exam 2 Flashcards

(35 cards)

1
Q

preload

A

End-diastolic pressure: when ventricles are filled and heart is relaxed
“stretch”

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2
Q

afterload

A

Work (or force) required to move (eject) blood into the aorta
“squeeze”

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3
Q

contractility

A

electrical contractions and muscle contraction of heart

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4
Q

alterations in SV

A

indicates issue in systole, diastole,peripheral disease

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5
Q

alterations in CO

A

could result in poor perfusion or overworking

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6
Q

systolic heart failure

A

decrease ability to pump due to increased afterload decreased EF
cant get blood through aorta and it backs up into the lungs

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7
Q

diastolic heart failure

A

decreased ability to fill leads to decreased SV and CO
cant get blood out of atrium and into venticle, backs into lungs
normal EF

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8
Q

left sided HF

A

blood can’t get from left side through aorta and to rest to body
-leads to increased pulmonary venous pressure (pulmonary congestion, edema)
-low organ perfusion
hypoxia, tissue death

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9
Q

right sided HF

A

results from progression of LSHF
-right side cant get blood to lungs bc of increased pulmon vasc pressure from LSFH
-back flow of blood back to other organs
-peripheral edema
-renal failure,hepatomegaly

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10
Q

pulmonary embolism

A

blockage of 1 or more pulmonary arteries
pt will not perfuse, check v/q scans

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11
Q

pulmonary hypertension

A

pressure in blood vessels from heart to lungs is too high
5 types
S/S: sob, fainting, chest pain, dizziness, tachycardia

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12
Q

Acute respiratory disorders

A

failure to add O2 and remove CO2 from blood

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13
Q

ARDS

A

-noncardiogenic pulmonary edema & hypoxemia caused by alveolar inflammation and infection
-requires mechanical ventilation
-

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14
Q

ARF

A

failure of the lungs for any reason that impairs ventilation, compromises v/q or impair gas diffusion
-manifests as PaO2 <50%, acidosis, cyanosis, tachypnea/tachycardia

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15
Q

COVID-19

A

inflammatory response isnt working
lung sustains acute injury, leading to ARDS

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16
Q

Pleural effusion

A

-“junk around lungs”
-excess fluid nw layers of pleura outside lung
caused by heart problems, cancer, pneumonia, pulmonary embolism

17
Q

pneumonia

A

-“junk in lungs”
-inflammation of bronchioles and alveoli
-wbc fight foreign invaders
-dead cell and debris build up creating ous filling parts of airway
-causes by infectious and non-infectious agents inhalation of irritants

18
Q

pulmonary edema

A

-“junk in lungs”
-lungs filled with fluid
-lung congestion, water on lungs
-increased WOB
-causes: fluid overload, hf

19
Q

Atelectasis

A

-“poor lung expansion”
-alveoli dont fill properly/incomplete lung expansion
-caused by immobility, mucus plug, external pressure (mass)

20
Q

pneumothoraces-collapsed lungs

A

-“poor lung expansion”
-air enters pleural space and disrupts negative pressure, causes lung collapse

21
Q

refractory asthma

A

due to allergic reactions
massive immune response
airway remodeling
wheezing on exhale

22
Q

COPD

A

caused my smoking, wood burning, occupational hazards

23
Q

emphysema

A

destruction of alveoli
responds to low O2 rather than CO2
pink puffer

24
Q

chronic bronchitis

A

long term inflammation of bronchi and hypersecretion of mucus
more comorbidities, sleep apnea, acidosis
blue bloater

25
bronchiectasis
permanent dilation of bronchi loose ability to contract and decrease elasticity associated with cystic fibrosis
26
cystic fibrosis
genetic condition causing mutation of transmembrane conductance regulator -cilia unable to push mucus out, build up, mucus plug -no real cure
27
intrinsic restrictive lung disease
problem w lungs themselves -stiff lungs due to thickened alveolar walls -decreased total lung volume -idiopathic pulmonary fibrosis -massive scarring impairs gas exchange, no cure
28
extrinsic restrictive lung disease
problem from something other than lungs -pressure placed on lungs or paralysis of muscle that helps with breathing
29
major functions of renal system
filter blood of toxins/waste and reabsorb needed molecules, regulates hemoglobin production, increase calcium absorption
30
urine formation
glomerular filtration, reabsorption, secretion
31
common causes of renal diesease
tubular obstructions that increase intratubular pressure, developmental defects, pregnancy, altered immune responses cancer
32
risk factors for renal disease
33
pre-renal
decreased perfusion to normal kidney reduced hydrostatic pressure-> reduced glomerular filtration hypovolemia reduced co reduced effective circulatory volume
34
intra-renal
glomerulonephritis tubular disease: acute tubular necrosis, most common in hospital interstitial disease: acute interstitial nephritis vascular: vasculitis
35
post-renal
obstruction along urinary tract renal stones prostate enlargement/cancer cervical cancer bladder cancer can lead to intra renal causes