Examinations Flashcards

1
Q

Examine this patient’s temporal lobe

A

Mnemonic = VOLMA 1. Introduce 2. Inspect for scars or features of any neurocutaneous conditions e.g. NF / tuberous sclerosis / Sturge-Weber 3. Visual field assessment to confrontation using a red hat pin 4. Object recognition / Facial recognition (BNT / Famous faces test). “Name these 3 objects…” 5. Language - receptive “fold this piece of paper in half, then half again and then place it under your chair” / conductive “repeat after me…” / expressive “how did you arrive here today?” / nominal “name as many animals as you can in one minute” (normal is 12) 6. Memory - short-term = recall of 3 items and long-term = dates of WW2 (1939-1945) 7. Auditory assessment with noise presented to the contralateral ear i.e. auditory extinction

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2
Q

Examine this patient’s parietal lobe

A

Mnemonic = VNS ALFA ADC Introduce and inspect for scars. Both: Visual fields to confrontation Neglect - line bisection test “draw a line bisecting these three lines half way along them”. Sensation - UL and LL, sensory extinction and agraphaesthesia. Dominant: Astereoagnosis - “Can you identify these objects that I am placing in your hands?” Left/right disorientation - “place you left hand on the table” Finger agnosia - “show me your ring finger” Alexia without agraphia - “Can you write a sentence for me?” “Can you read the sentence for me?” Acalculia - “Subtract 7 from 100 and keep going…” Non-dominant: Dressing apraxia “Can you show me how you put your jacket on?” Constructional apraxia “Can you draw a clock face for me?” “Can you draw these two intersecting hexagons?” “Can you show me how you use a key to open a lock?”

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3
Q

Examine this patient’s frontal lobe

A

Mnemonic = MPSRORACLEs Motor - power in the limbs Premotor -Luria’s hand test (fist, chop and palm), “Can you show me how you use a key to open a lock?” Smell - “Can you identify these smells please?” University of Pennsylvania Smell inventory Test (UPSIT - a booklet of 40 scratch and sniffs) Reflexes - Frontal release signs - palmar-mental, palmar-grasp and glabella tap. Orientation - Time person and place Recall - “Remember these 3 things…” Attention - “What are the months backwards” Concentration - “Spell WORLD backwards” Language - “Name as many animals as you can in one minute.” “How many words beginning with F can you name?” Eye movements - Frontal eye fields

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4
Q

How does macular sparing with a homonymous hemianopia help with identifying the lesion site?

A

Macular sparing is found with an occipital lobe lesion. No macular sparing suggests an optic radiation lesion.

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5
Q

Examine this patient’s head shape.

A
  1. Introduction 2. Inspection for shunts / scars / tracheostomy etc 3. Inspect head shape from the front, side and on top. 4. Comment on head shape, occipital flattening, frontal bossing, eyes - exophthalmos, midface hypoplasia, malocclusion of the teeth, ear position 5. Feel sutures 6. Feel fontanelle 7. Head circumference 8. To complete my examination I would inspect the growth chart, undertake a developmental milestone assessment and request fundoscopy.
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6
Q

Examine this patient’s autonomic function.

A
  1. Introduction 2. Inspect for scars (DBS) 3. Eyes - Horner’s syndrome / Pupillary response 4. Sweating 5. Lying and standing BP 6. Pulse - effect of Valsalva (should cause slight hypertension and bradycardia) 7. Bladder control
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7
Q

Examine this patient’s extrapyramidal system.

A
  1. Introduce - ask to walk 2. Inspect for Tremor, PD features - facial expression / dyskinesias, Scars for DBS - ask if it is switched on? 3. Eye movements 4. Head position 5. Torticolilis / titubation 6. Arm / leg movements - resting tremor, action tremor, intention tremor, finger-nose-finger test 7. Tone 8. Fast alternating movements (dysdiadochokinesia UL and foot tapping LL) 9. Writing / Archimedes spiral (frequency, amplitude and direction) / Vertical and horizontal line drawing
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8
Q

Examine this patient’s eyes

A
  1. Introduce 2. Inspect for scars, ptosis, resting position of the eyes (squint), square wave jerks 3. Ask if they wear glasses and if they can see out of both eyes 4. Visual acuity using a Snellen chart and colour vision with Ishihara 5. Pupillary response to light and accommodation 6. RAPD 7. Visual fields 8. Eye movements in H-shape asking for double vision. Side by side or diagonal. 9. Cover test if has a squint to distinguish a phoria (only with other eye covered) from a tropia (squint).
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9
Q

