Examination Features (Med Secrets p59) Flashcards

1
Q

Explain normal splitting of the second heart sound

A

S2 normally split into Aortic (A2) and pulmonary (P2). Degree of splitting varies with respiratory cycle

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

How does inspiration affect splitting of the second heart sound?

A

With inspiration, negative intrathoracic pressure INC venous return to RA and DEC to LHS. Thus:

  • P2 occurs slightly later
  • A2 to occur slightly earlier
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

How does expiration affect splitting of the second heart sound?

A

Negative intrathoracic pressure is eliminated and A2 and P2 occur almost simultaneously

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What is paradoxical S2 splitting?

A

Widening with expiration and shortening with inspiration (opposite of normal)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Causes of paradoxical S2 splitting?

A
  • Aortic insufficiency
  • Aortic stenosis
  • Hypertrophic cardiomyopathy
    Generally conditions that delay A2 by delaying ejection of blood from the LV
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is the significance of a loud P2?

A

Usually indicated pulmonary HTN

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What is S3?

A

Low frequency heart sound just after S2; also called ventricular gallop

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

How is S3 best heard?

A

With bell. Does not change with respiration

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is a pathologic S3?

A

S3 occurring in a variety of pathologic conditions including:

  • CHF
  • mitral valve prolapse
  • thyrotoxicosis
  • coronary artery disease
  • cardiomyopathies
  • pericardial constriction
  • mitral or aortic insufficiency
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is the mechanism behind S3?

A

Controversial: may be due to increase in the blood entering the ventricle (rapid ventricular filling). Usually indicates ventricular decompensation associated with heart disease.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is S4?

A

Sound occurring just before S1. Reflects decreased ventricular compliance (stiff ventricle).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What is an opening snap?

A

High frequency early diastolic sound associated with mitral or tricuspid valve opening.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What causes a pericardial knock?

A

Sudden slowing of LV filling in early diastole associated with the restriction of a rigid pericardium acting as a “rigid shell” such as in chronic constrictive pericarditis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What are the physical examination findings in MR?

A
  • Apical holosystolic murmur
  • S3
  • Quick upstroke and short duration of peripheral pulses
  • Widened pulse pressure
  • Hyperdynamic praecordium
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What are the 3 waves composing the JVP?

A
  • A wave: produced by RA contraction, occurring just before S1
  • C wave: caused by bulging upward of the close tricuspid valve during RV contraction
  • V wave: caused by RA filling just before opening of the tricuspid valve
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What are cannon A waves?

A

Very large and prominent A waves occurring when the atria contract against a closed tricuspid valve.

17
Q

Define pulsus paradoxus

A

Drop of more than 10mmHg in sBP during normal inspiration

18
Q

Why does pulsus paradoxus occur?

A
  • Fall in intrathoracic pressure during inspiration rapidly transmitted through pericardial effusion = exaggerated inc in venous return to R heart.
  • Inc return to R causes septal bulging to L
  • Smaller LV volume