Equinus Flashcards
Define ankle or talipes equines
- Sagitta plane deformity in which there is < than 10 degrees of DF at the AJ when the STJ neutral and MTJ max. Pronated at both axes
- may be Ossetia (very rare, congenital , significant trauma or significant OA) or muscular (gastric and/to soleus)
Outline the aetiology of ankle equines
- ongenital muscle shortage of part or all of triceps surae
- issues blockage of DF due to breaking of tibia or altered anatomy of talus (e.g. Eostosis)
- tight hamstrings or iliopsoas requiring compensatory DF at AJ
- posterior muscle contracture: spastic paralysis (UMNL e.g. Stroke or cerebral palsy); tonic mm spasm (in response to pain e.g. Sore AJ or STJ, can be overcome with constant force); dynamic mm imbalance (LMNL e.g. Polio affecting ant group, resulting in unopposed post contracture
- prolonged bed rest or wearing high heeled shoes
- excessive pronation of STJ and MTJ
Outline the pathomechanics of compensated ankle equinus
- if inadequate AJ DF in midstance as body passes over foot, foot remains planted, DF is achieved via STJ pronation
- foot may ‘break’ at MTJ which will also be unlocked, to allow DF of Ffoot on the Rfoot
- midfoot collapse over time
- active propulsion diminished, or abolished requiring lifting of the foot which may lead to overuse of leg muscles. (Inefficient gait)
Signs and sx. Of uncompensated ankle equinus
B- bouncy type gait, early heel lift, toe walking (if severe/kids)
I- increased WB in Ffoot may lead to HK on ball of feet and clawing of lesser digits
S- secondary hamstring contracture
P- proximal compensation. Recurvatum of knee, forward postural position, inc lordosis of lower back, and abd/add gait
Signs and sx. Compensated ankle equinus
“Chopen”
C- collapsed midfoot/break
H: HAV
O: one of the most destructive of pathologies (Esp. If long term and left untreated)
P: postural fatigue
E: excessive pronation of foot during gait
N: neuromas
Outline treatment for ankle equinus
- stretching of tight structures: stretching ( 5x 30 second stretches a day), casting (rolf says this ain’t shit)
- orthodox to control pronation
- heel lifts if symptomatology in triceps surae (e.g. If pt. has grand final on weekend: not for long term rx.)
- surgical lengthening
what are the pronation induced pathologies associated with compensated equinus?
- HAV (adductor hallucis has mechanical advantage)
- neuromas (unstable foot means bones crunch and grind)
- postural fatigue (floppy foot is harder to DF/PF esp. in propulsion
what sort of assessment do we do for suspected equinus cases?
- AJ ROM is assessed with patient prone
- With knee extended
STJ neutral or slightly supinated ( to limit the amount of motion there) - Repeat with knee flexed (to determine contribution of soleus v. gastrocnemius)
- If tight in extension and not flexion : the tightness is mostly from gastroc not soleus
-If tight in both scenarios then its soleus or both or a bony block.
-If endpoint is spongy then is muscular
-If the endpoint is hard then it’s a bony block
outline the treatment options for equinus
- Stretching of tight structures
- Casting [rolf says this is stupid]
- Heel lifts if symptomatology in triceps surae i.e. if patient needs to do a grandfinal over the weekend or something emergency style then add a heel lift in their shoe/to their orthotic or change shoes. NOT A LONG TERM SOLUTION
- Surgical lengthening [obviously last line]
outline the “ideal measurement criteria “ (from FPI lecture)
- WB
- multiplanar
- multisegmental
- reliable
- valid
- requiring minimal manipulation to subjective position