites (multiple species) - greasy pruritic dermatitis, ulceration
Dx - sticky tape prep
Tx - ivermectin
2) Fleas (Dog, Cat, Wild rodent fleas) - dull coat
Dx - visualisation
Tx - frontline (fibronil)
3) Lice (Pediculosis) - weight loss, alopecia
Dx - exclusion and treatment trail
Tx - ivermectin

55
Q

Neoplasia what type common in rabbits, ferrets, guinea pigs and rodents

A
  • Rabbits – none specifically more common
  • Ferrets – mast cell tumours common
  • Guinea pigs – trichofolliculoma
    ○ Benign basal cell epithelioma, surgical excision usually curative though not always needed as not likely to metastasise and rarely locally invasive
  • Rodents – Mammary neoplasia
    ○ Mice = malignant (usually) - not a good prognosis
    ○ Rats = benign
    ○ Both sexes can be affected
    ○ Vast distribution of mammary tissue means they can be almost anywhere on the body
56
Q

Ferret canine distemper virus clinical signs, diagnosis, treatment

A
Clinical signs 
- Conjunctivitis and rhinitis initially
- Pyrexia
- Mandibular and inguinal rash
- Hyperkeratotic lesions of the nose and foot pads with brown crusting
- Neurological signs prior to death
Diagnosis – can do serum antibody titres
Treatment – euthanasia is only humane option
57
Q

Alopecic syndrome in ferrets what are the two main ones and their cause

A

1) hyperoestrogenism (female) - persistent oestrus- need to be mated
2) adrenocortical disease (male) - early neutering, increased adrenal cortex, stimulation by FSH and LH, overproduction of androgens and oestrogens

58
Q

Hyperoestrogenism in female ferrets clinical signs, diagnosis and treatment

A

Clinical signs - swollen vulva, weight loss and lethargy, bilaterally symmetric alopecia
diagnosis - low PCV and pancytopenia
Treatment - spey prior to oestrus, mate to vasectomised male

59
Q

Adrenocortical disease in male or female ferrets clinical signs, diagnosis and treatment

A

CS - Swollen vulva (F)
Urethral obstruction (M)
Weight loss and lethargy
Bilaterally symmetric alopecia
D - low PCV and pancytopenia, adrenal mass
Treatment
Surgical - not without potential complication - can lead to haemorrhage
can be curative
Medical - Deslorelin implant (GnRH agonist)

60
Q

Hypovitaminosis C in guinea pigs what is the requirement, how receive, definciency leads to, clinical signs, diagnosis and treatment

A
  • Absolute requirement of 10mg/kg/day
  • Should source vitamin C from fresh fruits and vegetables
  • Deficiency predisposes to range of infectious disorders
    Clinical signs
  • Rough coat with scaley pinnae
  • Ecchymoses, petechiae and haematomas
  • Painful and swollen joints
    Diagnosis – not really necessary, just supplement
    Treatment – correct the diet and supply 50-100mg/kg/day during initial phase of dietary correction
61
Q

What drug avoid in rabbits and guinea pigs

A

corticosteroids

62
Q

Encephalitozoon cuniculi in rabbits transmission, clinical signs, diagnosis and treatment

A
  • Can be asymptomatic carriers
  • Speculated to be vertically acquired in utero
    Clinical signs
  • More commonly head tilt and neuro signs, renal signs
  • If ocular, then unilateral disease usually with cataract formation
  • Can see spontaneous lens rupture
  • Iris abscess, hypopyon possible
  • Acute uveitis (hyperaemic iris) with progression to chronic indicated by synechia or glaucoma
    Diagnosis - challenging! -> as already exposed so antibodies doesn’t help, if suspicious just treat
    Treatment
  • Fenbendazole for 30 days to kill parasite
  • Symptomatic treatment for inflammatory response - NSAIDS
63
Q

conjunctivitis in guinea pig common agent, diagnosis and treatment

A

Chlamydial conjunctivitis
○ PCR (Chlamydia caviae)
○ Tetracycline based ophthalmic preparation - chlorsig or ocuflox and oral doxycycline if severe
Always check for corneal ulceration as well