Examine these patients speech

A
  1. Introduce - ask name, age and occupation
  2. Inspect for scars (head and neck), tracheostomy, PEG
  3. Ask about hand dominance
  4. Dysphasia - Expressive, Receptive, Conductive and Nominal
  5. Dyshonia - ‘eee’ and ‘aaa’, hoarse voice and bovine cough suggest recurrent laryngeal nerve palsy
  6. Dysarthria - “Baby hippopotamus”, “Yellow lorry red lorry” and “British constitution”.
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10
Q

Examine the lower cranial nerves

A
  1. Introduce
  2. Expose and inspect - ACDF / retrosigmoid / suboccipital scars, tracheostomy, PEG

**Look for Horner’s syndrome, facial asymmetry

  1. Trigeminal nerve - sensation in the 3 divisions and onion bulb distribution

**Check the corneal reflex!

Motor - muscles of mastication: clench teeth = temporalis / masseter; open mouth = lateral pterygoids and mouth sided to side = medial pterygoids

  1. Facial nerve - asymmetry, smile, close eyes, show teeth, blow out cheeks and ask about taste (ant 2/3 of tongue)
  2. Vestibulocochlear nerve - nystagmus, vestibulo-ocular reflex and hearing (rub fingers over one ear and whisper in the other). Weber’s and Rinne’s 512Hz tuning fork test.
  3. IX and X - speech - say ‘eee’ and ‘aaa’ - uvula central? *moves away from the side of the lesion. Hoarse voice? Test gag reflex and cough (bovine?)
  4. XI - shrug shoulders and turn head to the left and right against resistance
  5. XII - tongue fasiculations, symmetry, power and atrophy.

To complete my examination I would request a PTA, FNE for vocal cord assessment, a videofluoroscopy for swallowing. I would also complete the rest of the cranial nerve as well as upper and lower limb examination.

I would like to see an MRI scan of the brain.

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11
Q

Examine this patients upper limbs

A
  1. Introduce - ask if any pain
  2. Expose and inspect the neck, the clavicle, the axilla, the arm and the hands for scars, any wasting, fasciculations or abnormal position such as clawing.
  3. Tone
  4. Maneouvres = Make a fist (Benediction sign), straighten out the fingers, OK sign, interossi movement and Froment’s test
  5. Power: Shoulder abd (C5), Elbow flex and wrist ext (C6), Elbow ext and wrist flex (C7), Finger flex (C8), APB and interossei (Median), Finger abd (T1)
  6. Reflexes - Biceps / Supinator (look for the inverted supinator reflex) / Triceps and Hoffman’s. Higher reflexes are Deltoid C5 > Medial pectoral C4 > Trapezius C2/3 > Jaw jerk (Trigeminal spinal nucleus)
  7. Sensation
  8. Proprioception
  9. Coordination - dysdiadochokinesia / dysmetria / intention tremor
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12
Q

Examine the lower limbs

A
  1. Introduction and ask if any pain
  2. Inspect for scars / wasting / trophic changes (venous / arterial skin changes). Look at the spine for scars! ?MMC repair
  3. Assess gait - Trendelenburg, ataxic, apraxic, antalgic, circumducting and high stepping
  4. Tone - Roll, lift knee, clonus
  5. Power - Hip flex L1/2 (Lumbar plexus to iliopsoas), Hip add L2/3 (Obturator n), Knee ext L3/4 (femoral n), Ankle dorsiflexion L5 both inversion (tibial n) and eversion (supfl. peroneal n), EHL L5 (deep peroneal n), hip abduction (L5 superior gluteal n) and planar flexion S1 (tibial n). Knee flexion (S1 sciatic n) Hip ext (S1 inferior gluteal n)
  6. Reflexes - Knee L4) and ankle jerk (S1) **Adductor reflex = L3 and Hamstring reflex = L5 but these are less reliable. Babinski
  7. Proprioception
  8. Sensation
  9. Coordination - run heal down the shin

To complete my examination I would assess perineal sensation and anal tone. I would also assess the upper limbs and obtain an MRI scan and NCS/EMG

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13
Q

Examine the gait

A
  1. Introduce
  2. Expose and inspect the head. spine and legs looking for scars, atrophy and fasciculations. Any cutaneous stigmata?
  3. Ask to walk across the room. Comment on Facial expression, arm swing, shuffling, tremor and Pace (mnemonic = FASTPACE) posture, step size, slapping, stability, turning and balance. Swing and stance phases.
  4. Walk heel toe
  5. Walk on heels
  6. Walk on tiptoes
  7. Rhomberg’s sign
  8. To complete my examination I would examine the upper and lower limbs, cerebellum and cranial nerves. Depending on my findings, I would request an MRI of the brain and spine.
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14
Q

