Exam 3- Knee/Ankle Patho Flashcards

(77 cards)

1
Q

what conditions do we need 2 of to be at risk of DVT

A

venous stasis
hypercoagulability
damage to venous wall

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2
Q

what is a DVT

A

partial or complete occlusion of a vein by a clot

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3
Q

what are situations that lead to a DVT

A

prior DVT
hx of cancer, CHF, or lupus
major infection, sx, or trauma
present chemo
oral contraceptive or hormone therapy
clotting disorder
> 60 yrs

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4
Q

where is DVT most common

A

LE deep veins

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5
Q

what is most preventable hospital related death

A

DVT

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6
Q

what is the most common cause of death/readmission after TKA/THA

A

DVT

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7
Q

what is the pathology of a DVT

A

greater exposure to platelets and clotting factors to damage venous walls

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8
Q

what can help prevent DVT

A

early and regular exercise
anticoagulants
compression stocking
avoid SAD
eliminate persistent smoking and drinking

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9
Q

what are S&S of DVT

A

gradual onset of dull ache, tightness, and pain in the calf with RF
worse with walking and dependent positions
less relief with rest and elevation

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10
Q

what are the review components of DVT

A

increased calf girth
calf pain and tenderness
redness and warmth

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11
Q

what could a DVT lead to

A

pulmonary embolism

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12
Q

what is the referral for DVT

A

emergency >3
urgent <2

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13
Q

what is a pulmonary embolism

A

DVT that moves and lodges into a smaller arteries the lungs

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14
Q

what are S&S of pulmonary embolism

A

SOB, wheezing, rapid breath
sudden sharp stabbing chest pain
mechanical motion

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15
Q

what is the referral for pulmonary embolism

A

urgent <2/6
emergency >2/6

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16
Q

what is PAD

A

ischemia leading to symptoms in the most distal area from blocked a

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17
Q

what are RF/etiology of PAD

A

> 45 yrs of age
family hx of MI or sudden death before 55
smoker
sedentary
metabolic syndrome

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18
Q

what is the pathology of PAD

A

narrowing of blood vessels limiting circulation

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19
Q

what are S&S of PAD

A

LE pain, calf, activity and elevated
unilateral or bilateral
relieved with rest and dependent position

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20
Q

what are the differences between DVT and PAD

A

DVT- pain with dependent, relief with elevation
PAD- pain with elevation, relief with dependent

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21
Q

what are the review components of PAD

A

loss of pulse
TTP
muscle atrophy and weakness- MMT
loss of hair- ob
cool and bluish skin- ob
bruit on auscultation
ankle brachial index

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22
Q

when is the ankle brachial index invalid

A

hx of HTN

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23
Q

how do you perform ankle brachial index

A

assess post tib and brachial systolic BP in all extremities
divide ankle/brachial

