exam 2 Flashcards
preload
End-diastolic pressure: when ventricles are filled and heart is relaxed
“stretch”
afterload
Work (or force) required to move (eject) blood into the aorta
“squeeze”
contractility
electrical contractions and muscle contraction of heart
alterations in SV
indicates issue in systole, diastole,peripheral disease
alterations in CO
could result in poor perfusion or overworking
systolic heart failure
decrease ability to pump due to increased afterload decreased EF
cant get blood through aorta and it backs up into the lungs
diastolic heart failure
decreased ability to fill leads to decreased SV and CO
cant get blood out of atrium and into venticle, backs into lungs
normal EF
left sided HF
blood can’t get from left side through aorta and to rest to body
-leads to increased pulmonary venous pressure (pulmonary congestion, edema)
-low organ perfusion
hypoxia, tissue death
right sided HF
results from progression of LSHF
-right side cant get blood to lungs bc of increased pulmon vasc pressure from LSFH
-back flow of blood back to other organs
-peripheral edema
-renal failure,hepatomegaly
pulmonary embolism
blockage of 1 or more pulmonary arteries
pt will not perfuse, check v/q scans
pulmonary hypertension
pressure in blood vessels from heart to lungs is too high
5 types
S/S: sob, fainting, chest pain, dizziness, tachycardia
Acute respiratory disorders
failure to add O2 and remove CO2 from blood
ARDS
-noncardiogenic pulmonary edema & hypoxemia caused by alveolar inflammation and infection
-requires mechanical ventilation
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ARF
failure of the lungs for any reason that impairs ventilation, compromises v/q or impair gas diffusion
-manifests as PaO2 <50%, acidosis, cyanosis, tachypnea/tachycardia
COVID-19
inflammatory response isnt working
lung sustains acute injury, leading to ARDS