Examine the cerebellum

A
  1. Introduce
  2. Expose and Inspect for scars over the head and spine
  3. Dysdiadochokinesia
  4. Ataxia - assess the gait. Truncal only = vermis/ Extremities = hemispheric
  5. Nystagmus
  6. Intention tremor
  7. Slurred speech
  8. Hypotonia
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15
Q

Examine the median nerve

A
  1. Introduce
  2. Expose and inspect - scars ?CTD ?spinal surgery
  3. Make a fist (benediction sign), ‘OK’ sign (AIN) and lumbricals.
  4. Power - APB, Wrist flexion (radial side vs ulnar side), pronation (pronator teres), finger flexion. **Wrist extension should be strong because this is radial nerve via the C6 root
  5. Sensation over the thumb and palm
  6. Tinel’s and Phalen’s signs
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16
Q

Examine the ulnar nerve

A
  1. Introduce
  2. Expose and Inspect - scars, wasting of the hypothenar eminence and first dorsal interosseous, fasciculations, clawing of the hand. Comment on the carrying angle and
  3. Make a fist and straighten fingers. Ulnar side lumbricals. Froment’s sign
  4. Tone
  5. Power - Wrist flexion (ulnar side vs radial side = median n), finger flexion (weakness in hypothenar eminence only). interossei (PAD and DAB), ulnar side lumbricals, thumb adduction.
  6. Sensation - little and 1/2 ring finger, ulnar aspect of the palm, forearm and arm.
  7. Tinels over Cubital tuneel and Guyon’s canal
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17
Q

Examine the radial nerve

A
  1. Introduce
  2. Inspect - scars, wasting, fasciculations
  3. Make a fist, straighten the fingers (PIN) and cock your wrist backwards (radial). Extend the elbow and abduct the shoulders (axillary involvement suggests posterior cord)
  4. Power in radial nerve muscles direct (triceps, brachioradialis, ext. carpi radialis and supinator), power PIN (finger extension and thumb abduction = abductor pollicis longus as well as thumb extension). Power in deltoids. If deltoids weak then check lat dorsi (both are posterior cord).
  5. Sensation over the dorsum of the 1st web space and check the regimental patch.
  6. Elbow reflex C7 and inverted supinator jerk C6 myeloradiculopathy
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18
Q

Examine the spine

A
  1. Introduce
  2. Expose and inspect - scars front of the neck, back of the neck, thoracotomy, spine, abdomen and wasting in the UL or LL. Look specifically for a Horner’s syndrome.
  3. Cervical spine - flexion, extension, lateral flexion and rotation - Spurling’s sign
  4. Thoracic spine - rotation
  5. Lumbar spine - flexion, extension, lateral flexion and rotation
  6. Do any deformities correct with bending?
  7. SLR
  8. Examination of the UL and LL.
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19
Q

What causes a homonymous horizontal sectoranopia?

A

LGN lesions. IF the horizontal meridian is spared then it is an anterior choroidal artery infarct. If the horizontal meridian is affected then it is a posterior choroidal artery infarct.

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20
Q

What is nystagmus, torticollis and head nodding called?

A

Spasms nutans. Seen in children with OPGs.

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21
Q

How would you examine a patient with facial pain / Examine this patient’s face.

A
  1. Introduce. Ask name, age and occupation.
  2. Inspect for asymmetry of the face, scars, rashes, excess lacrimation and abnormal movement. Ask where the pain is.
  3. Test sensation in V1/2/3 and from outside in (onion bulb) including corneal reflex
  4. Test masseter strength
  5. Test eye movements (Cavernous sinus involvement)
  6. Test facial power
  7. Test hearing
  8. Dentition
  9. I would complete my examination with fundoscopy looking for optic atrophy suggesting MS as well as an upper and lower limb examination.
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22
Q

What is the differential diagnosis of hemifacial spasm?

A

Blepharospasm

Facial dyskinesia

Facial myokymia

Tics associated with Tourettes

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23
Q

What is the differential diagnosis of Trigeminal neuralgia?

A

Trigeminal neuropathic pain

Post-herpetic neuralgia

Atypical facial pain

Trigeminal cephalalgias (SUNCT / SUNA)

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24
Q

Which nuclei contribute to the CN7?