64
Q

Pasturellosis how common in rabbits, transmission, zoonotic and what affects the disease

A
  • Gram -ve bacterium - most common cause of respiratory disease in rabbits
  • Commensal
  • Transmission - nasal, oral fomites, vertical
  • Very rarely may be zoonotic
  • Course of disease varies with
    ○ Strain virulence
    ○ Host immune response
    ○ Additional stressors
65
Q

Guinea pig respiratory disease what are the 4 main differential diagnosis and things within

A

○ Infectious
§ Bordetella (rabbit harbour this bacteria so don’t house together)
§ Adenovirus - sudden death
§ Fungal - less common
○ Inflammatory
§ Allergic suspected
○ Neoplastic
§ Primary respiratory adenocarcinoma
§ Metastatic mammary neoplasia - RARE
§ Lymphosarcoma and leukemia leading to dyspnoea as affect mediastinal lymph nodes
○ Diseases affecting other systems - heat stress, pain

66
Q

Ferret cardiac disease how common, normal HR, clinical signs, diagnostics and types

A
  • COMMON
  • Heart more caudal
  • Normal HR = 180-250bpm
  • Clinical signs - exercise intolerance, dyspnoea, weakness, pallor with prolonged CRT
  • Diagnostics - radiograph and echocardiography
    types - DCM common then endocardiosis (mitral valve)
67
Q

what is the most common respiratory pathogen in rats, transmission, leads to,

A
  • Mycoplasma pulmonis
    Transmission via direct contact, aerosolization and transplacentally - MOST RATS EXPOSED EXCEPT FOR SCIENTIFIC FREE
  • Usually sets up a persistent infection
  • Causes:
    ○ Acute: tracheobronchitis or pneumonia
    ○ Chronic: airway inflammation, bronchiectasis
    ○ Sometimes middle ear infection
  • Like asthma - potentiates neurogenic infection - get hypersensitivity
  • Like chronic bronchitis - ongoing pulmonary infeciton
  • Biofilm created allows bacterial colonisation
68
Q

Rodent respiratory disease what are the 6 main treatments needed

A
  1. Supportive care (oxygen, fluids, nutritional, pain relief)
  2. Antimicrobials
    ○ Doxycycline (anti-inflammatory action and penetrates the biofilm)
    ○ Enrofloxacin in combination with doxycycline
    ○ Azithromycin
  3. Bronchodilators (can be given orally or nebulised) – oral dose theophylline concurrently with enrofloxacin then reduce the theophylline dose by 30%
  4. Nebulisation (small particle size <3um) with
    ○ bronchodilators (aminophylline)
  5. Anti-inflammatories
    ○ Meloxicam
  6. Environmental management - important - good ventilation and husbandry
69
Q

What are the 6 main injection sites for marsupials and which marsupials use with each

A
  1. Lateral coccygeal vein in macropods (along either side of the tail).
  2. Ventral coccygeal vein (as for rodents) is useful for smaller marsupials e.g. possums, gliders, small carnivorous marsupials.
  3. Femoral vein/artery. (Direct needle at pulse felt in inguinal region.) Arterial blood is often obtained and digital pressure is required to prevent haematoma formation.
    ○ This vessel is good in koalas and bandicoots, but general anaesthesia is required to access it.
  4. Medial metatarsal vein (small vein along medial aspect of hindleg). Good for wombats.
  5. Cephalic vein can be used in macropods, koalas, wombats (with some difficulty).
  6. Jugular vein -> for those with necks (kangaroos), NOT WOMBATS
70
Q

What is important with diagnostics for GIT diseases in exotics

A

○ Blood work: WBCC (is it anaemic), glucose (high glucose worse prognostic), lactate (not as good), liver enzymes, electrolytes, protein
○ Faecal: float (rule out coccidia), direct smear, centrifuge, gram stain, culture
○ Imaging: ultrasound, radiographs (gut stasis not good), CT, MRI, fluoroscopy
○ Endoscopy/exploratory laparotomy: cytology, histopathology

