ER Nurse: Cardiovascular Emergencies 1 Flashcards

1
Q

what is the pathophysiology for a disrhytmia

A

1) rate of SA node D/c is 100><60
2) SA node fails to D/C
3) Conduction is delayed or blocked
4) Aberrant conduction is activated
5) Ectopic foci initiate impulse

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What are some pathologic conditions for disrhytmias

A

1) Metabolic derangement (hypoxia, acidosis, alkalosis)
2) e- imbalances (hypo/hyper K+, Hypo Mg/Ca)
3) chronic illness (COPD, CAD, ACS, CHF)
4) Meds (cardiac glycosides & Bronchodialaters)
5) Congenital defects
6) Heavy use of stimulants (nicotine, caffeine, amphetamines, cocaine)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Sinus dysrhythmia types

A

Bradycardia, tachicardia, arrhythmia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Atrial dysrhythmia types

A

1) premature atrial complexes
2) Atrial fibrillation (a-fib)
3) Atrial flutter
4) multiform atrial rhythm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Atrial ventricular blocks types

A

any block of the AV nodes, Bundle of His, Purkinje system
can be 1st, 2nd, 3rd degree blocks

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Junction dysrhythmia types

A

1) premature junction complex
2) junction escape beats
3) accelerated junction rhythm
4) Junction tachy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

ventricular dysrhythmia types

A

1) premature ventricular contractions
2) ventricular tachycardia
3) ventricular fibrillation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Sinus bradycardia

A

ID: SA node D/c <60 BPM
EKG: normal except rate
Causes: MI, athletes, sleep, vagal nerve stimulation (vomiting/straining), ICP>, anoxia, peds-hypoxia
S/S: AMS, syncope, CP, hypotension
Tx: Atropine, Dopamine drip, epinephrine, transcutaneous pacing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Sinus Tachy

A

ID: SA node D/c 100>
EKG: normal except rate 100>
Causes: homeostatic response that requires O2> ex// volume loss, fever, pain, anxiety, exercise, stimulants, ischemia
Tx: ID & Tx underlying cause

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Sinus arrhythmia

A

ID: rate of SA node varies slightly to moderate with at least a 0.12 sec difference between shortest and longest cardiac cycle
EKG: rate varies slightly, rhythm slightly irregular
Causes: common in pediatrics and young adults
Tx:

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Premature atrial complex (PAC)

A

ID:
EKG: irregular rhythm. P wave is narrower and earlier (rest of cycle looks good). PR interval can be normal or prolonged.
Causes: stimulants, digitalis toxicity, E- imbalances, hyperthyroidism, ischemia, CHF/pulmonary HTN (increase atrial pressure)
Tx: rarely treated unless accompanied by another more lethal disrhythmia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Premature atrial complex (overview)

A

PAC is an ectopic beat initiated by an irritable atrial focus. It originates in the atria but not the SA node.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Atrial Fibrillation

A

ID: uncontrolled rapid atrial quivering seen with drope in CO & clot risk
EKG: No identifiabole P waves or PR intervals. Irregular ventricle rate w/normal QRS compleces.
Causes: valve disease, cardiomyopathy, CHF, CAD, HTN, PNE, PE, pericarditis, cardiomyopathy
Tx: Meds if hemodynamically stable (CCB cardizem, BB brevibloc), synchronized cardioversion. Anticoagulation to prevent thromboembolism. Meds for new onset (amiodarone, ibutilide, procaniamide)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q
A

ID: uncontrolled rapid atrial quivering seen with drope in CO & clot risk
EKG: 4 atrial activations (p waves) occur before QRS. Appears as sawtooth waveforms.
Causes: valve disease, cardiomyopathy, CHF, CAD, HTN, PNE, PE, pericarditis, cardiomyopathy
Tx: Meds if hemodynamically stable (CCB cardizem, BB brevibloc), synchronized cardioversion. Anticoagulation to prevent thromboembolism. Meds for new onset (amiodarone, ibutilide, procaniamide)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Multiform atrial pacemaker

A

ID: 2 or more foci in AS node, atria, or AV jx compete for rhythm control
EKG: P-P & R-R intervals are irregular w/3 different P wave configurations present
Causes: normal in old or young & results from inhibitory vagal (parasympathetic effect of respiration on SA node & AV jx)
S/S: asymptomatic
Tx: withhold digoxin if rhythm is caused by digoxin toxicity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Paroxysmal Supraventricular Tachycardia (PSVT)

A

ID: reentracted tachycardia at AV jx leading to two conduction pathways leading to tachycardia
EKG: narrow QRS complext with ventricular rate 150-250 bpm. distorted P waves & PR interval is less than 0.12 sec or doesnt appear
Causes: e- imbalances, excess stimulants, amphetamines, emotional stress, hyperventilation
S/S:
Tx: stable vagal maneuvers (Valsalva cough, gag, apply ice in face) followed by meds adenoside, amiodarone, BB CCB, cardioversion & ablation

17
Q

Wolff Parkinson White (WPW syndrome)

A

ID: impulse conduction from atria to ventricle bypassing AV nodes
EKG: short PR interval, widened QRS with slurred upstroke
Cause: preexcitation syndrome w/congenital accessory pathway exist in the heart which permits
S/S: rarely until palpitations or CP occur due to rapid rate
Tx: ablation

18
Q

Premature Junctional dysrhythmias

A

ID:
EKG:
Cause:
S/S:
Tx:

19
Q
A

ID:
EKG:
Cause:
S/S:
Tx:

19
Q
A

ID:
EKG:
Cause:
S/S:
Tx: