Equine Health Management Y1 Flashcards

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1
Q

Health assessment observations?

A

Health assessment observations?

.Behaviour .Environment

.Temperature .Pulse

.Respiration .Mucous membranes

.Odour .Feet .Teeth .Skin

. Borborygmus (gut sounds)
- Too few = stasis/ colic
- Hypermotile = scouring, accompanied by a loose stool or
diarrhea.

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2
Q

Healthy horse has?

A

Healthy horse has?

.An alert expression - ears should prick forward as you move towards them. Eyes won’t be half closed. Nostrils should be relaxed. Are aware you’re there.

.Eyes - should be clear (unless you know the horse is prone to eye discharge), moucous membrane under eyes should be pale salmon pink.

.Nostrils - no consistent discharge, abnormal colour of discharge.

.Coat - should be glossy which lies flat.

.Skin - loose skin which moves easily across the underlying bones.

.Equal weight - distribution on each leg. No raising/ pointing front feet.

.Legs - cool with no unusual swelling.

.Sweating - no obvious sweating

.Urine - oraque pale yellow or colourless urine.

.Droppings - being passed several times a day.

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3
Q

Exam areas to look for in practical?

A

Exam areas to look for in practical?

.Alertness
-horse aware of surroundings
-Head over door, alert forward and relaxed? Not anxious (very alert), depressed (ears not forward when approached), flank watching (stomach pain/ colic), pawing (excitement when feeding or for no reason colic)

.Feeding
-eating immediately
-do go off food when fitter or covering (breeding).

.Drinking
-note alteration of regular drinking patterns, if bed wet with urine - too much or too little being consumed (cushing or diabetes).
-PPID (Cushing’s)
-abnormal smell (too much protein in diet)

.Posture
-should stand square i.e. injury or illness
-abnormal recumbency (make them feel better when ill).
-bedding in coat/ mane/ tail- indicates lying down.

.Pick up and look at feet (MUST exam).

.Safety protocols must be done when examining horse

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4
Q

Pulse?

A

Pulse?

.Adult horse at rest = 36 - 42bpm
.Higher in smaller younger animals
.Lower in older larger animals
.Increase in fear – 100bpm
.Increases in late pregnancy
.Exercise – fast work – 200-240 bpm in fit horses this should drop rapidly
.Increases with temp/fever
.Character varies with heart disease

.Pastern – laminitis pulse – bounding digital pulse

.Average 30-34 in a semi fit horse

.More fit the horse the lower the pulse

.Affected by: exercise, environment, excitement, disease, physical condition and age

.Pulse rates higher than 80bpm for a prolonged period of time may indicate colic

.Expect high HR in exercising horses – WHY – measure with a HR monitor

.With colic – Frank Andrews prof at university of Tennessee has noted the following:

'’90% survival rate if a colicky horse has HR below 60bpm
Between 60-80 horse has a 50% likelihood of survival
80-100bpm horse only has a 25% chance of survival
HR exceeding 100bpm is correlated with only a 10% survival rate’’.

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5
Q

Respiration?

A

Respiration?

Watch breath
Stand at shoulder and watch respiratory effort
1 = in & out
How many breaths/minute
8-14
Decreases when asleep
Increases with fever
Hyperthermia – panting and flaring nostrils, heaving sides

Inhalation and exhalation 10-14 breaths/minute

Exercise, high env. Temps, high humidity, excitement, pain and illness will inc. resp. rates

Sound and patterns of resp. – should be steady and effortless, producing little noise.
Irregular, shallow, laboured or noisy respiratory patterns can signify exhaustion or illness
Laboured breathing may indicate shock
Call a vet!

Can also watch for nostril flares – inaccurate –why, should not flare when horse at rest?
Also expansions of the rib cage or flank

Video = http://www.youtube.com/watch?v=1Ssx8KIrts8

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6
Q

Posture Abnormalities?

A

Posture Abnormalities?

Sitting on the wall
Indication of foot pain or irritation around anus

Head pressing
Liver disease, depression
Sign of colic or azoturia / ERS
Learned helplessness in environment

Attempting to strain
May indicate urine blockage (more common in geldings – calcium buildup
Unusual habits for that horse – remember every horse is an individual

Head-pressing is very bad – may be a sign of colic or azoturia – other names?, also worst behaviour to see in helpless horses that cannot cope with their environment

75-80% lameness is in the foot

Straining is more commonly seen in geldings, rare, may be due to a calcium buildup

Pointing front feet
Navicular syndrome, pus in the foot

Rocking back on the feet – laminitis

Acute navicular syndrome

75-80% lameness in the foot
No foot? No Horse!

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7
Q

Environment

A

Environment

Disturbed
May indicate colic, box walking
Pattern?

Scratch marks on wall
May indicate horse has been cast

Chew marks
Deficiency in diet, bad stable management, stereotypies
Evidence of having eaten, drank

Disturbed bedding, circular patterns may indicate box walking

Scattered bed, eaten bed – eg Rocket – pacing, playing, hungry

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8
Q

Skin

A

Skin

Good indicator of health

Coat
-Should be shiny, smooth and slightly greasy to touch
-Dull, staring, and dry indicates general ill health
-Pinch test – skin should return immediately. Indicates dehydration or rapid weight loss if remains peaked.

Sweating
-Pain, fever, overheating
-Spots, nodules and tumours

Dull coat or a pot belly could indicate worms

Colic and diarrhoea can dehydrate horses, especially foal scours
restricting a horse’s water intake for as little as two hours greatly increases the chance of colic

Horse is 70% water
Should have access to clean fresh water at all times
Horses drink around 45 litres of water a day and this can increase by up to 40% during warm weather.
Water is vital for fluid balance, tolerance for exercise and also helps digestion
Other signs of dehydration or heat stress include sunken eyes, a tucked up appearance, heavy breathing and a rapid pulse.
Dehydration can be caused by lack of fluid intake, fever, diarrhoea, sweating, blood loss and urination

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9
Q

Temperature

A

Temperature

Normal temperature ranges:
Adult horse
99.0 -100.5 °F
37.2-38.2 °C (sometimes as low as 36.5°C)
Foal
99.0 – 101.5 °F
37.5 – 38.6 °C

Higher or lower monitor and seek vet advice if not better

Internal body temp falls in a narrow range

Only a few degrees diff will indicate a problem

Higher in foals and younger horses – WHY? (foals cannot regulate their body temperature well, need proper protection against the weather/environmental conditions)– more influenced by ambient temps, and will vary more

Foal is considered hypothermic if the temp drops below 99°F, most newborn foals will not shiver due to the body’s inability to respond properly to the outside temperature. Similarly foals can easily become hyperthermic if their temperatutre rises above 103°F. They become heat stressed without shade and will hyperventilate to blow off extra heat, because the newborn does not reguate its temperature well it does not sweat in response to hot outside temperatures.

Env. Temp, exercise and excitement may increase body temp

Smaller, younger horses and active horses will have higher temps

Older, more sedentary animals have lower temps

Diurnal variation
Temp lowest at 3am, peaks at 6pm
Take temp at same time every day

Diurnal – daily variation – variation within the day

Insert thermometer into rectum
If possible, have an assistant
Stand tight to the horse’s quarters
Lubricate thermometer and hold against rectal mucosa
Hang on to the thermometer!!
Depending on the type of thermometer, leave in for 30-90 secs. – most modern ones will beep when they are ready to be removed

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10
Q

Factors Influencing Temperature

A

Factors Influencing Temperature

.Internal body temp falls in a narrow range.

.Only a few degrees differencd will indicate a problem.

.Temperature higher in foals and younger horses. Foal considered hypothermic if the temp drops below 99°F. Foals can easily become hyperthermic if their temperature rises above 103°F.

.Environment, temp, exercise/ arousal may increase body temp.

.Internal body temp falls in a narrow range. Only a few degrees diff will indicate a problem.

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11
Q

Temperature Variations

A

Temperature Variations

COLD
Reduction in temp – cold/hypothermia

Significant reduction in rectal temp potentially life threatening

Extremities normally become cold first when a horse is chilled

HOT
Moderate rise in temp - 1 °C mild fever – not generally a problem

Rise of up to 2 °C or over – call the vet

108 °F or 41 °C can lead to permanent brain damage - overheating

Low temps are indicative of shock, chilling or a very sick animal

Fevered foals will normally have a reduced temperature with an infection instead of a fever

Inc rectal temp may indicate disease, overheating, colic, if very high may lead to convulsions or death

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12
Q

Equine pulse rate

A

Equine pulse rate

.How many beats per minute at rest?
-20 to 40bpm at rest

Assessed at:
-Most easily at angle of jaw
-Either side of pastern - feel for groove along the pastern.
-Inside the forearm

.Should measure pulse in a relaxed horse

.When measuring TPR you should take the pulse first before the horse becomes too excited

.Every heart beat pushes blood through arteries – these are what we are feeling to monitor pulse and effectively HR

Video = http://www.youtube.com/watch?v=yFENXvYD5U0

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13
Q

Equine respiratory rate

A

Equine respiratory rate

.Normal at rest - 8 to 16 bpm

.Double effort – heaves/COPD/RAO

.Heaves - display of a heave line, horse uses diaphragm and abdominal muscles

HIGH respiratory rate
-Less severe cases resp. rate of 20 or above.
-Up to 40/50 breaths/min = obstruction – gasping, choking, snot pouring out of nose – call the vet

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14
Q

Recurrent Airway Obstruction (RAO)

A

Recurrent Airway Obstruction (RAO)

.Also known as Chronic Obstructive Pulmonary Disorder (COPD) or “broken wind” is a respiratory disease in horses. Also known as “heaves”.

.This is a chronic condition of horses involving an allergic bronchitis characterised by wheezing, coughing and laboured breathing.

.CAUSES - an allergic reaction to certain otherwise innocuous substances - allergens, typically dust and mold spores (e.g. Aspergillus spp.).

.It is therefore most common in horses fed on hay and bedded on straw.

.The condition is most common in the Northern Hemisphere - it is rare in the South. This is probably because northern horses are more likely to be overwintered in stables or barn, and therefore become sensitised more readily.

.NOTE: A similar condition, Summer Pasture Associated RAO also exists. In this case, the allergens are derived from fodder and pasture (pollen etc.). This is more common is summer, and management is reversed: horses should be stabled in well ventilated areas.
SYMPTOMS - Increased respiratory effort and shortness of breath, especially in response to strenuous exercise. In some cases, the horse may present acutely such that it really struggles to inspire sufficient air. This is a veterinary emergency.
In addition, a soft, moist cough may be seen, most commonly in association with exercise or eating. This cough may be productive, with thick, viscous sputum.
There may also be an audible wheeze.
In chronic cases a “heave line” may be visible on the ventral abdomen. This is caused by hypertrophy of the extrinsic respiratory muscles.

Treatment
As RAO is primarily a management condition, primary treatment is necessary. Where possible, the horse should be turned out to pasture. When removed from the allergens in the stable, the symptoms will usually subside, although they will recur if the horse is stabled again at a later date.
If this is not practical, certain alterations to stable routine may be effective.
Minimise dust and maximise air quality in the stable.
Soak hay or feed a low dust alternative such as haylage. The length of time to soak hay for the maximum benefit is debated; however, approximately 30 minutes appears to be the current consensus.
Feeding from the ground is oftenconsidered to be more beneficial than in a haynet, as this allows any mucous to drain out of the lungs.
Bed on a dust free bedding. Those horses that must continue to be stabled are normally bedded on paper, or a combination of rubber matting and paper, or low dust wood shavings. Straw contains dust and may irritate the condition further.
Make sure the stable is well ventilated.
Don’t muck out or brush up while your horse is in the stable to minimise the dust in the air.
Despite management changes, pharmacological intervention is often required, and almost invariably in severe cases. This breaks down into a number of categories: Bronchodilators: Often the mainstay of therapy. One of the most common is clenbuterol, either as an oral medication administered twice daily in feed, or via a nebulizer. Clenbuterol also has anti-inflammatory actions, and is therefore often preferred. In an emergency, intravenous clenbuterol or atropine may be used, but care must be exercised with atropine as it may predispose to adverse systemic side effects such as mydriasis (pupil dilation in the eye), excitement, GI stasis and colic.
Corticosteroids: Oral steroids such as prednisolone are commonly used; however, side effects are common, and the horse may be predisposed to laminitis. Therefore, the use of aerosolised steroids via an equine inhaler are becoming more common. This route of administration reduces the dose required, and the risk of side effects.
Mast cell stabilisers: Cromoglycate has been used, but this is may or may not be fully effective.
Care should be taken with these drugs in competition horses, as many of them are forbidden substances under racing and FEI rules.

For Pasture associated RAO depending on the allergen it may be controlled by Antihistamines such as Pititon and respiratory supplementation – menthol and Liquorice are the recommended ingredients by the vet

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15
Q

Mucous Membranes

A

Mucous Membranes

.Capillary Refill Time - measuring the amount of blood volume circulating and the blood pressure. Measures the cardiovascular status of the horse. Colour salmon pink.