71
Q

Gut stasis rabbits common TPR findings and 6 main treatments, what is most important

A

○ TPR: (HR <180bpm bradycardia, T <35.1 hypothermia, RR tachypnoea)
1. Rehydration - IV if possible, 2-3XM) - use ear vein -> INITIAL
○ Can also use subcutaneous fluids
2. Analgesia - opioids (GIT issues so only initially) vs NSAIDS (do well) - INITIAL
○ Buprenorphine, meloxicam
3. Prokinetic - varies on area of GI tract
○ Metaclopramide (proximal GI) vs Cisapride (whole GI)
○ Ranitidine (whole GI and inhibits acid secretion)
4. Food - syringe feed critical care - 3-4 big meals per day
○ 10-15 mins/kg QID-BID
5. Treatment -> for other concurrent disease - IMPORTANT
6. Decompress stomach if severe tympany
Will not get better without treatment but shouldn’t treat quickly

72
Q

What are the 3 main GIT problems that can have medical management and what are the options

A

1) trichobezoar
2) foreign body
3) caecal impaciton
Medical generally first options for above
- Fluids: 2-3X M
- Prostaglandin F2 alpha (0.2mg/kg)
- Pineapple juice for trichobezoars
- Mineral oil

73
Q

What is FRECV and FRSCV what animal effect, effects, diagnosis and treatment

A
  1. FRECV -> older ferrets, highly contagious!!!
    ○ Green mucoid diarrhoea (destroys gut lining - villus atrophy)
  2. FRSCV -> younger ferrets
    ○ Abdominal polyadenomegaly, weight loss, anorexia
    Both -> fever, hyperproteinaemia (WOULD THINK OTHERWISE), Anaemia
    Diagnosis - ultrasound, histopathology GI/lymph nodes
    Treatment - none, aim only to reduce inflammatory response and support, usually fatal weeks to months - euthanasia
74
Q

Antibiotic induced GIT disease in exotics what can result, affected species and main drugs

A
  • Fatal clostridial enterotoxaemia, enteritis, colitis
  • Affected species: rabbit, guinea pigs, chinchillas, hamsters
  • Main drugs: beta lactams: oral penicillins (can give injectables), oral cephalosporins, macrolides (erythromycin), lincosamides, tylosin, tetracyclines (GP)
75
Q

What are the 7 steps in the treatment of a dental abscess

A

1) identify the teeth involved
2) extract the tooth
3) instill proloxamer gel or marsupilise
4) check other teeth and other effects
5) pain relief - buprenorphine 3d, meloxicam 2 weeks
6) gut support - syringe feed
7) antibiotics - based on culture for 2 weeks min

76
Q

Rabbits what thinking with haematuria and discoloured urine

A
  • Haematuria
    ○ Think adenocarcinoma in entire females rabbits 4 years and older
    ○ Less common causes are UTIs
  • Discoloured urine
    ○ Pigments and minerals are a normal finding in rabbits urine
    ○ Need to differentiate if true haematuria or plant porphyrins by using cytology or UV light - porphyrins fluorece
77
Q

Guinea pigs main uroliths and causes of haematuria

A
  • Urolithiasis
    ○ Calcium carbonate and oxalate stones -> Required surgical removal
    ○ Common in both sexes over 2 years of age
    ○ Some are dissolvable by diet
  • Haematuria
    ○ UTIs, uroliths, pyometra
78
Q

3 ways to try to stop egg laying behaviour

A
  1. Superlorin (GnRH agonist) - initial stimulation then negative feedback of FSH and LH
    ○ Should last about 4-6 months
    ○ Goes IM and will self dissolve (cannot remove as get stuck to skin and causes more damage when try)
  2. leuprolide acetate (Lupron®)
    ○ a gonadotropin releasing hormone (GnRH) agonist, that acts by down-regulating pituitary GnRH receptors
    ○ Give on day 0,10,30 and then after that whenever needed
    ○ Injection - hard to get as small injection for large volume - avian vets
  3. Ensure photoperiod is regular
    ○ 12 hour photoperiod is idea?