A

The facial nucleus motor nucleus, superior salivatory nucleus (lacrimation) and nucleus tractus solitarius (taste to anterior 2/3 of tongue).

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25
Q

What are the segments of the facial nucleus?

A

Pontine (demylination / stroke / tumour / cavernoma) - syndromes are Gubler-Millard, Raymond-Cestan and Fovilles

Cisternal (CP angle) - (VS / meningioma / epidermoid / met / glomus tumour)

Meatal - (VS / meningioma)

Labyrinthine - (Trauma / Ramsey Hunt syndrome) - dry eye

Tympanic - (cholesteatoma) - hyperacusis

Mastoid - (trauma / surgery ) - loss of taste

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26
Q

What are the causes of conductive hearing loss?

A

Wax

Middle ear effusion

Otitis externa / media

TM rupture

Ossicular dislocation

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27
Q

What are the causes of sensorineural hearing loss?

A

Sensory - cochlear pathology = drugs / meningitis / TORCH infection / trauma

Neural = CN8/ brainstem = CP angle lesions, brainstem tumour, demyeliantion, stroke

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28
Q

What are the causes of vertigo?

A

I separate these into:

Peripheral causes = BPPV / Meniere’s / drugs / viral infections / vascular

Spontaneous semicircular canal dehiscence.

Central = CP angle tumours / vestibular neuronitis / demyelination, / stroke / brainstem tumours

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29
Q

Which Vagus nucleus is responsible for motor to the palate, pharynx and larynx?

A

Nucleus ambiguus

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30
Q

What is Hitzelberger’s sign?

A

Numbness in the posterior ear canal as a result of compression of nervus intermedius

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31
Q

What are the features of Wallenberg’s syndrome?

A

Facial numbness and loss of pain (spinal trigeminal) ipsilateral with loss of sensation over the contralateral body (dorsal columns)

Dysphagia (Nucl ambiguus)

Dysphonia (CN10)

Ataxia and Nystagmus (inferior cerebellar peduncle)

Horner’s syndrome (sympathetic disruption)

Palatal myoclonus (central tegmental tract - part of Mollaret’s triangle)

32
Q

What are the types of pain associated with trigeminal neuralgia?

A

Burchiel type 1 = classical episodic lancinating pain with no pain between attacks.

Burchiel type 2 = Episodc pain but with a constant background pain.

33
Q

What are the treatment options for TN?

A

Medical = AEDS = Carbamazepine (s/e diplopia / ataxia / myelosuppression / dizziness / drowsiness and siADH). Oxcarbazepine is a derivative of carbamazepine that has a better safety profile and does not need level monitoring. Gabapentine is less effective. Lamotrigine / botox / baclofen and phenytoin have also been used.

Antidepressants = amitriptyline

Analgesia = lignocaine nasal spray, Capsaicin cream and steroids.

Percutaneous = RF, Balloon compression and Glycerol

Radiosurgery

Open surgery = MVD

34
Q

What are the outcomes for percutaneous ablative treatments in TN?

A

Immediately after the procedure, all have a good response ranging from 91% with glycerol to 98% with RF/

At 5 years, however, outcomes are 50% with glycerol and 65% with RF.

GK is only 50-80% effective with 25% recurrence. Might be better for MS but has a latency of action (2-3 months).

RF is better for V3 and Balloon is better for V1/2 as there is less risk of corneal numbness and loss of the corneal reflex.

Glycerol is better for bilateral pain.

35
Q

What proportion of bilateral TN have MS?

A

20%

36
Q

Describe the sensation to the pinna?

A

Auriculotemporal (V3) anterior and superior helix

Greater auricular (C2/3) lower helix

CN7/9/10 supply the EAC

Lesser occipital (C2/3) supplies behind the ear

37
Q

What causes TN?

A

Ignition theory = Ephaptic transmission due to partial demyelination of A-beta fibres to A-delta and C-fibres so that normal touch sensation is transmitted as pain.

38
Q

What distributions do TN effect?

A

Most commonly a combination of V2 and V3.

V2>V3 alone.

V1 distribution is uncommon.

Involvement of all 3 distributions in 5% of cases.

39
Q

What are the complications for percutaneous TN treatment?