Abnormal MM colour
.Yellow– colic, constipation
.Very Yellow – jaundice
.Very White – shock or haemorrhage
.Brick Red – fever, post exercise
.Purple – cyanotic – seen in terminalcolic just before death

.Greater than 2 secs to refill indicates shock

.Purple/blue due to lack of oxygen in the blood – poor circulation, shock

.Yellow tinge reflects Jaundice, may be transient due to diminished appetite and food intake or a diet rich in alfalfa but it could be more lasting and serious as a result of liver disease

.Line body cavities open to the air – inside mouth, nostrils, eyelids, vagina, penis and sheath. Colour indicates quantity and condition of blood flowing to these areas. Be aware of lighting conditions – artificial lighting, dim, flashlights may distort true colour.

.Red Associated with septic or endotoxic shock when blood pools in the capillaries and small vessels.

Pale – white Indicates fever, blood loss, anemia and mild shock. Dehydration

“Performance horses, especially horses that work over extended time and distance, such as the endurance horse, not only contend with fluid shifts to demand tissues (such as heart and muscle for locomotion and brain and kidney for normal function), but they also must contend with fluid loss as sweat to maintain thermoregulation,” says Frazier.
He elaborates on what one might see with systemic changes as reflected in the mucous membranes: “The first aberration of the color of the mucous membranes as a horse dehydrates and/or responds to fluid shifts is a reddening of the mucous membranes above the tooth root with a corresponding paling of the rest of the tissue.” This is first seen on physical exam as a disparate color along the area where teeth meet gums and appears as a purplish or bluish tinge along the line of the teeth, with the membrane above appearing pale pink or normal pink.
Frazier explains the progression of circulatory compromise: “If these processes advance without compensatory adjustment, the mucous membranes will continue to pale, taking on a bluish tinge, and finally turn a purple, muddy color. Commonly, changes in mucous membrane color (paling) are accompanied by delay in the CRT of greater than one to two seconds. If dehydration and fluid shifts sufficiently compromise the perfusion of the cecum and/or large bowel, endotoxemia may affect the capillary beds and result in a ‘brick red’ color to the mucous membranes.”
Septic or endotoxic shock causes the peripheral vascular beds to open, with increased filling of blood in the capillaries and small vessels. As circulatory shock progresses, less oxygen is sent to the tissues, so the blood stagnates, loses its oxygen, and the membranes turn purplish or blue-tinged, then eventually turn to a muddy gray.

A horse suffering from systemic illness, such as gastrointestinal disease or colic, is at risk of deterioration of the circulatory system that is reflected by the character of the mucous membranes. Frank Andrews, DVM, MS, Dipl. ACVIM, a clinical professor at the University of Tennessee, has extensive experience with complicated colic cases referred to the Veterinary Teaching Hospital.
“If a colicky horse has pink or cyanotic (blue-tinged) mucous membranes, studies indicate that he has a greater likelihood of survival (55%), whereas a horse with toxic (gray or pale with dark red line at gum/teeth interface) or injected (dark red to ‘muddy’) mucous membrane color has a less likely chance of survival (44%),” notes Andrews. “This is not much of a statistical difference, but is more useful following surgery, when membrane color is better correlated with survival probability: If the color is pink after surgery, that horse will likely live.”
In determining the prognosis for survival in colic cases, Andrews reports that as a metabolic parameter, CRT has better positive correlation. Rough estimates of survival suggest that with CRT less than two seconds, there is a 90% survival rate; if CRT ranges between 2½ to four seconds, there is a 53% survival rate; while if CRT is greater than four seconds, there is only a 12% survival rate.

Non-steroidal anti-inflammatory drug toxicosis (as for example with phenylbutazone, flunixin meglumine, or ketoprofen) can create oral ulcers concurrent with the development ofgastric ulcers.

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16
Q

Elimination of Waste

A

Elimination of Waste

Generally, horses:
.Defaecate – every 2-3 hrs
.Urinate – every 4-6 hrs
.Look for changes in colour &/ consistency

.Frequent eliminations and straining are not normal.

.Coffee-coloured or bloody urine, diarrhoea and constipation – call the vet.

.Normal horse urine is cloudy. This is due to the calcium carbonate crystals present in urine.

.Normal horse urine is foamy. This is due to the presence of totally normal mucus in urine. The mucus helps to prevent the calcium carbonate crystals from forming stones.

.Normal horse urine can vary in color, from light yellow to dark yellow. The color can vary within the amount that is voided at one time.

.Sometimes, you may notice that after exercise the urine is darker. Usually no worries there, darker urine can be normal or it can be a sign of a tying up and some other serious diseases. You may also observe that there is dark urine at other times, or even bloody urine. That is the time to worry and call your Veterinarian.

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17
Q

Routine health checks

A

Routine health checks

TEETH
.Should be checked 6/12 months for;
-Uneven wear
-Require regular rasping
-Problems with teeth
-Eating difficulties
-Ridden problems
-Excessive saliva production

SIGNS of teeth problems
-Quidding may drop food from their mouth while eating
-Lose condition
-Excessive saliva dripping from the mouth
-Resist the bit when ridden or driven
-Undigested food in their droppings
-Bad breath where food is trapped in gaps between their teeth.
-Diastema – abnormal accumulation of food between the teeth

.An equine dental professional must be truly interested in equine dentistry, have an appropriate level of expertise, and possess the proper instrumentation. This person needs to be able to perform a complete dental examination and basic conservative, corrective procedures properly. Equine dentistry is not an area where more is better, as excessive tooth removal and/or excessive heat generation via rasping can damage teeth. Corrective dental procedures should allow horses to reach their full athletic potential and maintain good dental health, relieve pain, and extend tooth life.

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18
Q

Other routine health checks

A

Other routine health checks

FOOTCARE

.Regular farrier visits essential. Every 6 weeks but depends on the individual horse when it needs the farrier.

.Maintain stable hygiene where appropriate. Pick out feet regularly.

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19
Q

Shoeing

A

Shoeing

REASONS FOR SHOEING
.Prevent excessive wear of feet
.Provide grip?/Studs?
.Reduce concussion
.Combat the effects of wet conditions
.Correct defects and faulty movement
.Assist veterinary problems

.Amount of hoof they grow is equal to the amount of hoof they wear away.

.Domesticated horses are expected to carry riders, pull carriages and walk up and down concrete streets; this puts un-natural strain on the hoof.

.Without some form of shoe to protect the hoof the horse would soon be lame, as the amount of hoof they wear away would far outweigh the amount they grow.

.Can help to treat a disease, support the hoof, correct a gait abnormality and, in some cases, may even save a horse’s life (e.g. for a horse suffering fromlaminitis).

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20
Q

Farrier

A

Farrier

TRIMMING
.Can vary between 6-10 wks depending on the hoof growth rate of the horse/pony

SHOEING
.Average horse requires new shoes every 6-8 weeks (or refits)
Depends upon:
-Growth rate of the feet, how horse wears his shoes, workload

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21
Q

SIGNS TO INDICATE RE-SHOEING IS NECESSARY

A

SIGNS TO INDICATE RE-SHOEING IS NECESSARY

.Lost or loose shoe
.Thin shoes (slipping on road)
.Risen clenches
.The shoe/s have spread
.The foot is growing over the shoe
.The wall of the foot has grown - unbalanced

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22
Q

HORSES NOT NECESSARILY REQUIRING SHOES

A

HORSES NOT NECESSARILY REQUIRING SHOES

.Those doing little road work
.Native horses/ponies who have naturally hard wearing feet
.Horses and ponies turned out at grass
.Front shoes only needed
.Regular trimming still required
.Welfare issues

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23
Q

The Farriers (Registration) Act 1975

A

The Farriers (Registration) Act 1975

Within the Farriers (Registration) Act 1975,farriery means “any work in connection with the preparation or treatment of the foot of a horse for the immediate reception of a shoe thereon, the fitting by nailing or otherwise of a shoe to the foot or the finishing off of such work to the foot”.
This means in practice that trimming which is not going to result in the application of a shoe to the foot of an equine is not covered by the Act and is therefore not regulated by this Council.
However, horse owners should be aware that although, simple trimming and rasping of horses’ feet is allowed by lay persons under the Act to permit maintenance of foals’ and other unshod horses’ feet. Where more radical trimming and reshaping of horses’ feet is contemplated there is the potential for creating severe lameness. Experience has indicated this is particularly so where such ‘therapy’ is not undertaken by qualified farriers or veterinary surgeons.
Incorrect trimming of horses’ hooves over an extended period can also cause lameness. Horses suffering from significant foot disease such as laminitis should in any case be under the care of a veterinary surgeon. This is to ensure that appropriate treatment is carried out and that any necessary medication is prescribed, the prime object being the welfare of the animal concerned.
Both the practice of radical foot trimming by lay persons resulting in significant lameness, and/or failure to provide veterinary attention under these circumstances may engender suffering and consequent criminal proceedings under the Animal Welfare Acts enforced by the RSPCA and SSPCA.
It is recommend that owners should only carry out minor work on feet of a cosmetic or emergency nature and that any significant trimming should be carried out by a Registered Farrier or a veterinary surgeon, who will have been properly trained and strictly regulated by a code of conduct

Currently anyone can set themselves up as a barefoot trimmer, potentially without holding any qualification or having any relevant experience. This is clearly a serious welfare issue. It is not appropriate for someone with insufficient training and experience to trim a horse’s hooves. There are a number of significant welfare risks ranging from laming the horse by over-trimming or poor hoof balance to advising the owner that the horse is capable of work that it is not able to do without risking harm to the feet.

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24
Q

Vaccination against

A

Vaccination against

.Equine Influenza
.Tetanus
.Equine Herpes Virus
.Rotavirus
.Equine Viral Arteritis – breeding stallions
.Strangles?
.Grass Sickness??

.Influenza:
-Primary course 2 injections 21-92 days apart First booster 150-215 days after 2nd primary vaccination Subsequent boosters within 365 days of preceding booster

-Most influenza vaccinations also contain the tetanus vaccine combined in a single injection and if you follow the vaccination schedule using the combined vaccine your horse will also be protected against tetanus.
When using separate vaccines, the schedule for tetanus vaccination is as follows:

-Primary course 2 injections 4-6 weeks apart First booster within 12 months of the 2nd primary injection Subsequent boosters only needed every 2 years

.Newborn foals are frequently given an injection of tetanus antitoxin (an ‘antidote’ to the tetanus infection) as soon as possible to provide temporary cover for 3-4 weeks. Regular tetanus vaccination can start at 3 months old.

.Rotavirus – diarrhoea in foals

.Eva – only in stallions – shed virus in semen

.Grass sickness links to botchulism – vaccinate for B in USA

Equine Grass Sickness Vaccine Trial
October 17, 2013
Grass Sickness Vaccine Trial Pilot Study Announced
The Equine Grass Sickness Fund, The Animal Health Trust and the Royal (Dick) School of Veterinary Studies, University of Edinburgh are delighted to announce that a small pilot trial of a vaccine to protect against grass sickness has been approved by the Veterinary Medicines Directorate. A total of 100 horses and ponies have been enrolled for this pilot study, with thanks to the participating owners for their support.
This pilot study is in preparation for a full vaccine trial, which is likely to commence in 2013-2014 subject to securing sufficient funding, which will involve at least 1000 horses and ponies. We have great hopes that these vaccine trials will mark an enormous breakthrough in the prevention of this devastating disease.

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25
Q

Some Common Equine Ailments

A

Some Common Equine Ailments

.Colic
.Choke
.Equine Influenza
.Strangles
.Sarcoids
.Mud fever & Rain scald
.Laminitis

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26
Q

Important info

A

Important info

.Remember that all horses will respond differently and have different baseline values

.Some horses are naturally calm, others are very active and excitable

.Some are aggressive eaters, some are slow and picky

.Bear in mind if there is a lot going on at the yard, moving home, bringing in a new neighbour

.Mares in season, stallions during the covering season

.Pregnant mares, foaling, weaning

.Within the first 4 days of life, the normal foal temperature varies between 99 to 102°F. The resting heart rate is between 70 to 100 beats per minute or higher if excited or active. Resting respiration rate in the first weeks is between 20 to 40 breaths per minute

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27
Q

Signs of health

A

Signs of health

 Know your horse
 Know ‘normal’ parameters for the individual
 Be aware of his behaviour

From Week 1 theory

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28
Q

Appearance

A

Appearance

 Should have bright eyes, look alert and generally content

 Colour in the eyes, nose, other membranous areas
o Pink is good
o Deep red / inflammation – colic?
o White – debility – off colour?
o Yellow gums – liver disorder?
o Purple – really bad – circulatory problem?

