Endodontics Flashcards

1
Q

What is the root canal procedure steps?

A

Prerprocedural checks

Chief concern and history taking

Taking the PA

Consultation with the patient and tutor

EPI calculation

Procedural

Step 1: Initial acess

Apply anaesthetic and isolate the appropriate tooth using single clamp

Using a high speed, end cutting bur such as 838 to gain initial access to the pulp chamber – always refer back to the PA

Switch to the non-end cutting endo-z bur in order to expand the access cavity

Use endoprobe to locate the canal

Pre-curve size 10 file and insert it a few mm into the canal

Take a PA

Irrigate the canal with a pre-bent hypochlore 1%

Step 2: Expansion of the coronal aspect

Using size 2 and 3 gate glitten bur, brush it a few mm into the canal coronally to expand the access

Irrigate

Step 3: Working length determination

Determine the working length using electronic apex locator, attach the device, advance to 1mm away from apex and have a good refrence point, take a PA to confirm

When confirmed chart as correct working length

Irrigate

Step 4:

Pre-cruve files and set them to the correct working length

Starting with the smallest file (size 10) instrument using clokc winding technique

Irrigate

Ensure canal is very loose for the smallest file – move to file that is 1 size larger

Irrigate and recapitulate and irrigate again

Ensure canal is very loose for the size up file – move to the file that is 1 size larger

Continue until you reach file 30 – take a PA of master apical file at 25 to confirm

Step 5: Step back

Pre-cruve file 30 and 35, set to correct working length

Create a stepback of 1mm with file 30

Irrigate

Create a stepback of 2mm with file 35

Irrigate

Step 6: Medicaments

Use calcium hydroxide for non-symptomatic and odontopaste for symptomatic

Apply using a file or a lentulo spiral at low rpm

Step 7. Interim restoration

  1. Put a nice layer of cavit in the pulp chamber – use a small cotton pallet with water to engae it

Use RMGIC to restore – check occlusion and recall the patient in 4 weeks

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2
Q

Why is endodontics important?

A

1.Treatment of pain is the main factor in patient

2.Stopping the propagation of infections

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3
Q

How do you write a diagnostic statement in endodontics?

A

1.Pulp and root canal condition – necrotic pulp/irreversible pulpitis

2.Periapical status – chronic/acute apical periodontitis evident radiographically

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4
Q

What aspects should we consider for endodontic treatment?

A

1.Strategic value of the tooth

2.Periodontal factor – if the tooth is grade III mobility what si the point of endodontically treating it?

3.Patient factors – MHx, motivation, age, compliance with treatments

4.Restorability options – consider the entire mouth

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5
Q

What clinical test should you do during endo examination?

A

1.Percussion of tooth

2.Palpation around the soft tissue

3.Periodontal examination – cracks and fractures

4.Pulp sensibility tests – cold, hot or electric

5.Radiographs – PAs and BWs with or without use of GP with fistula

6.Special test – removal of restoration, selective LA, transillumination oe fracture finder on each cusp

7.Full data analysis and appropriate diagnosis presented to patient with options – two component diagnosis pulp+root canal AND periapical status – IT IS SEEN ON MY UNI REMMEBER IT

8.Patient need to be informed about the time, cost, step-by-step of each procedure, risks and need to consider a crown restoration

9.Ask the patient “is there anything you want to ask me, your opinion is essential for your own wellbeing”

10.Accurate record keeping

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6
Q

What is considered to be moderate diffuclty in the AAE classifications?

A

One or two of the following:
1. ASA class 3 patient
2. Vasoconstrictors intolerance
3. Anxiety
4. Limitation in opening
5. Gagging
6. Moderate pain or swelling
7. extensive differential diagnosis
8. Difficulty in obtaining radiographs
9. 1st molar
10. Moderate inclination - 10-30 degress
11. Soem trouble with rubber dam
12. Coronal distruction or complex restoration
13. Canal morphology is slightly more complex
14. Pulp stones
15. 3-5 mm near the IAN
16. Minimal apical resorption
17. Crown fracture
18. Previous access without complications
19. Endo-perio lesion

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7
Q

What is considered to be high diffuclty in the AAE classifications?

A

3 or more in moderate difficulty and at least one in the high diffuculty such as?
1. ASA 4
2. Can’t get anaesthesia
3. Uncooperative
4. Significant limitation in opening
5. Extreme gaggin
6. Sever pain
7. History of orofacial pain
8.2nd or 3rd molar
9. Extreme inclanation
10. Extreme rotation
11. Significant deviation from normal tooth/root form
12. C-shape morphology, extreme curvature or S-shape curve, rare root morphology, very long teeth
13. Pulp chaber not visible
14. extremly close to IAN (<3mm)
15. Extreme resopriton
14. Root fractures
15. Previous endo

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8
Q

What is the classification of cracked teeth according to severity from low to high?

A

1.Craze line – very common and only in enamel – use transillumination

2.Fracture cusp – usually from insufficient cusp support from deepest point of restorative material – sharp pain on biting usually on non-funcitonal cusps

3.Cracked tooth – centered, vertical crack that has pulpal symptoms and can be in none restored teeth – typically mandibular second mollars

4.Split tooth – progression of cracked tooth – hopeless prognosis

5.Veritcal root fracture – a longitudinally oriented fracture extending from the root canal to the periodontium – hopeless prognosis

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9
Q

What are pre-disposing factors from cracked teeth?

