Ex. Phys. Environmental Physiology Flashcards
basic human responses to environmental stressors
- the environment presents stressors that disrupt homeostasis in our internal environment
- each type of environment requires a unique set of adaptive responses
- people may lack or have sufficient hereditary abilities to adapt to any or all environments
- people vary in how they respond to the same environmental stressor, which produces different outcomes
- environmental stressors can positively or negatively affect a person’s ability to meet physical or psychosocial demands
- environmental stressors are strongly mediated by psychological factors
- -if the stressors are not viewed as harmful or alarming, they may produce small or even opposite physiological responses
- physiological responses to environmental stressors at times can be excessive, inappropriate, inadequate, or disordered
- physiological behavioral changes sometimes occur before an environmental stressor has been applied
what is the necessary adaptation time to any environment
-time for deacclimation
8-14 days for most people
people
deacclimation within 14-28 days following removal from environment
variation in how people respond to stressors
-depends on
bodies may show no strain, illness, or injury
depends on
-level of tolerance developed
-immune system competence
-age
-level of fitness
-number/intensity/type of previous exposures to an environmental stressor
what are excessive physiological stressors exaggerated by
other diseases and disorders (heart problems, arthritis, renal function)
thermoregulation
many adaptive responses to different environments affect thermoregulation
hypothalamus plays a large part
heat transfer mechanism
hypothalamus functions
acts as a central monitoring system, but receives feedback from the peripheral system
sensitive to temperature changes in the blood
attempts to maintain a core temp. of 37C (98.6F “normothermic)
-too hot: hyperthermic
-too cold: hypothermic
initiates efferent feedback via the SNS to dissipate or conserve heat
heat transfer mechanisms
radiation: direct transfer of heat
convection: fluid (air/water) movement over skin
conduction: contact with skin
evaporation: sweat vaporization
exercise in the heat acute responses: how do you keep cool?
increases blood flow to skin via vasodilation of cutaneous arterioles conducts heat away from core to environment
sweating releases fluid to skin for evaporation
blood plasma volume typically decreases
retain H2O and NaCl via kidneys to offset fluid losses
hormones typically do not play a role in cooling
results of exercise in the heat
core temperature increases
what happens when humidity rises above 50-70%
effectiveness of evaporation decreases and skin stays hot (and flushed)
exercise in the heat exercise/performance implications
as core temp increases, performance typically decreases (CV function, muscular endurance, metabolic function, CNS drive, etc.)
-maximal strength appears to be unaffected
other negative effects include dehydration, heat exhaustion, cramps, and syncope
heat acclimation
sweating response improves: more sweat, faster onset, occurs at lower temperatures, more dilute to spare electrolytes
blood volume increases to lower exercise HR while maximizing skin blood flow
decreased core temperature (hopefully)
improved exercise tolerance (but dehydration is still a major hurdle)
acute responses to exercise in the cold: how do you keep warm?
SNS initiates skeletal muscle tremors (shivering) and thyroxin released from thyroid to increase basal metabloic rate (BMR) and core temp.
increased blood flow to core via vasoconstriction of cutaneous arterioles concentrates and conserves heat around vital organs
decreased max HR and Q
decreased dissociation of O2 from Hb
skin and hair trap air and warm it to create a boundary layer of insulation
decreased blood plasma volume due to some evaporative cooling
retain H2O and Na+ via kidneys to offset dehydration
cold exercise/performance implications
weight and awkwardness of clothes may conserve heat, but increase exertion
shivering increases VO2 at submax intensities and decreases motor coordination
decreased Q and O2 delivery with increased muscular demand increases reliance on anaerobic energy production, which shortens time to fatigue
increased risk of hypothermia and frostbite
increased risk of dehydration
cool acclimatization
more efficient thermoregulation capabilities
greater reliance on FFA due to Epi and Norepi release from SNS
reduced skin blood flow increases venous return, which increases SV
lower lactate production due to glycogen sparing effect of FFA use
cold may actually ease exercise effort