 Eyes & nose
o Discharge can indicate fever, low grade virus – treatment?
o Don’t confuse with drippy nose post exercise

 Coat and skin
o Starring coat – long term - worm infestation?
o Skin test for dehydration

 Sweating
o Pain or discomfort
o More immediate sign

 Swellings
o Glands / throat – worst case Strangles

 Don’t confuse with grass glands
o Legs – check stance

 Resting forelimb not good

 Resting hindlimb may be ok

o Body
 Rash – seasonal – insects? Nettles?

 Allergies – medicines – feed: protein build up?

Lots of these things self correct or need minimal treatment. However, it is vital that you remain vigilant

From Week 1 theory

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29
Q

Observing the horse

A

Observing the horse

 Has he eaten?
 Is he drinking?
 He is unhappy or grumpy or displaying unusual behaviours
 Lying down away from the group?
 Check the bed
 Droppings
o Texture
o Colour
 Urine
o Urinating excessively or not at all
o Clear to yellow
o Dark / purpley with strong smell – problem

From Week 1 theory

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30
Q

Signs of good health

A

Signs of good health

Temperature 38
Pulse 36-42 beats/min
Respiration 8 – 15 breathes/min
Good condition, shine to coat
Bright eyes
Alert, mobile ears
Droppings; normal consistency &
amount (8-10 daily) & break upon
hitting ground
Supple skin, skin recoil test, should
recoil immediately demonstrating
elasticity
Mucos membranes – salmon pink,
normal response to capillary refill test
Eating, drinking normally

From Week 1 theory

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31
Q

Signs horse may be off colour

A

Signs horse may be off colour

.Rise or fall in TPR
.Dull staring coat
.Poor condition
.Loose droppings /too hard /lack of
.Skin tight
.Lameness /resting a limb
.Obvious signs of injury /heat /swelling
.Discharge from nose
.Inability /lack of will to eat /drink
.Looking uncomfortable /rolling excessively
.Sweating /coughing /scratching /rubbing

From Week 1 theory

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32
Q

Temperature

A

Temperature

The temperature is taken by inserting
the thermometer into the anus of the
horse and holding against the side of the
rectum. Low readings may be taken if
the thermometer is inserted into a ball
of faeces by accident.

 Rectally

 Shake mercury thermometers, digital better?

 High temp may indicate
o Flu or virus
o Inflammation
o Bacterial induced from an infection in a wound.

 Low temp may indicate
o Hypothermia
o Loss of blood
o Serious illness

From Week 1 theory

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33
Q

Pulse

A

Pulse

 Optimum lower in a fit horse
 Higher in an overweight horse
 Higher in foals (50-100bpm)
 Take at a time when the horse is calm
 Digital pulse
 Jaw
 Pastern
 Forearm
 Tail

From Week 1 theory

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34
Q

Respiration

A

Respiration

 Faster at rest indication of pain
 Heave lines
 Watch from the flank or side

From Week 1 theory

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35
Q

Basic illnesses

A

Basic illnesses

 Colic
 Laminitis
 Mud fever
 Sweet itch
 Flu / coughs / colds
 Azoutria
 Skin conditions
o Ring worm
o Rain scald
o Hives
The quicker ailments and conditions are reported, the more chance of a speedy
recovery and prevention of the ailment becoming more serious or spreading.

From Week 1 theory

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36
Q

Prevention

A

Prevention

 Good feeding
 Good exercise
 Good stable management
 Vaccinations – Flu and tet’ – yearly
 Feet – 6-8 weeks
 Teeth – 6-12 months
 Back – 3 months optimum (6-12?)
 Worming

From Week 1 theory

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37
Q

YOUR HORSE HEALTH
CHECKLIST DAILY

A

YOUR HORSE HEALTH
CHECKLIST - DAILY

Daily

HEART RATE
(normal range 28 – 44 beats per minute at rest)

RESPIRATORY RATE
(normal range 8 – 14 breaths per minute at rest)

TEMPERATURE
(normal range 37.5ºC – 38.5ºC)

DIGITAL PULSE

URINATION – FREQUENCY AND HABITS

APPETITE DRINKING

EYES

PICK OUT FEET AND CHECK SHOES (if appropriate)

ASSESS DROPPINGS

HOOF TEMPERATURE

MOVEMENT/STANCE

GENERAL DEMEANOUR

SKIN

From Week 1 theory

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38
Q

YOUR HORSE HEALTH
CHECKLIST MONTHLY

A

YOUR HORSE HEALTH
CHECKLIST - MONTHLY

BODY CONDITION SCORE

THOROUGH SKIN CHECK

From Week 1 theory

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39
Q

YOUR HORSE HEALTH
CHECKLIST ANNUAL

A

YOUR HORSE HEALTH
CHECKLIST - ANNUAL

ROUTINE VET CHECK

VACCINATIONS

DENTAL CHECK

From Week 1 theory

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40
Q

YOUR HORSE HEALTH
CHECKLIST - OTHER

A

YOUR HORSE HEALTH
CHECKLIST - OTHER

CHECK TACK AND EQUIPMENT
BEFORE USE

FWEC/WORMING TREATMENT FOLLOWING
VETERINARY ADVICE

From Week 1 theory

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41
Q

What is the maximum amount of time a wet poultice should be used for?

A

What is the maximum amount of time a wet poultice should be used for?

.You shouldn’t really use a wet poultice for more than three days as the foot needs chance to dry. You can dry poultice for a while though. Best to speak with you farrier if you are still concerned.

From
https://forums.horseandhound.co.uk/threads/foot-abscess-how-long-to-poultice.380432/#:~:text=Be%20careful%20of%20over%20poulticing%20the%20foot.%20You,but%20can%27t%20get%20it%20-%20any%20other%20suggestions%3F

Question from Pratical sheet Week 1 and 2

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42
Q

Why might you apply a wet poultice to a foot abscess?

A

Why might you apply a wet poultice to a foot abscess?

.Poultices keep the opening moist and encourage drainage. Changing bandages daily is recommended, as a poultice can dry the foot out if the abscess hasn’t broken.

.Soaking the hoof in warm water and Epsom salts (two cups to a gallon) between bandage changes is also a good practice.

From
Microsoft bing search

.A wet poultice (the most common way to apply) is more effective at drawing fluid to out, but a dry poultice could work well if an abscess has a lot of fluid coming out on its own.

From
https://www.petplanequine.co.uk/events/horse-hoof.asp#:~:text=A%20wet%20poultice%20%28the%20most%20common%20way%20to,Make%20sure%20the%20foot%20is%20clean%20and%20dry.

Question from Pratical sheet Week 1 and 2

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43
Q

Basic First Aid

A

Basic First Aid

First Aid = dealing with minor injuries OR being the first responder to major injuries. Have a first aid kit.

Good first aid needs to:
.Be administered promptly
.Be administered correctly
.Limit chances of further injury
.Recognise the need to call a vet

From
Week 1 and 2 practical = Practical First Aid and Bandaging - RECORDED Lecture NotesFile, slide 2

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44
Q

Wound Management

A

Wound Management

.Control the bleeding – apply pressure

.Clean, Clean, Clean!!!!!!

.Water / Saline Solution / DILUTED chlorhexidine (hibiscrub)

.DO NOT apply any creams / ointments / sprays

.Dress the wound

From
Week 1 and 2 practical = Practical First Aid and Bandaging - RECORDED Lecture NotesFile, slide 3

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45
Q

Dressing Wounds

A

Dressing Wounds

Have everything to hand before you start!

1) Dressings
-Melolin
-Allevyn

2) Soft cotton bandage
-Soffban

3) Knitted bandage

4) Cotton wool / Gamgee

5) Vet wrap

Refer to videos on Moodle to see how to apply dressings and bandages to leg wounds

From
Week 1 and 2 practical = Practical First Aid and Bandaging - RECORDED Lecture NotesFile, slides 4 to 8

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46
Q

Bandaging Awkward Areas: Knees and Hocks

A

Bandaging Awkward Areas: Knees and Hocks

.Risk to joints (carpal bone and point of hock)

.These areas need to be alleviated of pressure

.More likely to slip down

Refer to video on Moodle to see how to apply a hock bandage correctly

From
Week 1 and 2 practical = Practical First Aid and Bandaging - RECORDED Lecture NotesFile, slide 9

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47
Q

Do’s and Don’ts of Bandaging

A

Do’s and Don’ts of Bandaging

Do:
.Use clean sterile dressings
.Wrap evenly in one direction
.Wrap all dressing layers the same direction
.Overlap each layer by 50%
.Get everything ready before you start

Don’t:
.Wrap any layer too tight – room for a finger
.Have any wrinkles or points of uneven pressure
.Apply vet wrap directly to the leg
.Have too tight (or too loose)
.Leave gaps in the layers

From
Week 1 and 2 practical = Practical First Aid and Bandaging - RECORDED Lecture NotesFile, slide 12

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48
Q

Hoof Problems

A

Hoof Problems

Hot Hooves = Problems!!!!!!!!!!
-Laminitis
-Abscess

.Feel for heat in your horse’s hooves everyday – know what is normal

.Notice any difference in heat level between feet

.Feel for a Digital Pulse; BOUNDING = INFLAMMATION

From
Week 1 and 2 practical = Practical First Aid and Bandaging - RECORDED Lecture NotesFile, slide 13

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49
Q

What is a Hoof Abscess?

A

What is a Hoof Abscess?

.Also known as “pus in the foot” – EXACTLY that!! A build up of infected pus in the hoof.

Occurs when tiny cracks appear in the dry hoof wall that allow moisture, dirt, and bacteria to get in.

.This leads to infection and a build up of pus

.Very quickly becomes VERY painful for the horse

.They will be very lame and find it hard to put their foot to the floor.

.Watch out for swelling of the pastern - a sign that the infection is going up the leg. If this happens, call your vet immediately.

How to treat it???
-Farrier can dig it out if they can locate where it is
-Vet can apply a small hole to allow for drainage
-Hoof abscesses are routinely treated with a poultice “draw” it out

From
Week 1 and 2 practical = Practical First Aid and Bandaging - RECORDED Lecture NotesFile, slide 16

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50
Q

What is a poultice?

A

What is a poultice?

.Type of dressing that is put onto the horse hoof designed to draw out an abscess that has formed

.It normally comes in the shape of a horses hoof – or can be cut to size.

.Can be applied wet (placed in cooled boiled water) or dry

.A wet is more effective at drawing
fluid out, but a dry poultice could work
work well if an abscess has a lot of
Fluid coming out on its own.

.Used to draw out infection and
keep the area clean to prevent further
reinfection.

.Modern poultice dressings (such as Animalintex) contain boric acid and tragacanth, which work as an antiseptic and help draw out the pus.

From
Week 1 and 2 practical = Practical First Aid and Bandaging - RECORDED Lecture NotesFile, slide 17

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51
Q

Applying a Poultice

A

Applying a Poultice

1) Make sure the infected hoof has been cleaned thoroughly before you start. It must be clean and dry.

2) If you are using a wet poultice; remove from the cooled previously boiled water and squeeze the excess water out

3) Apply to the foot.

4) Wrap a layer of soft padding such asGamgee or Soffban bandage (or even a nappy) over the top of thepoultice and around the foot.

5) Focus yourattention to cushioning soft tissue areas like the heel bulbs and the coronary band.

6) Now apply the Vet wrap around the hoof to hold it all in place. Not too tight to cause pressure over the soft tissues, but not too loose either as it will fall off.

7) Use the heavy duty strips of duct taped – that you should have already cut and prepped - placing it over the sole of the foot and up the sides of the hoof wall.

8) This will create acreate a waterproof layer and to keep the dirt out.

9) Place Elastoplast and more duct tape over the top to ensure it is secure.

10) You can also use a poultice boot, or thick plastic haylage
bags or feed sacks to keep moisture and dirt out.

11) Change the poultice at least once a day, or more if
there’s a lot of pus coming out.

12) Only use a wet poultice for two to three days at a
time, then switch to a dry poultice.

.If you leave a wet poultice any longer the wound and
hoof will get waterlogged, which may weaken the foot.

.Refer to Moodle for a video showing how to apply a poultice

From
Week 1 and 2 practical = Practical First Aid and Bandaging - RECORDED Lecture NotesFile, slides 18 and 19

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52
Q

First Aid Kit (Equine)

A

First Aid Kit (Equine)

The following would be a suggested list of contents for a full equine first aid kit:

 Clean bowl or bucket
 Clean towel
 Large roll of cotton wool
 Round-ended curved scissors for trimming hair from wound edges
 Anti-bacterial scrub eg Hibiscrub or Pevidine
 Pack of sterile saline — very handy when on the move
 Ready-to-use poultice eg Animalintex, Poultex
 Wound gel eg Intrasite Gel
 Non-stick dressings eg Melolin, Rondopad
 Gamgee and large scissors for cutting it to size

 A selection of bandages including:
- Stretch cotton bandages eg Knit-firm, K-band and crepe
bandages
- Adhesive bandages eg Elastoplast
- Elastic conforming self-adhesive bandages eg Vetrap, Co-
plus
- Tubular bandage eg Tubigrip
- Synthetic orthopaedic bandage eg Soffban
- A set of stable bandages

 A roll of electrical insulating tape 2cm wide
 A roll of black PVC tape or silver duct tape 7.5 or 10cm wide
 A gentian violet or antibiotic spray
 Petroleum jelly eg Vaseline
 Wound powder containing fly repellent
 Wound gel such as Dermagel, intrasite gel or vetalintex
 Small pair of tweezers
 Thermometer
 Paper and pencil
 A bright torch for inspecting wounds in poor light

From
Practical week 1 and 2
Additional Reading/Resources:
Equine First Aid Kit - Checklist from pony club

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53
Q

Most bandages include the same two to three layers:

A

Most bandages include the same two to three layers:

.Topical dressing, which might be a liniment, medicated pad, ointment, or powder. These are generally used in horses with injuries or skin conditions.