A

1.Occlusal factors – posterior buccal crossbites, anterior open bites, steep cusps

2.Parafunction

3.Age

4.Width and depth of cavity

5.Use of rotary instrumental – common in vertical root fractures

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10
Q

How do we diagnose coronal cracks?

A

1.Patient symptomatology

2.Vision enhancers

3.Symptom reproducers – Very important

4.Radiographs

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11
Q

What is the mechanisms of pain in cracked teeth?

A

Due to peripheral location and low excitability threshold of A-beta and A-delta fibers, rapid sharp pain is generated. In cracked teeth, the increased rapid movement of dentine fluid in dentinal tubules during relaxation after prolonged biting result in such symptom. When bacterial toxins infiltrate the pulp, hyperalgesia can result. This is a state of pulpal inflammation, which lowers the threshold of stimulation of A-delta fibers. Thus patient feels pain easier. A second type of pulpal stimulation is occurs through activation of C-fibers. C-fibers have a higher threshold of excitability thus only respond to prolonged inflammation of pulpal tissues. C-fibers activation means more than likely, pulp treatment is needed.

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12
Q

What is the treatment of cracked tooth + symptoms of reversible pulpitis?

A

1.Removal of restoration + invastigate crack + remove or reduce any cracked cusps + restore

2.Provisional restoration and review in 3 months to confirm diagnosis

3.Consider crown

4.Reduce caueses of the crack

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13
Q

What is the treatment of a cracked tooth + symptoms of irreversible pulpitis?

A

1.Endodontic access + investigate crack

2.Temporize with ortho band and proceed with RCT or extract

3.Consider causes of this and try to reduce them

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14
Q

What is the prognosis from poor-to-good for cracked teeth?

A

1.Poor – segment separation, crack crossing pulpal floor, periodontal pocketing at fracture line, damaging habits not amenable to change

2.Guarded – crack extends to the floor of the pulp chamber

3.Good – crack into roof of the pulp chamber, does not reach pulpal floor

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15
Q

What is a clinical presentation of a vertical root fracture?

A

1.Broad-based soft tissue swelling in the mid-root

2.Sinus tract bilaterally on 2 aspects

3.Depp pockets on 2 bilateraly surface (eg buccal-lingual)

4.J-shapped lesion/defect on the radiographs

5.Presents of isolated horizontal/angular bone loss

6.Visual confirmation through raising the flap

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16
Q

What are the 3 main avenues of communication between periodontium and the pulp?

A

1.Dentinal tubules – only through patent dentinal tubules due to loss of cementum

2.Lateral and accessory canals – seen in 30-40% of the teeth usually around the apical 1/3 of the tooth

3.Apical foramen – possibly the main avenue – acts as entry and exit

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17
Q

How does pulpal pathothis effect the periodontal ligament?

A

1.Pulpal pathosiis occur

2.The bi-products of pulpal pathosis are release in proximity to the preidontal ligament through accessory canals/apical foramen, eliciting an inflammatory response from PDL

3.Increase in vascularity and creation of temporary periodontal pocket – know as retrograde periodontitis

4.In addition – use of endodontic macterials or procedural errors could create moderate to sever periodontal consequences

18
Q

What is the classification of endo-perio lesions?

A

1.Primary endo – secondary perio – drainage through sulcus – treatment: root canal

2.Primary perio – secondary endo – treatment: non-surgical periodontal therapy, control of local factors, control of systemic facots and endo-treatment if needed

3.True combined lesion – primary endo treatment then perio then finish treatment with obturation, also remedicate canal if periodontal treatment is not finished

19
Q

What procedures comprise endodontic microsurgery?

A

1.Periapical curettage

2.Apicectomy

3.Retrograde endodontic treatment

20
Q

What are indication for periapical microsurgery?

A

1.Presen of disease after treamenet/re-treatment

2.Re-treatment not viable

3.Adjunct to re-treatment – re-treatment and surgery

4.Preservation of adequate coronal restoration

5.Costs

21
Q

What are contraindications of periapical microsurgery?

A

1.Re-treatment viable

2.Poor coronal restoration

3.Medical contraindication – radiotherapy or bisphosphonates

4.Anatomical contraindication – sinus or IAN

5.Other patient factors – fear factor

22
Q

What are two different types of cysts in endodontics?

A

1.A true cyst – a cyst that does not communicate with the root canal and will not heal following treatment by endodontics only – this can only be determined retrospectivley AND NOT RADIOGRAPHICALLY

2.A pocket cyst – a cyst with communication with the root canal and can be healed after endodontic treatment – this can only be determined retrospectively AND NOT RADIOGRAPHICALLY

23
Q

What are the steps of a microsurgery procedure?