A

Bleeding

Infection - oral mucosal penetration

V3 weakness - malocclusion of the jaw

Loss of corneal reflex / corneal numbness

Dysaesthesia / anaesthesia Dolorosa

Hearing reduction (tensor tympani V3)

Visual dysfunction / cranial nerve palsy (IOF)

Carotid injury - stroke / CCF

Lower cranial nerve palsy (jugular foramen)

No response / recurrence of pain

Intracranial injury - bleed / seizures

Bradycardia / hypotension seen with 15% of balloon

HSV eruption

40
Q

Describe Hartel’s technique

A

I mark 2.,5 cm lateral from the angle of the mouth. I mark the midpupillary line and a point 3 cm anterior from the tragus. I aim for a target at the intersection of these points. I put LA in the cheek and use my finger in the mouth to prevent oral mucosal penetration. I utilise lateral fluoroscopy and aim for the intersection of the clivus and the petrous ridge. The jaw usually clenches upon entry into the F.Ovale (V3) and CSF egress can be seen from the needle.

41
Q

What stimulation parameters do you use for RF?

A

50Hz stimulation, 1 ms pulse starting at 0.1 mV and building up to 0.5 mV.

The impedance when in the nerve should be 200-300 Ohms

42
Q

What are the complications associated with MVD?

A

Bleeding

Infection

Cerebellar injury

Stroke - brainstem results in hemiparesis

Cranial nerve dysfunction including hearing loss (1%), vertigo, dysphagia, dysphonia

Numbness of the face

Facial weakness

Herpes reactivation

Loss of corneal reflex / anaesthesia dolorosa

CSF leak

Wound complications

43
Q

Draw the brachial plexus.

A
44
Q

Where is the long thoracic nerve usually damaged?

A

It passes over the middle scalene muscle so can be damaged by overhead weight lifting.

45
Q

What are the causes of winged scapula?

A
  1. Serratus anterior weakness - long thoracic nerve
  2. Trapezius weakness - spinal accessory nerve
  3. Rhomboids weakness - dorsal scapula nerve
46
Q

What are the C5 and C6 muscles?

A

C5 = rhomboids and supra/infraspinatus

C6 = biceps. brachioradialis and deltoids

47
Q

How do you distinguish radial from posterior cord injury?

A

In posterior cord, triceps is likely to be involved with latissimus doris (thoracodorsal n.) and deltoid (axillary n.) weakness

48
Q

What muscles are innervated by the radial nerve directly or via the PIN?

A

Radial nerve = triceps, brachioradialis, ext. carpi radialis longus and brevis and supinator

PIN = Ext. carpi ulnaris, ext digitorum / proprius / minimi, abductor and extensor pollicis longus

Supfl radial nerve = sensory only.

49
Q

Where is the site of compression with PIN?

A

Arcade of Frohse under the supinator tendon.

50
Q

What nerve is likely to be affected with a dislocated humerus?

A

Axillary C5/6 > deltoids weakness and numbness over the regimental patch

51
Q

Which muscles are supplied by the median nerve directly or by the AIN?

A

Median = Flex carpi radialis, FDS, pronator teres, palmaris longus and LOAF muscles

AIN = Flex pollicis longus, flex digitorum profundus 1+2 and pronator quadratus.

52
Q

Sites of median nerve compression?

A

Carpal tunnel > Pronator teres > Stuther’s ligament.

53
Q

What is Wartenberg’s sign?

A

Little finger abduction due to weakness in the palmar interossei / abductor digiti minimi (uilnar nerve) so catches when putting hands in pockets.

54
Q

Sites of ulnar nerve compression?

A

Cubital tunnel (Osborn’s ligament)

Struther’s arcade (between the median intermusclar septum and medial head of triceps)

2 head of FCU

Guyon’s canal (only motor deficit)

55
Q

Why does ulnar neuropathy cause clawing?

A

Due to weakness of the 4+5 lumbricals, which results in loss of flexion at the MCP joint and loss of extension at the PIP joint causing clawing.

56
Q

What is the Stewart classification for ulnar neuropathy?

A

Clinical classification

  1. mild = sensory loss only
  2. moderate = motor 4/5
  3. Severe = motor <4/5
57
Q

What are the EMG/NCS features of ulnar neuropathy?

A

CV <50 m/s

Drop >10m/s across the elbow

CMAP reduction >20% across the elbow - more severe suggesting axonal loss

58
Q

Medical causes of entrapment neuropathy?

A

Pregnancy

DM

Hypothyroidism

Acromegaly

Rheumatoid

PMR

Gout

59
Q

What test do you perform for thoracic outlet syndrome?

A

Adson’s test - rotate head to ipsilateral side and extend neck. Deep inspiration whilst feeling the pulse.

Treatment is scalaenectomy and resection of the C7 rib

60
Q

What is Parsonage-Turner syndrome?