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24
Q

what does >.9 and >1.4 ABI mean

A

> .9= PAD
1.4= poorly compressed vv

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25
how could you differentiate calf pain due to PAD and stenosis
bicycle test
26
what is the referral for PAD
urgent
27
what is osteochondritis dissecans
damage to subchondral bone
28
where is osteochondritis dissecans most common at
younger boys medial femoral condyle and talus
29
what can cause osteochondritis dissecans
jt RT or shear trauma
30
what is the pathology of osteochondritis dissecans
ischemia then separation of subchondral from convex and WB bones
31
what are S&S of osteochondritis dissecans
persistent severe pain jt locking, catching, and swelling hypermobility plus ARJC cannot progress to WB
32
what are review components of osteochondritis dissecans
ROM- limited and painful with ext RST- weak and painful end range ext comp + TTP at femoral condyle
33
what is the referral for osteochondritis dissecans
urgent
34
what factors can play a role in healing for osteochondritis dissecans
higher BMI passive restraints m imbalances impaired proprioception
35
what is the management for an unstable lesion for osteochondritis dissecans
sx for resurfacing or fixation
36
what is the management for stable lesion for osteochondritis dissecans
rx for instability with ARJC
37
what are RF for reactive arthritis
IV drug users high sexual activity infrequent pelvic exams weaker immune system
38
where does septic arthritis infection come from
site of primary infection
39
where are acute infection come from
remote site from primary infection
40
where is reactive arthritis most common
larger LE jt- knee and ankle
41
what can cause reactive arthritis
respiratory infection GI, urinary, and colon infection
42
what is the pathology of reactive arthritis
bacteria stimulates antibodies creating inflammation and tissue damage
43
what are S&S of reactive arthritis
infection S&S autoimmune S&S
44
what are review components of reactive arthritis
ob- redness, swelling vitals- temp ARJC S&S palpation- TTP, warm, swollen tender lymph nodes
45
what is the referral for reactive arthritis
urgent
46
what is diabetes mellitus
chronic systemic disorder characterized by hyperglycemia and abnormal metabolism
47
what is insulin
released from pancreas lowers blood sugar stores fat
48
what is type 1 diabetes
auto immunity affecting the pancreas that produces insulin deficiency of insulin production
49
what is type 2 diabetes
excessive dietary sugar and other simple carbohydrates limits effect of insulin
50
how can more sugar affect insulin
more sugar = more insulin production = more fat
51
what are cardinal S&S for diabetes
frequent urination dry mouth excessive thirst decreased skin turgor blurry vision weakness/fatigue
52
what are the neuropathies diabetes can cause
sensory motor autonomic
53
how is sensory affected by diabetes
peripheral n hyposensitivity or numbness less aware of heart attack
54
how is motor affected by diabetes
weakness of mm due to innervation of peripheral n
55
how is autonomic affected by diabetes
diminished pulses necrosis poor healing stroke cardiac dz
56
what can diabetes lead to if not managed well
kidney dz and blindness cognitive dysfunction- alzheimers (type 3)
57
what are the review components of diabetes
ob- charcot foot, dry mouth, cognitive decline, fruity breath ROM- limited and painful RST- possible weakness neuro- diminshed sensation, + dural palaption- diminished pulses
58
due to persistent inflammation and impaired circulation of diabetes, what are these pts more susceptible to
carpal tunnel syndrome adhesive capsulitis trigger finger delayed healing nociplastic pain
59
why should diabetics exercise 3 10 minute bouts
eat, burn sugar, eat, burn sugar
60
why should diabetics wait 1-2 hrs after a meal to workout
meals increase blood sugar and insulin and exercise work to decrease blood sugar
61
what education can be given to diabetic pts
wear accommodating shoes and socks examine feet avoid alcohol and cortisone shots
62
what is systemic inflammation a primary contributor to
diabetes HTN high triglycerides low HDL being overweight
63
what is gout
metabolic disorder with elevated levels of uric acid and deposition of urate crystals
64
where is the most common site for gout
1st MTP most common crystallopathy
65
what are the RF for gout
family hx decreased renal function high fructose SAD high nitrogen diets
66
what can cause gout
genetic
67
what is the pathology of gout
uric acid typically forms from breaking down cellular waste in the bloodstream kidneys unable to process uric acid
68
what are the symptoms of gout
sudden onset of severe jt pain night or morning increasing frequency
69
when do symptoms of gout start
10-12 yrs of hyperuricemia
70
what are the review components of gout
ob- redness, swelling palpation- warmth, fever ROM- limited and painful, ext RST- possible weak CM- consistent block
71
what can we do to help pt with gout
education on causes and RF
72
what is osteomyelitis
inflammation of bone
73
where is osteomyelitis most common
tarsal metatarsal tibia femur
74
what are RF for osteomyelitis
immunosuppression chronic illness (diabetes) IV drug use jt replacement
75
what can cause osteomyelitis
microorganism binds to cartilage spreads quickly through metaphysis
76
what are S&S of osteomyelitis
gradual onset of deep achy pain/stiffness infection S&S localized and progressive pain limits motion and WB
77
what are the review components of osteomyelitis
ob- painful asymmetric gait, red and swollen temperature- warmth, fever ROM- limited and painful, ext RST- possible weak CM- consistent block