A

1.Incision at the gingiva – submarginal or full thickness flap

2.Periosteal elevator to raise the flap

3.Retract the flap with retractor

4.Osteotomy using a round or flat fissure bur to create a window in bone to access the lesion

5.Endo-curette is use to remove the soft tissues of the lesion and resection is performed

6.Micro-mirrors are used to observe the resected root

7.Preparation of the root end with ultrasonic tip

8.Use haemostatic agent to control contamination of prepare root end tip

9.MTA is used with sterile water and inserted to the root end

10.Suturing is performed

24
Q

What intra-operative factors are associated to negative outcome in endo?

A

1.Iatrogenic perforation

2.Patency at apical terminus

3.Extrusion of root fillings

25
Q

What are some of the procedural erros that may occur in endo?

A

1.Loss of working length – BLOCKAGE – AMALGAM CAN FALL INSIDE

2.Ledging – larger the file, more likely it is to create a ledge because thick mettal bends less

3.Instrument separation (fracture) - common in rotary – always let the patient know and let the specialist know

4.Zipping/elbows - creation of triangle shape ledge at terminus of the canal

5.Stripping or lateral wall perforation – remember, the biggest bulge of dentine in the canal is on the corresponding site to the name I.e. buccal buldge is in the buccal canal

6.Shaping the canal beyond terminus

7.Excessive removal of root canal dentine – peri-cervical dentine is essential to prevent vertical root fractures

8.Failure to clean the canal properly – self explanatory, basically missed the goal

26
Q

How do we state perforations?

A

1.With or without communication with the oral cavity

2.Corona/apical

3.Size

4.Delay in treatment – need for repair

27
Q

What is the advantage nickle titanium files?

A

They are super elastic, minimising ledging, no zipping and no transportation.

28
Q

What is the disadvantage of nickle titanium files?

A

Sudden breakage, unlike SS files.

29
Q

What systems for rotary used in SADS?

A

ProTaper and K3

30
Q

How to avoid torsion fracture of file?

A

1.Continuous rotation – put the file while rotating – do not start the file if it is engaged

2.Pecking motion

3.Cown-down

4.Glide path

5.Do not increase in apical direction

31
Q

What is important to consider in endodontic and other emergencies?

A

A patient in pain wants immediate relief, the clinicians want to make a quick diagnosis. But do not lose systematic approach to patient case, think ahead.

32
Q

What are signs and symptoms of reversible pulpitis?

A

1.Pain is not spontaneous

2.Short duration

3.Localised

4.Sharp

5.Disappears immediately upon withdrawal of stimulus

6.Stimulated by: cld and occlusal interference

33
Q

What are the signs of symptomatic irreversible pulpitis?

A

1.Pain keeps patient at night and is lengthy

2.Localised

3.Aggravated by heat, spontaneous, when removed pain persists

4.Throbbing or dull pain

5.Analgesics do not word

34
Q

What is the treatment of odontogenic infection without severe or systemic factors?

A

1.Drainage of pus

2.Surgical or endodontic treatment

3.Oral antibiotics: Metronidozole 400mg, oraly 12hourly for 5 days + amoxicillin 500mg orally, 8hourly for 5 days

4.For patient sensative to penicilin: use clindamycin 300mg orallym 8 hourly for 5 days

35
Q

What are the advantages of internal bleaching?

A

1.Conservative of tooth structure

2.Natural translucency of the tooth

3.Prior to placement of a definite restoration

4.A return to normal color could occur

5.Inexpensive

36
Q

What are risk of internal bleaching?

A

1.Invasive cervical resorption

2.Coronal fracture

3.Chemical burns

37
Q

What are the indicators of internal bleaching?

A
  1. Adequate endodotntics and no periapical pathosis
  2. No vertical root fracture
  3. Caries free
  4. Evidence of intrinsic discolouration
  5. Disolouration of pulp chamber
  6. Dentine discolouration
  7. Can’t to external bleaching
38
Q

What materials are used in internal bleaching?

A
  1. Sodium perborate – in SADS – available in powder form
  2. Hydrogen peroxied – can burn tissues
  3. Carbamide peroxide – ususally used for external bleaching
39
Q

What can you use to remova sealant following obturation?

A

Endo dry

40
Q

What is the SADS protocol for internal bleaching?

A

1.Patient need to be elidgible – no EMERINT PROSGENINT OR EMERREPAIR COCs

2.Consent – multiple appoitments, replacemen of restoration, upredictable, not stable and retreatment may be possible. Cervical resorption may occur

3.Titanium – 117- application of internal bleaching, 990-subsequent application of internal bleaching

4.Remove extrinsic staining

5.Record pre-op shade

6.Rubber dam

7.Remove restorative mamterial from access cavity, keep stained dentine

8.Remove endodontic filling 1-2mm below CEJ

9.Seal the access to the endodontic filling wit 2mm of GIC or cavit

10.Etch pulp chamber, rinse and dry

11.Mix sodium perborate with water until stiff paste is formed

12.Place into the labila surface of the access cavity

13.Cover the bleach with cotton pellet and seal with cavit or GIC

14.Repeate steps of bleaching every 7 days untile desired colour is achieved

15.Remove all bleaching material an rinse throughly

16.Record post-op shade

17.No definite resoration for 7 days because enamel might have been weakened