.Thick cotton padding such as practical (roll) cotton, layers of sheet cotton, cast padding, or fabric quilt or pillow wraps.

.Compressive/securing layer such as stable/track bandage, Vetrap, gauze, polo wrap, elastic tape, or stockinette.

From
Week 1 and 2 further reading link below
https://thehorse.com/115469/horse-bandaging-fundamentals/

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54
Q

Bandaging tips

A

Bandaging tips

  1. Keep everyone safe. Preventing human injuries is just as important as treating or preventing equine ones. The person applying the bandage should avoid kneeling or sitting on the ground, says Dechant, and should instead crouch, ready to move out of the way if necessary. She also recommends having a competent handler hold the horse during the process. Bear in mind, too, that some horses initially resent wraps on the hind legs, especially over the hocks, so it’s best to apply these in an open area in case the horse kicks out.
  2. Don’t skimp on the padding. “Insufficient padding is going to cause a bandage bow,” says Hanson. Padding should be clean, dry, and in reasonable shape, Dechant adds. Since the idea of the padding is to protect the leg, it’s important to avoid incorporating frayed bits of padding or fill that contains wrinkles or bunches–these can cause pressure points under a bandage.
  3. Keep it even under pressure. Remember that “anything directly against the skin should not be applied with any tension at all,” Dechant says. But uneven tension in a bandage’s securing layers also can potentially cause tendon damage. “You want an even distribution of compression along the leg” with this layer, too, says Hanson.

“The key is to apply it firmly but not too tightly,” Dechant adds. If using Vetrap or a similar flexible bandage to secure the padding, she suggests applying enough tension to remove 80% of the wrap’s innate “wrinkles.” She also stresses the importance of overlapping layers of bandage by 50% to avoid having edges of the wrap material dig into the leg.

Using a neatly and tightly rolled bandage will ease application and reduce the need to pull against the horse’s leg and sensitive tendons to tighten the wrap. This will also help ensure the bandage is as smooth against the horse’s leg as possible to avoid uneven pressure.

  1. Choose your own direction. Despite barn lore to the contrary, neither sources believe the direction a wrap is applied is critical. “Counterclockwise vs. clockwise is less important than technique,” says Dechant. “I don’t think the tendons care if they’re rolled to the outside or to the inside. However, each layer should be rolled the same (direction).” Hanson agrees with Dechant, noting that he hasn’t come across anything in literature to suggest wrapping in one direction or the other is superior. It is, however, important to be consistent in your technique and not to pull too tightly across the tendons.
  2. Keep it clean. Shavings, straw, dirt, and moisture can irrate the skin and increase the risk of a wound becoming infected. Start with clean, dry materials and check the bandage frequently for damage, dirt, or moisture. To seal out debris, Dechant recommends securing the top and bottom of a disposable-type wrap with elastic tape such as Elastikon.

From
Week 1 and 2 further reading link below
https://thehorse.com/115469/horse-bandaging-fundamentals/

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55
Q

Foot abscess process

A

Foot abscess process

.A foot abscess is a localised infection within the hoof, which forms a pocket of pus within the hoof horn. This usually develops due to a tiny penetration of the foot, for example, by a sharp stone. Bacteria are implanted into the deeper layers of the sole, and the more superficial layers then close over the top, trapping the infection. This can happen up to several weeks before any signs are seen, and usually goes totally unnoticed.

.After an average of 10-14 days, the bacteria have grown, and enough pus has accumulated that there is a large pressure build up. This pressure pushes on nerves within the foot and causes often excruciating pain. There is also significant inflammation within the foot, which can be noticed as a bounding digital pulse, and a degree of heat in the hoof wall. Lameness will also develop at this point, which will often be so severe that the horse is unwilling to place their foot on the ground. This is the time at which the vet is often called.

From week 1 and 2 further reading link below
https://equine-vets.com/health/f/foot-abscess/

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56
Q

Identifying Lameness

A

Identifying Lameness

• Step 1: Look at the horse static and take history.

• Step 2: initial assessment. Walk and trot straight line,
(flexion tests, hoof and joint tests), lunging on both
firm and soft surface, palpation of limbs, back and
neck. Ridden exam.

• What are we looking and feeling for when we
palpate?
• Why different surfaces?
• Why circle?

• Step 3: Diagnostic procedures. –nerve blocks
then once area of concern located xrays/ultrasound potentially bone scans/MRI

From week 2 theory

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57
Q

Lameness protocol

A

Lameness protocol

• Before static assessment always ensure:
– The horse is standing square on a firm, even
surface
– View from cranial, lateral and caudal

• Look

• Feel
– Logical

• Dynamic assessment :
– View from cranial, lateral and caudal

• Looking for:
– Straightness
– Width
– Length of stride
– Travel

• Initially straight lines on a hard surface

• May also use soft surface and look at the horse ridden.

• LISTEN.

From week 2 theory

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58
Q

lameness in FL or HL?

A

lameness in FL or HL?

Forelimb
• Head nod on sound limb
• Louder landing on sound limb
• Increased fetlock extension on sound limb

Hindlimb
• Looking from behind the hip
is hitched and the lame limb
appears to be carried higher
than the non-lame limb.
• Occasionally head nod on
lame limb
• Louder landing and fetlock
extension on sound limb
• “Back pain”

From week 2 theory

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59
Q

Location of lameness

A

Location of lameness

• Ligament
• Tendon
• Foot
• Joint
• Muscle

From week 2 theory

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60
Q

Where Gait Phase for lameness

A

Where Gait Phase for lameness

Upper Limb Lameness
• Swing phase
• Muscular
• Joints above carpus/tarsus
• Bones

Lower Limb Lameness
• Stance phase
• Joints below carpus/hock
• Tendons
• Bones

From week 2 theory

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61
Q

Lameness Prevalence

A

Lameness Prevalence

• 8-13% of horses experience lameness each year
– 70% recover

• Distribution
– Load 60% FL
– 40% HL

• Up to 95% of FL lameness distal to carpus

• HL lameness less common, but not to be dismissed

From week 2 theory

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62
Q

Importance of a Thorough Assessment for lameness

A

Importance of a Thorough Assessment for lameness

• Ridden assessment more important than previously thought
– Estimated 12% of lameness is only apparent when ridden

• Rider ability important and should be standardised where
possible for assessment

• Assess in trot and on both diagonals.

• Subtle lameness may require more intensive work.

From week 2 theory

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63
Q

Lameness Assessment tips

A

Lameness Assessment tips

• Consider the relative quality of the trot and the canter.

• Pain often causes generalised restriction of movement
– The horse may fail to track up
– The horse may become heavy in
the rider’s hand
– Unwillingness/tension

• Low grade forelimb lameness
– The horse may take slightly lame steps on turns

• Assess body lean – should be symmetrical.

• Problems in canter often manifest first.

• With hindlimb lameness it is typical that there are difficulties in movements requiring more collection.

• Saddle slip may be indicative of lameness.
– Saddle slip with HL lameness >86% occurs to the side of the lame limb.

From week 2 theory

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64
Q

Grading Lameness

A

Grading Lameness

0 Lameness not perceptible under any circumstances

1 Lameness is difficult to observe and is not consistently apparent, regardless of circumstances

2 Lameness is difficult to observe at a walk or when trotting in a straight line but consistently apparent under certain circumstances

3 Lameness is consistently observable at a trot under all
circumstances

4 Lameness is obvious at a walk

5 Lameness produces minimal weightbearing in motion and/or at rest or a complete inability to move

From week 2 theory

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65
Q

Conditions of the Foot

A

Conditions of the Foot

Most common cause of lameness in forelimb. Multiple structures can be affected;
• Bruising
• Abscess
• Wall cracks
• Sole
- penetrations/punctures
• Laminar problems
• Collateral cartilage
• Coffin/pastern joints
• Navicular bone/bursa
• Hygiene issues

From week 2 theory

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66
Q

Soft Tissue Structures

A

Soft Tissue Structures

• Tendons
– Transfer of muscle contraction into skeletal movement
– Increase locomotory efficiency by storing and releasing energy

• Ligaments
– Align and stabilise adjacent bones
– Passive resistance to motion

From week 2 theory

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67
Q

Common Soft Tissue Injuries

A

Common Soft Tissue Injuries

• Discipline related injury types
– SDFT

• Increased risk factors; fast work; age; firmer surface;
weight and hoof conformation
– DDFT

• More commonly seen in SJ, Endurance and Dr (age?)
– Proximal suspensory desmitis – SJ, dressage
– Tendonitis

From week 2 theory

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68
Q

Tendons

A

Tendons

• Injury occurrence 11% -46%
• Re-injury in 43% - 93%
• Common reason for retirement
• Diagnosis?

From week 2 theory

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69
Q

Tendon Injury

A

Tendon Injury

• Extrinsic
– Sharp laceration
– Traumatic blow
– Damage to ECM and cells

• Intrinsic
– Degenerative condition
– Overuse injury

• Mechanical influences
– Cumulative microdamage
– Single acute overload

• Hyperthermia
– Loss of stored energy as heat
– Up to 45º in gallop

• Poor blood flow
– Limited during maximal loading
– Reperfusion injury

From week 2 theory

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70
Q

Tendon Treatment and Rehab

A

Tendon Treatment and Rehab

• Immediately – 10-14 days
– Reduce inflammation
– NSAIDs?
– Support
– Rest

• Cold therapy
– Gold standard?

• Controlled Exercise

• Additional treatments?

• Regimen individual to horse and situation

From week 2 theory

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71
Q

Muscle Injury

A

Muscle Injury

Phase 1: Necrosis and Inflammation
Phase 2: Regeneration, Repair, Remodel
Phase 3: Remodelling and Fibrosis

Predisposing factors?
Diagnosis?
Treatment – RICE?
Recovering time?

RICE principle?

.Rest
- First 24-48 hours critical treatment period

.Ice
- For first 48 hours post-injury
- 20 minutes every 3-4 hours

.Maybe Compression

.Maybe Elevation

From week 2 theory

72
Q

Joints

A

Joints

• Composed of
– Bone
– Cartilage
– Peri-articular soft tissues

• All involved in joint disease

• Cartilage probably most important
– Load-distribution
– Frictionless gliding
– Altered structure and function leads
to abnormalities in all tissues

From week 2 theory

73
Q

Joint Injury

A

Joint Injury

• Osteoarthritis
– Degeneration and loss of articular
cartilage
– Development of new bone on joint
surfaces and margins
– Number of potential causes

• Puncture injury near the site of a
joint

• Repetitive microtrauma
– Damage to healthy cartilage

• Subchondral bone sclerosis
Increased bone stiffness
Abnormal stresses on cartilage
Mechanical damage

From week 2 theory

74
Q

Joint Injury: Assessment

A

Joint Injury: Assessment

• Pain attributed to peri-articular soft
tissues and bone

• Limited range of motion

• Effusion
– Destabilisation of joint

From week 2 theory

75
Q

Week 2 Summary theory

A

Week 2 Summary theory

• Most tissues heal in a similar fashion starting with inflammation

• Mild lameness without obvious heat or swelling can initially be treated with rest

• More severe or chronic lameness or if obvious heat or swelling present
– Should be attended to by vet

• Prognosis for future athletic use depends on type and site of lesion

From week 2 theory

76
Q

What do we mean by biosecurity

A

What do we mean by biosecurity

.Dictionary definitions
–procedures or measures designed to protect the population against harmful biological or biochemical substances (i.e. biosecurity risks)

.Biosecurity is set of measures aimed at preventing the introduction and/or spread of harmful organisms (i.e. biosecurityrisks), in order to minimise the risk of transmission of infectious diseases to people, animals and plants.

.Main factors/routes of transmission
-Humans
-Weather
-Animals
-Food

From week 3 lecture

77
Q

Routes of transmission: animals

A

Routes of transmission: animals

Direct animal-animal contact:
• at markets or shows,
• new animals introduced to farm,
• fence-line contact,
• within farm contacts.

Fomites & vectors:
• Aborted foetus – Equine herpesvirus type 1
• pets – toxoplasmosis,
• wildlife – Birds (e.g. salmonella), Badgers (bTB),
• pests - Rats (leptospira), Flies & midges (salmonella, bluetongue, African Horse sickness, West Nile fever).

