Exam 1 Flashcards

(131 cards)

1
Q

brain stem

A

controls heart rate and breathing
begins where spinal cord swells after entering skull
brain’s oldest region, contains medulla, reticular formation, and pons

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2
Q

medulla

A

controls heart rate and breathing

damage usually results in death

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3
Q

pons

A

helps coordinate movements

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4
Q

reticular formation

A

nerve network running through thalamus and brain stem
filters and directs incoming sensory input
important in controlling arousal
contains cells producing most serotonin

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5
Q

thalamus

A

directs sensory messages (except smell) to cortex, processes input

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6
Q

cerebellum

A

“little brain”, enables nonverbal and skill learning
coordinates and smoothens voluntary movement
helps process and store info outside of awareness

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7
Q

limbic system

A

contains amygdala, hippocampus, hypothalamus

neural center bordering older parts of brain and cerebral hemispheres

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8
Q

amygdala

A

linked to fear and aggression emotions

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9
Q

hippocampus

A

consolidates short term into long term memory

loss results in anterograde and retrograde amnesia

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10
Q

hypothalamus

A

maintains homeostasis
controls endocrine system using pituitary gland
emotion and reward

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11
Q

cerebrum

A

2 hemispheres containing cerebral cortex and underlying sub-cortical structures
has 4 lobes

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12
Q

cerebral cortex

A

thin layer of interconnected neural cells covering cerebral hemispheres
same structure in all humans (grooves/valleys)
larger = increased learning, thinking, adaptation capacity

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13
Q

frontal lobe

A

contains motor cortex
speaking, muscle movements, planning, judgment, decision making
last part of brain to fully develop

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14
Q

Phineas Gage

A

RR tie destroyed most of frontal lobe of cortex

“wasn’t gage”, went from kind to profane and dishonest

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15
Q

parietal lobe

A

contains somatosensory cortex

integrates sensory info, manipulation of objects, numbers and their relationships, spatial vision

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16
Q

occipital lobe

A

receives input from eyes
smallest lobe
in the back

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17
Q

temporal lobe

A

auditory processing, language comprehension, memory, emotion

directly above hippocampus

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18
Q

motor cortex

A

rear of frontal lobes
controls voluntary movements
contralaterally oriented: right controls left
body parts with larger portion of motor cortex have more control

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19
Q

somatosensory cortex

A

parietal lobe
processes info from skin senses and movement of body parts
each part’s sensitivity determined by size in cortex

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20
Q

association areas

A

integrate, interpret, and act on sensory information while linking it with stored memories
found in all lobes of cortex, not involved in primary sensory or motor functions

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21
Q

brain plasticity

A

ability of brain to remap and make new connections to allow regions to perform atypical functions in case of damage to regular region
diminishes later in life

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22
Q

corpus callosum

A

large band of neural fibers connecting two hemispheres of cortex

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23
Q

split brain

A

cut corpus callosum to treat epilepsy
brain’s hemispheres cannot communicate
heart: says they saw art, points to he w/left hand

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24
Q

Henry Molaison

A

case study after most of temporal lobe and hippocampus removed to treat epilepsy
could not recall old memories or form new ones
could learn/improve at skills