A

Intense pain which results in weakness with 2 weeks (80% weakness is at the time)

Confined to the shoulder girdle in 50%

Due to a viral neuroopathy.

61
Q

What are the alternative reflexes to Babinski?

A

Schaeffer (Achilles pinch)

Gordan (Calf pinch)

Oppenheim (Knuckles down shin)

Stronsky (Little toe snapping out)

62
Q

What are the features of a spastic gait?

A

Spasticity is a velocity-dependent increase in tone in response to passive stretch. Assessed using the modified Ashworth scale.

Gait is scissoring, may walk on tiptoes, stiff lower limbs and scuff the bottom of the foot.

In cerebral palsy there is normal sensation.

63
Q

Why does cerebral palsy cause spasticity?

A

Due to periventricular WM lesions causing a loss of the descending inhibition to the spinal cord result in abbarent reflexes

64
Q

How do you grade function in cerebral palsy?

A

Using the Gross Motor Function Classification Score (GMFCS)

  1. Normal
  2. Difficulty walking long distances
  3. Walks with a stick
  4. Needs a wheelchair
  5. Powered wheelchair due to loss of axial tone
65
Q

What is the differential diagnosis of cerebral palsy?

A

Neuromyotonia

Hereditary spastic diplegia

(That is why you need to see the WM periventricular damage on MRI)

66
Q

What are the treatment options for cerebral palsy induced spasticity?

A
  1. Physiotherapy - orthotic devices
  2. Medications - Botox / Baclofen and Tizanidine
  3. Surgery - Orthopaedic tendon releases, SDR and Baclofen pumps

SDR selection criteria = Peacock’s criteria

67
Q

What is the Fasano / Park protocol for neurophysiological monitoring in SDR?

A

Sustained motor responses as a result of direct stimulation of sensory fibres

1 = single motor unit

2 = adjacent motor units

3 = all unilateral motor units

4 = contralateral motor

68
Q

What are the different operations for SDR?

A

Park procedure = T12 laminotomy to expose the bottom on the conus

Peacock = T12-L5 laminoplasty

69
Q

Describe the gait in PD.

A

Festinating gait / Marche a petite pas

Failure of arm swing

Mask like facies

Stooped posture

Small steps

En bloc turning (stiff)

Arm tremor

Imbalance

Freezing

Negative cueing from lines / doorways / shadows

70
Q

What is a Simian gait?

A

Flexed hip and knees with a boucing gait in a patient with PD. Suggests Multisystem atrophy.

71
Q

What gait is suggestive of PSP?

A

Upright posture (not stooped)

Broad based / ataxic gait

Loss of arm swing

Difficulty turning

72
Q

What is the Canterbury classification of CTS?

A
  1. Very mild - only with detailed tests
  2. Mild - DML <4.5 and SNAP CV <50 m/s
  3. Moderate - DML 4-5-6.5 SNAP present
  4. Severe - DML 4.5-6.5 SNAP absent
  5. Very severe - DML >6.5
  6. Extremely severe - DML >6.5 and CMAP amplitude <0.2 mA
73
Q

How do you distinguish C6 radiculopathy from CTS?

A

Spurling’s sign

Sensation over thenar eminence (spared in CTS)

Wrist extension is weak in C6 radiculopathy and strong in CTS

74
Q

What does an inverted supinator reflex mean?

A

Supinator reflex is mute due to C6 radiculopathy

There is flexion of the fingers due to hyperreflexia in C8

Suggests C6 myeloradiculopathy.

75
Q

What are the sites of compression of the radial nerve?

A

Axillary - saturday night palsy

Spiral fractures of the humerus (spare the trapzius)

Arcade of Frohse (supinator muscle) - PIN compression causing a finger drop

76
Q

What are the external (cutaneous) features of Spina bifida occulta?

A

Vary from pigmentation to hairy patch etc

Benign sacrococcygeal dimple - pit between natal cleft

Dermal sinus tract / LDM - skin punctum. Stains for GFAP. Should be explored and resected as risk of meningitis

Diastematomyelia - hairy patch - Type 1 = fibrous or bony spur, 2 dural sacs. Type 2 = 2 spinal cords within one dural sac. Risk of tethering

Sacrococcygeal teratoma - Currarhino triad (= sacral dysgenesis, tethered cord and presacral teratoma, MNX-1 mutation). Look for a cloacal anomaly!

Terminal myelocystocele - big caudal lump.

Spinal lipoma / lipomyelomeningocele - Pang classification - dorsal / caudal / transitional or chaotic.