Human Mechanical transmission:
• Carrying of disease causing
organisms
– hands
– clothing
– footwear
– equipment (e.g. head collar)
– vehicles

Weather:
Wind
• The viral plume from an animal infected with Foot and Mouth can spread 500 m down wind.
• Likely to be similar for equine influenza.
• Could carry infected insects (e.g. midges).
Rain
• Flooding – transfer of microorganisms in flood water.

From week 3 lecture

78
Q

Zoonosis, zoonotic disease

A

Zoonosis, zoonotic disease

.Diseases and infections which are naturally
transmitted between vertebrate animals and
humans (WHO 1959)

  1. Humans can get
    some animal
    diseases
  2. Animals can get
    human diseases (Reverse
    Zoonoses)

From week 3 lecture

79
Q

Biosecurity risk: Food

A

Biosecurity risk: Food

Not the main risk. However:
• lead, copper and other heavy metals
intoxication,
• ergot – cereal fungus (Claviceps purpurea),
• atypical myopathy (sycamore seeds, leaves and
seedlings, toxin),
• equine grass sickness (Clostridium botulinum
toxins),
• equine asthma (dust, mould, toxins…),
• guttural pouch mycosis (Aspergillus fumigatus).

From week 3 lecture

80
Q

Benefits of Biosecurity

A

Benefits of Biosecurity

• Reduced the risk and occurrence of diseases,
• improved animal health & welfare,
• reduced stress for owners,
• reduced need and costs of treatment (e.g.
antimicrobial resistance).

From week 3 lecture

81
Q

Benefits of reduced livestock disease

A

Benefits of reduced livestock disease

• Reduced disruption of farming and rural businesses,
• improved productivity and performance,
• improved quality of marketable produce,
• reduced risk of transmission of diseases that can
spread to humans, e.g. Salmonella spp, E.coli etc,
• protected export markets,
• potential to improve the quality of the environment
as a consequence of the reduced disposal of wastes.
From - (DARDNI, Department of Agriculture, Environment and
Rural Affairs is a government department
in the Northern Ireland, 2004)

From week 3 lecture

82
Q

Biosecurity: at what level to
apply?

A

Biosecurity: at what level to
apply?

• Herd/farm level (internal & external)
– Individual business issues.
• National level
– Individual country issues.
• Global level
– Between Country issues.

From week 3 lecture

83
Q

Farm biosecurity - internal

A

Farm biosecurity - internal

Between groups of animals :
• livestock batches,
• age groups
• ≠ susceptibility (e.g. foals for
rhodococcus equi infection),
• specific status (e.g. pregnant mares
for equine herpesvirus type-1
infection and risk of abortion),
• etc.

From week 3 lecture

84
Q

prevention of Rhodococcus equi infection
aka RATTLES)

A

prevention of Rhodococcus equi infection
aka RATTLES)

.Rhodococcus equi infection can induce abscesses
in the lung and abdomen, joint infection,
osteomyelitis, diarrhea, lymph nodes
inflammation et.

.This disease primarily affects foals less than 6
months of age.

.The bacteria is present in soil,manure and feces (e.g. horses).
Foals are infected by inhalation of contaminated
dust.

.Risk: pastures with low grass cover for mares and foals in the
suceptible period (equiways).

.The main vector is contaminated dust

.Preventive measures
-move regularly water troughs when it is possible or reseed or water regularly.
-Remove regularly droppings from paddocks with mares and foals at foot.
-Do not use dust ground for young foals - muck out regularly.
-Suitable floor for foals.
-Clean dust from Equipment.
-Identify area at risk and/or contaminated.

From Research project: LABÉO/Equiways (R.Paillot/C.Vercken)

From week 3 lecture

85
Q

Farm Biosecurity - external

A

Farm Biosecurity - external

.Vaccination
.Isolating incoming livestock and sick livestock
.Boot dips
.Pick up/drop off areas - so dust from vehicles not getting into
foal area.
.Dedicated clothing
.Washing /Disinfection facilities
.Separate herd/wildlife and neighbouring
livestock - by double fencing.

From week 3 lecture

86
Q

Farm biosecurity: multiples elements
to take into account

A

Farm biosecurity: multiples elements
to take into account

• Horse movement (e.g. stallion, mares, foals).
• Other animals (e.g. dogs).
• Human activities (e.g. veterinarians, farrier).
• Vehicle movement.
• Materials.

From week 3 lecture

87
Q

National Level Biosecurity

A

National Level Biosecurity

• preventing diseases entering the country,
• controlling the spread of infectious diseases across
the country.

From week 3 lecture

88
Q

Equine Influenza biosecurity

A

Equine Influenza biosecurity

• Endemic in most countries,
• highly contagious,
• most outbreaks are associated with movement.

Main elements of prevention:
• surveillance,
• vaccination,
• movement restriction,
• Containment (quarantine).

From week 3 lecture

89
Q

Notifiable Diseases

A

Notifiable Diseases

.What are they? A list of diseases with potential for rapid
spread and serious economic or public health implications,
which require legislation to co-ordinate control actions.

They have a significant impact on one of more of:
• International Trade (e.g. export health certificates),
• Public Health (zoonotic diseases),
• Animal Welfare (and human),
• Wider Society (e.g. restriction, reputation).

Taking into account:
• Cost to the community (needs to be reasonable),
• Availability of appropriate solutions.

From week 3 lecture

90
Q

Notifiable Diseases in the UK

A

Notifiable Diseases in the UK

.‘Notifiable’ diseases are animal diseases that you’re legally
obliged to report to the Animal and Plant Health Agency
(APHA), even if you only suspect that an animal may be
affected. Failure to report a suspect of notifiable animal
disease is treated as an offence!

.Some endemic and exotic diseases are also zoonotic.

Notifiable diseases can be:
• Endemic – already present in the UK, such as bovine TB
• Exotic – not normally present in the UK, such as foot
and mouth disease

Website - https://www.gov.uk/government/collections/notifiable-diseases-in-animals

From week 3 lecture

91
Q

Notifiable disease African Horse Sickness virus (AHSV)

A

Notifiable disease African Horse Sickness virus (AHSV)

.African Horse Sickness virus (AHSV) : Reoviridae family, Orbivirus genus ; spread via bitting midges (culicoides).

• Exotic disease (endemic in South Africa; outbreak in Thailandin 2020)

• Pulmonary form: Fever, incubation period of 3-5 days,
laboured breathing, foamy discharge. 90% mortality.

• Cardiac form: Swelling over face, eyes, death from heart
failure, incubation period of 7-14 days. 60% mortality.

website - https://thehorse.com/148892/researchers-digging-deeper-into-african-horse-sickness/

From week 3 lecture

92
Q

Zoonotic diseases (e.g. West Nile Fever)

A

Zoonotic diseases (e.g. West Nile Fever)

.In humans, West Nile virus (WNV) can cause a disease known
as West Nile fever. Around 80% of infected people
have few or no symptoms, around 20% of people develop mild symptoms (such as fever, headache, vomiting, or a rash). Less than 1% of people develop severe symptoms (e.g.
encephalitis or meningitis).
WNV can induce a neurological disease in horses.
In wild birds, from no signs to death.

.Yearly spread of WNV throughout the U.S. 1999–2002. Counties reporting WNV activity in humans (red) and
non-human e.g., birds, mosquitoes, equines, and other mammals (blue), as reported to CDC ArboNet.” (Roehrig J.T., Viruses, 2013).

From week 3 lecture

93
Q

Recognising Signs of
Distress

A

Recognising Signs of
Distress

• Key for more subtle injury/illness
• Assessment of health protocol
– TPR
– Behaviour
– Skin pliability
– Colour of mucous membranes
– Colour and consistency of feces and urine
– Presence of absence of gut sounds
– Evidence of lameness
– Bleeding, swelling or evidence of pain

94
Q

Ring up vet asap

A

Ring up vet asap

.Respiratory distress
.Inability to rise or stand
.Notably painful or severe diarrhea
.Gut pain with acute onset or colic
.Unwell foals
.Prolonged/abnormal sweating, high temperature, anxiety, restlessness or loss of appetite
.VERY serious injury involving deep wounds, severe
hemorrhage, suspected fractures or damage to the eyes
.Evidence of straining >30 mins in foaling mares
.Together with any other signs of acute pain or injury

From SLIDES - What is an Equine Health Emergency

95
Q

Immediate veterinary
attention

A

Immediate veterinary
attention

• Fractured limb
• A collapsed horse that is unable
to stand
• Non-weight bearing lameness
coupled with distress
• Non-weight bearing lameness
coupled with a wound
• Multiple limb non-weight bearing
lameness
• Wounds that require stitching
• Colic pain that is moderate or
violent and/or continuous
• Diarrhoea that is continuous
and/or painful
• Choke where coupled with
obvious distress
• Sudden or severe inability to
breathe normally
• Punctured or ulcerated eye or
sudden onset blindness
• Continuous bleeding from mouth,
nostrils, rectum, vagina, penis, or
an arterial bleed
• Sudden onset of severe
neurological dysfunction
• Difficulties at foaling
• A sick foal, especially if it will not
feed from the dam

From SLIDES - What is an Equine Health Emergency

96
Q

Waiting for the Vet to Arrive

A

Waiting for the Vet to Arrive

  1. Keep the horse calm.
  2. Secure horse and human safety.
  3. Arrange and coordinate help
  4. Contact your vet. Be ready with key
    information.
  5. Follow any instructions provided by the vet.
  6. Do not administer drugs unless specifically
    instructed to do so by your vet.

From SLIDES - What is an Equine Health Emergency

97
Q

What might the vet need to
know?

A

What might the vet need to
know?

IMPORTANT
• How lame is the horse?
• How much pain is a horse in?
• Can you take your horse’s temperature and pulse rate? What is their breathing like?
• If there is a wound, specifically where on the horses’ body is it? Do the edges of the wound pull apart? Is it clean or dirty?

From SLIDES - What is an Equine Health Emergency

98
Q

Prompt but not immediate vet
attention required

A

Prompt but not immediate vet
attention required

Lameness
– Marked lameness that has not responded to normal first
aid treatment
– Sudden onset lameness that is weight bearing

Injury
– Traumatic injuries and wounds that are superficial
– Injury that has not responded to normal first aid treatment

Signs of non-specific ill health
– Persistent weight loss/sustained loss of appetite
– Signs of disease that might be infectious
– Sub acute illness or injury

• Very mild colic*
• Potential lymphangitis
• Flare ups of chronic laminitis
• Flare ups of chronic inflammatory respiratory disease

Note that these conditions are likely to require
assistance from the vet in the form of an initial phone
call but are then frequently monitored to ascertain if in
person veterinary attention is required.

From SLIDES - What is an Equine Health Emergency

99
Q

Routine veterinary treatment
required

A

Routine veterinary treatment
required

• Intermittent and slight lameness
• Persistent dermatitis
• Intermittent and slight eye discharge
• Reduced appetite with no other clinical signs
• Nasal discharge with no fever or difficulty breathing
• Persistent coughing
• Anything else that sits outside the normal
behaviour and health for your horse

From SLIDES - What is an Equine Health Emergency

100
Q

Emergency Action Plan

A

Emergency Action Plan

Having a plan is KEY to avoid panic
1. Contact details of your vet.
2. Have a contingency contact.
3. Know the most direct route to your vets and how
long it takes.
4. Help and assistance.
5. Accessible and current first aid kit both on the
yard and in vehicles/trailers.

Owner Absence
• Clear instructions in case of emergency
– Who is responsible if you are not available
• Your horse’s passport preferably signed, etc
• Information regarding insurance cover
• Referral plan? Payment for emergency
treatment if insurance is not in place? Part of
a livery contract? Review yearly.

From SLIDES - What is an Equine Health Emergency

101
Q

Key Equine Diseases and Infections

A

Key Equine Diseases and Infections
Strangles
Equine Influenza
Ringworm
Tetanus

Key Disorders/Conditions:
Colic
Laminitis
Choke
‘Tying Up’

From week 4 lecture

102
Q

Strangles

A

Strangles

.Prevalent infectious respiratory disease
.Transmission via infected respiratory droplets but not airborne

Symptoms:
.High temp - first
.Lethargy/depression
.Reluctance to eat/drink, difficulty swallowing and/or lowered head and neck
.Thick and discoloured nasal discharge
.A cough
.Swelling of glands under the jaw
-Lymph node enlargement
-Abscess development

Strangles: Prevention
.Best Defence:Quarantine and test all new arrivals!
.Vaccination: Can be useful in open management systems
.Detailed prevention plan and policies!