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25
EEG
electroencephalogram readout of brain's electrical activity, shows function only superficial of cortex small time range (ms)
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PET
positron emission tomography, shows brain's consumption of glucose (hot spots) some structure and function, seconds long
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MRI
details soft tissues, gives structural information
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fMRI
functional MRI, shows function and structure by adding blood flow
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central nervous system
contains brain and spinal cord, make decisions
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peripheral nervous system
contains autonomic and peripheral nervous systems, everything but brain and spinal cord
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autonomic nervous system
contains sympathetic and parasympathetic parts | controls self-regulated action of internal organs and glands
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sympathetic vs. parasympathetic nervous systems
sympathetic: arousing, fight or flight, stress parasympathetic: calming
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somatic nervous system
controls voluntary movements of skeletal muscles
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spinal reflex
signal travels up sensory neuron to interneuron, then immediately to motor neuron (bypassing brain) movement before pain is registered in brain can be bypasses consciously by brain
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endocrine system
releases hormones in bloodstream that bind to receptors on cells longer-lasting and slower-acting than nervous system
36
first psychology lab
Wilhelm Wundt, 1879
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structuralism
pioneered by Titchener | uses introspection to search for mind's structural elements
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introspection
making observations on self while doing something, i.e. why or thoughts during abandoned as unreliable used to look for mind's structural elements
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functionalism
James studied higher-order thoughts, feelings, and behaviors looked for evolutionary functions ex. fight or flight response
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behaviorism
Watson and Skinner scientific study of observable behavior, rejected introspection stimuli > brain > response if it cannot be measured, cannot be studied conditioning
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Freudian psychology
emphasis on unconscious thoughts and emotional responses to childhood events that affect our behavior iceberg theory of conciousness
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cognitive revolution
return to interest in mental processes how info is perceived, stored, and remembered more scientific
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nature vs. nurture
behavior determined by genes vs. environment | usually answer is both
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3 factors influencing behavior
biological: genes and their expression, naturally selected traits, mutations psychological: learned fears and expectations, emotional responses sociocultural: presence of others, cultural, familial, peer expectations
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types of exploratory/descriptive research
case studies and naturalistic observation
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case studies
using individual cases of interest to look for universal principles can be misleading if individual is atypical
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naturalistic observation
unobtrusively observing behavior in a natural setting without affecting it describes, but does not explain, behavior
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correlation
how much two continuous factors vary together NOT distinct groups ex. age, weight, income not gender, race, or boss
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correlation coefficient
tells how related two variables are, from -1 to 1 positive: both increase, >0 negative: one increases while other decreases, <0 none: not correlated, about 0
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independent variable
what is being studied, altering it produces measurable effect on another variable
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manipulated variable
independent variable in true experimental design, manipulated by experimenters
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subject variable
independent variable in quasi design, subjects enter with this variable already determined
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When can causation be inferred?
True experiments, not quasi
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quasi experimentation
IV not manipulated, subject variables instead subject groups determined before study i.e. gender, race, weight can conclude differences exist between groups but not the cause
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true experimentation
"true" IV manipulated by experimenters employs random assignment, experimental and control groups CAN infer causation as groups are random
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how to control a true experiment
double-blind assignment: both participants and experimenter unaware of participant's group placebo: any effect caused by an inert substance that participant believes is active
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measures of central tendency
single score that represents a set of scores | mean, median, mode, range, st. deviation
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When is an observed difference reliable?
When it is representative of population (i.e. not biased) less variable observations are more reliable than more variable ones (smaller range) large sample size, n uses random sampling
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random sampling
group of people that represent your population of interest | everyone in the population has equal chance of participating in study
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phrenology
Gall, bumps on head revealed mental abilities or character traits beginning of idea of brain function localization
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resting potential
negative charge inside neuron at rest
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selectively permeable membrane
dynamic equilibrium allows ions to continuously flow in and out while overall charge of cell remains the same
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action potential
firing of neuron when sufficiently excited/depolarized EPSPs make cell more positive/closer to AP IPSPs make cell more negative/further from AP
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excitatory threshold
charge required for neuron to send signal | all or nothing response
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rate law
APs are all same strength | stronger stimulus causes higher firing rate, not stronger AP
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reuptake
excess neurotransmitters reabsorbed by sending neuron | SSRIS inhibit serotonin uptake, make it more active
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enzyme breakdown
excess neurotransmitters broken down by enzymes in synapse | acetylcholine esterase
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antagonists
block or inhibit postsynaptic effects, curare (poison darts)
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agonists
facilitate postsynaptic effects, morphine
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ligands
anything that can bind to cell receptors
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pharmacodynamic tolerance
brains overcompensate for exogenous ligands by reducing their effectiveness, even to endogenous ligands alcohol, GABA deficiency
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conciousness
our awareness of ourselves and our environment
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dual processing
info is often processed simultaneously on separate conscious and unconscious tracks
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high road
conscious, deliberate actions, problem solving, naming an object more accurate but slower, less efficient
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low road
unconscious, auto actions, walking, making memories | more efficient but less accurate
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visual perception track
thinking consciously about the world, reorganize and plan future actions
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visual action track
guides moment-to-moment movements
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selective attention
focusing conscious awareness on a particular stimulus spotlight analogy can only listen to one convo at a time
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cocktail party effect
