Exam 4 Flashcards
Oviperous
Egg laying-shelled
Viviparous
Live young no shelled eggs
Ovoviviparous
Eggs hatch inside the female
Young are born alive
Whip worm
Trichuris trichiura
Trichuris life cycle
- unembryonated eggs passed in feces (diagnostic)
- 2-cell stage
- advanced cleavage
- embryo after eggs are ingested (infective)
- larvae hatch in small intestine
- adults in cecum
Epidemiology of Trichuriasis
Rectal prolapse
Trichina worm
Trichinella spiralis
Trichinella spiralis
- among the smallest nematode parasites
- same animal or individual serves as both intermediate and primary host
Trichinosis
Stage 1 - penetration of adult female into the mucosa 12hours-2days after infection Low grade infection Worms migration in intestinal epithelium causes -traumatic damage to the host tissue - inflammation - nausea, vomiting, diarrhea -sweating -respiratory difficulties - red skin blotches Stage ends with facial edema and fever
Trichnosis prevention
Cook meat to safe temps Whole cuts- 145F Ground- 160F Clean meat grinders after each use Don’t let pigs and wild animals eat scrap, raw meat or carcasses
Giant kidney worm
Dioctophyme renale
Dioctophyme renale life cycle
- Unembryonated eggs are shed in urine
- Eggs become embryonated in environment
- Eggs ingested by intermediate host. Larvae develop to L3
- Paratenic host consume intermediate; L3 larvae encysted in tissue
- Definitive host becomes infected after ingesting intermediate or paratenic host contains the L3 - infective stage
- Humans become infected accidentally after consuming undercooked paratenic host
Adults in kidney-diagnostic stage
Rhabditiform larvae
L1 and L2 stages
Pathogenic form
Non infective
Feeding
Filariform larvae
Infective form L3 and L4 No skinny Isthmus in esophagus Nonfeeding L4- in body of host
Strongyloides stercoralis
Zoonotic Only the female is parasitic A) parasitic female- homogonic B) free living male C) free living female- heterogonic Female reproduces through parthenogenesis- eggs develop without male - Fecally contaminated soil
Strongyloides life cycle
-Excretion of Strongyloides larvae
-Infective filariform larvae enter through the intact skin
- visceral migration
- adult female living in intestinal mucosa produces eggs that hatch while still in the intestine
(Auto infection cycle)
- some rhabditiform larvae become filariform and renter intestinal mucosa or peri-anal skin and follow the usual infection cycle
Strongyloidiasis
1st phase- cutaneous- invasion of larval stage includes itching swelling
2nd phase- pulmonary phase- migration and bursting out of the lungs can cause lung infection and pneumonia
3rd phase- intestinal phase- female worm in intestinal mucosa cause abdominal pain and shedding of intestinal wall causing blood in fecal matter
Two types of hookworms?
Ancylostoma duodenale (old) Necator americanus (new)
Life cycle of hookworms
- Eggs are passed in feces
- Larvae hatch and develop in soil
- L3 larvae migrate to grass
- L3 larvae penetrate skin causing “ground itch” and enter bloodstream
- L3 larvae enter lungs and alveolar spaces causing coughing
- Coughed up larvae are swallowed
- Larvae reach small intestine, mature and start feeding
What is geophagy and what causes it?
The want to eat soil/dirt
Hookworms
Ascaris lumbricoides
Ascaris suum
Suum- pigs Lum- humans but probably started out with pigs Both can be parasitic for either Large size Probably been around as long as humans Eggs have a thick shell
Life cycle of Ascaris lumbricoides
Eggs in feces 2 cell stage Advanced cleavage 3~4 weeks Infective stage L2 Embryonated egg Ingested Larvae hatches in intestine Burrows into portal circulation Lungs L3 L4 trachea Pharynx Swallowed Adults in small intestine