From week 4 lecture

103
Q

Traffic Light System

A

Traffic Light System

On suspicion of infection, the yard should be divided into different areas:

.Red Zone: high risk horses with signs of the disease

.Amber Zone: medium risk – horses that have been in contact with infected horses but are not yet showingsigns of the disease
.
.Green Zone: low risk – horses which have not been in contact with infected horses

From week 4 lecture

104
Q

Strangles: An Outbreak

A

Strangles: An Outbreak

Initial Control
-Limit spread of infection within the premises

Containment
-Prevent further spread of infection to new premises

‘Clean-Up’
-Identify carriers and treat

From week 4 lecture

105
Q

Equine Influenza

A

Equine Influenza

.Highly contagious, viral, infectious disease

.Indirect and direct spread

.Unlike other infections the flu virus does not survive for long outside of the horse

.Airbourne - isolation of horses need 100 meters apart

From week 4 lecture

106
Q

Equine Influenza: Symptoms

A

Equine Influenza: Symptoms

.High Temperature (Pyrexia) - first sign
.Clear, watery discharge which may later (in a few days)
become thick and yellow/green in colour
.Coughing
.Reduced appetite
.Swollen and painful glands around the jaw

From week 4 lecture

107
Q

Equine Influenza: An Outbreak

A

Equine Influenza: An Outbreak

.Traffic Light System

Consider how it is spread!
-Isolation facilities
-Restrict movement on/off the yard

From week 4 lecture

108
Q

Ringworm (Dermatophytosis)

A

Ringworm (Dermatophytosis)

.Fungal skin infection. Two main strands of fungus:
-Trichophyton
-Microsporum

.Highly contagious!
.Zoonotic
.Incubation period of 2-3 weeks
.Spreads quickly by direct contact
.Can survive for many years onwooden surfaces

From week 4 lecture

109
Q

Ringworm: Symptoms

A

Ringworm: Symptoms

.Various presentations
-Skin lesions
-Hair loss
-Scabbing

.Can become extensive if not treated

From week 4 lecture

110
Q

Ringworm: Controlling an Outbreak

A

Ringworm: Controlling an Outbreak

.Separate grooming kit, tack, etc.
.Handler care – over clothes, dealt with last the horse that is ill
.Thorough disinfection of environment (grooming kit, rugs,stables, fencing etc.)

From week 4 lecture

111
Q

Tetanus

A

Tetanus

.Also known as tonic spasm, tetany and lock jaw
.Caused by the toxin of the clostridium tetani bacteria
-Attacks the horse’s CNS, pulling the tendons, ligaments and muscles taut
-Highly fatal infectious disease
.Incubation period: 7-21 days
.Prevention is better than cure!
-Poor prognosis unless diagnosed and treated aggressively early on. Highly fatal
.Not contagious

From week 4 lecture

112
Q

Tetanus: Symptoms

A

Tetanus: Symptoms

.Protrusion of the 3rd eyelid
.Stiff gait/stance
.Elevated tail
.Shaking
.Muscle spasms
.Difficulty swallowing (dysphagia)
.Sweating

From week 4 lecture

113
Q

Colic: Symptoms

A

Colic: Symptoms

.Sweating
.Lack of gut noises
.Abnormal behaviours:
-Kicking or biting at the stomach
-Lying down or rolling repeatedly
-Uncomfortable, reluctance to eat
.Reduced or no passing of droppings

From week 4 lecture

114
Q

Colic

A

Colic

.Abdominal pain which may range from mild to life-threatening.
-falls into various categories, depending on the specific underlying cause.

.Different Types of Colic

-Impaction - Caused by a blockage in the intestine- could be from worms. No gut sounds or reduced.

-Spasmodic - Characterised by increased intestinal contractions.

-Tympanic (gaseous) - A build-up of gas in the intestine. From eating sugary foods.

-Sand - Inflammation or blockage of the intestine resulting from ingested sand.

From week 4 lecture

115
Q

REACT to Colic

A

REACT to Colic

.BHS scheme run in conjunction with the University of Nottingham below

R Restless or agitated
E Eating less or dropping reduced
A Abdominal pain
C Clinical changes
T Tired or lethargic

.Vet attention needed

From week 4 lecture

116
Q

Laminitis

A

Laminitis

.Systemic condition that manifests in the foot

.Can be sub-divided into 3 main categories:
-Endocrinopathic
-Sepsis-related
-Supporting limb laminitis/mechanical overload

.Prevalence
- 1 in every 10 horses/ponies developing at least one bout of laminitis each year* (Pollard et al., 2018)
- >50% of cases waiting >10 days to seek veterinary intervention – why?

.High recurrence

From week 4 lecture

117
Q

Laminitis: Symptoms

A

Laminitis: Symptoms

.General signs of pain
.The feet may be hot
.Increased digital pulse
.Soreness in the feet/shifting of weight/laying down/unwillingness to move/lameness
.Simple sugar difficulty digesting and causes build up of toxins in blood and reduced blood supply in hoof causes lanimae to detatch from hoof wall

Most prevalent owner- reported signs:
Difficulty turning
Short/stilted or lame walk

From week 4 lecture

118
Q

Laminitis: Action Required

A

Laminitis: Action Required

.Call your vet immediately
-Serious changes associated with the disease occur within the first 72 hours.

• Any potential cause should be removede.g. high sugar food

• Chronic cases - mild attacks that come back frequently. Have a management plan

From week 4 lecture

119
Q

Choke

A

Choke

.Obstruction in the oesophagus
.Characterised by:
-Feed material coming out of the horse’s nose
-Discomfort and signs of distress
-Coughing, gaging or straining when trying to swallow

.Action required - stop horse from eating, massage area

From week 4 lecture

120
Q

Exertional Rhabdomyolysis Syndrome (ERS)

A

Exertional Rhabdomyolysis Syndrome (ERS)

Also known as:
-Azoturia
-Monday morning disease
-Tying-up
-Paralytic myoglobinuria
-Setfast

A description of a horse with muscle damage that could have numerous causes.

From week 4 lecture

121
Q

Exertional Rhabdomyolysis Syndrome (ERS) symptoms

A

Exertional Rhabdomyolysis Syndrome (ERS) symptoms

.Stiffness
.Sweating
.Reluctance to move
.Posture as if to urinate?
.Elevated pulse 100bmp at rest and respiration

Actions required
.Stop exercising, do not force the horse to walk
.Call the vet
.Rug the horse in cold weather (keep warm)
.Provide access to water

From week 4 lecture

122
Q

Endoparasites

A

Endoparasites

.A parasite that lives inside its host like a tapeworm

From week 5 lecture

123
Q

Strongyloides westeri Taxonomy (Threadworm)

A

Strongyloides westeri Taxonomy (Threadworm)

.Phylum: Nematoda.
.Family: Strongyloididae
.Superfamily: Rhabditoidea
.Genus: Strongyloides
.Species: westeri
.Common Name: Threadworm
.Size: <1cm x 1mm
.Importance: low
.Intestinal threadworm commonly infecting young foals
.Associated with intestinal enteritis
.The pre-patent period is 8-15 days
.Eggs have been found in the faeces of foals within 2 weeks of
experimental infection.
.Pathogenesis unclear – however unique amongst helminths
as 3 possible transmission routes:
-Lactogenic – most important for foals
-Oral
-Percutaneous

From week 5 lecture

124
Q

Strongyloides westeri Taxonomy (Threadworm) Epidemiology

A

Strongyloides westeri Taxonomy (Threadworm) Epidemiology

.Direct lifecycle
-no intermediate host

.Few large scale prevalence studies exist
-Typically found in faeces on 5-50% on managed foals < 2 months of age

.Mares harbour larvae in hypobiotic stage in tissues which are reactivated during parturition
-Migrate to mammary gland

.Larvae can be isolated in milk at 5 days post partum
-Highest concentration 10-14 days
-Last seen c. 45 days
-Can be identified in foals as young as 5 days

From week 5 lecture

125
Q

Strongyloides westeri Taxonomy (Threadworm) Lifecycle

A

Strongyloides westeri Taxonomy (Threadworm) Lifecycle

.Infection common in foals 2 weeks to 4 months
.Animals immune generally immune after 6 months
.Severe infection may cause diarrhoea
.Infected by larvae in the mare’s milk
.In the gut they migrate to lungs and then coughed up,
swallowed and on reaching the intestines mature into adults
.Easily controlled by most wormers

From week 5 lecture

126
Q

Strongyloides westeri Taxonomy (Threadworm) Pathogenesis

A

Strongyloides westeri Taxonomy (Threadworm) Pathogenesis

.Foals generally infected via dam’s milk, but percutaneous infection can also occur
→ threadworms in small intestine → diarrhoea

.Reservoir of larvae in mare’s tissue release larvae triggered to migrate to the mammary glands and thus pass to the foal in milk

.Alternatively, worms can penetrate the skin causing local erythema gaining access to the body before migrating to the intestine

.In the foal they develop into mature threadworms in the small intestine (where only the female worms are parasitic)

From week 5 lecture

127
Q

Strongyloides westeri Taxonomy (Threadworm) Transmission

A

Strongyloides westeri Taxonomy (Threadworm) Transmission

Here they cause catarrhal lesions with oedema and erosions of the mucosal epithelium
→ impairment of digestion and absorption → diarrhoea

Eggs are passed in faeces

One or several free-living generations may occur before a third stage infective larvae is produced

The pathology is due to worms causing inflammation of the anterior third of the SI with villous atrophy and increased numbers of lymphocytes in the lamina propria

From week 5 lecture

128
Q

Oxyuris equi Taxonomy (Pinworm)

A

Oxyuris equi Taxonomy (Pinworm)

Phylum: Nematoda.
Superfamily: Oxyuroidea.
Family: Oxyuridae
Genus: Oxyuris
Species: equi
Common Name: Pinworm
Size: ~1-6cm (thin pointed tail denouncing name)
Importance: Low

From week 5 lecture

129
Q

Oxyuris equi (Pinworm) Transmission

A

Oxyuris equi (Pinworm) Transmission

By ingestion of infective eggs off contaminated fomites

Eggs remain stuck to the perineal area for several days before dropping or being rubbed off onto fomites or bedding

Eggs are very susceptible to dessication and so those outdoors usually die

Heavy infections can occur in stabled horses where eggs are at high concentrations in damp areas such as mangers or bedding

Causes terrible itching and subsequent hair loss under, on and around the tail

From week 5 lecture

130
Q

Oxyuris equi (Pinworm) Pathogenesis

A

Oxyuris equi (Pinworm) Pathogenesis

Adult worms cause pruritus during egg-laying

Adult worms crawl out of anus to lay worms on the skin around the tail

Horses rub tail area causing damage to hairs of tail head, ‘rat-tailed’ appearance

L4 larvae may cause inflammation of intestinal mucosa as erosions caused by feeding (rare)
- Otherwise not a typical intestinal parasite

Local irritation around anus can be controlled by sponging anal area to remove eggs

From week 5 lecture

131
Q

Oxyuris equi (Pineworm) Management

A

Oxyuris equi (Pinworm) Management

.Long lifecycle – c. 5 months
- Immature stages less sensitive to wormers and may survive
treatment

Sellotape Test:
1.Collect the sample in the morning, ideally before 9am. Pinworm are most active at night so this will give the best chance of detecting any activity that is present.
2.Take a 4” length of clear sticky tape and press this firmly onto the skin around the anus (not the hair).
3.Fold the tape in half, sticky side to sticky side.
4.Look for eggs under the microscope

.Ensure fomites are regularly cleaned and disinfected
.Good “under tail” hygiene important

From week 5 lecture

132
Q

Habronema spp. Taxonomy (stomach worm)

A

Habronema spp. Taxonomy (stomach worm)

Phylum: Nematoda
Class: Secernetea
Order: Spidurida
Family: Habronematidae
Genus: Habronema
Common Name: Stomach worm
Size: 6-25mm dep on spp.
Importance: Low (rare)

From week 5 lecture

132
Q

Habronema spp. Taxonomy (stomach worm)

A

Habronema spp. Taxonomy (stomach worm)

Phylum: Nematoda
Class: Secernetea
Order: Spidurida
Family: Habronematidae
Genus: Habronema
Common Name: Stomach worm
Size: 6-25mm dep on spp.
Importance: Low (rare)

From week 5 lecture

133
Q

Habronema spp. (stomach worm) Epidemiology

A

Habronema spp. (stomach worm) Epidemiology

.Habitat
-Uses muscidae fly as an intermediate host
-Worldwide distribution but infection prevalence increases with warmer climates.
-Cutaneous lesions “summer sores” often found in spring and summer due to increased activity of the intermediate hosts

.Transmission
-Adult flies transmit larvae from faeces to horses.

From week 5 lecture

134
Q

Habronema spp. (stomach worm) Lifecycle

A

Habronema spp. (stomach worm) Lifecycle

Eggs/L1 are passed in faeces from infected horses.

