Equinus Flashcards

1
Q

Define ankle or talipes equines

A
  • Sagitta plane deformity in which there is < than 10 degrees of DF at the AJ when the STJ neutral and MTJ max. Pronated at both axes
  • may be Ossetia (very rare, congenital , significant trauma or significant OA) or muscular (gastric and/to soleus)
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2
Q

Outline the aetiology of ankle equines

A
  • ongenital muscle shortage of part or all of triceps surae
  • issues blockage of DF due to breaking of tibia or altered anatomy of talus (e.g. Eostosis)
  • tight hamstrings or iliopsoas requiring compensatory DF at AJ
  • posterior muscle contracture: spastic paralysis (UMNL e.g. Stroke or cerebral palsy); tonic mm spasm (in response to pain e.g. Sore AJ or STJ, can be overcome with constant force); dynamic mm imbalance (LMNL e.g. Polio affecting ant group, resulting in unopposed post contracture
  • prolonged bed rest or wearing high heeled shoes
  • excessive pronation of STJ and MTJ
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3
Q

Outline the pathomechanics of compensated ankle equinus

A
  • if inadequate AJ DF in midstance as body passes over foot, foot remains planted, DF is achieved via STJ pronation
  • foot may ‘break’ at MTJ which will also be unlocked, to allow DF of Ffoot on the Rfoot
  • midfoot collapse over time
  • active propulsion diminished, or abolished requiring lifting of the foot which may lead to overuse of leg muscles. (Inefficient gait)
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4
Q

Signs and sx. Of uncompensated ankle equinus

A

B- bouncy type gait, early heel lift, toe walking (if severe/kids)
I- increased WB in Ffoot may lead to HK on ball of feet and clawing of lesser digits
S- secondary hamstring contracture
P- proximal compensation. Recurvatum of knee, forward postural position, inc lordosis of lower back, and abd/add gait

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5
Q

Signs and sx. Compensated ankle equinus

A

“Chopen”
C- collapsed midfoot/break
H: HAV
O: one of the most destructive of pathologies (Esp. If long term and left untreated)
P: postural fatigue
E: excessive pronation of foot during gait
N: neuromas

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6
Q

Outline treatment for ankle equinus

A
  • stretching of tight structures: stretching ( 5x 30 second stretches a day), casting (rolf says this ain’t shit)
  • orthodox to control pronation
  • heel lifts if symptomatology in triceps surae (e.g. If pt. has grand final on weekend: not for long term rx.)
  • surgical lengthening
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7
Q

what are the pronation induced pathologies associated with compensated equinus?

A
  • HAV (adductor hallucis has mechanical advantage)
  • neuromas (unstable foot means bones crunch and grind)
  • postural fatigue (floppy foot is harder to DF/PF esp. in propulsion
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8
Q

what sort of assessment do we do for suspected equinus cases?

A
  • AJ ROM is assessed with patient prone
  • With knee extended
    STJ neutral or slightly supinated ( to limit the amount of motion there)
  • Repeat with knee flexed (to determine contribution of soleus v. gastrocnemius)
  • If tight in extension and not flexion : the tightness is mostly from gastroc not soleus
    -If tight in both scenarios then its soleus or both or a bony block.
    -If endpoint is spongy then is muscular
    -If the endpoint is hard then it’s a bony block
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9
Q

outline the treatment options for equinus

A
  • Stretching of tight structures
  • Casting [rolf says this is stupid]
  • Heel lifts if symptomatology in triceps surae i.e. if patient needs to do a grandfinal over the weekend or something emergency style then add a heel lift in their shoe/to their orthotic or change shoes. NOT A LONG TERM SOLUTION
  • Surgical lengthening [obviously last line]
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10
Q

outline the “ideal measurement criteria “ (from FPI lecture)

A
  • WB
  • multiplanar
  • multisegmental
  • reliable
  • valid
  • requiring minimal manipulation to subjective position
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