hearing your name will shift your attention from one convo to another
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selective inattention
we are "blind" to anything we are not looking at/paying attention to
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inattentional blindness
failing to see visible objects because our attention is directed elsewhere
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change blindness
occurs when we fail to notice a change in the environment while focusing on something else watch for cyclist, card trick
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multi-tasking
humans are bad at it our attention has limits, if 2 actions require same part of brain/similar activities can only do one at once walking and talking but texting and driving
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sleep
characterized by an absence of conscious behavior higher threshold for arousal by external stimuli 1/3 of lifespan but not sure how much we need, 8 hours is estimate
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theories of sleep
adaptation theory: sleep is a result of our internal "clock", evolved to protect us from dangers of nighttime recuperation theory: wakefulness causes deviation from homeostasis, sleep returns us to it
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EEG during sleep
shows neural oscillations, records excitation of neurons in cerebral cortex amplitude = # of neurons firing
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sinusoidal waves
reflect flow of current thru cell membranes close relationship to neuronal firing different wave types based on frequency
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alpha oscillations
awake, 8-12 hz and smooth, relaxation
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beta oscillations
awake, 13-30 hz and irregular, arousal
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stage 1 sleep
characterized by theta oscillations (3.5-7.5 Hz), transition between wakefulness and sleep
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stage 2 sleep
irregular neural activity | theta oscillations, K complexes, sleep spindles
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K complexes
sharp sudden EEG waves, thought to be involved in memory consolidation stage 2 sleep
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sleep spindles
short 12-14 Hz bursts of rapid activity
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stage 3 sleep
delta oscillations of less than 3.5 Hz | about 20-50% of all
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stage 4 sleep
more than half of oscillations are delta
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slow-wave sleep
less than 1 Hz, can collectively be classified as stage 3 up state: high firing of neocortex neurons down state: lack of firing, cell rest period
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sleep cycle
roughly 90 minutes after the start of sleep | 1 > 4 back to 1, includes REM
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REM sleep
rapid eye movement characterized by irregular activity, rapid lateral eye movement, lack of muscle tone theta and beta oscillations most spinal/cranial motor neurons inhibited increased blood flow, oxygen consumption in brain genital arousal (not necessarily sexual)
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Circadian Rhythm
roughly 24-hour cycle of biological rhythms controlled by light natural rise and fall of body temp (higher in morning), alertness, strength
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lark
people who physically and mentally peak in the morning, tend to do better in school, eat healthier
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owl
physical and mental peak at night, more creative
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SCN
suprachiasmatic nucleus of hypothalamus signaled by light activated retinal proteins controls melatonin secretion via pineal gland inhibited during morning, increased at night
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insomnia
recurring problems in falling or staying asleep self-report sleep for diagnosis but we usually underestimate aggravated by sleeping pills or alcohol
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narcolepsy
cause sleep attacks, cataplexy, sleep paralysis, hynagogic hallucination lapse directly into REM sleep hereditary autoimmune disorder, orexinergic neurons disturbed or destroyed treated with stimulants or antidepressants
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sleep attacks
irresistible urge of narcoleptics to sleep during day, 2-5 mins, awaken feeling rested
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cataplexy
complete paralysis while awake, caused by strong emotional reactions or sudden physical effort dog
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sleep paralysis
paralysis just before falling asleep
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hypnagogic hallucinations
vivid dreams occurring just before sleep
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sleep apnea
temporary cessation of breathing that causes repeated awakenings CO2 builds up in blood, wake up gasping for air treatable with CPAP or surgery
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orexin
hypothalamus peptide that prevents sleep
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CPAP
continuous positive airway pressure | mask that prevents airway from collapsing while sleeping
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REM rebound
when we have sleep debt/deprived of REM | reach REM faster and stay in it longer
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sleep deprivation
causes sleep debt: increased sleeping till repaid suppresses immune system activity can predict depression/life dissatisfaction increased cognitive errors i.e. auto accidents
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tolerance
taking more of a drug to achieve same effect over time | alcohol metabolism increases over time
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sensitization
opposite of tolerance, taking same amount creates larger effect
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addiction
dependency on drug to function and/or feel normal
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psychoactive drug
chemical substance that alters perceptions and moods
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GABA
inhibitory neurotransmitter
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glutamate
excitatory neurotransmitter
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alcohol
low dose: mild euphoria, reduce anxiety high dose: incoordination, sedation releases us from responses to averse stimuli + (euphoria) and - (anxiety) reinforcer increases dopamine production, release in nucleus accumbens (+) depressant
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2 major sites of action of alcohol
antagonist: indirectly at NMDA receptors for glutamate agonist: indirectly at GABA receptor, - reinforcement and sedative effect
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heroine
releases dopamine in nucleus accumbens at 150-300% higher levels produces analgesia, hypothermia, sedation, reinforcement passes thru placental barrier, newborns addicted high tolerance buildup means high addiction different opiate receptors in different locations produce different effects
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narcan
competitive antagonist to opiates by binding to opiate receptors
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cocaine
binds and deactivates dopamine transporters, preventing their reuptake also blocks voltage-gated sodium channels
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amphetamines
cause dopamine and norepinephrine channels to run in reverse, sending neurotransmitters into synapse and blocking reuptake meth is more potent
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mesolimbic dopamine system
essential for all forms of reinforcement drugs alter it blocking receptors causes loss of cocaine reinforcement effect
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cannabis
THC stimulates dopamine release | acts on cannabinoid 1 receptors in brain
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nicotine
more deaths than hard drugs releases many neurotransmitters diminishes appetite and anxiety, boosts alertness, reduces pain sensitivity stimulates nicotinic acetylcholine receptors increases activity of mesolimbic DA neurons, causing release of DA in nucleus accumbens
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caffeine
displaces adenosine in brain, which causes tiredness
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hallucinogens
drugs that distort perceptions and evoke sensory images without input LSD and marijuana
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addiction risk factors
high comorbidity with high stress, anxiety, personality/mood disorders self-medication etiological: high stress and addiction caused by same thing personality familial factors: copying parents