L1 are ingested by larval stages of intermediate hosts (house and stable flies)

L1 develop to L3 in synchrony with intermediate hosts development to maturity

When the adult flies feed around the muzzle of horses the L3 larvae pass from its mouthparts and are swallowed

L3 larvae burrow into the mucosa of the horses stomach forming nodules

L3 larvae develop to mature adults after around 8 weeks in the nodules and produce eggs

From week 5 lecture

135
Q

Habronema spp. (stomach worm) Pathogenesis

A

Habronema spp. (stomach worm) Pathogenesis

Adult helminths in stomach cause a (usually) sub-clinical gastritis

Larvae deposited on skin, open wounds or chronically wet areas on the horse can cause pruritic granulomous lesions

Occular granulomous lesions cause discharge and chronic conjunctivitis

Larvae deposited around male genitalia may cause oedema around the lesions, with occasionally massive lesions developing on male genitalia (Penis Habronemiasis)

Lesions in lungs due to migrating larvae have also been reported.

From week 5 lecture

136
Q

Dictyocaulus arnfieldi Taxonomy (Lung worm)

A

Dictyocaulus arnfieldi Taxonomy (Lung worm)

Phylum: Nematoda
Superfamily: Trichostrongyloidea
Family: Dictyocaulidae
Genus: Dictyocaulus
Species:
Common Name: Lung worm
Size: 6.5cm
Importance: Low (rare – in horses sharing grazing with donkeys only)

Lifecycle similar to that of the cattle lungworm

Difficult to diagnose but usually associated with donkeys grazing with horses
Donkey is a symptomless carrier

Causes coughing and poor condition in young horses

Chronic resp. diseases in older horses

Easily controlled

From week 5 lecture

137
Q

Cestodes

A

Cestodes

.Flatworms
.In the horses – tapeworms
.Specifically Anoplocephala perfoliata

From week 5 lecture

138
Q

Anoplocephala perfoliata Taxonomy (tapeworm)

A

Anoplocephala perfoliata Taxonomy (tapeworm)

Class: Cestoda
Family: Anoplocephalidae
Genus: Anoplocephala
Species: perfoliata
Common Name: Tapeworm
Size: 40-80cm dep on segments
Importance: Medium

3 main species affecting the horse
-Anoplocephala perfoliata
-Anoplocephala mamillana
-Anoplocephala magna

Two latter species are rare in managed horses

All responsible for ileal and intussusception colics

Clinical signs vary depending on severity of lesions

From week 5 lecture

139
Q

Anoplocephala perfoliata (tapeworm)

A

Anoplocephala perfoliata (tapeworm)

.The most important tapeworm in the UK

.Lives in the SI and Caecum
- ICJ main attachment point

.Only species with documented clinical impact

.Adults up to 40-80cm in length by 1.2cm wide

.Eggs irregularly spherical or triangular

.Cannot be detected by FEC

Intermediate host of orbatid mite

Life cycle: mature segment are
passed in the faeces, releasing eggs
- Ingested by orbatid (forage) mites in which the develop the cysticeroid stage in 2-4 months

Adult tapeworms are found in the intestine 1-2 months after the ingestion of infected mites

Horses of all ages affected, but mainly reported in animals up to 3-4 years

From week 5 lecture

140
Q

Anoplocephala perfoliata (tapeworm) Lifecycle

A

Anoplocephala perfoliata (tapeworm) Lifecycle

Adult tapeworms in equine intestine → mature segments passed in faeces → disintegrate to release eggs → eggs ingested by forage mites → develop into cysticercoids in 2-4 months in mites → infected mites ingested by horses → adult tapeworms develop over 6-10 weeks.

Numbers of mature worms increase in autumn with egg-laying adults most common in winter through spring.

A. perfoliata lives about 6 months.

Habitat
-Oribatid mites (intermediate hosts) on pasture harbour cysticercoids.
-Maximum numbers of infected mites in late summer/autumn

Prepatent period
6-10 weeks

Transmission
Faeco-oral via intermediate host (forage mite).

From week 5 lecture

141
Q

Gastrophilus spp. Taxonomy (bot fly)

A

Gastrophilus spp. Taxonomy (bot fly)

Phylum: Arthropoda
Class: Insecta
Family: Oestridae
Genus: Gastophilus
Species: intestinalis (for horses)
Common Name: Bot fly
Size: L3 ½ - ¾ inches
Importance: Low - (non-pathogenic, nuisance)

Obligate parasites of horses

Robust dark flies, 10–15 mm in length

The body is densely covered with yellowish hairs

In female the ovipositor is strong and protuberant

Wings have no characteristic cross-vein

From week 5 lecture

142
Q

Gastrophilus spp. (bot fly) Pathogenesis

A

Gastrophilus spp. (bot fly) Pathogenesis

Bot flies lay batches of150-200 eggs around lips, legs

The adult flies have a short lifespan and females can deposit all of their eggs within 2–3 hours if the weather is mild and a suitable host is available

The eggs are easily seen; they are 1–2 mm long and usually white/ yellow in colour

They either hatch spontaneously in about 5 days or are stimulated to do so by warmth, which may be generated during licking and self-grooming

Larvae stay and develop in stomach for 10–12 months
When mature in following spring or early summer, they detach and are passed in faeces

The larvae reattach in the rectum for a few days before being passed out

Pupation takes place on the ground, and after 1–2 months the adult flies emerge

They do not feed, and live for only a few days or weeks, where they mate and lay eggs

If suitable hosts are unavailable the flies move to high points to aggregate and mate, following which females initiate a longer-distance search for hosts

There is therefore only one generation of flies per year in temperate areas

From week 5 lecture

143
Q

Gastrophilus – Bot Fly signs and treatment

A

Gastrophilus – Bot Fly signs and treatment

Cause irritation to the gut and colic unless treated

Frost kills the bot fly so life cycle broken and no chance of re-infection until the next season

Noticeable in the form of white/yellow eggs on the horse – typically on the legs or shoulder

Removal using a bot knife

From week 5 lecture

144
Q

Gastrophilus (bot fly) Epidemiolgy and Transmission

A

Gastrophilus (bot fly) Epidemiolgy and Transmission

Lifecycle
.Adults
-Eggs laid on horse’s hair.

.Larvae:
-1st, 2nd and 3rd stage.
-Pupate on ground

Transmission
-Adult fly on wing in the summer months → lays eggs on
individual horses.
-L1-L3 spend 9 months in the horse.
-Pupa lasts about 6 weeks on the ground

From week 5 lecture

145
Q

Equipment needed to set up an effective quarantine area

A

Equipment needed to set up an effective quarantine area

.Treatment carts;
.Painters’ disposable coveralls;
.Disposable gloves;
.Rubber boots;
.Foot bath containers;
.Garbage bags;
.Garbage cans with secure lids;
.Disposable plastic shoe covers;
.Dedicated thermometer for each horse;
.Dedicated equipment for each horse; and
.Appropriate signage.

Anyone entering the isolation barn should follow appropriate sanitation measures, including:

.Wearing rubber boots dipped in a prescribed foot bath and/or disposable booties.
.Using disposable or dedicated gowns or coveralls for each horse stall.
.Changing disposable gloves between handling each horse.
.Wearing a treatment coat over the reusable coveralls.
.Showering or changing clothes before touching other horses if contaminated while treating an infected horse.
.Washing hands for 60 seconds (or singing “Happy Birthday” twice at normal tempo) before entering or leaving the isolation area. Using disposable towels and depositing them in a covered waste container at the hand-washing site.
.Setting up a perimeter around the stall area to limit entry and traffic. You can designate this perimeter with ropes, construction fencing, and so forth. Restrict random access to this area, and provide only one entry and exit.
.Installing appropriate lighting for easier assessment and treatment.

From
https://thehorse.com/112775/how-to-set-up-a-disease-isolation-unit-on-your-farm/

146
Q

Guidance on isolation

A

Guidance on isolation

Premises
1. The isolation facility should be a separate, enclosed building of sound, permanent construction, capable of being cleansed and disinfected effectively.
2. It must not be possible for other horses to approach within 100 metres of the isolation facility while it is in use.
3. An adequate supply of fresh, clean water must be available at all times for the isolated horses and for cleaning purposes.
4. Adequate supplies of food and bedding material for the whole of the isolation period must be made available and stored within the isolation facility before isolation commences.
5. Equipment and utensils used for feeding, grooming and cleansing must be used only in the isolation facility.
6. Protective clothing must be available at the entrance to the isolation facility and not be taken outside of this facility.
7. A separate muck heap should be used within the isolation facility.

Procedures
1. Before use, all fixed and moveable equipment and utensils for feeding, grooming and cleansing within the isolation facility must be disinfected using an approved disinfectant. A list of these is provided on the Defra website (select only ‘General’ for suitable products).
2. Attendants of the isolated horses must have no contact with any other horses during the isolation period.
3. The isolation period for all isolated horses shall be deemed to start from the time of entry of the last horse.
4. No person may enter the isolation facility unless specifically authorised to do so.
5. When no attendants are on duty, the facility must be locked securely to prevent the entry of unauthorised persons. If such strict measures are not possible in practice, the owner/manager of the premises where isolation is needed should devise their own isolation programme and procedures in conjunction with the attending veterinary surgeon and if appropriate with additional input from a recognised expert in equine infectious disease control.

From
https://codes.hblb.org.uk/index.php/page/130

147
Q

Pre-Purchase Exams:

A

Pre-Purchase Exams:

The five stages consist of:
 Stage 1: Clinical examination of the horse at rest. This is includes a thorough palpation of all the limbs, listening to the heart and lungs and an examination of the horse’s eyes in a dark stable with an ophthalmoscope.

 Stage 2: Observation of the horse walking and trotting in hand in a straight line on a hard, level surface, including flexion tests. The horse is normally also trotted on a small diameter
circle on a firm surface to detect subtle lameness issues.

 Stage 3: Observation of the horse performing strenuous exercise, normally under saddle. This allows further evaluation of the way the horse moves and assessment of the heart and
respiratory systems’ response to exertion.

 Stage 4: A period of rest in the stable to give time for any stiffness induced by the exercise to become apparent. The horse’s markings are normally recorded at this time and the
passport examined.

 Stage 5: A second examination of the horse at trot in hand. This is primarily to check that the strenuous exercise has not exacerbated a subtle underlying lameness problem. Flexion tests or trotting on a small diameter circle are sometimes repeated at this time. A blood sample will be taken which is stored for six months and can be used, if concerns arise after
purchase, to test for substances that may have masked certain conditions.

From
https://moodle.writtle.ac.uk/pluginfile.php/1190965/mod_resource/content/0/Pre-Purchase%20Examinations.pdf

148
Q
A

What does a lameness ‘work-up’ entail?

 We will allocate a vet to look at your horse/pony. He/she will take some history from you regarding your horse/pony’s background, such as:
o What your horse/pony is used for
o How he/she is managed
o Shoeing etc

 They will also ask questions about the nature of the current lameness:
o Duration
o Severity
o Previous treatment
o Response to rest/treatment etc

 We will then perform an initial lameness examination; this may include:
o Walk and trot in a straight line
o Flexion tests
o Lunging on both firm and soft surface
o Palpation of the limbs/back/neck
o Possibly a ridden examination, dependent on the history and nature of
the problem

 If, following this examination, there is something that suggests a cause of the lameness we may elect to perform the relevant diagnostic imaging procedure straight away
o X-rays and/or ultrasound examination

 If there is no obvious cause of lameness, then we commonly embark on performing a series of ‘nerve blocks’:
o We will place small volumes of local anaesthetic, using small needles, in specific places on the leg to numb the nerve supply to a specific area.
o If your horse/pony is sound after this nerve block then we know that the cause of lameness is coming from somewhere within the numbed region.
o If your horse/pony remains lame then we will perform the next nerve block up the leg, as we generally start at the bottom of the leg and work our way up.

From
https://moodle.writtle.ac.uk/pluginfile.php/1190966/mod_resource/content/0/Lameness_work-up_info_sheet.pdf

149
Q

Equine de-worming: a consensus on current best practice

A

Equine de-worming: a consensus on current best practice

.Despite increasing awareness within the veterinary profession and equine industry of the potential implications of anthelmintic resistance (AHR), there is a concern that insufficient measures are being taken to reduce its development and spread. This document was commissioned to provide veterinary surgeons with up to date information on worm control plans that will prevent clinical disease while minimising selection pressure for resistance.

From
https://www.magonlinelibrary.com/doi/abs/10.12968/ukve.2019.3.S.3?journalCode=ukve

150
Q

Worm testing in autumn

A

Worm testing in autumn

.Testing for tapeworm prior to the annual treatment for encysted redworm in late autumn/winter is an easy win. This will help to decide whether a moxidectin only (or where there is no resistance known, a 5-day fenbendazole) treatment is required or if a moxidectin + praziquantel combination wormer is advised to include treatment for tapeworm at the same time.

.At other times of year where it has been confirmed that treatment for tapeworm is needed, a worm egg count should be conducted. If treatment for redworm is indicated at the same time, then an ivermectin + praziquantel combination wormer should be the first choice.

