exam 2 Flashcards

1
Q

preload

A

End-diastolic pressure: when ventricles are filled and heart is relaxed
“stretch”

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2
Q

afterload

A

Work (or force) required to move (eject) blood into the aorta
“squeeze”

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3
Q

contractility

A

electrical contractions and muscle contraction of heart

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4
Q

alterations in SV

A

indicates issue in systole, diastole,peripheral disease

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5
Q

alterations in CO

A

could result in poor perfusion or overworking

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6
Q

systolic heart failure

A

decrease ability to pump due to increased afterload decreased EF
cant get blood through aorta and it backs up into the lungs

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7
Q

diastolic heart failure

A

decreased ability to fill leads to decreased SV and CO
cant get blood out of atrium and into venticle, backs into lungs
normal EF

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8
Q

left sided HF

A

blood can’t get from left side through aorta and to rest to body
-leads to increased pulmonary venous pressure (pulmonary congestion, edema)
-low organ perfusion
hypoxia, tissue death

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9
Q

right sided HF

A

results from progression of LSHF
-right side cant get blood to lungs bc of increased pulmon vasc pressure from LSFH
-back flow of blood back to other organs
-peripheral edema
-renal failure,hepatomegaly

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10
Q

pulmonary embolism

A

blockage of 1 or more pulmonary arteries
pt will not perfuse, check v/q scans

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11
Q

pulmonary hypertension

A

pressure in blood vessels from heart to lungs is too high
5 types
S/S: sob, fainting, chest pain, dizziness, tachycardia

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12
Q

Acute respiratory disorders

A

failure to add O2 and remove CO2 from blood

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13
Q

ARDS

A

-noncardiogenic pulmonary edema & hypoxemia caused by alveolar inflammation and infection
-requires mechanical ventilation
-

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14
Q

ARF

A

failure of the lungs for any reason that impairs ventilation, compromises v/q or impair gas diffusion
-manifests as PaO2 <50%, acidosis, cyanosis, tachypnea/tachycardia

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15
Q

COVID-19

A

inflammatory response isnt working
lung sustains acute injury, leading to ARDS

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16
Q

Pleural effusion

A

-“junk around lungs”
-excess fluid nw layers of pleura outside lung
caused by heart problems, cancer, pneumonia, pulmonary embolism

17
Q

pneumonia

A

-“junk in lungs”
-inflammation of bronchioles and alveoli
-wbc fight foreign invaders
-dead cell and debris build up creating ous filling parts of airway
-causes by infectious and non-infectious agents inhalation of irritants

18
Q

pulmonary edema

A

-“junk in lungs”
-lungs filled with fluid
-lung congestion, water on lungs
-increased WOB
-causes: fluid overload, hf

19
Q

Atelectasis

A

-“poor lung expansion”
-alveoli dont fill properly/incomplete lung expansion
-caused by immobility, mucus plug, external pressure (mass)

20
Q

pneumothoraces-collapsed lungs

A

-“poor lung expansion”
-air enters pleural space and disrupts negative pressure, causes lung collapse

21
Q

refractory asthma

A

due to allergic reactions
massive immune response
airway remodeling
wheezing on exhale

22
Q

COPD

A

caused my smoking, wood burning, occupational hazards

23
Q

emphysema

A

destruction of alveoli
responds to low O2 rather than CO2
pink puffer

24
Q

chronic bronchitis

A

long term inflammation of bronchi and hypersecretion of mucus
more comorbidities, sleep apnea, acidosis
blue bloater

25
Q

bronchiectasis

A

permanent dilation of bronchi
loose ability to contract and decrease elasticity
associated with cystic fibrosis

26
Q

cystic fibrosis

A

genetic condition causing mutation of transmembrane conductance regulator
-cilia unable to push mucus out, build up, mucus plug
-no real cure

27
Q

intrinsic restrictive lung disease

A

problem w lungs themselves
-stiff lungs due to thickened alveolar walls
-decreased total lung volume
-idiopathic pulmonary fibrosis
-massive scarring impairs gas exchange, no cure

28
Q

extrinsic restrictive lung disease

A

problem from something other than lungs
-pressure placed on lungs or paralysis of muscle that helps with breathing

29
Q

major functions of renal system

A

filter blood of toxins/waste and reabsorb needed molecules, regulates hemoglobin production, increase calcium absorption

30
Q

urine formation

A

glomerular filtration, reabsorption, secretion

31
Q

common causes of renal diesease

A

tubular obstructions that increase intratubular pressure, developmental defects, pregnancy,
altered immune responses
cancer

32
Q

risk factors for renal disease

A
33
Q

pre-renal

A

decreased perfusion to normal kidney
reduced hydrostatic pressure-> reduced glomerular filtration
hypovolemia
reduced co
reduced effective circulatory volume

34
Q

intra-renal

A

glomerulonephritis
tubular disease: acute tubular necrosis, most common in hospital
interstitial disease: acute interstitial nephritis
vascular: vasculitis

35
Q

post-renal

A

obstruction along urinary tract
renal stones
prostate enlargement/cancer
cervical cancer
bladder cancer
can lead to intra renal causes