.Alternatively, a double dose of pyrantel can be recommended for administration if;
- the treatment is for tapeworm only or
ascarids are also present (more likely in foals or yearlings) or
the redworm population in question is known to not be resistant to pyrantel.

.As a note of caution, in studies conducted by at Moredun Research Institute, small redworm resistance to pyrantel was found on up to 50% of yards, depending on the group of establishments tested.

From
http://equisal.co.uk/Worming-advice-after-discontinuation-of-Equitape

151
Q

FEC

A

FEC

.Fecal egg counts (FEC) will help you check the efficacy of your dewormers, determine which of your adult horses shed the most strongyle eggs, and monitor ascarid presence in foals and yearlings.

.To conduct an FEC, collect a fresh manure sample from each horse to be tested. About three “nuggets” of manure will suffice. Label, date, and refrigerate the samples. Ideally, have them tested by a veterinarian or an animal health laboratory within 24 hours and no later than seven days after collection. Seek advice from your veterinarian about collecting samples and where to send them.

.The FEC results enable you to categorize adult horses as low (0-200 eggs per gram [EPG]), medium (200-500 EPG), or heavy (>500 EPG) strongyle shedders.

.By identifying the horses that shed the most strongyle eggs in their feces and treating them with effective dewormers, you can effectively reduce strongyle egg-shedding in your pasture.

From
https://thehorse.com/169288/risk-and-reality-horse-parasite-control-and-anthelmintic-resistance/?utm_medium=Health+enews&utm_source=Newsletter

152
Q

Fecals Don’t Show Everything

A

Fecals Don’t Show Everything

.Fecal egg counts do not distinguish large strongyle (the most dangerous but least common parasite in horses) eggs from small strongyle eggs. Equine parasitologists recognize this as one of the limitations of FECs. Other constraints the American Association of Equine Practitioners (AAEP) lists include:

.They do not accurately reflect the horse’s total adult strongyle or ascarid burden; a higher egg count just means more eggs, not necessarily more worms;

.They do not detect immature or larval stages of parasites, including migrating large strongyles and ascarids, and/or encysted cyathostomins (small strongyles embedded as cysts in the large intestine wall);

.Standard fecal techniques often underestimate tapeworm infections, so a modified FEC, as well as serum and saliva tests, exist for detecting these parasites; and
They usually miss pinworm eggs, which adhere in packets around the anus rather than shedding in feces.

From
https://thehorse.com/169288/risk-and-reality-horse-parasite-control-and-anthelmintic-resistance/?utm_medium=Health+enews&utm_source=Newsletter

153
Q

MUST read for exam

A

READ Rendal 2019 worm resistance

154
Q

Why control parasites?

A

Why control parasites?

Damage to the intestines

Pass infection on to other horses

Colic (spasmodic, impaction, infarction, intusseption)

Main method of control:
Anthelmintics
Management

155
Q

Role of an SQP (suitably qualified person)

A

Role of an SQP (suitably qualified person)

POM-V: Prescription only medicine (Vet only)

POM-VPS: Prescription only medicine (Vet, Pharmacist or SQP)

NFA-VPS: Non-food producing animal (Vet Pharmacist or SQP)

AVM-GSL: Authorised Vet. Medicine General Sales List

156
Q

Anthelmintics

A

Anthelmintics

Have a broad spectrum of activity
.Showing acceptable efficacy against four target parasites (small/large stongyles, Parascaris, Oxyuris)

Be efficient against all parasitic stages of a particular spp.

Be non-toxic to the host

Be rapidly cleared and excreted by the host

Have an easy administration

Be reasonably priced

https://www.noahcompendium.co.uk/ - to look up drug info

157
Q

Wormers in uk

A

Wormers in uk

.Benzimidazole - panacur

.Moxidectin - equest - resistance and no other option for
redworm.

.Ivermectin - eqvalan

.Pyrantel - strongid-p - double dose treats tapeworm

.Praziquantel - for tapeworm and at vet only

Combination wormers
.Equimax - ivermectin and praziquantel
.Equest pramox - moxidectin and praziquantel

158
Q

Wormer Dose rate

A

Wormer Dose rate

If the dose is sub-optimal to that recommended on data sheets:

.Selection of resistant strains will be increased
.Therefore animal must be weighed (scales or weigh tape)
.Calibration of WE equipment should be carried out regularly

DO NOT UNDERDOSE
Over dosing safer

159
Q

Worm control

A

Worm control

.Gastro-intestinal nematodes of the horse controlled by dosing every 6-13 weeks depending on the drug used

.Different wormers have different length of action

.Spring – Autumn is the high risk period

.Nemotodes - okay in winter
.Bot fly - not okay in winter

.Winter dosing only required every 3 months (depending on weather patterns)

.If larval cyathostomiasis is a problem then treatment is important at the beginning of the winter period

160
Q

Benzimadazoles

A

Benzimadazoles

.Introduced to market in 1960s – revolutionised industry

.Expected FECR >90% - egg reappearance : 6-8 weeks (Rendle et al., (2019)

.Efficacy not considered effective unless FECRT undertaken – resistance ubiquitous

.BZD - large chemical family use to treat nematode infection in domestic animals

.Categorised by broad spectrum of activity

.For young or older horses normally as less aggressive

Mode of activity
.Disrupt energy metabolism at a chemical level
-Bind to protein tubelin and block polymerisation into microtubules
-Microtubules essential structural components of cellular structure – essential to energy metabolisation

.BZDs only marketed anthelmintic class that has anti-metabolic mode of activity

.Nematodes have limited energy storage organs so must consume continually
- BZDs make the nematode effectively starve

161
Q

Fenbendazole

A

Fenbendazole

.Used for foals up to 6 months

.FBZ firstly broad spectrum, equine anthelmintic
-Given at an elevated dosage of 10mg/KG shows high efficacy against migrating ascarid and large strongyle larvae
-Also effective against encysted cyathostomins

.Kaplan et al., (2004) found 95% of farms had indigenous populations of BZD resistant cyathostomins

.Resistance in these parasites taken as a rule rather than exception on managed horses

162
Q

Pyrantel embonate

A

Pyrantel embonate

Cause rapid, spastic paralysis of nematodes

Paralysed worms unable to conduct co-ordinated feeding and would starve if not excreted via intestinal peristalsis

Not registered for 4L (immature worms) cyathostomes as have shown to survive treatment (Reinemeyer, 2003)

No activity again encysted cyathostomes, migrating strongyles

Non-ovicidal - doesn’t kill the worms

6 week dosing

Broad spectrum with efficacy against large redworm @ 6.6mg/kg
-Variable against S. edentatus, Oxyuris and cyathostomes

Some resistance noted, not as widespread as BZD
-Including adult cyathostomes (Craven et al., (1998)

Also effective for A. perfoliata @ double dose rate of 13.2mg/kg – 95%

163
Q

Macrocylic lactones

A

Macrocylic lactones

.less resistance

Two distinct families: Both kill internal and external e.g. lice and worms
-Avermectins: Ivermectin - doesn’t kill encysted stage
-Milbemycin: Moxidectin - does kill encysted stage

.Mac lacs interfere with glutamate gated (GluCl) channels resulting in flaccid paralysis of the parasites
-Arrested larvae cannot feed and are expelled with intestinal peristalsis

.Migrating stages are killed by a cellular immune response

.Both endoctocides
-killing both ecto & endoparasites (both nematodes and insects).

.Onset of activity is rapid
-Typically occurring during 48hrs of treatment
-Ideally keep inside for 48 hours after treatment

.Kill all luminal stages of dvt

.Effective against migratory stages (S. vulgaris, Parasacris and Strongyloides)

.Efficacy against Gasterophilus spp. attached to stomach and duodenum
-Ivermectin also licensed for oral stages

.Also effective against Habronema and Draschia

164
Q

Ivermectin

A

Ivermectin

.Ivermectin came onto market in 1980s
-Broad in action and comprises both antibiotic and antiparasitic modes
-Mac. Lacs. revolutionised parasite control in horses
-low does rate
-low toxicity level
-8 week activity
-No resistance reported in first 20 years on the market – now more widespread - esp. in relation to Cyathostomes and Ascarids in all recent studies

165
Q

Moxidectin

A

Moxidectin/ equest

.More lipophilic (fat soluble) than ivermectin - don’t treat under 6 months as not much fat on foal

.Metabolised in fat stores of the horse and released gradually over 13 weeks
-Careful with underweight horses

.No efficacy against tapeworm

.Resistance now a growing concern

166
Q

vermectin / Moxidectin share similar properties
Main differences:

A

vermectin / Moxidectin share similar properties
Main differences:

Moxidectin
-is effective against encysted cyathostomes
-Efficacy ranging between 60-80% dep. on study
-Licensed for larvicidal treatment
-Supresses strongyle counts for 12-16 weeks post-treatment (Jacobs et al., 2015) (hence 13 week dosing schedule)
-Longest ERP (egg reappearance period) of any drug, c. 1.5x ivermectin

167
Q

redworm

A

redworm

.90% of all small redworm found in the horse will be encysted larvae.
.Equest only one to treat all stages of small redworm in one dose.
.Lower toxicity to dung dwelling insects than ivermectin based wormers. e.g. fly eating dung and getting wormer

168
Q

Praziquantel

A

Praziquantel

.Very effective anthelmintic

.No known resistance on efficacy in A. perfoliata

.Licenced for decades in companion animal medicine
More recently for horses

Mode of action
-Damages larval tegument (outer body covering) causing cell membrane permeability
-Results in spastic paralysis

169
Q

Combination wormers

A

Combination wormers

.Both moxidectin and ivermectin can be combined with praziquantel in the form of a combi wormer

Wormers
.Equest pramox
.Equimax

170
Q

Administration of medicines

A

Administration of medicines

Syringe
-Dosing syringe
-Cleaned wormers
.Mix powders with water to form paste
.Place in corner of mouth over tongue
.Raise muzzle
.Lips closed

In Feed
.Powder mixed in with dampened hard feed
.Soaked sugar beet or molasses
-Binds powder
-Masks taste
.Some horses will not eat powders =>
.Alternative dosing method

171
Q

What is Parasitic Resistance

A

What is Parasitic Resistance

.Anthelmintic Resistance is the term given to parasites evolved to be no longer affected by the chosen worming treatment
-Similar seen in antibiotic treatment
-Lack of modern options
-Egg reappearance times reduce

.Slowing resistance 2-fold:
1. Protection of Key Medicines
2. Reducing worm challenges in the environment
(Management)

172
Q

SMART Worming Programmes

A

SMART Worming Programmes

S imply
M onitor &
A ssess the
R isk &
T reat appropriately

173
Q

NEED to know for exam!!
10 Management Tips for Preventing Resistance

A

10 Management Tips for Preventing Resistance

1) Protect Key Medicines:
Utilise monitoring (FEC / Equisal). Resistance stems from exposure to meds.

2) Avoid underdosing
Exposing worms to not enough chemical will increase resistance. Use WB. All UK wormers have safe upper tolerance levels

3)Don’t worm and move
After worming, don’t change horse to new pasture. Stable for 48 hours after treatment

4)Maintain grazing companions
This will stop your horse ingesting parasites that may have been exposed to chemical and developed resistance

5) Poo Pick
Shed larvae hatch within 7 days and can migrate in pasture. Harrowing only effective over 40◦C – UK?

6) Muck Heap Placement
Worms will travel up to 1m in dry conditions. Correct placement can aid prevention of re-infestation

7)Cross Grazing
Utilising other animals (e.g. sheep) can help reduce worm burden in grazing. Helminths usually species specific (except some tapeworms, lungworm and liver flukes)

8) Isolate new horses
Keep new horses separate until FEC / Equi-Sal monitoring shows clear from parasite burden

9) Careful grazing away from home
Consider preserved forage instead. Competition venues have high risk of parasite burden. Horses stabled with travelling companions at ^ risk of developing infestations

10) Good Stable Management is Key
Keeps stables and shelters clear from faeces. Regularly empty and clean troughs and buckets to prevent re-ingestion

174
Q

ID of resistant worms

A

Identity of resistant worms

.Commercial resistance testing now available (c. £8) in form of FERT

.Ability to see if recently given treatment has been effective
-Especially important if using a Fenbendazole based wormer (Panacur Equine Guard) as high reported resistance

.FEC taken to assess parasite burden in the horse
-Treatment given if necessary
-10-14 days later another faecal sample taken and analysed

175
Q

ID of resistant worms

A

Identity of resistant worms

.If result not reduced by 85-95% then resistance is noted
- If identified, speak to vet or SQP for alternative treatment strategies.

.E.g. medium worm egg count between 200-1150e.p.g. should reduce to < 50 e.p.g.

.A high worm egg count of 1200 e.p.g. should reduce to < 100 e.p.g. on a follow up test

.Widespread resistance noted with Fenbendazole and Pyrantel Embonate
.Some resistance noted with Ivermectin based products
.Some new resistance noted with Moxidectin on parascaris equorum and cyathostomins
.No known resistance to Praziquantel